ライフサイエンスコーパス: vestibular schwannoma

1 crosurgical resection for acoustic neuromas (vestibular schwannomas).

2 rilymph (PL), cerebrospinal fluid (CSF), and vestibular schwannoma.

3 facial paralysis after surgical treatment of vestibular schwannoma.

4 veloped facial paralysis after treatment for vestibular schwannoma.

5 imary and diagnostic tumor type is bilateral vestibular schwannoma.

6 haracterized by the development of bilateral vestibular schwannomas.

7 nations were performed in nine patients with vestibular schwannomas.

8 th a reduction in the volume of most growing vestibular schwannomas.

9 t problem and the cardinal sign is bilateral vestibular schwannomas.

10 NF2m-IRF3 condensates were evident in human vestibular schwannomas.

11 haracterized by the development of bilateral vestibular schwannomas.

12 t, accurate AI for 3D volumetric analysis of vestibular schwannomas.

13 ary adenoma (1.21, 1.16-1.25, p<0.0001), and vestibular schwannoma (1.19, 1.04-1.35, p=0.0085), and i

14 ary adenoma (1.13, 1.05-1.22, p<0.0001), and vestibular schwannoma (1.48, 1.19-1.84, p<0.0001) when c

15 tary adenoma (1.80, 1.41-2.30, p<0.0001) and vestibular schwannoma (1.49, 1.10-2.04, p=0.011).

16 ary adenoma [1.20, 1.09-1.31, p<0.0001], and vestibular schwannoma [1.54, 1.16-2.04, p=0.0031]).

17 Diagnostic entities included vestibular schwannomas (1011 [20.6%] of 4905 patients),

18 26 years) with progressive target tumors (10 vestibular schwannomas, 8 nonvestibular schwannomas, 20

19 The CNN model detected and delineated vestibular schwannomas accurately on contrast-enhanced T

20 ith a newly diagnosed (<6 months) unilateral vestibular schwannoma and a maximal tumor diameter of le

21 canals of patients with Meniere's disease or vestibular schwannoma and measured the endolymph-to-peri

22 ial schwannoma had an LZTR1 mutation (3 were vestibular schwannomas and 1 was a nonvestibular schwann

23 omas, six gangliogliomas, 41 meningiomas, 10 vestibular schwannomas and 31 pituitary adenomas.

24 ors and the therapeutic targeting of PAK1 in vestibular schwannomas and argue against PAK1 and PAK2 e

25 e evaluations, particularly in patients with vestibular schwannomas and candidates for cochlear impla

26 redict the degree of tumor-brain adhesion of vestibular schwannomas and may provide a method to impro

27 nherited disorder characterized by bilateral vestibular schwannomas and meningiomas.

28 dominant disease characterized by bilateral vestibular schwannomas and other nonmalignant tumors of

29 ion of vascular endothelial growth factor by vestibular schwannomas and the role of Notch signaling i

30 VEGF was expressed in 100% of vestibular schwannomas and VEGFR-2 in 32% of tumor vesse

31 inform treatment decisions for patients with vestibular schwannoma, and further investigation of long

32 All 5 had multiple tumors in addition to vestibular schwannoma, and none had a positive family hi

33 Vestibular schwannomas are benign nerve sheath tumours t

34 Vestibular schwannomas are known to occur in the context

35 ate normal anatomic structures, evaluate for vestibular schwannomas, assess for inflammatory and/or i

36 Patients with bilateral vestibular schwannomas associated with neurofibromatosis

37 ilin-2, in paraffin-embedded samples from 21 vestibular schwannomas associated with neurofibromatosis

38 meningioma, glioblastoma, pituitary adenoma, vestibular schwannoma, astrocytoma, and oligodendrogliom

39 5% male) who underwent surgical resection of vestibular schwannoma at 66 reporting facilities, the me

40 c measures differed between individuals with vestibular schwannoma (at both time points) and healthy

41 movements were measured in individuals with vestibular schwannoma before and then 6 weeks after unil

42 genetic condition associated with bilateral vestibular schwannomas, benign tumors that arise from th

43 roughput miRNA expression profiling of human vestibular schwannomas by using an array representing 40

44 High surgical vestibular schwannoma case volume in a medical instituti

45 ssociation study in a cohort of 911 sporadic vestibular schwannoma cases collated from the neurofibro

46 lume, defined as the mean number of surgical vestibular schwannoma cases per year in the 2 years prec

47 a reduction in VOR gain for individuals with vestibular schwannoma compared to control subjects, that

48 phosphorylated forms were elevated in human vestibular schwannomas compared with normal human Schwan

49 tify novel genetic associations with risk of vestibular schwannoma development, we conducted a genome

50 rent management strategies for patients with vestibular schwannomas dominated the field.

51 enrolled at the Norwegian National Unit for Vestibular Schwannoma from October 28, 2014, through Oct

52 Here, we show that vestibular schwannomas from NF2 patients and human, merl

53 Patients with multiple schwannomas without vestibular schwannomas have been postulated to compose a

54 Vestibular schwannomas have long been treated as a homog

55 Neurofibromatosis 2-associated vestibular schwannomas have variable growth rates that t

56 The management of vestibular schwannoma may include observation, microsurg

57 s may not have symptoms until much later and vestibular schwannomas may be the only tumors.

