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1 ellular effects of retinoids (derivatives of vitamin A).
2 by antenatal or newborn supplementation with vitamin A.
3 s have led to an increase in deficiencies of vitamin A.
4  patients with measles includes provision of vitamin A.
5 espect to those introducing higher intake of vitamin A.
6 se in the prevalence of inadequate intake of vitamin A.
7 and brain, Stra6 expression was regulated by vitamin A.
8   Concurrently, fat and lung rely on dietary vitamin A.
9  ILCs were reduced in the absence of dietary vitamin A.
10  STGD and RP, especially for daily intake of vitamin A.
11 noic acid (atRA) is the active metabolite of vitamin A.
12 exaenoic acid, and docosapentaenoic acid and vitamin A.
13                           Optimal loading of vitamin A (1.46-1.48mg/100mgcasein) was found at 9.7mM p
14 tochrome P450 enzyme, cyp27c1, that converts vitamin A(1) into vitamin A(2) to produce a red-shifted
15 me, cyp27c1, that converts vitamin A(1) into vitamin A(2) to produce a red-shifted chromophore.
16 compared with deficiencies of zinc (60%) and vitamin A (34%).
17 23.2 mg/100 g), phosphorus (335.3 mg/100 g), vitamin A (486.7 ug/100 g) and alpha-tocopherol (174.5 u
18 complexes were further evaluated for unbound vitamin A, ability of milk protein to bind vitamin A and
19           Fat is found to be synergistic for vitamin A absorption.
20  critical role for STRA6 in the transport of vitamin A across blood-tissue barriers in the eyes, brai
21 e fruit was found to contain the potentially vitamin A-active keto-carotenoids sapotexanthin and cryp
22 ta support our hypothesis that low levels of vitamin A actively promote GI GVHD and are not simply a
23 Canthaxanthin is a carotenoid that lacks pro-vitamin A activity but is known to have antioxidant acti
24                                          The vitamin A activity of alpha-carotene-containing foods is
25 itamin A) and alpha-retinol (with negligible vitamin A activity).
26 tinoic acid, an active metabolite of dietary vitamin A, acts as a ligand for nuclear receptor transcr
27                         During this process, vitamin A aldehyde is shepherded within photoreceptors a
28  central 15-15' double bond to form retinal (vitamin A aldehyde).
29                                              Vitamin A (all trans retinoic acid, ATRA) treated leukem
30                                              Vitamin A (all-trans retinol) plays critical roles in ma
31 rh(-/-) mice had approximately 2-fold higher vitamin A (all-trans-retinol (all-trans-ROL)) in the neu
32 f the recommended dietary allowance (RDA) of vitamin A amongst children aged 3-10years.
33 vitamin A and was induced by the therapeutic vitamin A analog isotretinoin, which protected against s
34  was expressed in human skin, was induced by vitamin A analogs, and killed skin bacteria, indicating
35 rich source of beta-carotene, a precursor of vitamin A and a potential tool for fighting vitamin A de
36 kers by ameliorating oxidative stress, while vitamin A and beta-carotene may have additional antimyco
37  ratios for incident tuberculosis disease by vitamin A and carotenoids levels, controlling for other
38 We assessed the impact of baseline levels of vitamin A and carotenoids on tuberculosis disease risk.
39                               In conclusion, vitamin A and D deficiencies have opposing effects on mo
40           Most common deficiencies concerned vitamin A and D, iron, and zinc.
41 ls and MetMb percentage and higher levels of vitamin A and E.
42  Micronutrient deficiencies such as those of vitamin A and iron affect a third of the world's populat
43  that disrupt cognition are also linked with vitamin A and point to their possible treatment with vit
44 d vitamin A, ability of milk protein to bind vitamin A and solubility of protein and vitamin A as aff
45 ice to diets sufficient and insufficient for vitamin A and used heterozygous siblings as controls.
46 to determine concentrations of supplementary vitamin A and vitamin E esters and beta-carotene in infa
47        RELMalpha expression required dietary vitamin A and was induced by the therapeutic vitamin A a
48 sources contributing to excessive intakes of vitamin A and zinc among infants and toddlers may need f
49  excessive intakes (greater than the ULs) of vitamin A and zinc, respectively.
50                               The effects of vitamin A and/or vitamin D deficiency were studied in an
51 specific differences were observed regarding vitamins A and B12, zinc, and potassium.
