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1 small neuronal subsets provided a Golgi-like vital stain.
2 asure by using triphenyltetrazolium chloride vital staining.
3 k-up time, Schirmer test, and ocular surface vital staining.
5 regions were fully contractile and viable by vital staining and microscopy but demonstrated 25% reduc
8 munofluorescence, cytochemistry, fluorescent vital stains, and fluid-phase markers in conjunction wit
13 }-6-aminocaproyl-d-erythro-sphingos ine as a vital stain for chlamydiae proved to be a sensitive meth
15 n, or beta-COP (a specific golgi protein) or vital stains for endoplasmic reticulum (ER) and golgi.
16 orofluorescein diacetate (a vacuolar luminal vital stain), had a pronounced shift in red/green fluore
19 escape, we followed acidification using the vital stain LysoTracker red and acquisition of the proto
21 endplates (pi-junctions) were identified by vital staining of lateral plantar nerve (LPN) and sural
22 with high synaptic density as ascertained by vital staining of recycling synaptic vesicles, and were
24 , peeling of the internal limiting membrane, vital staining of the internal limiting membrane with in
28 This approach makes use of a lineage-tracing vital stain that is retained through cell generations an
32 nature of this compartment was confirmed by vital staining with a pH sensitive dye, LysoSensor yello
35 ata on new techniques and technology such as vital staining with methylene blue and protoporphyrin fl
39 duces muscle membrane damage, as revealed by vital staining (with Evans blue dye) of the diaphragm an