ライフサイエンスコーパス: virus RNA

1 quantitative assay for 5-5000 copies of zika virus RNA.

2 on to form the viral replicase and replicate virus RNA.

3 SSPE were tested for the presence of measles virus RNA.

4 mized using a synthesized fragment of Dengue virus RNA.

5 ttle or no sequence similarity to the helper virus RNA.

6 o be involved in cyclization of yellow fever virus RNA.

7 does not stimulate the export of hepatitis B virus RNA.

8 en 1915 and 1919 were tested for influenza A virus RNA.

9 e replication and transcription of influenza virus RNA.

10 surface antigen (HBsAg) and hepatitis delta virus RNA.

11 d less efficiently than genomic Rous sarcoma virus RNA.

12 GM-CSF was +0.14 log human immunodeficiency virus RNA.

13 fficient packaging of avian leukosis-sarcoma virus RNA.

14 ng the 3' end of the minus strand of Sindbis virus RNA.

15 ive binding assay with tRNA and brome mosaic virus RNA.

16 emen specimen that tested positive for Ebola virus RNA.

17 imed with other PAMPs, including hepatitis C virus RNA.

18 l samples-in spite of high concentrations of virus RNA.

19 h DI RNAs but not in synthesis of the helper virus RNAs.

20 e transcription and replication of influenza virus RNAs.

21 3'-untranslated region of pea enation mosaic virus RNA 2.

22 spectively; all had undetectable hepatitis C virus RNA 48 weeks after the end of therapy.

23 primary endpoint was absence of hepatitis C virus RNA 6 months posttreatment.

24 Although the virus RNA accumulates to high levels within infected cel

25 proteases p29 and p48 contributes to optimal virus RNA accumulation.

26 pse patients (undetectable serum hepatitis C virus RNA after 24 weeks of peginterferon-ribavirin and

27 t of which included detection of hepatitis D virus RNA among anti-hepatitis D virus seropositive part

28 teroviruses possess specialized functions in virus RNA amplification, virus invasion, and cell-to-cel

29 for ribosome shunting in cauliflower mosaic virus RNA and are well conserved in cIAP2 orthologs.

30 Plasma human immunodeficiency virus RNA and CD4 lymphocyte response to nucleoside reve

31 y reproducible quantification of hepatitis C virus RNA and displayed a nearly 600-fold dynamic range

32 infection, RIG-I-like receptors (RLRs) sense virus RNA and induce MAVS to form prion-like aggregates

33 L segment minigenome systems for analysis of virus RNA and protein features involved in replication.

34 the 3' untranslated region (UTR) of Sindbis virus RNA and relocalizes from the nucleus to the cytopl

35 l; after several years, both hepatitis delta virus RNA and serum HBsAg became undetectable.

36 nts in spleen necrosis virus and human foamy virus RNA and support the model that divergent retroviru

37 nt-independent expression of bovine leukemia virus RNA and supports the hypothesis that SNV RNA conta

38 f 519 donors who were positive for West Nile virus RNA and the removal of more than 1000 potentially

39 C infection, close monitoring of hepatitis C virus RNA and treatment of patients with persistent vire

40 mplexes (RNPs) formed by the hepatitis delta virus RNAs and protein, HDAg, perform critical roles in

41 n of a cellular protein binding to influenza virus RNAs and, importantly, suggest that influenza viru

42 ol dehydrogenase, satellite tobacco necrosis virus RNA, and alfalfa mosaic virus (AMV) 4, were used i

43 transferases, the level of serum hepatitis C virus RNA, and histologic necroinflammatory scores all d

44 NASBA) technique with which small amounts of virus RNA are amplified using a simple water bath.

45 Infection was assessed by measuring plasma virus RNA as well as T and B cell responses.

46 (undetectable [<125 IU/mL] serum hepatitis C virus RNA at 24-week follow-up).

