ライフサイエンスコーパス: vermis

1 ventral uvula (lobules X-IX of the posterior vermis).

2 r/ataxia syndrome was found in a part of the vermis.

3 the transmitter used by many neurons in the vermis.

4 hombic lip and overmigrate into the anterior vermis.

5 antly restricted to the cerebellar posterior vermis.

6 ntal cortex, corpus callosum, and cerebellar vermis.

7 with subsequent loss of Atoh1 expression in vermis.

8 o of these are ongoing within the cerebellar vermis.

9 f8 gene dose, resulted in loss of the entire vermis.

10 ), and paravermis, but with decreases in the vermis.

11 II/EAAT4-negative PC subsets in the anterior vermis.

12 ols had greater activation in the cerebellar vermis.

13 us, insula, frontal operculum and cerebellar vermis.

14 al right cerebellum, as well as the anterior vermis.

15 obules I, IV, V, IX, and X of the cerebellar vermis.

16 operculum bilaterally and in the cerebellar vermis.

17 vascular resistance in the dorsal cerebellar vermis.

18 posterior-inferior lobules of the cerebellar vermis.

19 globus pallidus, cerebellum, and cerebellar vermis.

20 ctivity in both the hippocampus and anterior vermis.

21 creases in p38 MAPK activity in the anterior vermis.

22 RKs) and p38 MAPK in the anterior cerebellar vermis.

23 frontal brain, basal ganglia, and cerebellar vermis.

24 observed, most notably lobes IV and V in the vermis.

25 ol of cells that develop into the cerebellar vermis.

26 ularly in the cerebral cortex and cerebellar vermis.

27 amus and premotor cortex, and the cerebellar vermis.

28 s expressed transiently in the anterior lobe vermis.

29 thin the cerebellum, especially the inferior vermis.

30 section of the posterior inferior cerebellar vermis.

31 striatum, olfactory tubercle, and cerebellar vermis.

32 ing to the vestibulocerebellum and posterior vermis.

33 he left prefrontal cortex and the cerebellar vermis.

34 tal lobes but not in the superior cerebellar vermis.

35 A-A/BDZ receptors in the superior cerebellar vermis.

36 nd DV bilaterally in the superior cerebellar vermis.

37 diate and lateral cerebellum, as well as the vermis.

38 visual cortex, right cuneus, and oculomotor vermis.

39 rallel fiber synapses in the mouse posterior vermis.

40 sented across multiple lobules of the rodent vermis.

41 ns, except in the cerebellar hemispheres and vermis.

42 the vermis most particularly in the anterior vermis.

43 sorders arising from damage to the posterior vermis.

44 , and hypoplasia of brainstem and cerebellar vermis.

45 the paracentral motor cortex and cerebellar vermis.

46 ociated with smaller regional volumes in the vermis.

47 g orthodromic identification from the caudal vermis.

48 medial wall of the hemisphere project to the vermis.

49 3)mum(2)/sec; right, 1.09 x10(3)mum(2)/sec), vermis (1.26 x10(3)mum(2)/sec), thalami (left, 1.17 x10(

50 0(3)um(2)/sec; right, 1.09 x10(3)um(2)/sec), vermis (1.26 x10(3)um(2)/sec), thalami (left, 1.17 x10(3

51 c relatives had lower metabolism in anterior vermis (12%; P = 0.01) and hippocampus (19%; P = 0.002)

52 ecreased functional connectivity between the vermis 3 and the bilateral supplementary motor area, and

53 short midbrain and small inferior cerebellar vermis (38% each) as well as hypertrophy of the clava (2

54 al supplementary motor area, and between the vermis 4,5 and the occipital cortex.

55 ellar hemispheres (14%, P < 0.001), anterior vermis (40%, P < 0.001) and fusiform gyrus (20%, P < 0.0

56 nectivity of the cerebellum (lobule 4, 5 and vermis 6) with the precuneus.

57 ated with decreased cerebellar (lobule 8 and vermis 8, 9) connectivity with fronto-polar, superior te

58 imulus specificity, including the oculomotor vermis, a key area associated with eye movement control.

59 gray matter decline in anterior lobules and vermis, accelerated vermian white matter expansion, and

60 uncle and bilateral portions of the superior vermis also showed persistent decrease in FA over time.

61 Lobules 10 and 9 in the caudal posterior vermis [also known as nodulus and uvula (NU)] are though

62 In the posterior vermis, although the zebrin II+ bands are wider and mult

63 ased regional brain volumes in the posterior vermis, amygdala, and hippocampus.

