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1 ations suggest that R0=1.6 in the absence of virus integration.
2 la188 contact analogous tDNA bases to effect virus integration.
3 esent in the adjacent intron or generated by virus integration.
4 nces, as is characteristic for avian sarcoma virus integration.
5 and anonymous cDNAs disrupted as a result of virus integration.
6 e investigate potential oncogenic impacts of virus integration.
7 e leukemia virus, and avian sarcoma/leukosis virus integrations.
8 hat the transcription factor Friend leukemia virus integration 1 (Fli-1) is a target of miR-145a and
9 vious data demonstrated that friend leukemia virus integration 1 (Fli-1), an erythroblast transformat
10               The ETS factor Friend leukemia virus integration 1 (FLI1) is a key modulator of lupus d
11                              Friend leukemia virus integration 1 (FLI1), a critical transcription fac
12                              Friend leukemia virus integration 1 (FLI1), an Ets transcription factor
13            TSA also restored Friend leukemia virus integration 1, an inhibitor of the type I collagen
14 how similarities with human immunodeficiency virus integration and provide a unifying mechanism for d
15 g cytokine regulators, that are disrupted by virus integration, and we determined mechanisms through
16                             We conclude that virus integration can contribute to carcinogenesis in a
17 e findings reveal another mechanism in which virus integration can ignite tumorigenesis and a promisi
18 , and we determined mechanisms through which virus integration causes deregulation of cellular gene e
19        Despite the invariant position of the virus integration event, the three ABPL tumor cell lines
20 l cancers by whole-genome sequencing detects virus integration in 77%, revealing five statistically s
21                 We generated murine leukemia virus integrations in human HepG2 and K562 cells and sub
22 ction of RIG-I in suppressing DNA repair and virus integration into the host genome, and meanwhile en
23 stablishment of a persistent reservoir after virus integration into the host genome.
24                                              Virus integration into the human genome occurs frequentl
25 p52, and Rep40) are pleiotropic effectors of virus integration, replication, transcription, and virio
26 including wingless-type murine mammary tumor virus integration site (WNT) pathway subtype, Sonic Hedg
27      B-cell-specific Moloney murine leukemia virus integration site 1 (BMI1) is a component of the po
28  To address the cellular origin of ecotropic virus integration site 1 (EVI1)-expressing aggressive KM
29 tures of Rep68 bound to the adeno-associated virus integration site 1 in different nucleotide-bound s
30  into the safe-harbor locus adeno-associated virus integration site 1 in human embryonic stem cells.
31 i-1 (B-cell-specific Moloney murine leukemia virus integration site 1) as a regulator of human epider
32  D1 and wingless-related mouse mammary tumor virus integration site 10b (Wnt10b) in 3T3-L1 cells.
33 MDS and EVI1 complex protein EVI1 [ecotropic virus integration site 1], in 5 patients) or PRDM16 (pos
34 ducible by wingless-type mouse mammary tumor virus integration site family member (WNT)/beta-catenin
35 nt refers to the wingless-type mammary tumor virus integration site family of proteins), that are reg
36 esenchymal Wingless-type Mouse Mammary Tumor Virus integration site family, member 10B (Wnt10b)/beta-
37 the cellular gene at the mouse mammary tumor virus integration site in the int-5 locus is aromatase.
38 Meis (named for a myeloid ecotropic leukemia virus integration site), respectively, are encoded by mu
39 cancer-associated breakpoint and a papilloma virus integration site.
40 f Wingless-related MMTV (mouse mammary tumor virus) integration site 3 (WNT3) by ingrowing axons from
41 n Wingless-related MMTV (mouse mammary tumor virus) integration site 3 (WNT3).
42 ical wingless-type MMTV (mouse mammary tumor virus) integration site family (WNT) signaling pathway i
43             For the discovery of hepatitis B virus integration sites from probe capture data, the ver
44                                              Virus integration with host genome sequence is a type of