47 efined as undetectable levels of hepatitis C virus RNA at 4 weeks) and were eligible to participate.

48 , reverse transcriptase PCR identified Ebola virus RNA at a higher level in CSF (cycle threshold valu

49 e (2 log10 reduction in level of hepatitis C virus RNA at week 12; n=466), and undetectable hepatitis

50 eek 12; n=466), and undetectable hepatitis C virus RNA at weeks 20 (n=320), 48 (end of treatment, n=2

51 replication, internal polyadenylation of B19 virus RNAs at (pA)p is favored in cells which are both p

52 microwave heating of a suspension of a model virus, RNA bacteriophage MS2, 13 chemical digestion prod

53 samples to screen blood donors for West Nile virus RNA began in July 2003.

54 For example, the human immunodeficiency virus RNA-binding protein Rev associates with the solubl

55 nt relationships with human immunodeficiency virus RNA blood levels.

56 These data show that heterologous virus RNAs (BSMV) can serve as primer donors for MStV mR

57 er unrelated IRES (from encephalomyocarditis virus RNA), but did not affect the 5'-end-dependent tran

58 he packaging signals for all eight influenza virus RNAs, but it lost the ability to independently rea

59 cipants' semen samples were tested for Ebola virus RNA by real-time RT-PCR and participants received

60 Duration of detection of Ebola virus RNA by real-time RT-PCR varies by individual and m

61 s from all five cases were positive for Zika virus RNA by RT-PCR, and sequence analyses showed highes

62 ) with a less than 2-log drop in hepatitis C virus RNA by week 4.

63 prevent the free reassortment of influenza A virus RNAs by rewiring their packaging signals.

64 Virus RNA clearance time lengthened with disease progres

65 tis C virus infection with serum hepatitis C virus RNA concentrations of at least 5 log10 IU/mL, crea

66 10.6-fold even at relatively low hepatitis C virus RNA concentrations.

67 Rous sarcoma virus RNA contains a negative regulator of splicing (NRS

68 ts of virus load >500 human immunodeficiency virus RNA copies/mL.

69 Virus RNA could be detected for up to 33 days in vaginal

70 ylcytidine (1) was designed as a hepatitis C virus RNA-dependent RNA polymerase (HCV RdRp) inhibitor.

71 The influenza virus RNA-dependent RNA polymerase (RdRP) cleaves the 5'

72 subgenomic RNA synthesis by the brome mosaic virus RNA-dependent RNA polymerase (RdRp), provided that

73 For the brome mosaic virus RNA-dependent RNA polymerase (RdRp), we determined

74 rs (IC(50) = 0.08-3.8 microM) of hepatitis C virus RNA-dependent RNA polymerase (RdRp).

75 By using a purified dengue virus RNA-dependent RNA polymerase and a subgenomic 770-

76 Influenza virus RNA-dependent RNA polymerase consists of three vir

77 ion stability and poor activity of the avian virus RNA-dependent RNA polymerase in human cells.

78 More specifically, we show that the Nodamura virus RNA-dependent RNA polymerase interacts with the ou

79 The hepatitis C virus RNA-dependent RNA polymerase NS5B is responsible f

80 The Sindbis virus RNA-dependent RNA polymerase nsP4 possesses an ami

81 would support the notion that the influenza virus RNA-dependent RNA polymerase undergoes a conformat

82 cessing of heterotypic segments by influenza virus RNA-dependent RNA polymerase, an inhibitory effect

83 Influenza A virus RNA-dependent RNA polymerase, purified from virion

84 erstood but is suggested to target influenza virus RNA-dependent RNA polymerase.

85 We find that in this group of viruses, RNA-dependent RNA polymerases do not direct gen

86 hildren had recombinant vesicular stomatitis virus RNA detectable in saliva.