64 head movements in the rat caudal cerebellar vermis, an area essential for graviceptive functions.

65 genital brain malformation of the cerebellar vermis and brainstem with abnormalities of axonal decuss

66 ed by hypoplasia/dysplasia of the cerebellar vermis and by ataxia, hypotonia, oculomotor apraxia, and

67 (vestibular nuclear area, cerebellar ventral vermis and floccular lobe), cardiorespiratory control (m

68 kinje cells recorded from the posterior lobe vermis and hemisphere have high simple spike firing freq

69 ion in anterior subregions of the cerebellar vermis and hemisphere in the asymptomatic premutation gr

70 proliferation was similar in the cerebellar vermis and hemispheres in all patients with ciliopathy a

71 /dysfunction was indicated in the cerebellar vermis and hemispheres in both diseases by lower total N

72 d number and size of Purkinje neurons in the vermis and hemispheres, molecular defects, and reduced f

73 sterior lobe were profoundly reduced in both vermis and hemispheres.

74 observed in multiple lobules throughout the vermis and hemispheres.

75 Our finding of decreased metabolism in vermis and hippocampus of asymptomatic relatives suggest

76 results challenge the classical view of the vermis and indicate that it no longer should be consider

77 ing crucial to producing the distinct medial vermis and lateral hemisphere foliation patterns in mamm

78 We found that the right cerebellar vermis and left lobule V of cerebellar anterior lobe wer

79 The vermis and left thalamus matured earliest (1.3 years).

80 te nucleus, cerebellar peduncles, cerebellar vermis and lobules V and VI, and corpus callosum.

81 We recorded PC activity in the vermis and lobus simplex of head-fixed mice while monito

82 sence of posterior vermis with some anterior vermis and nodulus present), to severe (the absence of p

83 myo-inositol was higher than controls in the vermis and pons in AOA2 and in the vermis in FRDA.

84 eural computations performed by the anterior vermis and posterior vermis (nodulus/uvula).

85 g FGF signaling perturbs the balance between vermis and roof plate development in rhombomere 1.

86 ttributed to the reduction of the cerebellar vermis and some regions of the hemispheres.

87 and ventral striatum, and midline cerebellar vermis and subgenual cingulate cortex.

88 inactivation data showing that the posterior vermis and the caudal fastigial nucleus, to which it pro

89 co-nuclear zone consisting of the cerebellar vermis and the fastigial nucleus.

90 A similar increase in the cerebellar vermis and the left thalamus likewise suggests a role of

91 for total cerebral volume, the volume of the vermis and the midsagittal area and volume of the inferi

92 ed rabies virus into lobules VB-VIIIB of the vermis and used retrograde transneuronal transport of th

93 ecursor cells, resulting in complete loss of vermis and variable hypoplasia of cerebellar hemispheres

94 l processing (sensorimotor cortex, striatum, vermis) and an increased influence of the CbTC circuit.

95 differences were significant in cerebellum (vermis) and thalamus.

96 motor output (anterior and dorsal cerebellar vermis) and the maintenance of equilibrium (vestibular n

97 rin II+ cell mass is absent from the central vermis, and analysis of the anterior lobe reveals severa

98 ment and separately for the whole brainstem, vermis, and cerebellar hemispheres.

99 as well as the caudate, thalamus, cerebellar vermis, and cerebrum in 20 first-episode psychosis patie

100 palsy in the putamen, caudate, thalamus, and vermis, and decreased in the superior cerebellar peduncl

101 ellar network comprising the inferior olive, vermis, and deep cerebellar nuclei including the dentate

102 fossa lesion, including that in the nodulus, vermis, and deep cerebellar structures.

103 the posterior lobe of the cerebellum and the vermis, and in some cases they were the most noticeable

104 x, lingual gyrus, striate cortex, cerebellar vermis, and left thalamus.

105 trophy of the cerebral cortex and cerebellar vermis, and mild atrophy of the cerebellar hemispheres.

106 thalamic, amygdala, hippocampal, cerebellar vermis, and regionally specific uptake in the cerebral c

107 thalamus, left cerebellar regions, anterior vermis, and right cuneus.

108 us, globus pallidus, hippocampus, cerebellar vermis, and very low expression was detected in the stri

109 ibution, and also in the superior cerebellar vermis; and the non-ACD group had significant reductions

110 arized progression of differentiation in the vermis anlage.

