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1 tion of focal liver lesions and diagnosis of vesicoureteral reflux.
2 o potentially milder manifestations, such as vesicoureteral reflux.
3 ebrile urinary tract infection commonly have vesicoureteral reflux.
4  kidney after bladder warming would indicate vesicoureteral reflux.
5 s, to potentially milder conditions, such as vesicoureteral reflux.
6 velopment of renal scarring in children with vesicoureteral reflux.
7 reased the risk of urologic complication and vesicoureteral reflux.
8 s are ureteropelvic junction obstruction and vesicoureteral reflux.
9 he two approaches in low-grade (grades I-II) vesicoureteral reflux.
10 tive value for ruling out high-grade (III-V) vesicoureteral reflux.
11 l need to reexamine treatment modalities for vesicoureteral reflux.
12 cystourethrography has been used to rule out vesicoureteral reflux.
13 place in the algorithm for the management of vesicoureteral reflux.
14 III genes result in the phenotype of primary vesicoureteral reflux.
15 hildren (112 of 117) had grade I, II, or III vesicoureteral reflux.
16 ant to prevent further damage, infection and vesicoureteral reflux.
17 ortical scarring, and long term follow-up of vesicoureteral reflux.
18 er of the 23 piglets with surgically created vesicoureteral reflux.
19 d 235 participants (80.5%) had grade IV or V vesicoureteral reflux.
20 , 2.47; 95% CI, 1.19-5.12), and grade 4 to 5 vesicoureteral reflux (0.60 per person-year; HR, 4.38; 9
21 ale (90%), 375 were white (78%), and 375 had vesicoureteral reflux (78%).
22 nderwent cystourethrography (117 of 302) had vesicoureteral reflux; 96 percent of these children (112
23                          Among children with vesicoureteral reflux after urinary tract infection, ant
24 f pathology is lessened, however the risk of vesicoureteral reflux and consequent febrile urinary tra
25  to 5 months of age with grade III, IV, or V vesicoureteral reflux and no previous UTIs to receive co
26          In infants with grade III, IV, or V vesicoureteral reflux and no previous UTIs, continuous a
27  have greatly increased our understanding of vesicoureteral reflux and provide a promise of novel non
28 ients as bulking agents for the treatment of vesicoureteral reflux and urinary incontinence.
29 ing obstruction, may also be associated with vesicoureteral reflux and/or obstruction of the bladder
30  incontinence, decreasing surgical rates for vesicoureteral reflux, and decreasing recurrent urinary
31 phric blastema leads to renal hypodysplasia, vesicoureteral reflux, and ectopic ureters to name a few
32  of urinary leak, stricture, compression, or vesicoureteral reflux, and hospital costs were analyzed.
33 nary tract infections, indwelling catheters, vesicoureteral reflux, and immobilization hypercalcuria
34 ection (UTI) requiring antibiotics, grade of vesicoureteral reflux, and posttransplant bladder capaci
35 nary tract problems including recurrent UTI, vesicoureteral reflux, and renal scarring, the mechanism
36  circumcision debate, dysfunctional voiding, vesicoureteral reflux, and the diagnosis and follow-up o
37 reteroceles, along with the recognition that vesicoureteral reflux associated with ureteroceles can b
38 nfants <= 2 years of age with a diagnosis of vesicoureteral reflux between 2009 and 2022 were recruit
39 bnormal screening renal sonograms often have vesicoureteral reflux, but a normal sonogram does not re
40  demonstrated moderate success in correcting vesicoureteral reflux, but little is known about its imp
41 el noninvasive molecular diagnostic tests of vesicoureteral reflux by proteomics methodology.
42 y1 knockin mice also present megaureters and vesicoureteral reflux, caused by failure of ureters to s
43 ch infants should be referred for endoscopic vesicoureteral reflux correction in their first visits.
44 le nature of bladder imaging in children for vesicoureteral reflux detection makes the search for non
45 chard Wahl reviews urinary tract infections, vesicoureteral reflux, dysfunctional voiding, and approp
46                    Traditional management of vesicoureteral reflux focuses on preventing renal compli
47 s in 298 individuals with confirmed UTIs and vesicoureteral reflux from the Randomized Intervention f
48 phylaxis decreased by 47% when adjusting for vesicoureteral reflux grade and bowel and bladder dysfun
49 trasonography imaging of the bladder to find vesicoureteral reflux has yet to be refined enough to be
50                   Both knockouts experienced vesicoureteral reflux, hydronephrosis, renal dysfunction
51 ement of urinary tract infections (UTIs) and vesicoureteral reflux in children and examine new altern
52                             The treatment of vesicoureteral reflux in children has seen a shift from
53  an increasingly popular method for managing vesicoureteral reflux in children.
54 or detection of genetic mutations leading to vesicoureteral reflux in humans by studying differential
55 ion generates excessive apoptosis leading to vesicoureteral reflux in newborns, which underscores the
56 n may play an etiological role in congenital vesicoureteral reflux in otherwise normal children.
57                     The current diagnosis of vesicoureteral reflux involves voiding cystourethrograms
58                     The incidence of primary vesicoureteral reflux is about 1% to 2% of the general p
59 est that in some infants and older children, vesicoureteral reflux is associated with congenital stru
60 on (UTI) in infants with grade III, IV, or V vesicoureteral reflux is controversial.
