ライフサイエンスコーパス: vitamin

1 le strategies to synthesize and salvage this vitamin.

2 plants were evaluated including minerals and vitamins.

3 During this process, vitamin A aldehyde is shepherded within photoreceptors a

4 In conclusion, vitamin A and D deficiencies have opposing effects on mo

5 Pro-vitamin A biofortified (yellow) cassava has the potentia

6 in but no effect on anthropometry or iron or vitamin A deficiencies.

7 Vitamin A deficiency results in a deterioration of these

8 vitamin A is an efficient strategy to combat vitamin A deficiency.

9 Vitamin A deficient (VAD) mice fared worst with more rap

10 gher frequencies among CD4(+) splenocytes of vitamin A deficient vs. sufficient mice.

11 that (1) stem cells are kept inactive by the vitamin A derivative retinoic acid, which is synthesized

12 Food supplementation with vitamin A is an efficient strategy to combat vitamin A d

13 thus can be utilized to discover defects in vitamin A metabolism during the regeneration of the visu

14 Vitamin A regulates the adaptive immune response and a m

15 efficacy of biofortified cassava to improve vitamin A status of Nigerian preschool children.

16 ntial to contribute significantly to improve vitamin A status, especially in populations that are dif

17 ration treatment of diarrhoea, and receiving vitamin A supplementation) and household characteristics

18 nicotinamide adenine dinucleotide (NADH) and vitamin A were scanned on sturgeon samples kept at 4 deg

19 ere associated nominally with decreased risk-vitamin A, vitamin B6, beta-carotene, lutein and zeaxant

20 <= 0.0005) with decreased risk of late AMD: vitamin A, vitamin B6, vitamin C, folate, beta-carotene,

21 et included measures of serum ferritin (SF), vitamin A, zinc, and CRP measured using different assays

22 ns on proteolysis, lipolysis and calcium and vitamins A and D3 bioaccessibility in salmon, sardine, s

23 nterplay between biosynthesis and salvage of vitamins and cofactors in apicomplexans.

24 The associations between B vitamins and lipid concentrations across trimesters were

25 he essential contribution of micronutrients (vitamins and minerals) to nutrition is often overlooked.

26 Because B vitamins are essential for survival, null mutations ofte

27 Vitamin B(1) is an essential dietary component, and defi

28 The predominant source of dietary vitamin B(1) is plant-based foods.

29 Although Mtb can synthesize vitamin B(12) (cobalamin) de novo, uptake of cobalamin h

30 d a suitable medium to provide water soluble vitamin B(12) and fat soluble vitamin D(3) in single pro

31 Vitamin B(12) and other cobamides are essential cofactor

32 entally benign solvent, and the synthesis of vitamin B(12) component on the gram scale.

33 Vitamin B(12) is the only known essential human micronut

34 Supplementation of cultures with cobalamin (vitamin B(12)) increased nitrite oxidation rates and sti

35 Hemoglobin, iron, ferritin, folate and vitamin B(12), in addition to many other laboratory indi

36 ethod to enable enhancement of researches on vitamin B(2) content of milk and its variation factors.

37 m 0.4 to 7%), RBP (35%), selenium (70%), and vitamin B-12 (0.1%), yet for maternal intakes only a pos

38 Vitamin B-12 and folate deficiencies in women and childr

39 Low plasma vitamin B-12 and low plasma folate were each associated

40 and alpha1-acid glycoprotein (AGP) and serum vitamin B-12 and serum and RBC folate among nonpregnant

41 We could not examine the interaction for vitamin B-12 deficiency and high plasma folate, because

42 include elevated folate and, less commonly, vitamin B-12 deficiency, or other nutritional deficienci

43 se health effects other than those linked to vitamin B-12 function?

44 The measurement of bioavailability of vitamin B-12 is etiologically important in deficiency bu

45 ss the effect of parenteral replenishment of vitamin B-12 on the bioavailability.

46 on of the coexistence of high folate and low vitamin B-12 status with cognitive function, utilizing v

47 h folic acid exposures are combined with low vitamin B-12 status.

48 Parenteral replenishment of the vitamin B-12 store in deficient individuals prior to the

49 before and after parenteral replenishment of vitamin B-12 stores, from the kinetics of its plasma app

50 Deficient compared with higher vitamin B-12 was significantly associated with lower cog

51 Among those with "normal" vitamin B-12, higher UMFA or serum total folate was prot

52 whole grains, and legumes, supplemented with vitamin B-12, is nutritionally superior to diets includi

53 ons < ~20 nmol/L, a recognized threshold for vitamin B-6 deficiency.

