1 attributed to a genotype; however, in simple virilizing CAH, we observe wide phenotypic variability,

2        However, many affected girls are born virilized, despite low circulating androgens.

3  98% reduction in enzyme activity and simple virilizing disease.

4 stosterone levels in women, thus producing a virilizing effect.

5                       However, this does not virilize female fetuses because androgens are aromatized

6 the salt-wasting form and 13 with the simple virilizing form), 39 age-matched normal subjects, and 20

7 sorder (P = .04), including 79.7% for simple virilizing forms and 32.4% for nonclassic forms.

8                               Female fetuses virilize if their adrenals secrete excessive levels of a

9 wn forms of CAH, namely salt wasting, simple virilizing, or nonclassical CAH.

10 ed either with severe salt-wasting or simple virilizing phenotypes or with milder nonclassical phenot

11 potent hAR binding and agonist activity, low virilizing potential, and good pharmacokinetic profiles

12 lly, affected female newborns are profoundly virilized (Prader score of 4/5), and both genders displa

13         A patient carrying this mutation has virilized spontaneously at puberty, so in vivo evidence

14  the patient with cortisol-secreting tumor), virilizing symptoms (in the patient with testosterone-se

15 antiandrogen for inoperable prostate cancer, virilizing syndromes in women, and the inhibition of sex

16         In the pediatric population, though, virilizing tumors carry a better survival than non-funct