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1 uman influenza to systematically investigate viral interference.
2 equired for degradation of target-RNA during viral interference.
3 ic cell activation that can be the target of viral interference.
4 activity by AAV2 constitutes a novel form of viral interference.
5  six exhibited an amphotropic host range and viral interference.
6 : (1) birth rates, (2) temperatures, and (3) viral interference.
7 ysregulated host immune responses and active viral interference.
8 on of viruses were important determinants of viral interference.
9  primary virus infection was associated with viral interference after the secondary challenge.
10 x, by p12(I) represents a novel mechanism of viral interference and disrupts the intracellular traffi
11 irus and virus-virus interactions, including viral interference and genetic recombination, which cann
12                                              Viral interference and innate antiviral immune mechanism
13 of RNA interference (RNAi) as a mechanism of viral interference and is a demonstration of ERV exaptat
14 orm enables rapid discovery of mechanisms of viral interference and the identification of potential t
15 various applications such as gene knockdown, viral interference, and diagnostics.
16                                         This viral interference appears to be independent of any anti
17 al resistance include host genetic variants, viral interference, cross-protective natural antibodies,
18                                              Viral interference did not occur in mosquito cells.
19 by monovalent immunizations, suggesting that viral interference did not occur in recipients of the te
20 at the pandemic H1N1 virus has the strongest viral interference effects with RSV, aligning with a pre
21 mic infection, modulated by pseudotyping and viral interference, facilitates a stepwise mechanism of
22 and comparing different models, we find that viral interference from influenza is the only mechanism
23 d) or FRA, was characterized with respect to viral interference group, host range, complete genome se
24                   Previous evidence based on viral interference has strongly suggested that the type
25 e same cells-a therapeutic strategy known as viral interference-has recently generated a lot of inter
26                                Mechanisms of viral interference have not been characterized for human
27  sustained viral response (SVR), implicating viral interference in host lipid metabolism.
28 f HBV/HDV infection to exploit mechanisms of viral interference in human hepatocytes and to test the
29 s genome may result in efficacious molecular viral interference in mosquito cells and, more important
30 ce to ecotropic MuLVs appears to result from viral interference involving binding of the endogenously
31                                              Viral interference is characterized by the resistance of
32                      We found that efficient viral interference is established within 24 h of infecti
33                                              Viral interference is important in understanding respira
34 rus interactions, particularly manifested by viral interference mechanisms at different scales.
35 indicated that immune mechanisms rather than viral interference mediated protection.
36  inhibiting a coexisting oncogenic virus via viral interference or immune cross-reaction.
37 ing population susceptibility, mobility, and viral interference should be examined in future studies.
38                                              Viral interference was observed when the interval betwee
39                                     To study viral interference, we evaluated cases of RSV and HRV co
40 r data reveal a new cbVG-driven mechanism of viral interference where cbVGs induce PKR-mediated trans
41 clearance from the nasopharynx and allow for viral interference with antibacterial immune responses,
42                            Here, we focus on viral interference with antigen presentation; in particu
43 nduced through these signaling pathways, but viral interference with critical proximal receptor inter
44 ning the activation of IRF-3 as a target for viral interference with ISG induction.
45                       This function overcame viral interference with MHC class I antigen presentation
46 his finding represents a novel mechanism for viral interference with NKG2D and sheds light on interce
47 hood of reinfection raise the possibility of viral interference with other mouse models of disease.
48                                              Viral interference with secretory cargo is a common mech
49 rporating both an innate immune response and viral interference with that response.
50 -priming overrides the effects of cis-acting viral interference with the class I Ag presentation path
51 hallenging previously proposed mechanisms of viral interference with the immune system.
52                                 We evaluated viral interference with the ultimate step in cytotoxic T
53 is of genes encoding antiviral proteins, and viral interference with this pathway.