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1  involve changes in the autonomic control of visceromotor activity, including during cognitively dema
2                                              Visceromotor and cardiovascular responses to colonic dis
3 ctivity in areas also controlling autonomic, visceromotor and skeletomotor responses.
4 ience makes sense, we propose that implicit (visceromotor and somatomotor) emotional processes are di
5 ed of two main classes, termed branchiomotor/visceromotor and somatomotor.
6 hood (8-10 weeks) at which point behavioral, visceromotor, and great splanchnic nerve responses to gr
7 aviors involving integration of respiratory, visceromotor, and somatomotor activity.
8 ced stimuli and to integrate the sensory and visceromotor aspects of emotional behavior.
9                          In contrast, facial visceromotor axons selectively required SEMA3A/NRP1.
10 bx20 is expressed by migrating branchiomotor/visceromotor (BM/VM) neurons within the hindbrain during
11  corticotropin-releasing factor and in other visceromotor cell types of the paraventricular hypothala
12 in and are regulated, in turn, by descending visceromotor commands from our brain and by actions such
13  and organ-specific regulation to descending visceromotor commands, whereas the output from M2 could
14 infralimbic area, as major components of the visceromotor cortex that directly project to hypothalami
15 , and motor neurons of the somatic motor and visceromotor cranial nerve nuclei and the ventral horn o
16                                              Visceromotor electromyographic responses increased withi
17                                       If the visceromotor functions of M1 and M2 reflect their skelet
18  medial areas involved in cognitive (PL) and visceromotor (IL) functions.
19 o the paraventricular nucleus, including the visceromotor (infralimbic) cortex, median preoptic nucle
20 as 10, 14, and 25 are heavily connected to a visceromotor network.
21                                              Visceromotor neurons in the dorsal vagal and ambigual nu
22 mediate acute stress effects on hypothalamic visceromotor neurons, (2) comprise targets for corticost
23  motor neuron populations, branchiomotor and visceromotor neurons.
24 t of cranial nerves II, V, VII, and VIII and visceromotor nuclei of nerves VII, IX, and X as well as
25 nd ventral octavolateral nuclei, vagal lobe, visceromotor nuclei, and reticular formation, including
26 rolateral periaqueductal gray); hypothalamic visceromotor pattern generator network (five of six know
27 tricular and supraoptic nuclei, hypothalamic visceromotor pattern generator network), and thalamocort
28 entricular hypothalamic nuclei, hypothalamic visceromotor pattern generator network), orofaciopharyng
29 al gray, Barrington's nucleus), hypothalamic visceromotor pattern-generator network (five of six know
30 ontrol network, neuroendocrine, hypothalamic visceromotor pattern-generator network, thalamocortical
31 ed to identify and characterize hypothalamic visceromotor populations responsive to acute and chronic
32  colorectal distention (CRD) in the rat: the visceromotor reflex (vmr) and L6-S1 dorsal horn neuron a
33                Pain behavior was assessed by visceromotor reflex activity in response to noxious stim
34 l SR140333 injection diminished the enhanced visceromotor reflex to colorectal distention at day 11 i
35 ion of the NK(1)R antagonist SR140333 on the visceromotor reflex to colorectal distention in both gro
36 les in facilitating the acoustic startle and visceromotor reflexes.
37 that the PAG may play a more general role in visceromotor regulation, even in the absence of threat.
38                               Thus, a map of visceromotor representation appears to be embedded withi
39 biological stimulus with its somatomotor and visceromotor representation.
40                                          The visceromotor response (VMR) evoked by noxious colorectal
41 y reduces colorectal distention (CRD)-evoked visceromotor response (VMR) in mice.
42  recordings of pACC neurons and examined the visceromotor response (VMR) to colorectal distention (CR
43 ral nociceptive behavior was measured as the visceromotor response (VMR) to colorectal distention (CR
44                                            A visceromotor response (VMR) to graded colorectal distens
45                    The nociceptive response (visceromotor response [VMR]) to CRD was recorded in norm
46 ght activation of ExPANs induced a pain-like visceromotor response and expression of Fos in spinal PP
47  effects of fractalkine were assessed on the visceromotor response in rats exposed to minocycline or
48   There was a significant enhancement of the visceromotor response in zymosan-, but not saline-treate
49 y afferents significantly blunted the evoked visceromotor response to bladder distension and led to s
50                                          The visceromotor response to colon distension (15-60 mm Hg)
51                                          The visceromotor response to colorectal distension was measu
52         Visceral nociception, modeled by the visceromotor response to colorectal distension, and colo
53 avoidance stress significantly increased the visceromotor response to colorectal distention (20-80 mm
54                                          The visceromotor response to colorectal distention at baseli
55 nt of visceral hypersensitivity by measuring visceromotor response to colorectal distention in rats.
56 PK signaling is responsible for the enhanced visceromotor response to colorectal distention in STZ-D
57 tive colon as demonstrated by an exaggerated visceromotor response to colorectal distention in the F3
58 produces hyperalgesia (i.e., facilitates the visceromotor response to colorectal distention) mediated
59 r saline was given intracolonically, and the visceromotor response to noxious colorectal distention (
60                               An exaggerated visceromotor response, referred pain to mechanical stimu
61 ngs of the abdominal musculature, termed the visceromotor response.
62                                              Visceromotor responses (VMR) to CRD were recorded in rat
63 ceral nociception was evaluated by measuring visceromotor responses (VMR), afferent nerve mechanosens
64 l colorectal distention (fCRD) to drive both visceromotor responses (VMRs) and aversion.
65                 Nociception was evaluated by visceromotor responses and afferent nerve recordings to
66 There was a highly significant difference in visceromotor responses between the phases of the estrous
67                                          The visceromotor responses measured during the phase of proe
68 of these cell groups in driving hypothalamic visceromotor responses to a given stressor, and (iii) de
69                                              Visceromotor responses to colorectal distension and colo
70                 Intraluminal Cat-S amplified visceromotor responses to colorectal distension and indu
71                                  We measured visceromotor responses to colorectal distension in roden
72                                              Visceromotor responses to colorectal distension, an indi
73 increased fecal water content, and increased visceromotor responses to colorectal distension.
74 natants generated a 3- to 4-fold increase in visceromotor responses to colorectal distension; this wa
75                               Hypersensitive visceromotor responses to colorectal distention in STZ-D
76                                              Visceromotor responses to distension were significantly
77                               In awake rats, visceromotor responses to intragastric acid are quantifi
78    Gastritis and gastric ulcers enhanced the visceromotor responses to intragastric acid.
79 was measured in guinea pig distal colon, and visceromotor responses were recorded in a rat model of c
80 gions that control reproductive behavior and visceromotor responses, confirming a similar analysis by
81 al area of the BST - cell groups involved in visceromotor responses.
82 areas that comprise this network represent a visceromotor system, distinct from the sensory related "
83 ending axonal projections that may influence visceromotor systems and visceral "mind-body" symptoms.