111 cell proliferation in the medial cerebellar (vermis) anlage after E11.5.

112 en syndrome), liver fibrosis, and cerebellar vermis aplasia (Joubert syndrome) in approximately 10% o

113 liopathy giving rise to NPHP with cerebellar vermis aplasia and retinal degeneration.

114 ion with retinal degeneration and cerebellar vermis aplasia in Joubert syndrome are poorly understood

115 Decreased volume of the superior cerebellar vermis appears to represent an important substrate of th

116 n samples, show that the hemispheres and the vermis are affected in JS/MKS and provide evidence of a

117 onal nystagmus revealed that the nodulus and vermis are common areas of injury.

118 pallidus, sensorimotor cortex and cerebellar vermis, as well as increases in the precuneus (BA 7).

119 g the cerebellum, in particular the superior vermis, as well as the medial and inferior frontal corte

120 t these FGFs are not required to pattern the vermis at this stage of development.

121 isorder marked by agenesis of the cerebellar vermis, ataxia, hypotonia, oculomotor apraxia, neonatal

122 in patient-derived brain tissue, cerebellar vermis atrophy (5/20), and callosal hypoplasia (4/20).

123 e associated with more pronounced cerebellar vermis atrophy (lobules I-V beta = -1.06, P < 0.001; lob

124 gressive gait ataxia with tremor, cerebellar vermis atrophy, and optic-nerve thinning.

125 early infancy and are affected by cerebellar vermis atrophy, ataxia, and peripheral neuropathy.

126 ngulate cortices (ACC) as well as cerebellar vermis, bilateral cerebellar hemispheric lobule VI, and

127 (increased rCBF) were observed in cerebellar vermis, brainstem and right anterior cingulate.

128 to eye movements, suggesting that the caudal vermis can also directly influence vestibulo-ocular path

129 Optical stimulation of the oculomotor vermis caused saccade dysmetria.

130 The vermis classically is thought to be included within the

131 Purkinje neurons in the caudal cerebellar vermis combine semicircular canal and otolith signals to

132 Gray matter volume in the anterior superior vermis correlated with lifetime alcohol consumption in t

133 f left VIIIa, relative decrease of VIIIb and vermis Crus I volume) and white matter microstructure in

134 ole-cell recording from slices of cerebellar vermis derived from juvenile (P18-25) or adult (P60-83)

135 trast to the considerable posterior lobe and vermis disease burden identified in C9orf72 mutation car

136 efore, suppression of P-cells in the lingual vermis disrupted the forces that would normally decelera

137 mother-reared monkeys, we found an enlarged vermis, dorsomedial prefrontal cortex, and dorsal anteri

138 tion patterns were present in the cerebellar vermis during bimanual coordination tasks, with greater

139 lus) and IXc,d (ventral uvula) of the caudal vermis during vestibular stimulation.

140 IXc,d (ventral uvula) of the macaque caudal vermis during vestibular stimulation.

141 posterior lobule which echoes the posterior vermis DW 'tail sign' observed in human imaging studies.

142 In contrast, the posterior vermis, encompassing the nodulus and uvula, integrates v

143 Therefore, many P-cells in the oculomotor vermis exhibit changes in SS activity specific to adapte

144 ad, responses in ancient cerebellar regions (vermis, fastigal nucleus, archicerebellum) may be more d

145 in one case with epilepsy, which also showed vermis folial malformation.

146 re additionally required to pattern anterior vermis foliation.

147 nd ventral uvula (lobules X and IXc,d of the vermis) for vestibular processing has been strongly sugg

148 ese results indicate that diverse cerebellar vermis functions could be mediated by modular synaptic c

149 The cerebellar posterior vermis generates an estimation of our motion (translatio

150 ulator cohesin in anterior dorsal cerebellar vermis granule neurons in adult mice disrupts enhancer-p

151 In addition, the right cerebellar vermis had enhanced connectivity with motor and cognitiv

152 lic activity and the supramarginal gyrus and vermis had significantly more metabolic activity.

153 activation in the anterior dorsal cerebellar vermis has a crucial role in a delay tactile startle lea

154 Also, in preclinical studies, the vermis has been shown to modulate forebrain dopamine sys

155 These results establish that the oculomotor vermis helps control the characteristics of normal ipsiv

156 Therefore, we show that the caudal vermis hosts a re-encoded, gravitationally polarized rep

157 Unlike the cerebellar vermis, however, MSTd neurons also carry a spatial orien

158 ior fossa malformations including cerebellar vermis hypoplasia (CVH), mega-cisterna magna (MCM) and D

159 rt Syndrome, a ciliopathy causing cerebellar vermis hypoplasia and ataxia.