52           Greater nutrient intake densities (vitamins A and B6, calcium, protein, and zinc) were nega
53                           Mice deficient for vitamins A and D (VADD) had disease progression similar
54 al fat, cholesterol, fatty acids, vitamin C, vitamins A and D, and 21 mineral elements (including tot
55  high-GHGE diets contained higher amounts of vitamins A and D, choline, calcium, iron, and potassium.
56 a at the University of Wisconsin, working on vitamins A and D, respectively.
57 ns on proteolysis, lipolysis and calcium and vitamins A and D3 bioaccessibility in salmon, sardine, s
58 lises selective dual-channel fluoresence for vitamins A and E and visible absorbance for beta-caroten
59           This study evaluated the intake of vitamins A and E of infants from 'ready-to-feed' foods a
60 A carotenoid, is cleaved to produce retinol (vitamin A) and alpha-retinol (with negligible vitamin A
61 e report that retinoic acid (RA) or retinol (vitamin A) and ascorbate (vitamin C) act as modulators o
62 corbate (vitamin C), beta-carotene, retinol (vitamin A), and urate.
63                                        Iron, vitamin A, anemia, malaria, and anthropometric measures
64                  Low and deficient levels of vitamin A are common in low- and middle-income countries
65                              Most effects of vitamin A are exerted by its metabolite, retinoic acid (
66 bind vitamin A and solubility of protein and vitamin A as affected by complexation.
67 at LAL is required for efficient nutritional vitamin A availability.
68 irements) were >40% for calcium, niacin, and vitamins A, B-6, and B-12.
69 roperties of encapsulated functional lipids--vitamin A, beta-carotene and omega-3 fish oil--on the st
70  order to increase polyunsaturated lipid and vitamin A bioaccessibility and avoid formation of toxic
71  aim was to estimate iron, zinc, protein and vitamin A bioavailability from individual diets, and inv
72                                          Pro-vitamin A biofortified (yellow) cassava has the potentia
73 ect refines approaches to interpret iron and vitamin A biomarker values in settings of inflammation a
74 ght to inform the interpretation of iron and vitamin A biomarkers (ferritin, serum transferrin recept
75 red serum concentrations of certain iron and vitamin A biomarkers, confirming the need to consider ad
76 n STH infection intensity and limitations of vitamin A biomarkers.
77                                              Vitamin A bound to retinol binding protein 4 (RBP4) cons
78       The liver is the main storage organ of vitamin A, but activation of the retinoic acid receptors
79 e receptor STRA6 mediates cellular uptake of vitamin A by recognizing RBP-retinol to trigger release
80              The regulation of iNKT cells by vitamin A by the P2X7 pathway is important to prevent ab
81 ly, they are good sources of carotenoids and vitamins A, C and B(6) and showed good antioxidant poten
82 ions that use of any antioxidant supplement (vitamins A, C, and E; carotenoids; coenzyme Q10) both be
83 ship between intake of antioxidant vitamins (vitamins A, C, D, and E) and individual carotenoids (alp
84 ource of phenolic compounds, fatty acids and vitamins A, C, E.
85 e measured directly to accurately assess the vitamin A capacity of alpha-carotene-containing foods.
86 y retinoic acid 6), is the RBP4 receptor and vitamin A channel; however, the role of STRA6 in vitamin
87  relations between bone health and different vitamin A compounds.
88         In controls, this regulation reduced vitamin A consumption when the dietary supply was limite
89 extendable to whole milk powders where total vitamin A content data can be calculated by summing the
90  breeding vitreous kernel varieties and high vitamin A content in maize.
91  acids (SCFAs) induced the expression of the vitamin A-converting enzyme RALDH1 in intestinal epithel
92 he CD103(+)CD11b(+) DC subset expressing the vitamin A-converting enzyme retinaldehyde dehydrogenase
93 helial cells correlated with the activity of vitamin A-converting enzymes in mesenteric lymph node de
94 nding that several key micronutrients (e.g., vitamin A, copper, manganese, and zinc) support iron's f
95        Additionally, we found that LSFF with vitamin A could protect nearly 3 million children per ye
96 manganese, zinc, phosphorous, boron, cobalt, Vitamins A, D, B6, thiamine, riboflavin, niacin and coba
97 ltiple proximal risk factors (e.g., iron and vitamin A deficiencies, inflammation, malaria, and body
98 ses to calculate changes in anemia, iron and vitamin A deficiencies, stunting, wasting, and underweig
99 in but no effect on anthropometry or iron or vitamin A deficiencies.
