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1 slated at the inclusion sites rather than in virus factories.
2 mbly sites, where the protein surrounded the virus factories.
3 bled into virus particles within cytoplasmic virus factories.
4                                              Virus factories actively producing virus capsids on thei
5 ia interaction with the capsid protein L3 to virus factories and virions to antagonize TRIM5alpha; th
6 beginning to reveal the relationship between virus factories and viroplasm and the cellular structure
7 the transpoviron replicates within the giant virus factory and accumulates in high copy numbers insid
8 ane proteins that are synthesized within the virus factory and lack COPII or other binding sites that
9          Here, we show that the Birnaviridae virus factories are not membrane bound, demonstrate prop
10 th A30 and G7 presumably occurred within the virus factory areas of the cytoplasm, where each was con
11                               In reassortant viruses, factory costaining for cUb mapped primarily to
12 al replication and, thus, the possibility of virus factories in this region.
13               Intriguingly, the CaMV-induced virus factory inclusions seem to protect against autopha
14 n vivo and ubiquitin colocalizes with p28 to virus factories independently of an intact RING domain.
15 fer of nucleic acid from the interior of the virus factory into the nearly completed capsids.
16 acity of the TC-muNS fusion proteins to form virus factory-like (VFL) structures and colocalize with
17                                              Virus factories of ARV are composed of nonstructural pro
18 enerate subcellular microenvironments called virus factories or 'viroplasm'.
19 tration of viral transcripts within distinct virus factories or superinfection exclusion also could i
20 h specific intracellular compartments called virus factories or virioplasm.
21 ruses replicate in cytoplasmic compartments (virus factories or viroplasms) composed of viral and cel
22                           The development of virus factories proceeded over 3 to 4 h postinfection an
23             The first feature to appear on a virus factory surface when a new capsid is born is the c
24 e X-body, which comprises a highly organized virus "factory." TGB1 is both necessary and sufficient t
25  assembly often occur in virus inclusions or virus factories that form at pericentriolar sites close
26 the host cell that has been converted into a virus factory, thus disrupting the viral life cycle.
27 ay from the endoplasmic reticulum within the virus factory to nascent viral membranes and demonstrate
28 operative pathway from ER domains within the virus factory to the viral membrane.
29 ically replicate within discrete cytoplasmic virus factories (VFs) that may represent a barrier to ge
30 information about the nature of Birnaviridae virus factories (VFs), we used a recombinant infectious
31 virus-encoded cytoplasmic structures, termed virus factories (VFs), where viral transcription, transl
32 enome that replicate in discrete cytoplasmic virus factories (VFs).
33        A35R localized intracellularly to the virus factories, where the first stages of morphogenesis
34 ls infected in the absence of inducer showed virus factories with abnormal electron-dense viroplasms