54 lored the association between plasma folate, vitamin B-6 in the form of pyridoxal 5'-phosphate (PLP),

55 e of Kyns as potential markers of functional vitamin B-6 status across 2 large cohorts.

56 Vitamin B-6 status is routinely measured as pyridoxal 5'

57 When aiming at engineering the thiamine (vitamin B1) pathway in plants, the availability of tools

58 Vitamin B12 (B12) is a micronutrient essential for one-c

59 ir clinical utility, it has been unclear how vitamin B12 and folic acid (FA) function at the molecula

60 characterized by intestinal malabsorption of vitamin B12 and in some cases proteinuria.

61 l biogenesis or acidification factors causes vitamin B12 deficiency.

62 For nonantioxidants, vitamin B12 use both before and during chemotherapy was

63 hey protein, beta-Lactoglobulin (betaLG) and vitamin B12, was studied using different spectroscopic t

64 croenvironment of betaLG was affected by the vitamin B12.

65 ein (RBP), 25-hydroxy vitamin D, folate, and vitamin B12; and a panel of immune response markers.

66 le-light, the substrates interacted with the vitamin B2 and different ROS were generated.

67 mphocytes activated by bacteria that produce vitamin B2 metabolites.

68 ing GAD dysfunction such as schizophrenia or vitamin B6 deficiency.

69 livery vehicle for the controlled release of vitamin B6 over a prolonged period of observation.

70 eration, and pharmacological blockade of the vitamin B6 pathway at both PDXK and PLP levels recapitul

71 linked to tumor growth, but the relevance of vitamin B6 remains uncertain.

72 erences in the abundance of genes related to vitamin B6 synthesis and branched-chain amino acid synth

73 ted nominally with decreased risk-vitamin A, vitamin B6, beta-carotene, lutein and zeaxanthin, magnes

74 ot synthesise themselves, such as pyridoxine/vitamin B6, taurine, some essential amino acids, and a c

75 with decreased risk of late AMD: vitamin A, vitamin B6, vitamin C, folate, beta-carotene, lutein and

76 ounced release in the small intestine with a vitamin bioaccessibility of 56%.

77 rtant for host fitness, and pathways for key vitamin biosynthesis and export were identified across M

78 The water-soluble vitamin biotin is essential for cellular growth, develop

79 CC fruits had high level of vitamin C (48-108 mg/100 g), malic acid (104-375 mg/100

80 ries had a higher content of polyphenols and vitamin C and antioxidant capacity, but lower values of

81 s suggest that UVC helps in the retention of vitamin C and phenolic content in acerola by altering as

82 tone dehydrogenase (GalDH), a key enzyme for vitamin C biosynthesis, and altered the composition of p

83 wearable sensor that can selectively measure vitamin C concentration in biofluids, including sweat, u

84 nd vegetable-based baby foodstuffs (declared vitamin C fortified) at gastric pH 1.5 and 4, respective

85 y was to investigate the bioaccessibility of vitamin C in fruit-, vegetable-, and cereal-based baby f

86 sor allows monitoring of temporal changes in vitamin C levels.

87 The bioaccessibility of vitamin C ranged from 10.4 to 43.4%, and from 0.4 to 19.

88 tal acidity of the vinegar, ascorbic acid in vitamin C tablets, and chloride in soy sauces and saline

89 The content of vitamin C was 0.98-3.65 g.kg(-1) in berries and 22.81-46

90 The highest vitamin C was for the FA5 fruit.

91 Ascorbate (Asc; vitamin C) plays essential roles in development, signali

92 imary metabolites (sugars, organic acids and vitamin C) were determined in the juices.

93 ar disease, frailty, atrial fibrillation and vitamin C).

94 sed risk of late AMD: vitamin A, vitamin B6, vitamin C, folate, beta-carotene, lutein and zeaxanthin,

95 Vitamin C, or L-ascorbic acid (AsA), is the most abundan

96 compared and analyzed the concentrations of vitamin C, vitamin E, zinc, and copper in both national

97 e samples tested, the measured quantities of vitamin C, vitamin E, zinc, and copper were slightly hig

98 g/day of zeaxanthin, and other antioxidants (vitamin C, vitamin E, zinc, copper) for 16 weeks.

99 fasting-mimicking diet selectivity reverses vitamin C-induced up-regulation of heme-oxygenase-1 and

100 Mounting evidence indicates that vitamins C and D are linked to tumor growth, but the rel

101 especially with regard to their fat-soluble vitamin contents.

