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1 esult that was positive for varicella-zoster virus DNA.
2  than the representation of dGMP in vaccinia virus DNA.
3 ved peptide pools and absence of torque teno virus DNA.
4 tion PCR were performed to screen for herpes virus DNA.
5  patient's lesion tested positive for cowpox virus DNA.
6 nged lowering of serum levels of hepatitis B virus DNA.
7 h conventional measures of serum hepatitis B virus DNA.
8 ed onto unintegrated Moloney murine leukemia virus DNAs.
9 assays that can detect as low as 16 pM Ebola Virus DNA, 0.01ng/mL carcinoembryonic antigen (CEA), and
10  the 328 patients with data for Epstein-Barr virus DNA, a detectable viral DNA titre was an independe
11 polymerase chain reaction (PCR) assay for BK virus DNA, a retrospective analysis was done of sequenti
12                            However, for both viruses, DNA-A:DNA-B ratios acquired by whiteflies diffe
13                 Thus, B12 restricts vaccinia virus DNA accumulation in part by repressing the ability
14 s autointegration of Moloney murine leukemia virus DNA, also plays an indirect role in generating the
15 erived from the terminal regions of vaccinia virus DNA and contained in a superhelical plasmid, into
16 sed on restriction enzyme digest patterns of virus DNA and growth rates both in vitro and in vivo.
17               The peak levels of hepatitis B virus DNA and hepatitis B core-related antigen after ces
18                       Rebound of hepatitis B virus DNA and hepatitis B core-related antigen was assoc
19 10(4) cellular genomes, whereas Epstein-Barr virus DNA and HIV-1 DNA were detected in 16 and 22 sampl
20 patitis B virus infection, serum hepatitis B virus DNA and liver biochemical test levels should be or
21   (v) Despite the reduced recoveries of LAT- virus DNA and productive-cycle transcripts in reactivati
22                                    Borealpox virus DNA and serologic evidence for past orthopoxvirus
23 d capsid has formed, it is packaged with the virus DNA and transported to the cytoplasm where further
24 ems, in which an immune response eliminating virus DNA and/or RNA is launched first, but then, if it
25 mutation was transferred into wild-type (wt) virus DNA, and the UL25 mutant ts1249 was isolated and c
26 wide variety of antigens including bacteria, viruses, DNA, and proteins due to the simplicity of thei
27 sease has been made using cloned hepatitis B virus DNA as a transgene in a severe combined immunodefi
28                   In arrested cells no foamy virus DNA band was detected in cells harvested at 1 or 7
29 ransactivator IE62, ORF 29 encodes the major virus DNA binding protein, and ORF 21 encodes a protein
30 iation with sorting nexin 17, to ensure that virus DNA bound to L2 is recycled through the trans-Golg
31 igned specifically for detection of smallpox virus DNA, but all viruses of the genus Orthopoxvirus te
32     Weekly plasma samples were tested for BK virus DNA by polymerase chain reaction (PCR).
33 y testing lesions swabs for varicella zoster virus DNA by polymerase chain reaction).
34             Once "uncoated" in this way, the virus DNA can be transcribed and replicated.
35 e host range among haloarchaea, and purified virus DNA can cause an infection after transformation in
36 herpesvirus 6 and human herpesvirus 7 and BK virus DNA, CMV disease, and acute rejection.
37    There were doses of 01/PEME for which the virus DNA concentration in the tumor increased over time
38  virus replication by >1000-fold increase in virus DNA copies over time.
39 e ability to detect and monitor Epstein-Barr virus DNA copy number in a variety of settings.