87 Hepatitis A virus RNA detected in clinical specimens was sequenced t

88 Median duration of Ebola virus RNA detection was 158 days after onset (73-181; ma

89 The duration of mumps virus RNA detection was studied during a mumps outbreak

90 assuming an eclipse of 7 d from infection to virus RNA detection.

91 the 5' end of type 1 human immunodeficiency virus RNA dimerize spontaneously in vitro in a reaction

92 Picorna-like insect virus RNAs direct an unorthodox form of translation init

93 py (AFM) was used to detect HCV (hepatitis C virus) RNA directly and to quantitatively analyse it wit

94 oat globulin, and satellite tobacco necrosis virus RNA displayed the strongest dependence on eIF4B.

95 ts with a >2-log(10) decrease in hepatitis C virus RNA during prior PEG-IFN/RBV therapy: 11% (4/38) i

96 K cells in the presence of bafilomycin A(1), virus RNA enters the cell and is translated, but replica

97 ctions as a positive effector of hepatitis B virus RNA export.

98 , by use of in situ hybridization, to detect virus RNA expression before and after in vitro T cell ac

99 of short, terminally labeled probes to Ebola virus RNA followed by click assembly and analysis of the

100 The two ribozymes found in hepatitis delta virus RNA form related but non-identical secondary struc

101 mprove amplification-free detection of Ebola virus RNA from blood.

102 Intracellular dengue virus RNA from cells infected with transcript-derived vi

103 -mediated initiation on encephalomyocarditis virus RNA from purified components and used primer exten

104 at 50% and 90% of male survivors clear Ebola virus RNA from seminal fluid at 115 days (90% prediction

105 s data showed a mean clearance rate of Ebola virus RNA from seminal fluid of -0.58 log units per mont

106 INTERPRETATION: Time to clearance of Ebola virus RNA from seminal fluid varies greatly between indi

107 . have claimed to have recovered influenza A virus RNA from Siberian lake ice, postulating that ice m

108 fibrosis and almost complete elimination of virus RNA from the heart by day 28 after infection.

109 bjects who achieved undetectable hepatitis C virus RNA from weeks 4 and 12, known as extended rapid v

110 scription and replication of the influenza A virus RNA genome are mediated by the viral RNA polymeras

111 n 5A (NS5A) encoded by the human hepatitis C virus RNA genome is shown here to induce the activation

112 volved in the packaging of Rift Valley fever virus RNA genome segments, L, M, and S.

113 he U5-IR loop of the feline immunodeficiency virus RNA genome suggests a novel intermolecular interac

114 stent and significant vPAR-CL signals across virus RNA genome, indicating a clear tropism of the enca

115 its effects on gene expression by the killer virus RNA genome.

116 -based sensors for the detection of the Zika virus RNA genome.

117 region, which is responsible for capping the virus RNA genome.

118 anscription and replication of the influenza virus RNA genome.IMPORTANCE Influenza A viruses are a ma

119 low mosaic (TYMV) and kennedya yellow mosaic virus RNAs had activities in all three properties simila

120 d mortality in the affected areas, and Ebola virus RNA has been found in the semen of the survivors a

121 Ebola virus RNA has been noted in the following body fluids da

122 The unusual structure of hepatitis delta virus RNA has previously been shown to enhance its stabi

123 n bias that favipiravir induces in influenza virus RNAs has not been described.

124 Vaccinia virus RNA helicase (NPH-II) catalyzes nucleoside triphos

125 tions (ts10, ts18, and ts39) of the vaccinia virus RNA helicase nucleoside triphosphate phosphohydrol

126 The data suggest that hepatitis C virus RNA helicase, and therefore viral replication, cou

127 The hepatitis C virus RNA helicase, NS3, is an important model system fo

128 ferase) RNA or naturally capped brome mosaic virus RNA, however, was not affected by the presence of

129 shedding, as defined by detection of measles virus RNA in > or =1 specimen obtained 30-61 days after

130 ring the 3 weeks following diagnosis yielded virus RNA in 93% of tests.