102 Rats (N = 38) injected with vitamin D (days 1-3) to induce CVC were infused with sal

103 to treatment with vitamin D compared with no vitamin D (HR: 0.79; 95% CI: 0.49, 1.27), but there was

104 t, and they include topical corticosteroids, vitamin D analogues, calcineurin inhibitors, and keratol

105 Additional adjustment for vitamin D and bisphosphonate use in the previous month r

106 Vitamin D and calcium for the prevention of fracture: a

107 and use of corticosteroids, bisphosphonates, vitamin D and calcium supplements (OR, 1.9; 95% CI, 1.2-

108 r between children receiving 1000 or 2000 IU vitamin D and children receiving 600 IU.

109 In this study, we evaluated vitamin D and mineral (iron, zinc, magnesium) transfer t

110 (FGF23) and Klotho, which normally regulate vitamin D and mineral homeostasis, on testicular functio

111 The randomized placebo-controlled Vitamin D and Omega-3 Trial suggested a possible benefit

112 A major goal of VITAL (Vitamin D and Omega-3 Trial) was to fill this knowledge

113 men aged 50 years or older in the VITAL-DEP (Vitamin D and Omega-3 Trial-Depression Endpoint Preventi

114 In the rescue experiments, we confirmed vitamin D and VDR inhibited LPS- or activated CD4(+) T c

115 matory biomarkers, and to assess the role of vitamin D as a potential mediator in the association bet

116 n of patients who reduced the dose of active vitamin D at Month 6 (31% vs. 10% in the placebo group).

117 bility, while phytates and tannins decreased vitamin D bioaccessibility.

118 CC incidence was unrelated to treatment with vitamin D compared with no vitamin D (HR: 0.79; 95% CI:

119 Vitamin D deficiency (<12 ng/mL) is associated with redu

120 this study, we examined the hypothesis that vitamin D deficiency (VDD) during early life stage devel

121 Obese children are vulnerable to vitamin D deficiency and impaired cardiovascular health;

122 der adults with a low prevalence of profound vitamin D deficiency at baseline.

123 Vitamin D deficiency causes pro-inflammatory macrophage

124 Vitamin D deficiency has been associated with increased

125 Vitamin D deficiency is associated with obesity-related

126 The prevalence of vitamin D deficiency is high in African populations.

127 nclude efforts to prevent, detect, and treat vitamin D deficiency, especially in newborn babies, wome

128 d intact PTH only among patients with severe vitamin D deficiency.

129 d markers of systemic inflammation underlies vitamin D effects.

130 Vitamin D exerts a regulatory role over mucosal immunity

131 Vitamin D exerts its actions through the vitamin D recep

132 the trial was 31.0 ng per milliliter in the vitamin D group and 10.7 ng per milliliter in the placeb

133 participants reported >=1 ARI: 74.1% in the vitamin D group versus 73.7% in the placebo group.

134 Vitamin D has a key role in stimulating calcium absorpti

135 The bioactivity of vitamin D in hPDLCs was assessed based on the gene expre

136 nalysis were performed to assess any role of vitamin D in mediating a causal effect of BMI on inflamm

137 No interactions between calcium and vitamin D in relation to all-cause mortality were observ

138 measurement and completed a questionnaire on vitamin D intake and ultraviolet light exposure.

139 We found no associations between vitamin D intake skin cancers, except positive associati

140 iometabolic risk markers, but double-blinded vitamin D intervention studies in children are scarce.

141 es of a double-blind, randomized, milk-based vitamin D intervention trial conducted during late fall

142 Vitamin D is the precursor to 25(OH)D but is analyticall

143 showed significantly lower serum 25-hydroxy vitamin D levels than inactive uveitis patients and loca

144 mong children with persistent asthma and low vitamin D levels, vitamin D3 supplementation, compared w

145 Activation of liver macrophage VDRs by vitamin D ligands ameliorates liver inflammation, steato

146 Calcium alone or in combination with vitamin D may reduce the risk of SCC, but not BCC.

147 cipants (n = 151) underwent serum 25-hydroxy vitamin D measurement and completed a questionnaire on v

148 that both dietary and endogenously produced vitamin D metabolites were under polygenic control in Af

149 er, data are limited regarding the impact of vitamin D on breast cancer subtypes among African-Americ

150 mega-3 Trial suggested a possible benefit of vitamin D on cancer incidence among black individuals.