40 tem lymphoma, and monitoring of Epstein-Barr virus DNA copy number in spinal fluid may be useful in a
41                       The quantity of latent virus DNA correlates with and may be a major determinant
42 infected cell cultures and tissues, smallpox virus DNA could be detected in a background of human DNA
43                  The utility of Epstein-Barr virus DNA detection and quantification in the serum or p
44                   Urine was collected for JC virus DNA detection.
45 d pretreatment levels of plasma Epstein Barr virus DNA (EBV-DNA), as determined by quantitative real-
46 linical presentation and demonstration of JC virus DNA either in the CSF or by histopathology, is an
47 9 which binds to integrated murine leukaemia virus DNA elements and recruits KAP1 to repress them.
48 A binding through specific contacts with the virus DNA ends in the 3'-processing and ST reactions.
49 ve detection of DNA, including a hepatitis B virus DNA fragment.
50              Since the size range of sheared virus DNA fragments governs the limit of accurate protei
51                  Terminal repeat analysis of virus DNA from a KS lesion suggests a monoclonal expansi
52 ability of Rep40 to package adeno-associated virus DNA in a tissue culture-based assay.
53 rase chain reaction [PCR] for herpes simplex virus DNA in cerebrospinal fluid) were randomized to rec
54 chain reaction amplification of Epstein-Barr virus DNA in cerebrospinal fluid, 18F-fluoro-deoxyglucos
55  demonstrated 2.75 times less herpes simplex virus DNA in cyclic-AMP treated BeWo cells, but 2.0 to 7
56 B patients with detectable serum hepatitis B virus DNA in European tertiary referral centers.
57 n plants that did become infected, levels of virus DNA in mutants and wild-type did not differ signif
58  on magnetic resonance imaging regressed; JC virus DNA in plasma, likely originating from the brain b
59 It is concluded that quantitative PCR for BK virus DNA in serum is useful both for identifying transp
60                                 Epstein-Barr virus DNA in serum or plasma has been found in infectiou
61  association of the presence of Epstein-Barr virus DNA in systemic lupus erythematosus patients compa
62                                           JC virus DNA in the CSF and peripheral blood was quantified
63 rospinal fluid positivity for herpes simplex virus DNA in the polymerase chain reaction analysis.
64 ticle reporting the presence of Epstein-Barr virus DNA in the vitreous of a patient with Vogt-Koyanag
65  full-blown AIDS without evidence of vaccine virus DNA in tissues.
66 , but 2.0 to 7.4 times more adeno-associated virus DNA in treated cells.
67 was negative for human polyomavirus 2 (or JC virus) DNA in the cerebrospinal fluid.
68 was negative for human polyomavirus 2 (or JC virus) DNA in the CSF.
69                                          The virus DNA inserts were amplified, quantitated, and spott
70 s study, we investigated whether hepatitis B virus DNA integration occurs preferentially at sites of
71 enetics and biochemistry of avian and murine virus DNA integration, but the pace of discovery increas
72                     By transfecting vaccinia virus DNA into cells infected with ectromelia virus and
73  frequent integration of mouse mammary tumor virus DNA into chromosomes, implicating the gene in the
74                 The entry of murine leukemia virus DNA into the nucleus occurs only upon dissolution
75 e immunized individuals over age 60, vaccine virus DNA is shed in saliva up to 4 weeks.
76                 In this suppressed state the virus DNA is vulnerable to mutagenic events that affect
77                                      In many viruses, DNA is confined at such high density that its b
78 not catalyze base changes in murine leukemia virus DNA, it can be recovered from these virus particle
79 ot on treatment but with a serum hepatitis B virus DNA level >200,000 IU/mL during the third trimeste
80 minotransferase and 37% had >/=1 hepatitis B virus DNA level assessed annually.
81                         Elevated hepatitis B virus DNA levels in patients in their 40s with perinatal
82         We therefore compared herpes simplex virus DNA levels in specimens before and after 16 months
83 containing only the 298-amino acid Chlorella virus DNA ligase (a 'minimal' eukaryotic ATP-dependent l
84                                    Chlorella virus DNA ligase (ChVLig) has pluripotent biological act
85                                    Chlorella virus DNA ligase (ChVLig) is a minimized eukaryal ATP-de
86                                    Chlorella virus DNA ligase (ChVLig) is an instructive model for me
87 the conformational dynamics of the Chlorella virus DNA ligase (ChVLig), a minimized eukaryal ATP-depe
88 nt ligation of RNA-splinted DNA by Chlorella virus DNA ligase (PBCV-1 DNA ligase).
