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1 ntravitreous injection of Vitrase for severe vitreous hemorrhage.
2 ed with patient sex, age, or the presence of vitreous hemorrhage.
3 nd safe modalities for treatment of diabetic vitreous hemorrhage.
4 viable treatment approaches for PDR-related vitreous hemorrhage.
5 of vision in his left eye caused by a dense vitreous hemorrhage.
6 rectomy, central-involving macular edema, or vitreous hemorrhage.
7 r, inferior pre-retinal hemorrhage, and mild vitreous hemorrhage.
8 dates, and the development of a self-limited vitreous hemorrhage.
9 clearing was associated with the density of vitreous hemorrhage.
10 patients initiating therapy for PDR-related vitreous hemorrhage.
11 arance of floaters, along with a retinal and vitreous hemorrhage.
12 (adjusted for baseline VA) and clearance of vitreous hemorrhage.
13 rhegmatogenous retinal detachment along with vitreous hemorrhage.
14 Retinal and vitreous hemorrhage.
15 R), vitrectomy was resorted for non clearing vitreous hemorrhage.
16 relation between rivaroxaban and spontaneous vitreous hemorrhage.
17 agulation may be associated with spontaneous vitreous hemorrhage.
18 sk of retinal detachment in fundus-obscuring vitreous hemorrhage.
19 compared with observation alone in eyes with vitreous hemorrhage.
20 Forty-eight patients had bilateral vitreous hemorrhage.
21 of contraction-promoting growth factors, or vitreous hemorrhage.
23 adverse event from baseline to week 100 was vitreous hemorrhage (0.9% vs. 6.8% in the IAI 2Q4 + PRN
26 age (1), central retinal vein occlusion with vitreous hemorrhage (1), and proliferative vitreoretinop
27 st frequent serious ocular adverse event was vitreous hemorrhage (1.3%, 0.7%, and 1.9%, respectively)
28 nts with epimacular proliferation (1.22) and vitreous hemorrhage (1.40) were also significantly eleva
29 quired vitreoretinal surgery for nonclearing vitreous hemorrhage, 1 in each 30 ms group; insignifican
30 se events were cataract (17.76%) followed by vitreous hemorrhage (12.10%) and retinal detachment (5.6
31 postoperative IVIs had a higher incidence of vitreous hemorrhage (18.5% vs. 3.2%, P = 0.039) than the
32 d at presentation; however, 8.0% of isolated vitreous hemorrhages, 19.2% of retinal breaks without de
35 resolving) included choroidal effusion (1), vitreous hemorrhage (3), Descemet detachment (1), and pe
36 66% (80%); neovascular glaucoma, 15% (22%); vitreous hemorrhage, 35% (42%); and secondary enucleatio
39 lar AESI; most common were cataract (11.4%), vitreous hemorrhage (6.1%), and conjunctival thickening
40 (9.1%, n=12), sterile vitritis (7.6%, n=10), vitreous hemorrhage (6.8%, n=9), choroidal detachment (3
41 5), sluggish pupil reactivity (6.9%, n = 4), vitreous hemorrhage (6.9%, n = 4), early hypofluorescenc
42 ses, 2 (0.8%) retinal detachments, 13 (5.2%) vitreous hemorrhages, 6 (2.4%) conjunctival erosions, an
43 to have PFV (49%), Coats' disease (20%), or vitreous hemorrhage (7%); those 2 to 5 years of age were
44 lications included IOL decentration (12.5%), vitreous hemorrhage (7.5%), IOL slippage (5%), IOL tilt
45 etinal detachment (12.5%), cataract (10.5%), vitreous hemorrhage (7.9%), and endophthalmitis (3.9%).