151 daily, aspirin (75 mg) or placebo daily, and vitamin D or placebo monthly.

152 ne, and African Index Medicus for studies on vitamin D prevalence, published from database inception

153 tigations assessing the impact of melanin on vitamin D production have produced contradictory results

154 This is followed by discussion of the vitamin D receptor (VDR) that mediates the cellular acti

155 Vitamin D exerts its actions through the vitamin D receptor (VDR), the expression of which was re

156 essed based on the gene expression levels of vitamin D receptor (VDR)-regulated genes osteocalcin and

157 egulatory role over mucosal immunity via the vitamin D receptor (VDR).

158 also describe the expression of a functional vitamin D receptor in IDEC.

159 The vitamin D receptor is highly expressed in the gastrointe

160 crophage miR-106b-5p secretion from impaired vitamin D receptor signaling causes inflammation-induced

161 Vitamin D receptor, oestrogen receptor and mineralocorti

162 marily by lithocholic acid signaling via the vitamin D receptor.

163 tic macrophages express the highest level of vitamin D receptors (VDRs) among nonparenchymal cells, w

164 ficiency and impaired cardiovascular health; vitamin D replenishment might improve their cardiovascul

165 Although adults with low vitamin D status are at increased risk of acute respirat

166 lated conditions, but the role of early life vitamin D status on the development of obesity is poorly

167 Observational studies have linked low vitamin D status to unfavorable cardiometabolic risk mar

168 ther as a main effect or in interaction with vitamin D status.

169 s with symptoms, markers of inflammation and vitamin D status.

170 evaluate the effect of different doses of a vitamin D supplement on cardiometabolic risk markers in

171 Vitamin D supplementation attenuated expression of key m

172 Monthly high-dose vitamin D supplementation does not prevent ARI in older

173 olites with T2D and argue against the use of vitamin D supplementation for the prevention of T2D.

174 Whether dosage of vitamin D supplementation has a differential effect on B

175 ction (ARI), randomized controlled trials of vitamin D supplementation have provided inconsistent res

176 dies are warranted to examine the effects of vitamin D supplementation in early life on long-term car

177 blem while examining the association between vitamin D supplementation initiated after breast cancer

178 Vitamin D supplementation may prevent falls in older per

179 There was no effect of maternal vitamin D supplementation on asthma and recurrent wheeze

180 There was no effect of prenatal vitamin D supplementation on most of the prespecified se

181 om RCTs, do not support a beneficial role of vitamin D supplementation on obesity-related inflammatio

182 ere triangulated with a literature review of vitamin D supplementation trials.

183 Vitamin D supplementation was associated with lower epig

184 ing for age, sex, season of measurement, and vitamin D supplementation.

185 This enables vitamin D to regulate epidermal differentiation and hair

186 Low vitamin D was also independently associated with thicker

187 Vitamin D was associated with nonsignificant increases i

188 a 20-epi-Inhoffen-Lythgoe diol derived from vitamin D(2).

189 how in this article that biologically active vitamin D(3) [1,25(OH)(2)-D(3)] significantly downregula

190 tal ligament cells (hPDLCs) are regulated by vitamin D(3) and play a fundamental role in periodontal

191 Although vitamin D(3) deficiency is considered as a risk factor f

192 cipants were randomly assigned to 1 of the 4 vitamin D(3) doses, and the best noncontrol dose for pre

193 risk of hypercalcaemia (three events in the vitamin D(3) group and two events in the placebo group;

194 water soluble vitamin B(12) and fat soluble vitamin D(3) in single product.

195 The vitamin D(3) regimen did not increase the risk of hyperc

196 roxyvitamin D(3) (25[OH]D(3)) as a marker of vitamin D(3) status.

197 d, double-blind, placebo-controlled trial of vitamin D(3) supplementation among adults living with HI

198 s, which might mitigate the effectiveness of vitamin D(3) supplementation during periodontal treatmen

199 2001 patients were randomly assigned to the vitamin D(3) supplementation group, and 1999 to the plac

200 1:1 to receive either weekly oral 50 000 IU vitamin D(3) supplements (cholecalciferol) for the first

201 first month of ART followed by daily 2000 IU vitamin D(3) supplements or a matching weekly and daily

202 th and biochemical composition of the cells (vitamin D(3), PUFAs and carotenoids) was evaluated.

203 to study the bioavailability of calcium and vitamin D(3), the W/O/W double emulsions were subjected

204 Fish have the highest natural content of vitamin D(3), which is suggested to originate from zoopl

205 hich microalgal species may be the source of vitamin D(3).