89 NA) detection method that utilizes Chlorella virus DNA ligase (SplintR((R)) Ligase).
90                          Using the Chlorella virus DNA ligase as a proof of principle, we recapitulat
91                  The 552 amino acid vaccinia virus DNA ligase consists of three structural domains de
92  roles of conserved amino acids of Chlorella virus DNA ligase during the third step of the ligation p
93                                     Vaccinia virus DNA ligase has an intrinsic nick-sensing function.
94         Our findings indicate that Chlorella virus DNA ligase has the potential to affect genome inte
95 ion analysis of the 298 amino acid Chlorella virus DNA ligase indicates that motif VI plays a critica
96                                    Chlorella virus DNA ligase is the smallest eukaryotic ATP-dependen
97                                    Chlorella virus DNA ligase is the smallest eukaryotic ATP-dependen
98                                     Vaccinia virus DNA ligase repairs nicked duplex DNA substrates co
99 )) in the nick joining reaction of Chlorella virus DNA ligase, an exemplary ATP-dependent enzyme.
100  to the mitochondria or expressing Chlorella virus DNA ligase, the minimal eukaryal nick-sealing enzy
101 progressive decrease in the median plasma BK virus-DNA load, and undetectable levels at the last foll
102 CPE, visible intracellular virions, and high virus DNA loads.
103  according to the VR definition (hepatitis B virus DNA &lt;200, < 2000, < 20,000 IU/mL) or duration of o
104                                           JC virus DNA may harbor in CD34+ cells in bone marrow that
105 ion inhibitor, partially reversed the helper virus DNA methylation and restored a portion of vector p
106                                        A pan-virus DNA microarray (Virochip) was used to detect a hum
107 each containing >/=105 copies herpes simplex virus DNA/ml collected a median of 5 months apart (IQR:
108 tained within those 322 samples were variola virus DNA, obtained from purified viral preparations, at
109 ic assay for the rapid detection of smallpox virus DNA on both the Smart Cycler and LightCycler platf
110 oligodendrocytes, which were positive for JC virus DNA on in situ hybridization.
111  varicella (by detection of varicella zoster virus DNA or epidemiological link) from 42 days after th
112  in cells transgenomic for EBV (Epstein Barr Virus) DNA or for the telomerase gene, the large number
113 y been described which use inactivated whole virus, DNA or RNA that express the virus' Envelope (E) g
114    Since this assembly step is unique to the virus, DNA packaging is a novel target for the developme
115 f exonuclease activity of the herpes simplex virus DNA polymerase (Pol) on DNA replication fidelity w
116      The catalytic subunit of herpes simplex virus DNA polymerase (Pol), a member of the B family pol
117 to the role of D4 as a co-factor of vaccinia virus DNA polymerase and allows a better understanding o
118                           The herpes simplex virus DNA polymerase catalytic subunit, which has intrin
119                               Herpes simplex virus DNA polymerase consists of a catalytic subunit, Po
120                                 The vaccinia virus DNA polymerase E9 requires two viral proteins, A20
121  seen even in the presence of herpes simplex virus DNA polymerase inhibitors, but not in the absence
122 he catalytic subunit, Pol, of herpes simplex virus DNA polymerase interacts via its extreme C terminu
123                               Herpes simplex virus DNA polymerase is a heterodimer composed of a cata
124                               Herpes simplex virus DNA polymerase is a heterodimer composed of UL30,
125                                 The vaccinia virus DNA polymerase is inherently distributive but acqu
126                        Although the vaccinia virus DNA polymerase is inherently distributive, a highl
127 n of the catalytic subunit of herpes simplex virus DNA polymerase with the processivity subunit, UL42
128 rase beta (Pol beta) and African swine fever virus DNA polymerase X (ASFV Pol X) with one-nucleotide
129 cently demonstrated that African swine fever virus DNA polymerase X (Pol X) is extremely error-prone
130                      The African swine fever virus DNA polymerase X (pol X), a member of the X family
131 low-fidelity polymerase, African swine fever virus DNA polymerase X (Pol X), and showed that they fol
132 rase beta (Pol beta) and African swine fever virus DNA polymerase X (Pol X).
