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1 nding of the pH-driven alteration of Sindbis virus structure.
2 on the viral envelope, and disruption of the virus structure.
3 he protein involved in its assembly into the virus structure.
4 ation of the Trp-38 side chain in the native virus structure.
5 tion shows PKD1 bound with minimal change in virus structure.
6 ndicating that the vp3 gene is important for virus structure.
7 tricacies, interactions, and beauty of these virus structures.
8 ranged major capsid proteins yet observed in virus structures.
9 ement has not yet been observed in any known virus structures.
13 ies have led to the determination of several virus structures at near-atomic resolution (3.3 - 4.6 A)
16 equivalence are then examined in some larger virus structures containing multiple subunit types and a
17 principle allow researchers to perform X-ray virus structure determination for single particles at ro
20 nular areas devoid of cellular organelles or virus structures except for occasional short crescent-sh
22 At the time of this writing, there are 931 virus structures from 62 different virus families in the
24 ly genetically encoded, and most pleomorphic virus structures have no selective advantage in vitro.
27 nce as a pathogen, little is known about the virus structure, in part because of its intrinsic struct
28 ext-generation vaccines target less variable virus structures, including the haemagglutinin stem.
31 differences in pandemic versus non-pandemic virus structures, replication properties and virus-host
32 ion electron microscopy images and the known virus structures showed high affinity and mutual orderin
33 inding permitted a more detailed view of the virus structure than was previously possible, leading to
34 lecular virology, immunity and pathogenesis, virus structure, the viral use and abuse of cellular pat
37 Comparison between our infectious bronchitis virus structure with published RTC structures from other
38 tion of these cell lines with respect to (i) virus structure within and reactivated from the cell lin