61                            The management of vesicoureteral reflux is evolving, with advocacy ranging
62  tract infection (UTI) risk in children with vesicoureteral reflux is largely unknown.
63                                              Vesicoureteral reflux is neither necessary nor sufficien
64        It has been shown that the finding of vesicoureteral reflux is variable and that single studie
65 enged the traditional paradigm of aggressive vesicoureteral reflux management with surgery or antibio
66                                    Bilateral vesicoureteral reflux of infected urine was induced in 1
67 thral valves (four), urethral atresia (one), vesicoureteral reflux (one), and megacystis (one).
68 ecent studies have shown that in grades I-IV vesicoureteral reflux, open surgical intervention compar
69 g and bladder dysfunction were predictors of vesicoureteral reflux outcomes when the infant was recei
70 inating COU from hydronephrosis secondary to vesicoureteral reflux, particularly when diagnostic imag
71 y tract infections and/or renal scarring and vesicoureteral reflux persistence, respectively, in chil
72 y tract infections and/or renal scarring and vesicoureteral reflux persistence, the random forest was
73 ng UTIs but has not been investigated in the vesicoureteral reflux population.
74 enesis, vesicoureteral junction obstruction, vesicoureteral reflux, posterior urethral valve, genital
75 sed to treat stress urinary incontinence and vesicoureteral reflux provide for similar rates of succe
76                                      Primary vesicoureteral reflux (pVUR) is one of the most common c
77  voiding cystourethrogram to investigate for vesicoureteral reflux remains controversial.
78 e of MRU for the assessment of urolithiasis, vesicoureteral reflux, renal trauma, and fetal urinary t
79 eteral injection therapy, for patients whose vesicoureteral reflux requires correction, remains uncle
80 ications and seven (4.8%) patients developed vesicoureteral reflux requiring reoperation.
81 he Randomized Intervention for Children with Vesicoureteral Reflux (RIVUR) Study and 295 controls, an
82 he Randomized Intervention for Children With Vesicoureteral Reflux (RIVUR) study, which will evaluate
83                                Consequently, vesicoureteral reflux screening in siblings and offsprin
84 ve been tried as bulking agents to eliminate vesicoureteral reflux since the technique was introduced
85 he Randomized Intervention for Children With Vesicoureteral Reflux Study, a 2-year randomized clinica
86 he Randomized Intervention for Children with VesicoUreteral Reflux Study.
87 phritis and renal scarring may be related to vesicoureteral reflux that is missed by standard voiding
88 controlled trial involving 607 children with vesicoureteral reflux that was diagnosed after a first o
89 ance of VCUG in a pediatric porcine model of vesicoureteral reflux, total radiation exposure can be r
90 f continuous antibiotic prophylaxis (CAP) in vesicoureteral reflux, train a model to predict the outc
91 he Randomized Intervention for Children With Vesicoureteral Reflux trial and the Careful Urinary Trac
92 he Randomized Intervention for Children with Vesicoureteral Reflux trial or the Careful Urinary Tract
93 porary data regarding the natural history of vesicoureteral reflux, urinary tract infection and renal
94 nts of irreversible bladder dysfunction with vesicoureteral reflux, urinary tract infection and resul
95                                              Vesicoureteral reflux (VUR) (OMIM %193000), a common cau
96 INDINGS: The causal relationship between UTI-vesicoureteral reflux (VUR) and renal scarring has been
97  (UTI) and urinary tract abnormality such as vesicoureteral reflux (VUR) are given prophylactic antib
98 d an intermediate prevalence of GD-CNVs; and vesicoureteral reflux (VUR) had the fewest GD-CNVs but w
99 struction [ureteropelvic junction (UPJ)] and vesicoureteral reflux (VUR) have been identified and hav
100                                              Vesicoureteral reflux (VUR) is a common, familial genito
101                                              Vesicoureteral reflux (VUR) is a risk factor for acute p
102                                              Vesicoureteral reflux (VUR) is diverse in its natural hi
103                                      Primary vesicoureteral reflux (VUR) is one of the most common ur
104                                      Primary vesicoureteral reflux (VUR) is the most common congenita
105  in predicting acute pyelonephritis (APN) or vesicoureteral reflux (VUR) using the data of 288 patien
106 al-specific gene can therefore cause primary vesicoureteral reflux (VUR), a hereditary disease affect
107 d to ureteric bud (UB) induction defects and vesicoureteral reflux (VUR), although the mechanisms wer
108 the kidney and urinary tract (CAKUT) include vesicoureteral reflux (VUR).
109                                              Vesicoureteral reflux was most common (15 patients).
110                                              Vesicoureteral reflux was simulated in four pigs, and 48
111       Although the presence of grade IV or V vesicoureteral reflux was the strongest predictor of ren
112 vel of greater than 40 mg/L, and presence of vesicoureteral reflux were all associated with the devel
113                         Sex and grade 1 to 3 vesicoureteral reflux were not associated with risk of r
114                           Early detection of vesicoureteral reflux will be valuable for prevention of

 
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