160 ent are associated with posterior cerebellar vermis hypoplasia and Dandy-Walker malformation.

161 elay, epilepsy, cortical atrophy, cerebellar vermis hypoplasia and ocular impairment.

162 Finally, we report cerebellar vermis hypoplasia in 35% of CHARGE syndrome patients wit

163 nd link reduced FGF signalling to cerebellar vermis hypoplasia in a human syndrome.

164 regression of the rhombic lip and posterior vermis hypoplasia in Lmx1a(-/-) mice.

165 hh signaling defects could contribute to the vermis hypoplasia observed in the human syndromes.

166 n presented congenital ataxia and cerebellar vermis hypoplasia with elongated superior cerebellar ped

167 tisystem disease characterized by cerebellar vermis hypoplasia with prominent superior cerebellar ped

168 thy Joubert syndrome is marked by cerebellar vermis hypoplasia, a phenotype for which the pathogenic

169 l brain disorder characterized by cerebellar vermis hypoplasia, abnormal eye movement, ataxia and men

170 ysgenesis of the corpus callosum, cerebellar vermis hypoplasia, and facial dysmorphism.

171 on axial brain MRI, together with cerebellar vermis hypoplasia, ataxia, and psychomotor delay.

172 al signal), thin corpus callosum, cerebellar vermis hypoplasia, optic nerve hypoplasia and mild ventr

173 disorder featuring absence of the cerebellar vermis (i.e. midline).

174 on in anterior cerebellar regions, including Vermis III and Cerebellum IV-V.

175 Purkinje cells (PCs) in Rcrus1 and posterior vermis improved social preference impairments and repeti

176 e image analysis, we measured the cerebellar vermis in 125 normal individuals with a broad age range

177 is preliminary study supports a role for the vermis in ADHD and suggests that further research is nee

178 each lobe of the cerebellar hemispheres and vermis in children with ADHD and comparison subjects and

179 ontal cortex, corpus callosum, and posterior vermis in children with autism and further suggest that

180 ls in the vermis and pons in AOA2 and in the vermis in FRDA.

181 iform gyri and reduced GMV in the cerebellar vermis in FXS at both timepoints, suggesting early, poss

182 endent loss of the most anterior lobe of the vermis in mice lacking Fgf17 and in mice lacking Fgf17 a

183 Lesions of the vermis in particular were associated with dysregulation

184 the pons, the cerebellar hemisphere and the vermis in patients with FRDA and AOA2 to identify potent

185 Injury to the cerebellar vermis in patients with mTBI and anxiety may indicate un

186 after stimulation of the right neocerebellar vermis, in contrast to all other conditions.

187 ent), to complete (the absence of the entire vermis including nodulus).

188 ogenetic inhibition of Purkinje cells in the vermis increased the frequency of attacks.

189 e in AOA2 than controls were observed in the vermis, indicating different mechanisms possibly leading

190 as defined by connectivity to the cerebellar vermis, inferior cerebellum (bilateral lobule X), and th

191 Volumes of the vermis, inferior posterior lobe, fourth ventricle, and t

192 dulus/uvula (NU) in the posterior cerebellar vermis is known to integrate canal and otolith vestibula

193 he plasticity at the level of the oculomotor vermis is more fundamentally important for forward adapt

194 Cerebellar fusion and absence of cerebellar vermis is often associated with supratentorial findings.

195 e projection from the cerebral cortex to the vermis is part of the neural substrate for anticipatory

196 c, the cerebellum is underdeveloped, and the vermis is severely reduced.

197 hree groups based on primary tumor location: vermis, left hemisphere, or right hemisphere.

198 ittent theta burst stimulation (iTBS) to the vermis lobule VII or right lateral cerebellar Crus I/II,

199 e mice, acute perturbation of the cerebellar vermis (lobule 4/5) or simplex produced reliable and spe

200 Enhanced rs-fc between the cerebellar vermis (lobule 6) and the left precentral gyrus was asso

201 hippocampal volumes, and smaller cerebellar vermis lobules VI and VII, in comparison with his brothe

202 lar cerebellum: the floccular lobe, anterior vermis (lobules I-V), and nodulus and ventral uvula (lob

203 l types in the vestibular part of the caudal vermis (lobules IX and X) to understand their role in th

204 volumes in the posterior-inferior cerebellar vermis (lobules VIII-X; effect size, 0.54; P =.04), even

205 vity of identified P-cells in the oculomotor vermis, lobules VIc and VII.

