ライフサイエンスコーパス: viral gene product

1 d expression of an essential immediate-early viral gene product.

2 ility that LAT and miRNAs were degraded by a viral gene product.

3 -initiated DNA damage-signaling pathway by a viral gene product.

4 s diversity on the structure and function of viral gene products.

5 -dependent promoters in the absence of other viral gene products.

6 genesis apart from infectious virus or other viral gene products.

7 TSCs frequently only express immediate early viral gene products.

8 characterized by differential expression of viral gene products.

9 imary human tumors; and it is inactivated by viral gene products.

10 ism against viruses that can be subverted by viral gene products.

11 tically underestimate the true complexity of viral gene products.

12 in transduced cells in the absence of other viral gene products.

13 mount a detectable antibody response to the viral gene products.

14 appaB is that it blocks apoptosis induced by viral gene products.

15 f the symptoms of MCD may be attributable to viral gene products.

16 ransfection assays in cooperation with other viral gene products.

17 zed cores and the consequent accumulation of viral gene products.

18 article assembly in the absence of all other viral gene products.

19 Current antiviral strategies target viral gene products.

20 on of RNAi and identify VA1 RNA as the first viral gene product able to inhibit RNAi in human cells.

21 ency-associated transcript (LAT) is the only viral gene product abundantly expressed in latently infe

22 An early, IE1-regulated viral gene product acts on a necroptosis step that follo

23 In infected cells, viral gene products alter the activities of cellular pro

24 nism responsible for the suppression of late viral gene products, an important step in viral carcinog

25 (rdHSV) mutants lack at least one essential viral gene product and are propagated in host cells that

26 fection elicits T-cell responses to multiple viral gene products and antibodies capable of neutralizi

27 V is capable of expressing eight of the nine viral gene products and infected cells release immature

28 /V gene mutant (rSV5-P/V-CPI-) overexpresses viral gene products and is a potent inducer of IFN, proi

29 7 requires one or more delayed early or late viral gene products and may be associated with the inhib

30 1) indicate the presence of productive cycle viral gene products and persistent immune response, sugg

31 tend the current nomenclature to include all viral gene products and provide a genome browser that vi

32 s displayed a decreased antibody response to viral gene products and reduced proviral copies in perip

33 HBV infection, possibly in helping stabilize viral gene products and suppressing antigen presentation

34 d attention to the complex interplay between viral gene products and the host innate immune responses

35 ns, viral gene expression (IE1/IE2 and other viral gene products) and viral replication proceeded eff

36 This interaction does not require other viral gene products, and deletion of the sole candidate

37 that PGE(2) increased production of multiple viral gene products, and NS-398 inhibited production of

38 riptional template for the production of all viral gene products, and thus, it is the molecular basis

39 Only a few viral gene products are expressed by the latent virus, a

40 Viral gene products are generally required in widely dif

41 es of assembly and release of HRSV and which viral gene products are involved in the directional matu

42 the time of infection suggests that specific viral gene products are responsible for modification of

43 expressed in infected human cells as a late viral gene product, as suggested by RNA analysis of KSHV

44 corresponding to seven ORFs known to encode viral gene products associated with lytic replication.

45 ata argue that one or more newly synthesized viral gene products block the induction of antiviral pat

46 ytic and latent phases that are regulated by viral gene products, but very little is known about the

47 ution of the cell death program triggered by viral gene products, by the effectors of the immune syst

48 Our findings reveal that a viral gene product can function in distinct cellular sub

49 replicating state in which the production of viral gene products cannot be detected.

50 to HCMV UL148.IMPORTANCE In myriad examples, viral gene products cause striking effects on cells, suc

51 gg into their insect host, and expression of viral gene products causes several physiological alterat

52 suggest that expression of immunoregulatory viral gene products could be a potential strategy to pro

53 LTR) circles, integrated provirus, and early viral gene products, demonstrating susceptibility to HIV

54 During VZV skin infection, viral gene products down-regulated interferon-alpha to p

55 levated expression of DNMT1, Notch1, and the viral gene product E1insertion markE4 in CD66(high) cell

56 sis generally have come from the analysis of viral gene products, either by studying their biochemica

57 HIV-1-tat, a viral gene product essential for HIV replication, has be

58 vivo when Nef is the predominant or the only viral gene product expressed.

59 Therefore, viral gene products expressed following infection with A

60 f viral proteinases and required substantial viral gene product expression.