100 , in Kenya, a lower prevalence of folate and vitamin A deficiencies.
101 andardized prevalence per 100,000 persons of vitamin A deficiency (25,155 to 19,187), undercorrected
102 eficiency (ferritin <15 ng/mL or 32 pmol/L), vitamin A deficiency (retinol-binding protein <14.7 mug/
103                                              Vitamin A deficiency (VAD) and soil-transmitted helminth
104                                              Vitamin A deficiency (VAD) has been hypothesized to play
105  vitamin A and a potential tool for fighting vitamin A deficiency (VAD) in developing countries.
106 The accurate estimation of the prevalence of vitamin A deficiency (VAD) is important in planning and
107                                              Vitamin A deficiency (VAD) is one of the most prevalent
108 ta-carotene accumulation that will alleviate vitamin A deficiency among people who rely on sorghum as
109 P flour could be used for the eradication of vitamin A deficiency as they were found to meet 29 and 8
110                                              Vitamin A deficiency continues to be a major public heal
111                                              Vitamin A deficiency impairs epithelial integrity, incre
112 n in improving vitamin A status and reducing vitamin A deficiency in children.
113                                              Vitamin A deficiency increases susceptibility to skin in
114                                              Vitamin A deficiency is a public health problem in sub-S
115                                              Vitamin A deficiency remains a nutritional concern in su
116                                              Vitamin A deficiency results in a deterioration of these
117 upport of programs for the global control of vitamin A deficiency still face vocal opposition by some
118                                              Vitamin A deficiency strongly predicted the risk of inci
119 possible confounders, we found that baseline vitamin A deficiency was associated with a 10-fold incre
120 iron and zinc deficiency were comparable but vitamin A deficiency was lower by the probability method
121                                           In vitamin A deficiency, iNKT cells fail to express P2X7 an
122 ealth strategies to prevent ocular injuries, vitamin A deficiency, perinatal infections and retinopat
123  with a plant-food-based approach to address vitamin A deficiency, reports the analysis of total caro
124 er the effects, treatment, and prevention of vitamin A deficiency, while faced with intense criticism
125 tect nearly 3 million children per year from vitamin A deficiency.
126 nd cardiac edema, phenotypes associated with vitamin A deficiency.
127 vitamin A is an efficient strategy to combat vitamin A deficiency.
128  be used as food-based supplements to reduce vitamin A deficiency.
129 cient (odds ratio [OR]: 0.981 (0.961-1.001), Vitamin A deficient (OR: 0.813 (0.794-0.833)), and have
130                                              Vitamin A deficient (VAD) mice fared worst with more rap
131 gher frequencies among CD4(+) splenocytes of vitamin A deficient vs. sufficient mice.
132  spermatogonial differentiation identical to vitamin A-deficient (VAD) mice; (2) the blockage of sper
133 s in the small intestine of mice raised on a vitamin A-deficient diet.
134                                              Vitamin A-deficient mice infected with S. mansoni had di
135 d development of enteric lymphoid tissues in vitamin A-deficient mice.
136                                              Vitamin A degradation was high and occurred rapidly: mor
137 beta was required for the development of key vitamin A-dependent adaptive immune responses, including
138                 Epithelial RARbeta activated vitamin A-dependent expression of serum amyloid A (SAA)
139 is known about how epithelial cells regulate vitamin A-dependent intestinal immunity.
140 cid receptor beta (RARbeta) is essential for vitamin A-dependent intestinal immunity.
141 n antimicrobial protein that is required for vitamin-A-dependent resistance to skin infection.
142 that (1) stem cells are kept inactive by the vitamin A derivative retinoic acid, which is synthesized
143 A proteins circulate in association with the vitamin A derivative retinol, suggesting that SAAs trans
144 e photoreceptors, contains 11-cis-retinal (a vitamin A derivative) and light isomerizes it to all-tra
145 s high, because it assesses translocation of vitamin A derivatives across the retinal pigment epithel
146 eroxy- and 4-hydroxy-2-alkenals, and several vitamin A derived metabolites were generated.
147 t consists of opsin covalently linked to the vitamin A-derived chromophore, 11-cis-retinaldehyde.
148 lated with vitamin A levels, indicating that vitamin A did not protect against mucosal injury.