206 The cerebellar vermis, long associated with axial motor control, has be

207 ggest that cell death is not responsible for vermis loss, but rather that it fails to develop because

208 hese data suggest that an abnormality in the vermis may contribute to the pathophysiology of schizoph

209 Evidence suggests that the cerebellar vermis may regulate aggressive behavior, though the cere

210 These mice show cerebellar hypoplasia with a vermis-midline fusion defect early in development.

211 Purkinje neurons that was most severe in the vermis most particularly in the anterior vermis.

212 pmental links between frontal and cerebellar vermis neural abnormalities were supported, in that impa

213 gnificantly, the differentiation of anterior vermis neuroepithelium was shifted rostrally and mediall

214 imilar to lobules 9 and 10 of the cerebellar vermis (nodulus and uvula), MSTd neurons respond selecti

215 ve shown that cells in the caudal cerebellar vermis (nodulus and ventral uvula, NU) reflect the outpu

216 rformed by the anterior vermis and posterior vermis (nodulus/uvula).

217 ctions of postmortem samples from cerebellar vermis of 10 patients with schizophrenia and 13 control

218 two-photon Ca(2+) imaging in the cerebellar vermis of awake behaving mice.

219 ed Purkinje-cell discharge in the oculomotor vermis of behaving rhesus monkeys (Macaca mulatta) and f

220 HO-1 was induced in Bergmann glia in the vermis of cerebellum.

221 The vermis of lobule VII receives a prominent input from the

222 ies of hundreds of P-cells in the oculomotor vermis of marmosets during saccadic eye movements and fo

223 low doses of kainic acid into the cerebellar vermis of mice elicited reliable and reproducible dyston

224 sitometric units [P=.003]) in the cerebellar vermis of schizophrenic subjects.

225 density of Purkinje cells in the cerebellar vermis of subjects with and without schizophrenia.

226 antly lower mean diffusivity in the inferior vermis of the cerebellum (patients: 7.71 x 10-4 mm2/s; c

227 ateral putamen and insula, anterior lobe and vermis of the cerebellum and superior colliculus).

228 hippocampus (CA1 and dentate), thalamus, and vermis of the cerebellum as early as 3 h post injury.

229 on steady-state blood volume in the midline vermis of the cerebellum in boys with attention deficit

230 The posterior vermis of the cerebellum is considered to be critically

231 The anterior vermis of the cerebellum is thought to play a key role i

232 fects (Purkinje cell loss) manifested in the vermis of the cerebellum.

233 sked whether Purkinje cells (P-cells) in the vermis of the oculomotor cerebellum, lobules VIc and VII

234 We studied this in the vermis of the spinocerebellar lobule V and the vestibulo

235 es from normal involved anterior lobules and vermis of youths who initiated substantial drinking.

236 e in the cerebellum (hemispheres and midline vermis) of 10 boys with ADHD who were administered place

237 The oculomotor vermis (OMV) of the cerebellum is necessary for the gene

238 tion of gaze called saccades, the oculomotor vermis (OMV) of the cerebellum must be intact.

239 -expressing Purkinje cells in the oculomotor vermis (OMV) of two male macaque monkeys.

240 art of the vermal cerebellum [the oculomotor vermis (OMV)] in the control of visually guided saccades

241 The influence of the cerebellar vermis on prefrontal and striatal circuitry should be ex

242 each case, the surgery destroyed the midline vermis only (ranging from lobules VI-X).

243 quired to ensure that folia exclusive to the vermis or hemispheres form in the appropriate mediolater

244 resulting from dysfunction in the cerebellar vermis or the basal ganglia.

245 profiling and anatomical circuit analyses of vermis output neurons in the mouse fastigial (medial cer

246 in right amygdala/hippocampus and cerebellar vermis (P < 0.001), relative to global brain.

247 had diminished fractional anisotropy in the vermis (P = .04).

248 The cerebellar vermis plays an essential role in maintaining posture an

249 which arises predominantly in the cerebellar vermis, preferentially affects children between the ages

250 We found that caudal cerebellar vermis Purkinje cells and cerebellar nuclei neurons sele

251 ly and temporally matched in mouse posterior vermis Purkinje cells and that Purkinje cell responses c

252 he motor-related signals encoded by anterior vermis Purkinje cells explain their altered sensitivity

253 recorded the activity of individual anterior vermis Purkinje cells in alert monkeys during passively

254 icant intersubject variability (decreases in vermis ranged from 18% to 60%).

