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1 ice with an AgRP neuron-specific deletion of vesicular GABA transporter.
2 icle proteins, such as synaptophysin and the vesicular GABA transporter.
3 size GABA (gamma-aminobutyric acid), and the vesicular GABA transporter.
4 2 and does not involve presynaptic component vesicular GABA transporters.
5 teins: the biosynthetic enzyme for GABA, the vesicular GABA transporter, a transcription factor that
6 by approximately 40%, whereas the density of vesicular GABA transporter and bassoon coimmunoreactive
7 tion revealed coexpression of SNAP-25, VGAT (vesicular GABA transporter), and GAD65/67 (glutamic acid
9 through delivery of saporin-conjugated anti-vesicular GABA transporter antibodies (SAVAs) in vitro a
10 sessed vesicular monoamine transporter 2 and vesicular GABA transporter, but they lacked immunostaini
11 suggest that transcriptional control of the vesicular GABA transporter by NO regulates GABA transmis
12 vesicular monoamine transporter 2) and VGAT (vesicular GABA transporter), consistent with vesicular s
13 es in glutamate acid decarboxylase (GAD) and vesicular GABA transporter expression, these findings pu
14 ber of GABAergic interneurons, and GABA(A)-, vesicular GABA transporter-, GAD65-, and GAT3-immunoreac
16 TRN neurons through conditional deletion of vesicular GABA transporter has no effect on spontaneous
18 areas as indicated by the colocalization of vesicular GABA transporter immunoreactivity predominantl
20 decarboxylase) 65 and 67 isoforms, and VGAT (vesicular GABA transporter) in interneurons from the str
21 2), but not in inhibitory neurons expressing vesicular GABA transporter, in the spinal cord of FK506-
22 e vesicular glutamate transporter 3 with the vesicular GABA transporter, indicating that GABA, glycin
23 sicular glutamate transporter 2), and VGAT- (vesicular GABA transporter) mRNA in specific subregions
24 either by cell type-specific deletion of the vesicular GABA transporter or by expression of botulinum
25 Rare co-localizations between pSTAT5 and vesicular GABA transporter or vasopressin were observed,
26 ndicated by elevated GABA synthetic enzymes, vesicular GABA transporter, perineuronal nets, and enhan
27 reduced by blocking action potentials or the vesicular GABA transporter, phasic and tonic currents de
28 vo neural activity recordings, we found that vesicular GABA transporter-positive (VGAT(+)) dPAG neuro
29 -positive presynaptic endings; GAD-positive, vesicular GABA transporter-positive inhibitory endings a
31 analysis identified a selective reduction of vesicular GABA transporter punctae on parvalbumin positi
32 singDyrk1A We also identified a reduction of vesicular GABA transporter punctae specifically on parva
34 gulated the expression of Agrp, Npy, and the vesicular GABA transporter Slc32a1, and impeded leptin s
39 , with a preference for those expressing the vesicular GABA transporter VGAT and primary afferent A-f
40 We found that CLC-3 colocalized with the vesicular GABA transporter VGAT in the CA1 region of the
41 prefrontal cortex (mPFC), expression of the vesicular GABA transporter VGAT was unchanged; however,
42 amino acid transporter, VIAAT (also known as vesicular GABA transporter VGAT) transports GABA or glyc
43 l organ neurons, marked by expression of the vesicular GABA transporter VGAT, drastically suppresses
45 lation of MPOA neurons that express ESR1 and vesicular GABA transporter (VGAT) (MPOA(ESR1 VGAT) neuro
47 f two other inhibitory synapse constituents, vesicular GABA transporter (vGAT) and gephyrin, in the N
48 utons were identified by the presence of the vesicular GABA transporter (VGAT) and NPY was found in 1
50 taken from male and female donors that lack vesicular GABA transporter (Vgat) expression disperse an
52 esicular glutamate transporter-2 (VGluT2) or vesicular GABA transporter (VGAT) neurons in the spinal
53 o found that septal OXTr neurons express the vesicular GABA transporter (vGAT) protein and optogeneti
54 rthermore, co-staining for synaptophysin and vesicular GABA transporter (VGAT) revealed a group of sm
55 fication of a unique amino acid motif in the vesicular GABA transporter (VGAT) that controls its syna
56 GABA transporters (GAT-1 and GAT-3), and the vesicular GABA transporter (VGAT) was evaluated by using
59 NO) signaling elevates the expression of the vesicular GABA transporter (VGAT) within recurrent colla
60 n of hippocampal interneurons, expression of vesicular GABA transporter (vGAT), and extracellular GAB
61 luR, NMDA receptor (NR) subunits, GAD65, the vesicular GABA transporter (VGAT), and the neuronal glut
62 ing spontaneous withdrawal, those expressing vesicular GABA transporter (VGaT), glutamate transporter
63 , as well as glutamic acid decarboxylase and vesicular GABA transporter (VGAT), markers of GABAergic
64 genetic and optogenetic activation of septal vesicular GABA transporter (vGAT)-containing neurons or
66 ntrast, TBS induced LTD and LTP in 12-16% of vesicular GABA transporter (VGAT)-expressing inhibitory
67 tials were significantly more depolarized in vesicular GABA transporter (VGAT)-expressing inhibitory
68 2 (VGluT2)-expressing excitatory neurons and vesicular GABA transporter (VGAT)-expressing inhibitory
69 e expressing channelrhodopsin-(ChR2)-EYFP in vesicular GABA transporter (VGAT)-expressing neurons.
70 ) and the dorsal horn of the spinal cord, or vesicular GABA transporter (Vgat)-positive GABA neurons
76 mmunoreactivity (IR) (approximately 27%) and vesicular GABA transporter (VGAT)/p38 IR (approximately
78 nsities of ChC cartridges immunoreactive for vesicular GABA transporter (vGAT+), which is present in
80 rojecting neurons in the LH that express the vesicular GABA transporter (VGAT; a marker for GABA-rele
81 wo transcripts not altered in schizophrenia: vesicular GABA transporter (VGAT; a marker of all GABA n
82 rters responsible for packaging either GABA (vesicular GABA transporter [vGAT]) or glutamate (vesicul
83 ly and temporally target the deletion of the vesicular GABA transporter, Vgat, in developing neurons.
85 shown that VGLUT3 is found together with the vesicular GABA transporter (VIAAT) on synaptic vesicle m