149 lesterol accumulation in the RPE, induced by vitamin A dimers or oxidized LDL, inhibits these defense
150                             The stability of vitamin A during cooking and storage is, however, low.
151 naldehyde is further metabolized to retinol (vitamin A), esterified and packaged into triacylglycerol
152  calculated by summing the innate long-chain vitamin A esters with the added esters.
153                Feeding the hep-LAL-ko mice a vitamin A excess/high-fat diet (VitA/HFD) further increa
154 CE ATRA, a biologically active metabolite of vitamin A, exerts pleiotropic biological effects, includ
155 etinoic acid (RA), a bioactive derivative of vitamin A, exhibits diverse effects on gene transcriptio
156 atter would meet nearly a half of the RDA of vitamin A for pregnant and lactating women.
157      beta-Carotene is an important source of vitamin A for the mammalian embryo, which depends on its
158                         Mice maintained on a vitamin A-free diet lose HSCs and show a disrupted re-en
159 d the total daily supply of provitamin A and vitamin A from diet and supplements was equivalent to 22
160 Retinoic acid (RA), a metabolite of retinol (vitamin A), functions as a ligand for nuclear RA recepto
161                               The history of vitamin A goes back over one hundred years, but our real
162                                              Vitamin A has essential but largely unexplained roles in
163     Retinoic acids, which are metabolites of vitamin A, have been shown to be involved in multiple T
164   Despite the enrichment of hepatocytes with vitamin A, HCV still establishes an efficient viral life
165                  Thus, STRA6 is critical for vitamin A homeostasis and the adaption of this process t
166                                              Vitamin A homeostasis is critical to normal cellular fun
167 between diet and immunity and indicates that vitamin A homeostasis must be tightly controlled by ISX
168 min A channel; however, the role of STRA6 in vitamin A homeostasis remains to be defined in vivo We s
169            There were significant changes in vitamin A homeostasis.
170 ed in the prevalence of inadequate intake of vitamin A if rice biofortified with beta-carotene were c
171 gest an evolutionarily conserved function of vitamin A in animal photoperiodism.
172 tions might provide insight into the role of vitamin A in cancer etiology.
173 iations may provide insight into the role of vitamin A in cancer etiology.
174 ogether, our data show an important role for vitamin A in controlling innate lymphoid cells and, cons
175 nd its relative contribution to postprandial vitamin A in humans after consumption of raw carrots.Hea
176                        Estimation of unbound vitamin A in milk protein-Vit A complexes was carried ou
177        Our findings identify a novel role of vitamin A in regulating iNKT cell homeostasis in many ti
178 testinal homeostasis, are also influenced by vitamin A in the small intestines.
179  in a cohort of children who were exposed to vitamin A in utero or at birth.The aim of this study was
180 0% substitution) decreased the prevalence of vitamin A inadequacy from baseline 78% in women and 71%
181  In the multivariate analysis, inflammation, vitamin A insufficiency, socioeconomic status, and age w
182        Interestingly, STGD patients with low vitamin A intake (<600 microg RAE/day) showed significan
183  TB was observed for the highest quartile of vitamin A intake (hazard ratio = 0.71, 95% confidence in
184 Software was used to estimate usual rice and vitamin A intake for the simulation analyses.
185 ndividual level to 1) determine the rice and vitamin A intake in nonpregnant, nonlactating women of r
186                                         Mean Vitamin A intake in the last six months was 0.63 (0.626-
187 orld impact of LSFF with key micronutrients (vitamin A, iodine, iron, folic acid) on improving micron
188 uacy ratio calculations: calcium, vitamin C, vitamin A, iron, fiber, and protein.
189                                              Vitamin A is a dietary component that is essential for t
190                                We found that vitamin A is a dominant factor that controls the populat
191                                              Vitamin A is a multifunctional vitamin implicated in a w
192                                              Vitamin A is a potent regulator of adaptive immunity.
193                                              Vitamin A is absorbed and converted to its bioactive der
194                    Food supplementation with vitamin A is an efficient strategy to combat vitamin A d
195  key at the intestinal border, where dietary vitamin A is first absorbed.
196                                        Since vitamin A is food-derived, tissue-specific uptake and st
197 iviral treatment for measles; treatment with vitamin A is recommended for younger children to decreas
198                            Here we show that vitamin A is required for the phenotypic conversion of i
199                                              Vitamin A is susceptible to light and heat and thus requ
200  hydrophobic retinol, the main form in which vitamin A is transported.