131 ed that the number of men positive for Ebola virus RNA in affected countries would decrease from abou

132 r methods allow reliable detection of rabies-virus RNA in biological fluids or tissue before death.

133 d transaminitis and had prolonged detectable virus RNA in blood and semen, suggesting that the possib

134 We monitored Ebola virus RNA in CSF and plasma, and sequenced the viral gen

135 osensor allows the rapid detection of Dengue virus RNA in only 15 min.

136 compared with the ability to detect rubella virus RNA in oral fluids by reverse transcription-PCR (R

137 in reaction assay was used to detect measles virus RNA in peripheral blood mononuclear cells, urine,

138 Detectable human immunodeficiency virus RNA in peripheral blood was associated with lower

139 had been discharged with undetectable Ebola virus RNA in peripheral blood.

140 er concentrations of simian immunodeficiency virus RNA in plasma, and those engaging in affiliation h

141 significantly reduced detection of influenza virus RNA in respiratory droplets and coronavirus RNA in

142 data showed the long-term presence of Ebola virus RNA in semen and declining persistence with increa

143 r time, we found that concentration of Ebola virus RNA in semen during recovery is remarkably higher

144 We report the presence of Ebola virus RNA in semen in a cohort of survivors of EVD in Si

145 itability in detecting the presence of Ebola virus RNA in semen.

146 posttransplant, with detectable hepatitis C virus RNA in serum and features of hepatitis C on liver

147 ty, Calif.) for the detection of hepatitis C virus RNA in serum and plasma.

148 inely detected in deer mice which maintained virus RNA in the blood and lungs.

149 technique was used to detect La Crosse (LAC) virus RNA in the central nervous system (CNS) tissues of

150 This study modeled the presence of Ebola virus RNA in the semen of male Ebola survivors participa

151 h the use of consensus primers detected Zika virus RNA in the serum of the patients but no dengue vir

152 In addition, the inhibition of brome mosaic virus RNA in vitro translation in wheat germ lysates by

153 (EUA) and are routinely used to detect Ebola virus RNA in whole blood and plasma specimens at the Lib

154 nostimulatory effects due to the presence of virus RNA in wild-type particles, indicating their suita

155 To study the encapsidation of Sindbis virus RNAs in infected cells, we designed a new assay th

156 We probed the structure of murine leukemia virus RNA inside virus particles using SHAPE, a high-thr

157 h NH(4)Cl did not prevent the penetration of virus RNA into the cell cytoplasm or translation of the

158 lpha) production is triggered when influenza virus RNA is detected by appropriate pattern recognition

159 Accumulation of unspliced Rous sarcoma virus RNA is facilitated in part by a negative cis eleme

160 Sequencing of the RT gene from plasma virus RNA isolated at peak viremia confirmed that both o

161 Barley yellow dwarf virus RNA, lacking a 5' cap and a 3' poly(A) tail, conta

162 A dramatic reduction in hepatitis C virus RNA level was observed after 2 days of treatment w

163 nts was evaluated and effects on hepatitis C virus RNA level, quasispecies evolution, and liver histo

164 Ebola virus RNA levels (virus load) in PBMC specimens were fou

165 Hepatitis E virus RNA levels also remained detectable in the serum a

166 patients had significantly lower hepatitis C virus RNA levels and more favorable changes in hepatic h

167 l-1 led to a >10,000-fold reduction in Orsay virus RNA levels, which could be rescued by ectopic expr

168 Patients with baseline hepatitis C virus-RNA levels (bHCV-RNA)>6 log IU/mL or cirrhosis hav

169 e quantitation of the human immunodeficiency virus RNA load in blood.

170 modelling to describe the dynamics of Ebola virus RNA load in seminal fluid, including clearance par

171 D4(+) T lymphocytes in uninfected AGMs, milk virus RNA load in SIV-infected AGMs was comparable to th

172 protein cleavage is sufficient for mediating virus RNA maturation.