133 yields of interleukin-12 p40, herpes simplex virus DNA polymerase, and interferon-gamma PCR products
134 e processivity subunit of the herpes simplex virus DNA polymerase, interacts with DNA and promotes pr
135 e processivity subunit of the herpes simplex virus DNA polymerase, UL42, is a monomer and has an intr
136 e processivity subunit of the herpes simplex virus DNA polymerase, UL42, is a monomer in solution.
137 e processivity subunit of the herpes simplex virus DNA polymerase, UL42, is essential for viral repli
138    The processivity factor of herpes simplex virus DNA polymerase, UL42, unlike "sliding clamp" proce
139 f the processivity subunit of herpes simplex virus DNA polymerase, UL42, which is crucial for interac
140  critical for horizontal transmission of the virus, DNA priming/MVA boosting was evaluated for the ab
141       One vaccination with 0.5 microg of CEE virus DNA provided protective immunity for at least 2 mo
142  containing packaging signals from these two viruses, DNA proviruses containing genetic information f
143 e 43 samples that contained purified variola virus DNA ranging in concentration from 1 fg/ microl to
144    Of the 206 samples that contained variola virus DNA ranging in concentrations from 100 fg/ microl
145 a indicated that a host pathway initiated by virus DNA replication and acting through instability mot
146 tiviral role for ANKLE2 in limiting vaccinia virus DNA replication and progeny release through regula
147 s origin-binding domain in the initiation of virus DNA replication by analyzing the biochemical activ
148    Thus, the activity of replicative lefs or virus DNA replication is sufficient to trigger apoptosis
149 The identification of BocaSR and its role in virus DNA replication reveals potential avenues for deve
150                             Visualization of virus DNA replication showed that a UL37-minus mutant wa
151 n of host protein synthesis, suggesting that virus DNA replication triggers inhibition of host biosyn
152   TNF-alpha is shown to suppress hepatitis B virus DNA replication without cell killing by disrupting
153 virus uracil DNA glycosylase is required for virus DNA replication, coupled with an inability to isol
154 D4, and D5) with essential roles in vaccinia virus DNA replication.
155 st DNA damage response (DDR), which promotes virus DNA replication.
156 ively impact virus multiplication, including virus DNA replication.
157 e for cellular genome damage in facilitating virus DNA replication.
158 ocked late gene expression without affecting virus DNA replication.
159                            Inactivated whole virus, DNA, RNA, and vectored vaccine approaches to prev
160                              The hepatitis B virus DNA sequence around the precore region was determi
161 s to interact with naked mouse mammary tumor virus DNA somewhat differently than with chromatin and A
162 substrates typically utilized the end of the virus DNA substrate as the point of integration, whereas
163 anscripts disappeared prior to initiation of virus DNA synthesis (considered early), and 100 transcri
164 enome allowed gamma-H2AX accumulation during virus DNA synthesis and impaired both very late viral ge
165 lef-11, p143, dnapol, and ie-1/ie-0) blocked virus DNA synthesis and late gene expression in permissi
166 ocalization of EAP and ICP4 did not occur if virus DNA synthesis and late gene expression were preven
167 nd 100 transcripts were still detected after virus DNA synthesis begins (considered early/late); (v)
168 ) 133 (36%) of the CDSs were expressed after virus DNA synthesis begins (considered late); and (vi) e
169  227 (62%) of the CDSs were expressed before virus DNA synthesis begins; (iv) these 227 CDSs were gro
170 he effect of the HIV-2IN(IN(2)) mutations on virus DNA synthesis was analyzed by packaging IN(2) muta
171 synthesis is then shut off and replaced with virus DNA synthesis.