201                                    Retinoid (vitamin A) is an essential diet constituent that governs
202 -trans retinoic acid (ATRA), a derivative of vitamin A, is a common component in cosmetics and commer
203 tinoic acid (atRA), the active metabolite of vitamin A, is a critical signaling molecule during embry
204 tinoic acid (atRA), the active metabolite of vitamin A, is an essential signaling molecule in all cho
205                             Furthermore, the vitamins (A, K and B group) and mineral contents (N, P,
206 isease risk with each decreasing quartile of vitamin A level.
207 well documented association between abnormal vitamin A levels and renal malformations in humans, and
208 m infections were increased in patients with vitamin A levels below the median (24% vs 8% at 1 year,
209       GI GVHD was increased in patients with vitamin A levels below the median (38% vs 12.4% at 100 d
210 er transplant was increased in children with vitamin A levels below the median (r = -0.34, P = .03).
211  except in vacuum-stored meat, ii) decreased vitamin A levels in the LT, iii) decreased vitamin E lev
212                                We determined vitamin A levels of the eyes, brain, and testis, which h
213                                         Free vitamin A levels were measured in plasma at day 30 postt
214                  We hypothesized that higher vitamin A levels would reduce the risk of graft-versus-h
215  mucosal injury, but was not correlated with vitamin A levels, indicating that vitamin A did not prot
216                                              Vitamin A loss is accompanied by stellate cell activatio
217 etinol, the most biologically active form of vitamin A, may influence cancer-related biologic pathway
218 nol dehydrogenase 10 (Rdh10), which perturbs Vitamin A metabolism and retinoid signaling, exhibit ful
219  thus can be utilized to discover defects in vitamin A metabolism during the regeneration of the visu
220 ut also further suggests a potential role of vitamin A metabolism in terminal differentiation of the
221 ne through this important branching point of vitamin A metabolism.
222                                          The vitamin A metabolite all-trans retinoic acid (ATRA) indu
223 s regulated by diverse inputs, including the vitamin A metabolite retinoic acid (RA).
224 ell autonomous manner and is elicited by the vitamin A metabolite, retinoic acid.
225 etinol, the most biologically active form of vitamin A, might influence cancer-related biological pat
226 nutritional supplements, such as vitamin B6, vitamin A, multivitamins, antioxidants, and iron and die
227 ga-6 lipids and, for the first time, that of vitamin A, naturally present in cod liver oil.
228 ns existed between maternal usual intakes of vitamin A, niacin and riboflavin and milk retinol, nicot
229 her maternal or newborn supplementation with vitamin A on intelligence, memory, and motor function.
230 f antenatal and newborn supplementation with vitamin A on the cognitive function of children at 8 y o
231  Our results highlight the impact of dietary vitamin A on the regulation of cell-cycle-mediated stem
232 A and point to their possible treatment with vitamin A or RA.
233 e genetic evidence that the clock-controlled vitamin A pathway mediates photoperiod responsiveness in
234 clock-deficient mutants, suggesting that the vitamin A pathway operates downstream of the circadian c
235 that included several genes belonging to the vitamin A pathway.
236 otene contributing 12-35% of newly converted vitamin A (predicted contribution = 25.5%).
237 meostasis and is involved in the pathway for vitamin A production.
238                                              Vitamin A promotes development of mucosal tolerance and
239        Our findings provide insight into how vitamin A promotes resistance to skin infection.
240  found that after a period of depletion, pro-vitamin A (PVA) carotenoids were preferentially diverted
241             However, the mechanisms by which vitamin A regulates skin immunity remain unclear.
242                                              Vitamin A regulates the adaptive immune response and a m
243 ntribution of OFSP-wheat composite breads to vitamin A requirements were evaluated.
244 we report that ATRA, an active metabolite of vitamin A, restores mechanical quiescence in PSCs via a
245 e combined influence of different factors on vitamin A retention and the oxidative status of wheat fl
246                                              Vitamin A retention was related to the extent of oxidati
247 torage, and the factors that mostly affected vitamin A retention were the storage duration, the type
248 s study, the estimated total daily intake of vitamin A (retinol equivalents) and vitamin E (alpha-toc
249                        Retinoic acid (RA), a vitamin A (retinol) derivative, has pleiotropic function
250 sociated with minimal increases in preformed vitamin A (retinol).