173 isk may not be underestimated in hepatitis D virus RNA negative hepatitis D patients with advanced li

174 11 of the A complementation group of Sindbis virus RNA-negative mutants.

175 0 donations that were positive for West Nile virus RNA, of which 362 (67 percent) were IgM-antibody-n

176 any age who had a positive result for Ebola virus RNA on reverse-transcriptase-polymerase-chain-reac

177 s disease was confirmed by a finding of Zika virus RNA or a specific neutralizing antibody response t

178 The crystal structure of an alfalfa mosaic virus RNA-peptide complex reveals that conserved AUGC re

179 a from 4 subjects had human immunodeficiency virus RNA pol T215Y/F mutant and 4 had codon 215 wild ty

180 nserved amino acids in bovine viral diarrhea virus RNA polymerase (BVDV RdRp) and RdRps from related

181 sm of initiation of replication by influenza virus RNA polymerase and establish whether initiation of

182 An interaction between the influenza A virus RNA polymerase and the C-terminal domain (CTD) of

183 e the principle of the assay using influenza virus RNA polymerase and yeast PAP as examples.

184 hate (T-705-RTP), is recognized by influenza virus RNA polymerase as a substrate competing with GTP,

185 alyzed the elongation properties of vaccinia virus RNA polymerase during a single round of transcript

186 and that the error rate of the yellow fever virus RNA polymerase employed by the chimeras for genome

187 tion of the vRNA promoter with the influenza virus RNA polymerase heterotrimeric complex is likely to

188 The PB2 subunit of the influenza virus RNA polymerase is a major determinant of viral pat

189 The PB2 subunit of the influenza virus RNA polymerase is a major virulence determinant of

190 The vaccinia virus RNA polymerase is a multi-subunit enzyme that cont

191 ing that the PB2 627 domain of the influenza virus RNA polymerase is not involved in core catalytic f

192 of N-end rule substrates such as the Sindbis virus RNA polymerase nsP4 (bearing N-terminal Tyr) and t

193 vidence that the H4L subunit of the vaccinia virus RNA polymerase plays a direct role in transcriptio

194 We have previously shown that Sindbis virus RNA polymerase requires an N-terminal aromatic ami

195 Vaccinia virus RNA polymerase terminates transcription downstream

196 Vaccinia virus RNA polymerase terminates transcription in respons

197 Vaccinia virus RNA polymerase terminates transcription in respons

198 Thus, influenza virus RNA polymerase uses different initiation strategie

199 The YLD virus RNA polymerase was able to express genes regulated

200 ates the remarkable flexibility of influenza virus RNA polymerase, and aids our understanding of the

201 d partially purified recombinant influenza A virus RNA polymerase, in the absence of influenza virus

202 says using a surrogate respiratory syncytial virus RNA polymerase.

203 uclear-cytoplasmic large double-stranded DNA virus RNA polymerases, and plant plastid RNA polymerases

204 cluded 167 participants who were hepatitis C virus RNA-positive and had injected drugs during the pri

205 e infected with SIN/beclin had fewer Sindbis virus RNA-positive cells, fewer apoptotic cells, and low

206 high levels of m(5)C in the murine leukemia virus RNA, precisely mapped its location, and showed tha

207 gulatory T cells reduced the amount of Seoul virus RNA present in the lungs and the proportion of ani

208 g interaction with and regulation of rubella virus RNA processing.

209 in the initiation but not the maintenance of virus RNA propagation and also contributes to the regula

210 double mutant of the 26 nt potato leaf roll virus RNA pseudoknot.

211 <10,000 copies/mL of human immunodeficiency virus RNA (range, <500-1250).

