ライフサイエンスコーパス: viral replication compartment

1 rs assisting VRC assembly and genesis of the viral replication compartment.

2 nrichment of phosphatidylethanolamine in the viral replication compartment.

3 ic reticulum-derived COPII vesicles into the viral replication compartment.

4 ymerase and redistributes to the perinuclear viral replication compartment.

5 ces and optimal lipid composition within the viral replication compartment.

6 hijack the Rab5-positive endosomes into the viral replication compartments.

7 -fold more rapidly than the wild type within viral replication compartments.

8 inetics, and localizes in the nucleus within viral replication compartments.

9 is-dependent manner and localized to nuclear viral replication compartments.

10 early-late kinetics and localized to nuclear viral replication compartments.

11 1 in normal human cells and are localized to viral replication compartments.

12 ruit cdk4 initially to ND10 and later to the viral replication compartments.

13 ear translocation of the Mediator complex to viral replication compartments.

14 0 chaperone machinery are formed adjacent to viral replication compartments.

15 ansfected and infected cells and is found in viral replication compartments.

16 rly in infection and is later recruited into viral replication compartments.

17 ed genomes preferentially progressed to form viral replication compartments.

18 tributed cotransfected cellular p53 into the viral replication compartments.

19 Formation of viral replication compartments also appeared normal.

20 ilarly interfering with the formation of the viral replication compartment and also targeting a prote

21 of the cell nucleus including formation of a viral replication compartment and chromatin marginalizat

22 n to elicit the formation of a large nuclear viral replication compartment and marginalization of the

23 To investigate the relationship between viral replication compartments and genetic exchange, we

24 FANCD2 relocalized to viral replication compartments, and FANCI-D2 interacted

25 In sequence, the ND10 bodies become viral replication compartments, and ICP0, a viral E3 lig

26 ment of beta-catenin to the host nucleus and viral replication compartments, and is required for late

27 not express late genes, while cells without viral replication compartments are incapable of both DNA

28 Hsp90 is found within viral replication compartments as well as in the Hsp70/H

29 Our data showed that the formation of the viral replication compartment at late infection resulted

30 d PK generates high levels of ATP within the viral replication compartment at the expense of a reduct

31 nitially become juxtaposed to ND10, and then viral replication compartments develop from the ND10-ass

32 we found that the UL79 protein localized to viral replication compartments during HCMV infection.

33 e assembly of ICP8 filaments in vitro, block viral replication compartment formation and virus produc

34 machinery proteins in the initial stages of viral replication compartment formation.

35 e in HSV-infected cells and are recruited to viral replication compartments; furthermore, short hairp

36 tion and ultimately become incorporated into viral replication compartments, (ii) each of the D cycli

37 ncy; a subset of RSK1/RSK2 is present in the viral replication compartment in the nucleus.

38 t UL84 also colocalized with UL44 and IE2 in viral replication compartments in infected cells.

39 eins and co-opted host proteins within large viral replication compartments in the cytosol of infecte

40 A number of these proteins accumulate in viral replication compartments in the infected cell nucl

41 9 h postinfection (hpi), UL32 accumulated in viral replication compartments in the nucleus of the hos

42 t of essential host transcription factors in viral replication compartments in the nucleus, away from

43 All these cellular proteins accumulate in viral replication compartments in the nucleus, indicatin

44 omatin remodeling factors are recruited into viral replication compartments indicates that chromatin

45 We also show that enrichment of PE in the viral replication compartment is assisted by actin filam

46 In addition, the number of viral replication compartments is significantly higher i

47 full HSV-1 reactivation and was detected in viral replication compartments of reactivating neurons.

48 pends on recruitment of host components into viral replication compartments or organelles.

49 genome composition, structure, size, or the viral replication compartment play a less obvious role.

50 During Herpes Simplex Virus infection, viral replication compartments (RCs) efficiently enrich

51 ed, or otherwise unwanted proteins away from viral replication compartments, sites of robust transcri

52 ntration of eIF4E and eIF4G within cytosolic viral replication compartments surrounded by PABP.

53 forms of both prereplication foci and active viral replication compartments that display an appearanc

54 eling of nascent viral DNA to image aberrant viral replication compartments that form in the presence

55 r solubilization and localization within the viral replication compartment, (v) was essential for the

56 nse proteins, which are localized in nuclear viral replication compartments, were reduced in the siX3

57 scaffolding protein, ATRIP, are recruited to viral replication compartments, where they play benefici

58 endosomes allows TBSV to build a PE-enriched viral replication compartment, which is needed to suppor