ライフサイエンスコーパス: viper venom

1 factor, factors IXa and VIIIa, and Russell's viper venom.

2 ex or by a purified activator from Russell's viper venom.

3 the lethal effects of honeybee or Russell's viper venom.

4 The X-activating protein from Russell's viper venom activates fXSt. Louis II completely but at a

5 re and 48 hours after injection of Russell's viper venom, an endothelial toxin.

6 Plaque rupture was triggered with Russell's viper venom and histamine.

7 aque disruption was triggered with Russell's viper venom and histamine.

8 ger) inducing plaque disruption with Russell viper venom and histamine.

9 markedly procoagulant in a modified Russell viper venom assay.

10 GDFX and was also activated by the Russell's viper venom at similar rate, but it cleaved the chromoge

11 are capable of broadly neutralizing distinct viper venom bioactivities in vitro by inhibiting differe

12 peptide, was isolated from the Azemiops feae viper venom by combination of gel filtration and reverse

13 phospholipid-initiated and diluted Russell's viper venom clotting time, which could be partly rescued

14 Because viper venom CTLs exhibit a high degree of sequence simil

15 degrees of sequence similarity to published viper venom CTLs.

16 In conclusion, we report a new, potent viper venom-derived inhibitor of alpha2beta1 integrin, w

17 Echistatin is a viper venom disintegrin containing RGD loop maintained b

18 Russell's viper venom factor X activator (RVV-X) triggers the casc

19 oresceinated antibody, and enzyme (Russell's viper venom factor X activator)-labeled antibody is allo

20 Factor X was activated with Russell's viper venom factor X activator, and single-chain unactiv

21 Viper venom induced haemorrhagic, coagulant and anticoag

22 olecules rescued the myotubes from Russell's viper venom-induced atrophy.

23 The cobra and viper venoms-induced sterile inflammatory molecules (IL-

24 the ApoE(-/-) (-22.22+/-7.95%) and Russell's viper venom-injected (-10.37+/-17.60%) mice compared wit

25 0.54, R(12 weeks) 3.83+/-0.52) and Russell's viper venom-injected wild-type mice (R(1)=4.57+/-0.86).

26 s, such as cyclic RGD peptides isolated from viper venom, may prove to be useful as anti-inflammatory

27 We characterized HUVEC GPIb using viper venom proteins: alboaggregins A and B, echicetin,

28 Russell's viper venom reduced the total number of cells, their mig

29 are small non-enzymatic proteins present in viper venoms reported to modulate hemostasis of victims

30 antigen can enhance defense against Russell viper venom (RVV) and determined whether such responses

31 e was then induced with the use of Russell's viper venom (RVV) and histamine.

32 An enzyme from Russell's viper venom (RVV) cleaves human factor V at Arg1018 and

33 Russell's viper venom (RVV) contains protein(s) that destabilize A

34 of coagulation factors V and X by Russell's viper venom (RVV) has been implicated in the development

35 showed that factor X activator of Russell's viper venom (RVV-X) contains six N-linked oligosaccharid

36 f three of the four largest gene families in viper venom, showing that complexity evolution is a conc

37 coagulable state, we have used the Russell's viper venom test (RVV) to show that red blood cells (RBC

38 ation by the factor X activator from Russell viper venom, the mutants were characterized with respect

39 rogram/mL) did not prolong a modified Russel viper venom time, suggesting no significant inhibition o

40 Disintegrins are peptides found in viper venoms which bind to platelets through the glycopr