212 a plasmid DNA vaccine incorporating Sindbis virus RNA replicase functions (pSINCP) and expressing an

213 rans-acting proteins, L and NP, required for virus RNA replication and gene expression were exchangea

214 trans-acting viral factors required for both virus RNA replication and gene transcription, requires t

215 e host factors is revealing basic aspects of virus RNA replication and helping to define the normal f

216 membrane association of positive-strand RNA virus RNA replication complexes is implicated in their f

217 Using a system to study Sindbis virus RNA replication in Drosophila melanogaster, we fou

218 cal, efficient synthesis of 1, a hepatitis C virus RNA replication inhibitor, is described.

219 Positive-strand RNA virus RNA replication is invariably membrane associated

220 PF-429242 did not affect virus RNA replication or budding but had a modest effect

221 nt insights into the early events in Sindbis virus RNA replication suggest a requirement for either t

222 e genome and encodes the enzymes involved in virus RNA replication).

223 HCV-induced dsRNA foci, the likely sites of virus RNA replication, and propose that HCV genome synth

224 inhibition was due not to interference with virus RNA replication, gene expression, or budding but r

225 67K mutations did not affect GPC processing, virus RNA replication, or gene expression.

226 Here, we show that BeAn virus RNA replication, translation, polyprotein processi

227 he OTU protease activity was dispensable for virus RNA replication.

228 bis virus genome are significant for Sindbis virus RNA replication.

229 cell lines transfected with a Semliki Forest virus RNA replicon encoding a single viral structural pr

230 uman papillomavirus type 16 E7, in a Sindbis virus RNA replicon vector.

231 These included DNA encoding Sindbis virus RNA replicons (pSINCP), cationic poly(lactide-co-g

232 Using engineered Sindbis virus RNA replicons expressing puromycin acetyltransfera

233 solation and characterization of hepatitis C virus RNA replicons resistant to a novel ketoamide inhib

234 accine platform consisting of Semliki Forest virus RNA replicons that express the vesicular stomatiti

235 Amplification of replication-competent alpha-virus RNAs (replicons) can be initiated by RNA or DNA tr

236 e that amplification of turnip yellow mosaic virus RNA requires aminoacylation, but that neither the

237 ly that frameshifting on Barley yellow dwarf virus RNA requires viral sequence located four kilobases

238 t the colinear mRNA derived from influenza C virus RNA segment 6 serves as the mRNA for CM2.

239 ear mRNA transcript derived from influenza C virus RNA segment 6.

240 The coding region of influenza A virus RNA segment 7 from the 1918 pandemic virus, consis

241 The virus RNA segment S, M, and L untranslated regions were

242 RNA species are derived from the influenza C virus RNA segment six, (i) a colinear transcript contain

243 c packaging signals for individual influenza virus RNA segments are located in the 5' and 3' noncodin

244 us, including modern-looking avian influenza virus RNA sequences from an archival goose specimen coll

245 ion substudy tested male survivors for Ebola virus RNA shedding in the semen.

246 ses studied for the presence of Epstein-Barr virus RNA showed scattered positivity in 2-7% of lymphoi

247 CSF Ebola virus RNA slowly declined and was undetectable following

248 inhibit the synthesis of all three influenza virus RNA species, block Crm1-dependent nuclear export,

249 formed during the interaction of a West Nile virus RNA stem loop structure with the human T cell-rest

250 ranscription of specific portions of measles virus RNA, such as the nucleocapsid gene, appears able t

251 + T215rev showed full human immunodeficiency virus RNA suppression while receiving a TDF- or tenofovi

252 ing activity but retained their functions in virus RNA synthesis and assembly of infectious particles

253 rk advances our understanding of influenza A virus RNA synthesis and identifies the initiation platfo

254 rk advances our understanding of Influenza A virus RNA synthesis and identifies the initiation platfo

255 iral infectious cycle in switching influenza virus RNA synthesis from transcription mode to replicati

256 nant purified hnRNP C proteins can stimulate virus RNA synthesis in vitro and that depletion of hnRNP

257 Influenza A virus RNA synthesis is catalyzed by the viral polymerase

258 y to analyze the effects of mutations on the virus RNA synthesis machinery in cells of both mammalian

259 e production of infectious virus and blocked virus RNA synthesis when added prior to infection.