58 Patients with hearing loss (134 positive for vestibular schwannoma [mean age +/- SD, 54 years +/- 12;

59 w-grade tumors affecting the cranial nerves (vestibular schwannomas), meninges (meningiomas), and spi

60 47), pituitary adenoma (n=27 506; n=87 772), vestibular schwannoma (n=11 525; n=30 745), astrocytoma

61 Vestibular schwannomas, neurogenetic syndromes such as s

62 tion syndrome that is manifested by multiple vestibular schwannomas, nonvestibular schwannomas, menin

63 aring loss (ARHL); (2) neuropathy related to vestibular schwannoma or neurofibromatosis of type 2; (3

64 imaging is the study of choice to exclude a vestibular schwannoma or other neoplasm of the cerebello

65 Neurofibromatosis 2 (NF2) features bilateral vestibular schwannomas, other benign neural tumors, and

66 association between hearing loss in sporadic vestibular schwannoma patients and the proteome of peril

67 Head movements were measured in vestibular schwannoma patients before, and then 6 weeks

68 icate chronic inflammation in the cochlea of vestibular schwannoma patients with severe to profound h

69 live, mature utricles from organ donors and vestibular schwannoma patients, and present a validated

70 However, the majority of vestibular schwannomas present sporadically without iden

71 idelines for treating small- to medium-sized vestibular schwannoma recommend either upfront radiosurg

72 a rare form of tinnitus that may arise after vestibular schwannoma removal.

73 ic reorganization in the brainstem following vestibular schwannoma removal.

74 as performed in 32, 32, and 20 patients with vestibular schwannoma, respectively.

75 ort study found that among adults undergoing vestibular schwannoma surgery, a higher facility case vo

76 f adverse outcomes among patients undergoing vestibular schwannoma surgery.

77 Patients diagnosed with sporadic vestibular schwannoma surgically treated at Washington U

78 cohort study found that, among patients with vestibular schwannoma, symptoms present at initial evalu

79 ive sampling of PL and of CSF, and biopsy of vestibular schwannoma tissue, was performed in 32, 32, a

80 adult patients aged 18 years or older with a vestibular schwannoma treated with surgery.

81 rgery following failed primary SRS/FSRT in 7 vestibular schwannoma treatment centers across the US an

82 21.3 region is truly associated with risk of vestibular schwannoma tumorigenesis.

83 Bilateral vestibular schwannomas, tumors on cranial nerve VIII, ar

84 with newly diagnosed small- and medium-sized vestibular schwannoma, upfront radiosurgery demonstrated

85 ch are unexpectedly 14-fold more abundant in vestibular schwannoma utricles, demonstrating the existe

86 Patients with vestibular schwannoma (VS) commonly present with neurolo

87 he older adult patient group, diagnosed with vestibular schwannoma (VS) has increased.

88 ts before resection of a sporadic unilateral vestibular schwannoma (VS) in order to assess function r

89 Vestibular schwannoma (VS) is an intracranial tumor aris

90 Vestibular schwannoma (VS) is an intracranial tumor that

91 Vestibular schwannoma (VS) is the most common tumor of t

92 In vestibular schwannoma (VS) patients hearing outcome and

93 advanced age on surgical outcomes following vestibular schwannoma (VS) resection, few if any large-s

94 elf-reported occupational noise exposure and vestibular schwannoma (VS), found in several studies, re

95 ent option for small-sized (Koos I up to II) vestibular schwannoma (VS).

96 ervous system neoplasms, including bilateral vestibular schwannoma (VS).

97 is an established, effective therapy against vestibular schwannoma (VS).

98 pe 2 (NF2)-related schwannomas, 104 sporadic vestibular schwannomas (VS) and 38 schwannomatosis-relat

99 utcomes following microsurgical resection of vestibular schwannomas (VS) and meningiomas.

100 Vestibular schwannomas (VS) are benign tumors that lead

101 Vestibular schwannomas (VS) are the most common tumor of

102 coordination impairments in individuals with vestibular schwannomas (VS) remains a challenge, particu

103 nherited disorder characterized by bilateral vestibular schwannomas (VS) that arise from neoplastic S

104 type 2 (NF2) is a rare disorder that causes vestibular schwannomas (VS), meningiomas and ependymomas

105 rates after radiotherapy in actively growing vestibular schwannomas (VS).

106 Vestibular schwannomas (VSs) are the most common tumours

107 position syndrome characterized by bilateral vestibular schwannomas (VSs) resulting in deafness and b

108 NF2 is characterized by bilateral vestibular schwannomas (VSs) that cause progressive and

109 of neurofibromatosis type 2 (NF2) caused by vestibular schwannomas (VSs).

110 urofibromatosis type 2 (NF2) and progressive vestibular schwannomas (VSs).

111 head-on-trunk kinematics in individuals with vestibular schwannoma were actually comparable before an

112 ssic type 2 neurofibromatosis with bilateral vestibular schwannomas were analyzed for mutations in th

113 54 years +/- 12;64 men] and 80 negative for vestibular schwannoma) were randomly assigned to a train

114 heral nerve schwannomas, including bilateral vestibular schwannomas which are associated with substan

115 udy of adults (>=18 years old) with sporadic vestibular schwannoma who underwent salvage microsurgery

116 ith neurofibromatosis type 2 and progressive vestibular schwannomas who were not candidates for stand

117 Management of sporadic vestibular schwannoma with radiosurgery is becoming incr