ライフサイエンスコーパス: vertebral artery

1 otected areas (ie, not dependent on the left vertebral artery).

2 n the left internal carotid artery and right vertebral artery.

3 nostic neuroangiography even beyond the left vertebral artery.

4 egulation in the internal carotid artery and vertebral artery.

5 ressure assessed at the internal carotid and vertebral arteries.

6 e catheterization of the internal carotid or vertebral arteries.

7 aged 53-86 years; mean, 73 years) had normal vertebral arteries.

8 ery (52 patients, 15 bilateral) intracranial vertebral artery (40 patients, 12 bilateral), basilar ar

9 commonest occlusive sites were: extracranial vertebral artery (52 patients, 15 bilateral) intracrania

10 ries, including the extracranial carotid and vertebral arteries and intracranial arteries, is increas

11 Imaging data on the patency of the vertebral arteries and posterior communicating arteries,

12 avian stenosis proximal to the origin of the vertebral artery and in the other case a coronary fistul

13 on the left side and dissection of the right vertebral artery and no ischemic changes within the brai

14 uplex ultrasound of the internal carotid and vertebral arteries, and transcranial Doppler ultrasound

15 d mild to severe ostial stenosis of a single vertebral artery, and eight patients (including four men

16 We conclude that endovascular stenting of vertebral artery atherosclerotic disease is safe and eff

17 ssive heat stress provoked ~16% increases in vertebral artery blood flow, independent of changes in e

18 ging, they were less likely to have a single vertebral artery dissection (aOR, 0.37; 95% CI, 0.25-0.5

19 using validated diagnosis codes for carotid/vertebral artery dissection.

20 The treatment of carotid and vertebral artery dissections is based on rather incomple

21 many patients being treated for carotid and vertebral artery dissections with percutaneous angioplas

22 The presence of unilateral delayed vertebral artery enhancement was significantly associate

23 ral artery stenosis and eight controls, both vertebral arteries filled simultaneously.

24 lavian stenosis or occlusion with retrograde vertebral artery flow confirmed with time-of-flight MR a

25 vertebral asymmetry influence mixing of the vertebral artery flow contributions.

26 Vertebral artery flow reversal is often found among pati

27 the human study, 50 internal carotid and 49 vertebral arteries from 25 subjects (mean age +/- standa

28 In the past 5 years, BCVI (carotid and vertebral arteries) has been recognized with increasing

29 Vertebral artery hypoplasia (VAH) is more common in hype

30 with hypertension have higher prevalence of vertebral artery hypoplasia (VAH), which is associated w

31 ence of congenital cerebrovascular variants; vertebral artery hypoplasia, and an incomplete posterior

32 Eight studies that examined 5704 carotid or vertebral arteries in 1426 trauma patients met inclusion

33 ranial carotid arteries in 251 patients, and vertebral arteries in 82 patients.

34 sient 20 min occlusion of common carotid and vertebral arteries in rats caused a dramatic (3-fold) in

35 Spontaneous dissections of the carotid and vertebral arteries in the neck are a common cause of str

36 uries (43 bilateral), and 79 patients had 97 vertebral artery injuries (18 bilateral).

37 .6, 2.4]; P < .001), carotid injuries versus vertebral artery injuries (49 of 420 [11.7%] vs 35 of 66

38 d artery injuries (CAI) and 43 patients with vertebral artery injuries (VAI) for an overall screening

39 No patients with isolated vertebral artery injuries had positive transcranial Dopp

40 injuries, but monitoring was not useful for vertebral artery injuries.

41 Traumatic vertebral artery injury (TVAI) can have a varied clinica

42 elayed stroke among patients who sustained a vertebral artery injury with or without additional vesse

43 ion (CeAD), a mural hematoma in a carotid or vertebral artery, is a major cause of ischemic stroke in

44 ak contrast enhancement in the right or left vertebral arteries may, in the appropriate clinical sett

45 ulted from carotid injury and 4 (26.7%) from vertebral artery occlusion (P = .03).

46 chanism of brain infarction in patients with vertebral artery occlusive disease.

47 le) of an occlusion in the V4 segment of the vertebral artery; proximal, middle, or distal segment of

48 ture-4 cases, perivascular hematoma-2 cases, vertebral artery puncture-1 case, pneumothorax-1 case) a

49 delay in peak enhancement in the ipsilateral vertebral artery ranged from 2 to 4 seconds (mean, 2.5 s

50 normal flow (P < .01) and those with ostial vertebral artery stenosis (P < .01).

51 ty of catheter-based therapy for symptomatic vertebral artery stenosis (VAS).

52 In eight of nine patients with ostial vertebral artery stenosis and eight controls, both verte

53 Vertebral artery stenosis can be treated with stenting w

54 Symptomatic vertebral artery stenosis is associated with a high risk

55 e studies of the exceedingly rare rotational vertebral artery syndrome have been added to the literat

56 including vertigo associated with rotational vertebral artery syndrome, as well as whiplash and degen

57 than the 'less-reactive' CA measured at the vertebral artery that was associated with WMH severity.

58 uplex ultrasound of the internal carotid and vertebral arteries to determine cerebral exchange kineti

59 e dogs received glucose via both carotid and vertebral arteries to maintain cerebral euglycemia (H-EU

60 In the Carotid and Vertebral Artery Transluminal Angioplasty Study (CAVATAS

61 ort the long-term results of the Carotid and Vertebral Artery Transluminal Angioplasty Study (CAVATAS

62 sonography at the internal carotid (ICA) and vertebral arteries (VA).

63 tid arteries (ICA and ECA, respectively) and vertebral artery (VA) (Duplex ultrasound) was measured.

64 red at the internal carotid artery (ICA) and vertebral artery (VA) and CBF velocity at the middle cer

65 the internal carotid arteries (ICAs) and the vertebral arteries (VAs); 3) atherosclerosis of the larg

66 ae leads to a complete loss of the bilateral vertebral arteries (VTAs) that extend along the ventrola

67 signal intensity between the right and left vertebral arteries was compared among the three groups b

68 blood flow through the internal carotid and vertebral arteries was performed to calculate global cer

69 The internal carotid and vertebral arteries were examined in relatively healthy s

70 Using a volume-rendered angiogram, vertebral arteries were measured along the curvature of

71 hundred fourteen (73%) carotid and 65 (67%) vertebral arteries were restudied with arteriography 7 t

72 inflammation within the aorta, carotid, and vertebral arteries with histologic validation in humans

73 e, and vessel type (internal carotid artery, vertebral artery) with BCVI-associated stroke.