260 By in situ analyses of vesicular stomatitis virus RNA synthesis, specific activities of viral RNA sy

261 signaling is required for maximal influenza virus RNA synthesis, viral ribonucleoprotein (vRNP) nucl

262 athway can differentially regulate influenza virus RNA synthesis, which may also offer some new persp

263 specifically impede intracellular influenza virus RNA synthesis.

264 NA, transcribed from the double-stranded CTF virus RNA template by reverse transcriptase PCR.

265 tant to gain wider knowledge about influenza virus RNA to create new strategies for drugs that will i

266 has shown a differential effect on influenza virus RNA transcription and replication.

267 inhibitors specifically diminished influenza virus RNA transcription from the cRNA promoter but not f

268 e the inhibition but also activate influenza virus RNA transcription from the cRNA promoter.

269 We propose that N(pro) is involved with virus RNA translation in the cytoplasm for virus particl

270 Chlorella virus RNA triphosphatase (cvRtp1) is the smallest member

271 reviously shown to be essential for vaccinia virus RNA triphosphatase activity inactivated the tripho

272 oposal that protozoan, fungal, and Chlorella virus RNA triphosphatases belong to a single family of m

273 In some eukaryotic viruses, RNA upstream of the coding region forms an inte

274 p every 3-6 weeks, which we tested for Ebola virus RNA using quantitative real-time RT-PCR.

275 that ICP27 also mediates the export of some virus RNAs via a Crm1-dependent pathway and present evid

276 The replication of many positive-strand RNA viruses [(+)RNA viruses] depends on the cellular protein

277 Replication of plus-strand RNA viruses [(+)RNA viruses] is performed by viral replicase

278 All well-characterized positive-strand RNA viruses[(+)RNA viruses] induce the formation of host mem

279 ment-specific packaging signals of influenza virus RNAs (vRNAs) are located in the 5' and 3' noncodin

280 Mumps virus RNA was characterized directly from cerebrospinal

281 Influenza virus RNA was detected as well or better after 6 months

282 Ebola virus RNA was detected in 86 semen specimens from 19 (73

283 Hepatitis C virus RNA was detected in Gammagard, and the risk of tra

284 More virus RNA was detected in the brain and spinal cord of I

285 Ebola virus RNA was detected in the semen of all 7 men with a

286 c biliary atresia in the 10 patients in whom virus RNA was detected.

287 The presence of Zika virus RNA was identified in a subgroup of these women on

288 However, the Delta p48 mutant virus RNA was rescued when p48 was provided in trans.

289 Levels of virus RNA were reduced in HLA-DR transgenic mice compare

290 vels of infectious virus, virus antigen, and virus RNA were similar in both groups.

291 ne chip, serum samples containing the target virus RNA were simply added by automatic flow from disti

292 Eggplant mosaic virus RNA, which has a differently constructed acceptor

293 alylated 3'-fragment of turnip yellow mosaic virus RNA, which has a pseudoknotted amino acid acceptor

294 of sustained clearance of serum hepatitis C virus RNA, which is influenced, in turn, by the patient'

295 Erysimum latent virus RNA, which lacks an identifiable anticodon domain,

296 ancing the hybridization of the target Ebola virus RNA with capture probes bound to the beads.

297 e detection of Zika, dengue, and chikungunya virus RNA with high target specific and low limits of de

298 F3), if a >2-log(10) decrease in hepatitis C virus RNA with previous PEG-IFN/RBV treatment was achiev

299 45-base synthetic fragment of Harvey sarcoma virus RNA with recombinant or synthetic HIV-1 NC protein

300 od donations were being tested for West Nile virus RNA with the use of investigational nucleic acid a