206 evant mechanistic studies regarding n-3 FAs, vitamin D, and vascular disease, and summarize recent me

207 r, retinol-binding protein (RBP), 25-hydroxy vitamin D, folate, and vitamin B12; and a panel of immun

208 The benefits of vitamin D, omega-3 fatty acids, and exercise in disease

209 For supplemental vitamin D, we observed possible inverse associations bet

210 The aims were to determine, in vitamin D-deficient overweight and obese children, wheth

211 In a subanalysis of vitamin D-deficient participants, sun exposure was assoc

212 coded by AD- and psychosis-related genes and Vitamin D-perturbed genes.

213 o use as well as serum levels of calcium and vitamin D.

214 ling pathways including Smads, TGF-beta, and vitamin D.

215 d whole body BMD compared to participants on vitamin D/calcium supplementation and exercise alone.

216 variability: -0.37 mm Hg) compared to 800 IU vitamin D3 (0.11 mm Hg; difference: -0.48 mm Hg; 95% CI:

217 obese children, whether supplementation with vitamin D3 1000 or 2000 IU/d is more effective than 600

218 Serum and SUBQ adipose tissue vitamin D3 concentrations increased proportionally to do

219 A total of 36 participants (37.5%) in the vitamin D3 group and 33 (34.4%) in the placebo group had

220 ean time to exacerbation was 240 days in the vitamin D3 group vs 253 days in the placebo group (mean

221 These findings do not support the use of vitamin D3 in adults to prevent depression.

222 noma, were randomly assigned to 1000 IU/d of vitamin D3 or placebo and 1200 mg/d of calcium carbonate

223 Compared with placebo, vitamin D3 supplementation did not significantly improve

224 ary study there was no significant effect of vitamin D3 supplementation on serum cholesterol profile

225 persistent asthma and low vitamin D levels, vitamin D3 supplementation, compared with placebo, did n

226 Vitamin D3 supplementation, compared with placebo, likew

227 of placebo or 4,200, 16,800, or 28,000 IU of vitamin D3 throughout pregnancy.

228 y 1, mean change (baseline to 3 mo) in serum vitamin D3 was -0.1 ng/mL in the placebo group and 6.8 n

229 Human vitamin E (alpha-tocopherol) catabolism is a mechanism f

230 contain 95 (Tilia cordata) and 50.6 mg/100 g vitamin E (Tilia platyphyllos).

231 Vitamin E (VitE) deficiency results in embryonic lethali

232 Additionally, the pyrolysis of vitamin E acetate also produces carcinogen alkenes and b

233 Vitamin E acetate was associated with EVALI in a conveni

234 metry to measure several priority toxicants: vitamin E acetate, plant oils, medium-chain triglyceride

235 ically, we compared them with those of other vitamin E analogs and the soy isoflavone genistein, a na

236 lf of the cows received a supplementation of vitamin E and plant extracts rich in polyphenols during

237 Unlike for genistein and the other vitamin E compounds, the GA-induced restoration of ApoE

238 Riboflavin and Vitamin E concentrations were not affected by maceration

239 berries and 22.81-46.32 g.kg(-1) in leaves, vitamin E content was 6.98-29.91 g.kg(-1) in berries and

240 Vitamin E improves liver histology in adults with nonalc

241 2 and rs79400241, respectively), involved in vitamin E metabolism, were associated with the levels of

242 acid (GA or delta-T3-13'COOH), is a natural vitamin E metabolite that has preliminarily been identif

243 ated with the levels of octadecanedioate and vitamin E metabolites (gamma-CEHC and gamma-CEHC glucuro

244 es appear to be distinct from those of other vitamin E metabolites and of genistein.

245 ron restriction and antioxidant therapy with vitamin E prevented liver disease.

246 ding carotenoids, phytosterols, squalene and vitamin E than PO, postulating its protective health ben

247 Vitamin E users had higher adjusted transplant-free surv

248 ration of small amounts of alpha-tocopherol (vitamin E) in blends with the cellobiose-triazole-linked

249 to investigate the effects of DHA (22:6n-3), vitamin E, and their probable interactions in men with a

250 nd analyzed the concentrations of vitamin C, vitamin E, zinc, and copper in both national and regiona

251 ested, the measured quantities of vitamin C, vitamin E, zinc, and copper were slightly higher than la

252 axanthin, and other antioxidants (vitamin C, vitamin E, zinc, copper) for 16 weeks.