255 cterized by severe defects of the cerebellar vermis, ranging from hypoplasia to aplasia.

256 the surprising result that a portion of the vermis receives dense input from the cerebral cortex.

257 ing mediolateral expansion of the cerebellar vermis, reduced thickness of the granule cell layer and

258 n of Spry2 alone caused loss of the anterior vermis, reducing FGF signaling further, by decreasing Fg

259 est Purkinje cell activity in the cerebellar vermis regulates aggression, and further support the imp

260 ysis of cerebellar sections from the midline vermis revealed that during development, the expression

261 l stress tasks, increased rCBF in cerebellar vermis, right anterior cingulate and right insula covari

262 Given the anterior vermis's role in maintaining balance during voluntary be

263 self-motion, Purkinje cells in the anterior vermis selectively suppress responses in the vestibulosp

264 the CS activity of P-cells in the oculomotor vermis signals the direction but not the magnitude of ey

265 Increasing Purkinje cell activity in the vermis significantly reduced the frequency of attacks in

266 RS had significantly higher MD values in the vermis than did healthy subjects (P < .05) and patients

267 inly in cortical structures, cerebellum, and vermis) that could be attenuated by pretreatment with ha

268 loss of zebrin II/EAAT4-negative PCs in the vermis, the densities of microglia and the Bergmann glia

269 isexpression also affects development of the vermis, the part of the cerebellum that spans the midlin

270 lume was observed in the superior cerebellar vermis; the volume loss persisted regardless of clinical

271 al areas Right crus 1 (Rcrus1) and posterior vermis through the cerebellar nuclei and ventromedial th

272 ume trajectories of 10 cerebellar lobule and vermis tissue constituents in 548 no/low drinking youths

273 of the right amygdala and midline cerebellar vermis to nonemotional as opposed to emotional body lang

274 l absence of nodulus, anterior and posterior vermis), to moderate (the absence of posterior vermis wi

275 ion of patients (PwMS and PwCIS), GM loss in vermis VI (R(2)=0.36; p<0.05 when considering age and T2

276 me decrease in posterior lobules (especially vermis VI) was associated with reduced IPS.

277 IIA and right HVI) and portions of posterior vermis (VI and superior VIIA) exhibited increased activa

278 n the posterior lobe (lobule VIIB, crus II), vermis (VI, VIII), flocculonodular lobe (lobule X), and

279 resonance imaging to measure cerebellum and vermis volume in 15 patients with schizophrenia and 15 n

280 They found that 1) vermis volume was greater in patients with schizophrenia

281 l prefrontal cortex, amygdala, and posterior vermis volumes were significantly associated with the se

282 in that impaired neuronal functioning in the vermis was associated with impaired neuronal functioning

283 on; specifically, lobule VII in the anterior vermis was missing.

284 The inferior vermis was significantly smaller in the schizophrenic gr

285 The cerebellar vermis was strongly activated by capsaicin, whereas ligh

286 terior quadrangular lobule, lingula, and the vermis were activated.

287 autistic twins, and volumes of the posterior vermis were altered in both autistic twins and co-twins.

288 hippocampus, corpus callosum, and cerebellar vermis were compared between mother-reared (n = 15) and

289 Blocks of alcohol-fixed cerebellar vermis were dissected at autopsy from the brains of 14 e

290 levels of the MAP kinases in the cerebellar vermis were linked to additional downstream targets of t

291 l cerebellum and the midsagittal area of the vermis were measured manually.

292 The blocks of vermis were sectioned and stained with 1% cresyl violet.

293 expansion in the cerebellar hemispheres and vermis were, on average, 3.15 and 2.72 times greater rel

294 In the vermis, which supports a range of behavioral functions,

295 ophrenia than in normal subjects, 2) greater vermis white matter volume in the patients with schizoph

296 ormation characterized by missing cerebellar vermis with apparent fusion of the cerebellar hemisphere

297 y partial or complete loss of the cerebellar vermis with fusion of the cerebellar hemispheres, in 8/1

298 p of Purkinje cells in the caudal cerebellar vermis with responses that reflect an estimate of head t

299 rmis), to moderate (the absence of posterior vermis with some anterior vermis and nodulus present), t

300 evere (the absence of posterior and anterior vermis with some nodulus present), to complete (the abse