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1 c network rewiring to promote survival under vitamin deficiency.
2 p between various micronutrient, protein and vitamin deficiencies.
3 nt populations can be a harbinger of other B-vitamin deficiencies.
4 result from dementia-related nutritional and vitamin deficiencies.
5 why homocysteine is not a good biomarker for vitamin deficiencies.
8 sical and recent evidence for stress-induced vitamin deficiencies and for plant responses that counte
9 ori reasonable that stressed plants suffer B vitamin deficiencies and that certain stress symptoms ar
10 /L) and severe (>100 umol/L) HHcy related to vitamin deficiencies and/or inherited disorders with CVD
11 wever, given known relations between B-group-vitamin deficiency and both hyperhomocysteinaemia and ne
12 ents substantially reduces the prevalence of vitamin deficiency and depletion markers before and duri
13 isrupt bile acid amidation cause fat-soluble vitamin deficiency and growth failure, indicating the im
15 cysteine; (ii) high methionine intake with B vitamin deficiency and hyperhomocysteinemia; and (iii) n
18 hether they correlated with other markers of vitamin deficiency; and whether they changed with cobala
20 lk or on how they are affected by maternal B vitamin deficiencies, antiretroviral therapy, or materna
21 n which much of the clinical descriptions of vitamin deficiencies are based and summarise current ava
25 inated intravascular coagulation, or certain vitamin deficiencies have been ruled out may require eva
26 mia, such as gastrointestinal bleeding, iron/vitamin deficiencies, hemolysis, and drug toxicities, we
31 D] concentrations and functional outcomes of vitamin deficiency in young children and breastfed and n
32 The percentage of nutrition items related to vitamin deficiencies increased from 1986 to 1993 on both
35 some vitamins, above levels causing classic vitamin deficiency, is a risk factor for chronic disease
37 h may result from gastrointestinal bleeding, vitamin deficiency, or liver-damaging diseases, such as
39 is or later onset of unexplained fat-soluble vitamin deficiency should be screened for defects in bil
40 on in 10 pediatric patients with fat-soluble vitamin deficiency, some with growth failure or transien
41 ng neurologic and psychologic dysfunction in vitamin deficiency states and in cases of congenital def
44 of poor pregnancy outcome, including other B vitamin deficiencies that result in homocysteinemia, ant
45 opathy occurs most frequently secondary to B-vitamin deficiencies, which is suspected to increase in
46 try of B vitamins and explore the concept of vitamin deficiency with the help of information from mam