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1 cies (n = 30) and genera (n = 14), including viridans group streptococci.
2 ci and group D streptococci from non-group D viridans group streptococci.
3 coccus bovis, and 110 strains of non-group D viridans group streptococci.
4 entiated Streptococcus pneumoniae from other viridans group streptococci.
5 genus/species or organism group level (e.g., viridans group streptococci), 11% positivity with discor
6 The most frequent causal microorganisms were viridans group streptococci (18.6%), Staphylococcus aure
7 y strains within the sanguis group and other viridans group streptococci, 42 strains from seven speci
8    Lectin-mediated interactions between oral viridans group streptococci and actinomyces may play an
9 the occurrence and expression of fimA in the viridans group streptococci and enterococci.
10           The LraI family of lipoproteins in viridans group streptococci and other bacteria have been
11 erium as is currently the case, for example, viridans group streptococci and Staphylococcus aureus.
12 lture other than Staphylococcus epidermidis, viridans group streptococci, and Corynebacterium spp.
13 es of penicillin resistance, particularly in viridans group streptococci, and resistance to multiple
14 nvasion of the cardiovascular endothelium by viridans group streptococci are integral events in the p
15 ization of the cardiovascular endothelium by viridans group streptococci can result in infective endo
16        During episodes of dental bacteremia, viridans group streptococci encounter platelets.
17 tritionally variant streptococci, a class of viridans group streptococci first isolated from patients
18                                         Oral viridans group streptococci, growing in chemically defin
19               Hydrogen peroxide generated by viridans group streptococci has an antagonistic effect o
20 k, streptococcus-infective endocarditis, and viridans group streptococci-infective endocarditis), whe
21                                              Viridans group streptococci interact with host cells by
22 tion of the tooth surface by actinomyces and viridans group streptococci involves the attachment of t
23 trate that mannosidase production within the viridans group streptococci is more widely distributed t
24 bited platelet aggregation by two strains of viridans group streptococci known to induce aggregation
25 arities between Streptococcus pneumoniae and viridans group streptococci may result in misidentificat
26 y the misidentification of pneumococcus-like viridans group streptococci (P-LVS) as Streptococcus pne
27                                              Viridans group streptococci (VGS) are a heterogeneous gr
28                                          The viridans group streptococci (VGS) are a heterogeneous gr
29      Due to the difficulty in distinguishing viridans group streptococci (VGS) by phenotype, analysis
30                                          The viridans group streptococci (VGS) have been suggested to
31                The incidence of IE caused by viridans group streptococci (VGS) in the United States a
32 rious infection due to beta-lactam-resistant viridans group streptococci (VGS) is a major factor driv
33                            Identification of viridans group streptococci (VGS) to the species level i
34 solates; 396 group B streptococci [GBS]; 369 viridans group streptococci [VGS]; 290 beta-hemolytic st
35 by all currently recognized species of human viridans group streptococci was determined using an assa
36 eroids, Micrococcus spp., Bacillus spp., and viridans group streptococci) when isolated from blood cu
37                       The alpha-hemolysin of viridans group streptococci, which causes greening of in