ライフサイエンスコーパス: viral membrane protein

1 ere replaced only by TM domains from related viral membrane proteins.

2 age or glycosylation, in any of the numerous viral membrane proteins.

3 domains (TMDs) of cell surface receptors and viral membrane proteins.

4 phosphorylation alters N's interaction with viral membrane proteins.

5 In addition, A27 associates with the viral membrane protein A17 to anchor to the viral membra

6 rc-dependent tyrosine phosphorylation of the viral membrane protein A36R.

7 ever, axonal entry and localization of other viral membrane proteins and endogenous cellular proteins

8 nvelopment depends upon interactions between viral membrane proteins and tegument proteins that encru

9 teine-containing domains of the E10R and L1R viral membrane proteins and the glutaredoxin are in the

10 Three viral membrane proteins are required for efficient anter

11 outside-in signaling cascade induced by the viral membrane protein B5R is required to potently activ

12 tein controls axonal localization of diverse viral membrane proteins but not that of capsid or tegume

13 lization and nanoparticle incorporation of a viral membrane protein complex from the virus membrane.

14 The viral membrane protein composition of HSV-1 DeltagC is e

15 Fusion is mediated by E1, a viral membrane protein containing the putative fusion pe

16 Immunization with influenza and HIV viral membrane proteins displayed on WAEVs elicits produ

17 xed with the genomic RNA, interacts with the viral membrane protein during virion assembly, and plays

18 ow pH in the endosome and is mediated by the viral membrane protein E1.

19 ells but did result in specific decreases in viral membrane protein expression and assembly, leading

20 fluenza virus entry but play direct roles in viral membrane protein expression and assembly.

21 rticle, which results in dissociation of the viral membrane protein from the ribonucleo-protein core.

22 We also show that the viral membrane proteins gE, gI, and US9, which have impo

23 At least three viral membrane proteins (gE, gI, and Us9) are necessary

24 The influenza viral membrane protein hemagglutinin (HA) is required at

25 Entry is mediated by the viral membrane protein hemagglutinin (HA), which trigger

26 was linked to the capsid by many copies of a viral membrane protein in the mature infectous virus.

27 read of infection and may disrupt packing of viral membrane proteins in lipid rafts, an essential ste

28 lustrate exciting opportunities to visualize viral membrane proteins in their native and possibly tra

29 undly decreased the stability of a subset of viral membrane proteins including those comprising the e

30 ontaneously "uncoated" with diffusion of the viral membrane proteins into the host plasma membrane an

31 poration of cellular as well as heterologous viral membrane proteins into the SIV envelope and may be

32 nervous system correlated with the amount of viral membrane proteins localized to axons.

33 ional assays, we show that the nonstructural viral membrane protein nsp4 is the key pore organizer, s

34 ormation changes and reorganization of these viral membrane proteins occur during the transition from

35 dies suggest that antibodies to two or three viral membrane proteins optimally derived from the outer

36 s repressed, A11 did not colocalize with any viral membrane proteins or associate with membranes.

37 The viral membrane proteins P3 and P6 are organized into a l

38 For a number of viral membrane proteins, Th cell epitopes are localized

39 virus (PRV) glycoprotein E (gE) is a type I viral membrane protein that facilitates the anterograde

40 tein (F) is highly conserved and is the only viral membrane protein that is essential for infection.

41 erpesvirus envelope protein Us9 is a type II viral membrane protein that is required for anterograde

42 s are short ( approximately 100 amino acids) viral membrane proteins that form oligomers of a defined

43 Fusion is mediated by viral membrane proteins through their acid-dependent con

44 oprotein B, required for membrane fusion, or viral membrane protein Us9, required for sorting virions

45 e selective as this mutant did not prevent a viral membrane protein, VSVGtsO45 or wild-type pendrin f

46 We showed that the highly conserved A9 viral membrane protein was inserted into the ER of uninf

47 he TM domains (TMDs) of immune receptors and viral membrane proteins, we have established a robust pr

48 singly, capsid and tegument proteins but not viral membrane proteins were detected in axons.