172                                  Hepatitis B virus DNA targets in human serum are simultaneously prec
173 ine aminotransferase and 44% had hepatitis B virus DNA testing; hepatitis B e antigen and hepatitis B
174 ition to packing the approximately 103 kb of virus DNA, the PhicrAss001 virion has extensive storage
175                                     Vaccinia virus DNA topoisomerase catalyzes resolution of syntheti
176             We demonstrate that the vaccinia virus DNA topoisomerase, a eukaryotic type I enzyme, cat
177 on resolution reaction catalyzed by vaccinia virus DNA topoisomerase.
178                 Information on the extent of virus DNA transcription and translation in infected tiss
179 article (LNP)-formulated Andes virus or Zika virus DNA vaccines are elevated over unformulated vaccin
180                     We developed two Sindbis virus DNA vaccines encoding the measles virus hemaggluti
181  diverse insects through events triggered by virus DNA (vDNA) replication.
182                                  The Sindbis virus DNA vectors expressed reporter genes in transfecte
183 dent muscle following injection with Sindbis virus DNA vectors.
184 OVID-19 pandemic, which included inactivated virus, DNA viral vectors and mRNA vaccines.
185 , and the prevalence of infection types (RNA virus, DNA virus, bacteria, and fungus).
186 patitis C virus, West Nile virus, and dengue virus), DNA viruses (vaccinia virus and human adenovirus
187 nfection by a range of viruses including RNA viruses, DNA viruses and retroviruses.
188 ution, as well as their determinants, in RNA viruses, DNA viruses and retroviruses.
189                                           JC virus DNA was detectable within cell compartments of nat
190                             Varicella zoster virus DNA was detected 2 months after the outbreak in en
191                                    Monkeypox virus DNA was detected by PCR from vaginal swab samples
192                                        HHV-8 virus DNA was detected by polymerase chain reaction (PCR
193                                    Monkeypox virus DNA was detected in 29 of the 32 persons in whom s
194                                 Epstein-Barr virus DNA was detected in a lymph node initially and the
195 revealed a normal cell count, herpes simplex virus DNA was detected in all samples by polymerase chai
196  this increased to 20 patients (71.4%) as JC virus DNA was detected in cerebrospinal fluid of two add
197                                           BK virus DNA was detected in serum samples from all 4 case
198                              John Cunningham virus DNA was detected in urine of seronegative individu
199 eim, Germany) PCR identification of smallpox virus DNA was developed and compiled in a kit system und
200 t brain, although selection of rearranged JC virus DNA was favored.
201                              John Cunningham virus DNA was found in 75% of genitourinary tissue sampl
202 ents without toxoplasmosis, and Epstein-Barr virus DNA was found in 9 of 14 patients with central ner
203 ys postinfection and remained low even after virus DNA was lost from the cells.
204 reas the size distribution of herpes simplex virus DNA was markedly different.
205                                           JC virus DNA was not detected in CSF or peripheral blood fr
206  cell depletion, showing that the persisting virus DNA was reactivatable.
207 ght hemispheric and brainstem lesions and JC virus DNA was undetectable in his cerebrospinal fluid.
208 short (<10 min) circulating half-life of the virus DNA, we could monitor virus distribution to the tu
209            Two regions of cauliflower mosaic virus DNA were designed as markers to study pathogen tra
210    Serum qHBcrAg, qAnti-HBc, and hepatitis B virus DNA were obtained at TDF initiation and every 6-12
211 mit of detection of 50 aM toward hepatitis B virus DNA with the capability of discriminating a single
212  ends of microtubules and ultimately deposit virus DNA within the host nucleus.

 
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