251  simultaneously quantifying iron (ferritin), vitamin A (retinol-binding protein), and inflammation (C
252                          The alcohol form of vitamin A (retinol/ROL) can be oxidized to all-trans-ret
253 foods, whereas the legumes, dairy, eggs, and vitamin A-rich fruit and vegetable food groups were each
254 urce foods, fruits, and vegetables including vitamin A-rich ones was higher in the adequate than in t
255 ewly absorbed alpha-carotene to postprandial vitamin A should not be estimated but should be measured
256 [ferritin or soluble transferrin receptor or vitamin A status (retinol-binding protein or retinol)] a
257 alaria infection, and biomarkers of iron and vitamin A status and compare adjustment approaches, and
258 knowledge, the associations between maternal vitamin A status and offspring bone development have not
259 ug beta-carotene/g) consumption in improving vitamin A status and reducing vitamin A deficiency in ch
260 nce global knowledge with regard to iron and vitamin A status assessment in women and preschool child
261 t it must be assessed in surveys of iron and vitamin A status for valid interpretation of micronutrie
262 entrations may have the potential to improve vitamin A status in maize-consuming populations.
263  efficacy of biofortified cassava to improve vitamin A status of Nigerian preschool children.
264 how the intestinal epithelium senses dietary vitamin A status to regulate adaptive immunity, and high
265  often used in population surveys to measure vitamin A status, but its interpretation is challenging
266 ntial to contribute significantly to improve vitamin A status, especially in populations that are dif
267 ation in Kenyan schoolchildren with marginal vitamin A status.
268 n be an efficacious, new approach to improve vitamin A status.
269 ing gene and protein expression profiles and vitamin A storage, resembled that of hepatic stellate ce
270 cutaneous anaphylaxis (PCA) model on VAD and vitamin A supplementation (VAS) model in wild-type mice
271                                              Vitamin A supplementation among individuals at high risk
272                                              Vitamin A supplementation might improve transplant outco
273                          The impact of early vitamin A supplementation on neurodevelopmental function
274 ration treatment of diarrhoea, and receiving vitamin A supplementation) and household characteristics
275 tinoic acid, the biologically active form of vitamin A that acts as a transcription factor ligand to
276 otein 1 (CRBP1) is essential for trafficking vitamin A through the cytoplasm.
277      Stability can be improved by binding of vitamin A to milk protein.
278 ient than other target tissues at converting vitamin A to retinoic acid (RA), which activates retinoi
279 hway also governs the metabolism of retinol (vitamin A) to its transcriptionally active metabolite, r
280 e protects the labile retinoid moiety during vitamin A transport.
281                                  Smoking and vitamin A use was not associated significantly with base
282                            We detected serum vitamin A (VA) concentration in different phenotypes of
283                    In some regions, multiple vitamin A (VA) interventions occur in the same target gr
284                                              Vitamin A (VA; retinol) supplementation is used to reduc
285                        Effects on folate and vitamin A varied between studies.
286 ere used for the preparation of milk protein-Vitamin A (Vit A) complexes.
287 itate has been found to be the most abundant vitamin A vitamer, but retinyl oleate is the prevalent f
288 on estimates, we estimated the production of vitamin A, vitamin B12, folate, iron, zinc, calcium, cal
289 ere associated nominally with decreased risk-vitamin A, vitamin B6, beta-carotene, lutein and zeaxant
290  <= 0.0005) with decreased risk of late AMD: vitamin A, vitamin B6, vitamin C, folate, beta-carotene,
291                           Mean adequacies of vitamin A, vitamin C, folate, calcium, iron, and zinc an
292 s between CRP, AGP, and biomarkers for iron, vitamin A, vitamin D, vitamin B-12, and folate from 0 to
293 Requirement or the Adequate Intake level) of vitamin A, vitamin K, magnesium, zinc, and copper was as
294 ts consisting of beta-carotene (precursor to vitamin A), vitamins C and E and the mineral magnesium (
295 pendence of lymphoid tissue inducer cells on vitamin A was furthermore illustrated by impaired develo
296 nicotinamide adenine dinucleotide (NADH) and vitamin A were scanned on sturgeon samples kept at 4 deg
297 nthelmintics, antibiotics, probiotics, zinc, vitamin A, withholding breastfeeding, extra doses, or va
298  of 24 (25%) STGD patients a daily intake of vitamin A within the recommended range while 14/24 (58.3
299                       MNPs (containing iron, vitamin A, zinc, and 11 other micronutrients) and other
300 et included measures of serum ferritin (SF), vitamin A, zinc, and CRP measured using different assays

 
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