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1 survival, represents a novel property for a viral structural protein.
2 substrate, or the molecular interactions of viral structural proteins.
3 ses an antibody response primarily targeting viral structural proteins.
4 ed by dramatic conformational changes within viral structural proteins.
5 icles (VLVs) that contain VSV G but no other viral structural proteins.
6 patches and extensions contained all of the viral structural proteins.
7 d does not require the coexpression of other viral structural proteins.
8 revealed they lacked gB but contained other viral structural proteins.
9 d macrophages by regulating the stability of viral structural proteins.
10 block viral DNA replication and synthesis of viral structural proteins.
11 rus assembly site for interaction with other viral structural proteins.
12 omeric state and spatial relationships among viral structural proteins.
13 d cells, requiring the nuclear import of all viral structural proteins.
14 ng in a brief and limited synthesis of HIV-1 viral structural proteins.
15 ar masses similar to those of phosphorylated viral structural proteins.
16 ber mutants were defective for processing of viral structural proteins.
17 the cellular and humoral immune response to viral structural proteins.
18 l assembly, and depends on the expression of viral structural proteins.
19 d hemagglutinin protein along with the other viral structural proteins.
21 lation in the 0 and +1 frames to produce the viral structural proteins and a +1 overlapping open read
22 t cells require dynamic interactions between viral structural proteins and a variety of cellular fact
25 In contrast to vRNA hybridization results, viral structural proteins and glycoproteins, evaluated b
26 idence for a potential mechanism linking the viral structural proteins and host PS receptor usage dur
27 gE-null mutants produce wild-type levels of viral structural proteins and infectious virions in the
28 and primary neuronal cells that concentrates viral structural proteins and is a major site of capsid
29 tein that is essential for axonal sorting of viral structural proteins and is highly conserved among
30 A virus (IAV), the virus encodes eight main viral structural proteins and multiple accessory nonstru
32 rus replication cycle is the assembly of the viral structural proteins and the packaging of the eight
34 D4(+) T cells that recognize epitopes in the viral structural proteins and thus can provide direct he
35 ers the cellular immune responses to a human viral structural protein, and that these effects may con
36 promoter, which regulates expression of the viral structural proteins, and a second internal promote
37 l, these genomes produce together all of the viral structural proteins, and cells release a combinati
38 replicates its genome, expresses all of the viral structural proteins, and releases viral particles
40 ble-membrane vesicles for RNA synthesis, and viral structural proteins assemble virions at the ER-Gol
41 hance the immunogenicity of this nonsecreted viral structural protein at the B and T cell level, we c
42 synthesis and limiting the production of the viral structural proteins but had little effect on viral
43 code the viral genome and the genes encoding viral structural proteins, by binding to and oligomerizi
45 merization of the Gag polyprotein, the major viral structural protein capable of forming virus-like p
47 Virus-like particles (VLPs), comprised of viral structural proteins devoid of genetic material, ar
48 try blocks the translation of ORF6 and other viral structural proteins due to inefficient subgenomic
49 IgG), IgG avidity, and IgG response to the 3 viral structural proteins (E1, E2, and C), reflected by
50 antiviral agents could be applied to certain viral structural proteins, enzymes, and other factors or
52 otein, and contains 60 copies of each of the viral structural proteins F and G, which comprise the sh
56 interact with the nucleocapsid domain of the viral structural protein Gag and is incorporated in sign
57 on diminishes nanoscale coclustering between viral structural protein Gag and the three transmembrane
59 1 (HIV-1) particle assembly mediated by the viral structural protein Gag occurs predominantly on the
60 been observed as its colocalization with the viral structural protein Gag or its accumulation in viru
62 B sequences for the genes encoding the major viral structural protein (Gag) and two regulatory protei
63 ugh a specific interaction between the major viral structural protein, Gag, and an RNA packaging sign
65 bis virus genomes (replicons) which lack the viral structural protein genes and contain heterologous
68 ng may effect cellular immune responses to a viral structural protein in the context of genetic immun
69 ruits the ribosome and drives translation of viral structural proteins in a factor-independent manner
70 gE is required for wild-type localization of viral structural proteins in axons of infected neurons.
72 e interaction of envelope protein with other viral structural proteins in the virus assembly and matu
73 an lymphocytes, while reducing the yields of viral structural proteins, infectivity, and tumor necros
74 However, it is not understood how Gag (the viral structural protein) interacts with these signals t
75 protein is essential for the organization of viral structural proteins into biomolecular condensates.
77 (ET) domains of gE/gI promote the sorting of viral structural proteins into proximal axons to begin a
78 ts indicate that reduced axonal targeting of viral structural proteins is a compelling explanation fo
80 and by enzyme immunoassays with recombinant viral structural protein (K8.1) and latent protein (late
81 of plasmids expressing all of the remaining viral structural proteins led to a substantial increase
83 likely that the cellular immune responses to viral structural proteins may be important for eradicati
84 ased cellular immune response to hepatitis B viral structural proteins may be important for recovery
85 lar model is that deposition of ubiquitin on viral structural proteins mediates class E machinery rec
86 infection, and neither the synthesis of the viral structural proteins nor the presence of the other
87 y mutant p12 show prolonged association with viral structural proteins nucleocapsid (NC) and capsid (
90 cycle, further highlighting the paradigm of viral structural proteins playing additional functional
92 -strand molar ratio of approximately 8:1 and viral structural proteins S, M, and N, and 65% were in a
93 sults provide a rare example of an oncogenic viral structural protein, show that interaction of the v
94 uring SARS-CoV-2 infection, 3DB recruits the viral structural proteins spike (S) and membrane (M) and
96 ile retaining the normal complement of other viral structural proteins such as the neuraminidase as w
99 This appears to be the first example of a viral structural protein that is also involved in the tr
101 SARS-CoV-2 Nucleocapsid protein (N) is a viral structural protein that packages the 30 kb genomic
102 assembled structures that are made up of the viral structural proteins that mimic the morphology of v
104 Forest virus RNA replicon encoding a single viral structural protein, the vesicular stomatitis virus
105 y assays that measure only antibodies to the viral structural proteins, the majority of such patients
106 us that lacks the open reading frames of two viral structural proteins: the envelope (E) and membrane
107 and to differentially regulate expression of viral structural proteins, their replication was made de
108 on in differentiated neurons by facilitating viral structural protein translation.IMPORTANCE Mosquito
110 imeric viruses were used to demonstrate that viral structural protein VP1 determines growth in the sp
111 ckaged within an AAV capsid made up of three viral structural proteins (VP1, VP2, and VP3) in an appr
113 avage of the viral outer capsid, the fate of viral structural proteins was assessed by sodium dodecyl
116 g specific antibody and T cell responses for viral structural proteins, with their T cell responses d