253 onhepatic cancers were not different between vitamin E-exposed patients and controls.

254 Folate is a fundamental vitamin for metabolism in plants and humans.

255 Vitamin H (biotin) is delivered to the fetus transplacen

256 atios at dual wavelengths for the individual vitamins in all tested samples confirmed the method's st

257 Based on deficiency of vitamins in AVNFH, an enzyme-cofactor network was genera

258 ent sequences against a panel of fat-soluble vitamins in canola oil, identifying a sensor which respo

259 routine assay analysis of all water-soluble vitamins in their most common forms, including the analy

260 determination of all nine main water-soluble vitamins, in addition to two commonly used vitamers, was

261 Vitamin K activates both hepatic coagulation factors and

262 treat analysis, with 80 allocated to receive vitamin K and 79 to receive placebo.

263 vention had less bleeding with apixaban than vitamin K antagonist (VKA) and with placebo than aspirin

264 Trial to Evaluate the Safety of Apixaban vs Vitamin K Antagonist and Aspirin vs Aspirin Placebo in P

265 reater reductions with edoxaban were seen in vitamin K antagonist naive patients, patients with CHADS

266 The advent of non-vitamin K antagonist oral anticoagulants, which attenuat

267 plus P2Y12 inhibitor) versus triple therapy (vitamin K antagonist plus aspirin and P2Y12 inhibitor) i

268 ndard anticoagulants (heparin or switched to vitamin K antagonist).

269 Patients were treated with vitamin K antagonists (48.4%), parenteral heparins (27.7

270 Vitamin K antagonists (VKAs), although commonly used to

271 ring pregnancy, and anticoagulation (LMWH or vitamin K antagonists [VKAs]) should be continued until

272 Thus, vitamin K antagonists act through mimicking the key inte

273 anticoagulants (DOACs) have largely replaced vitamin K antagonists in many indications for anticoagul

274 7) and 30 on OAC (direct anticoagulants: 26, vitamin K antagonists: 4), with no differences in baseli

275 tamin K transintestinal efflux and a biliary vitamin K efflux were observed, but the specific involve

276 to explore whether ABCB1 is also involved in vitamin K efflux.

277 Vitamin K is a cofactor for proteins involved in prevent

278 wal of VKAs and high-dose vitamin K2 improve vitamin K status in patients on hemodialysis, but have n

279 To determine if vitamin K supplementation might improve arterial stiffne

280 Finally, a vitamin K transintestinal efflux and a biliary vitamin K

281 asures of fasting circulating phylloquinone (vitamin K-1) and confirmed CVD events and mortality.

282 Vitamin K-dependent proteins in vascular tissue affect v

283 10 mg daily, or rivaroxaban 10 mg daily plus vitamin K2 2000 ug thrice weekly during 18 months.

284 Collectively, these findings reveal that Vitamin K2 could induce metabolic stress and trigger AMP

285 Withdrawal of VKAs and high-dose vitamin K2 improve vitamin K status in patients on hemod

286 ssigned participants to receive 400 mug oral vitamin K2 or matching placebo once daily for a year.

287 tion and lactate generation, indicating that Vitamin K2 promotes PI3K/AKT and HIF-1alpha-mediated gly

288 ession of PI3K/AKT and HIF-1alpha attenuated Vitamin K2-increased glucose consumption and lactate gen

289 enuation of glycolysis significantly blocked Vitamin K2-induced AMPK activation and subsequently prev

290 Importantly, upon glucose limitation, Vitamin K2-upregulated glycolysis markedly induced metab

291 er (HAT) pathways between an anti-tumor drug vitamin-K3 (MQ) and a nucleobase adenine (ADN) in the pr

292 other bacteria, including iron chelators, B vitamins, methionine, lycopene, squalene and polyketides

293 s beyond the need for protein and relates to vitamins, minerals, and numerous often-overlooked nutrie

294 and their cofactors, including amino acids, vitamins, oxygen, nitrite, and sulfate.

295 wn molecular mechanisms of iron, zinc, and B vitamin processing by human-associated bacteria, compari

296 The B vitamins provide essential co-factors for central metabo

297 s (FAs), purine nucleotides (AMP and GMP), a vitamin (pyridoxal-5P), and a cofactor (heme) in both th

298 ere supplemented with pancreatic enzymes and vitamins resulting in blood concentrations almost within

299 lic consequences of sub-optimal (functional) vitamin status.

300 Of 35 studies that evaluated high-dose vitamin treatment for COVID-19, 27 (77%) excluded pregna