61 ymphocytes, during which a limited subset of viral gene products facilitates maintenance of the viral

62 study, we examined the relative roles of two viral gene products for the ability to promote loss of t

63 tency establishment, an understanding of how viral gene products function in specific B cell subsets

64 Whether and how these viral gene products function in specific cells of the im

65 This viral gene product further inhibits the ability of p53 t

66 and the control of 4E-BP phosphorylation by viral gene products, growth-inhibitory cytokines and the

67 l replication and pathogenesis in plants, no viral gene product has as yet been shown to inhibit RNAi

68 Second, Nef, an early viral gene product, has been shown to alter the activati

69 nancies are latently infected, and different viral gene products have been identified in association

70 owledge demonstrating the role of a specific viral gene product (HTLV-I Tax) on the expression of gen

71 phosphorylation in a manner dependent on the viral gene products ICP0, unique short 3 (U(S)3), and un

72 Three viral gene products-IE1, pp71, and UL26-were shown to in

73 Thus, false-negative PCR results for a viral gene product in patients under prophylaxis/treatme

74 Detection of viral gene products in renal tubules and excretion of JC

75 for the genetic analysis of the roles of the viral gene products in the complete viral life cycle.

76 lls and reduced the accumulation of specific viral gene products, including the U(S)3 protein kinase,

77 In addition, the expression of the JCV early viral gene products increased NFAT activity to further a

78 RTA is an essential viral gene product involved in the initiation of gammahe

79 of sensory ganglia, where the only abundant viral gene product is a non-coding RNA, the latency asso

80 derstanding how expression of this essential viral gene product is regulated may identify new strateg

81 at presentation of RSPWFTTL from its natural viral gene product is TAP dependent.

82 primary CD4(+) T cells expressing autologous viral gene products, it was found that 1 to 13% of CD8(+

83 ath by necroptosis requires the detection of viral gene products late in infection; mu1 limits cell d

84 In this way, we identified a new viral gene product, M84, that conferred protection again

85 however, an immune response directed against viral gene products made by the vector results in toxici

86 ts of the complex are the functions of other viral gene products made later in infection.

87 at accumulation of immediate-early and early viral gene products might be the major stimulus for its

88 Immediate-early viral gene products of human cytomegalovirus (HCMV) are

89 We further found that viral gene products of IE1, pp71, and UL26 play roles in

90 ies of TGB1 function to compartmentalize the viral gene products of PVX infection.

91 novel means not only to study the effects of viral gene products on overall KSHV gene expression and

92 hat independently block apoptosis induced by viral gene products or exogenous agents.

93 a key role in blocking apoptosis induced by viral gene products or exogenous agents.

94 scription in the presence and absence of any viral gene products or viral DNA replication and determi

95 scription in the presence and absence of any viral gene products or viral DNA replication, (ii) the r

96 ous designation of het DNA as the source for viral gene products potentially encoded by both.

97 T cell responses to epitopes in m139 and M38 viral gene products predominate.

98 Consequently, E4 viral gene products present in DeltaE1 or DeltaE1 DeltaE

99 In this work, we identify two viral gene products required for postentry tropism in en

100 rves as the transcriptional template for all viral gene products required for replication.

101 transcriptionally active and to identify the viral gene product responsible for stabilization and ina

102 pe I latency, raising the possibility that a viral gene product(s) expressed in type III latency migh

103 he host immune response by encoding specific viral gene product(s).

104 These data describe a latent phase viral gene product targeted by CTL that may be relevant

105 ced MHC class II expression primarily by the viral gene products targeting CIITA and additionally by

106 however, several studies have implicated the viral gene product, Tax.

107 ucleocapsid (N) protein is a multifunctional viral gene product that encapsidates the RNA genome and

108 coreceptor, the viral envelope, and another viral gene product that govern postentry steps of virus

109 y in individual baboons, the identity of the viral gene product that is the major target of cellular

110 It also carries a version of gamma(1)34.5 (a viral gene product that promotes the dephosphorylation o

111 KK3 is the first viral gene product that subverts the trafficking of a ho

112 es simplex virus type 1 (HSV-1) are the only viral gene products that accumulate to abundant levels i

113 properties and interactions between host and viral gene products that can be exploited for the develo

114 culture evolution approach to identify other viral gene products that functionally interact with pUL3

115 he ability to enhance viral transmission) of viral gene products that interfere with antigen presenta

116 ominent feature of HCMV is the wide range of viral gene products that it encodes which function to mo

117 ent betaherpesvirus that encodes a number of viral gene products that modulate cellular antiviral sig

118 ecause it prevents excessive accumulation of viral gene products that trigger cell death.

119 e generally believed to depend upon a single viral gene product, the nuclear protein EBNA-1.

120 Among the seven known viral gene products, the envelope glycoprotein (GP) alon

121 ture-function studies of genetically diverse viral gene products, the generation of subtype-specific

122 Among the influenza viral gene products, the hemagglutinin (HA) glycoprotein

123 Although Nef is the viral gene product used by most simian immunodeficiency

124 -1, viral infection or expression of certain viral gene products, UV irradiation, B or T cell activat

125 uces apoptosis, we examined whether specific viral gene products were able to induce cell death.

126 Vpr is a 15-kDa late viral gene product, which is assembled in the virion and

127 viruses results in extensive interactions of viral gene products with macromolecular pathways of the

128 The interaction of viral gene products with the MHC class II pathway, howev

129 irus is produced, is mainly regulated by the viral gene product, Zta.