46 e (187.8), posterior capsule rupture (80.1), vitreous hemorrhage (76.9), and retinal detachment (20.8
47 most common postoperative complications were vitreous hemorrhage (8.2%, 0-41%), elevated intraocular
48 edema (14.4%), neovascular glaucoma (0.5%), vitreous hemorrhage (8.4%), and tractional retinal detac
53 ccurrence of a retinal tear, pre-retinal and vitreous hemorrhages after completing a session of whole
54 etic retinopathy in both eyes presented with vitreous hemorrhage and 6/60 visual acuity in his left e
55 rom a young boy with a history of idiopathic vitreous hemorrhage and a female infant with familial ex
57 Physician treatment algorithms for diabetic vitreous hemorrhage and non-proliferative diabetic retin
59 hole or epiretinal membrane), 1 patient had vitreous hemorrhage and the remaining 4 patients had rhe
61 of retinal detachment, choroidal hemorrhage, vitreous hemorrhage, and disorganized intraocular conten
62 urgical time, early (<1 month) postoperative vitreous hemorrhage, and mean change in best-corrected v
63 (tractional retinal detachment, nonclearing vitreous hemorrhage, and neovascular glaucoma) and 111 c
64 ion retinopathy, radiation optic neuropathy, vitreous hemorrhage, and neovascular glaucoma) were anal
65 mplete vascularization of peripheral retina, vitreous hemorrhage, and persistence of massive intravit
66 s after implant surgery, including cataract, vitreous hemorrhage, and retinal detachment, were relati
70 well as retinal/vitreous detachment, retinal/vitreous hemorrhage, and retinal tear (FAERS: ROR = 2.44
75 pathy, and concomitant retinal detachment or vitreous hemorrhage, and who were already on antidiabeti
78 ultivariable analyses identified presence of vitreous hemorrhage at baseline, increasing age, absence
82 ck hours, number/size of breaks, presence of vitreous hemorrhage, axial length, or grade/extent of PV
83 who presented with nontraumatic, nonsurgical vitreous hemorrhage between 2002 and 2012 were reviewed.
84 Only 1 of 57 eyes (1.8%) showed a transient vitreous hemorrhage, biopsy yield was 100% for genetic a
85 oup, but rates of retinopathy complications (vitreous hemorrhage, blindness, or conditions requiring
86 noblastomas include Coats' disease, PFV, and vitreous hemorrhage, but the spectrum varies depending o
88 cular hypertension, hemorrhagic retinopathy, vitreous hemorrhage, combined traction and rhegmatogenou
90 tachment, proliferative retinal disease, and vitreous hemorrhage contain varying amounts of growth fa
91 es with findings that would bias toward PPV (vitreous hemorrhage, dense cataract, proliferative vitre
93 etween-group differences in the incidence of vitreous hemorrhage (DEX, 3 eyes [4%]; IVT, 1 eye [1%];
96 a randomized clinical trial among eyes with vitreous hemorrhage due to proliferative diabetic retino
98 glaucoma, macular edema, retinal detachment, vitreous hemorrhage, epiretinal membrane, and band kerat
99 edema, macular ischemia, foveal hemorrhage, vitreous hemorrhage, epiretinal membrane, and retinal de
101 our patient is the third documented case of vitreous hemorrhage following whole-body vibration train
102 safe and efficacious in preventing recurrent vitreous hemorrhage for PDR in the early postoperative p
103 d adults with vision loss due to PDR-related vitreous hemorrhage for whom vitrectomy was considered.
106 ed with vitrectomy) compared with saline for vitreous hemorrhage from PDR that precludes placement or
108 including 205 adults with vison loss due to vitreous hemorrhage from proliferative diabetic retinopa
111 hemorrhage density was scored using a 0 to 4 vitreous hemorrhage grading scale in 12 radial segments
115 kin-8 and TNFalpha occurred in patients with vitreous hemorrhage; however, the only statistical diffe
116 t were associated with a lower occurrence of vitreous hemorrhages (HR, 0.74; 95% CI, 0.68-0.80), neov
117 or the secondary surgery included persistent vitreous hemorrhage in 1.9% (14/739), rhegmatogenous ret
118 n 15%, ocular hypertension in 10%, transient vitreous hemorrhage in 10%, retinal detachment in 5%, an
120 ient ocular hypertension in 44 eyes (11.3%), vitreous hemorrhage in 31 eyes (7.9%), and transient hyp
123 prescription (aDelta: -1.00, P = 0.024), and vitreous hemorrhage in at least 1 eye (aDelta: -1.92, P
124 d retinopathy of prematurity may suffer from vitreous hemorrhage in the absence of retinal tears, det
125 r RRDs occurred in 12.4% of patients who had vitreous hemorrhage, lattice degeneration, or a history
126 resence of lattice degeneration, presence of vitreous hemorrhage, location of retinal breaks, macular
127 acuity, lens status, presence or absence of vitreous hemorrhage, myopia, lattice degeneration, and s
128 embrane (n = 3), retinal hemorrhage (n = 2), vitreous hemorrhage (n = 1), retinal neovascularization
129 = 9) proliferative vitreoretinopathy (PVR), vitreous hemorrhage (n = 10), vitreous opacities (n = 8)
131 hage (n = 3), preretinal hemorrhage (n = 1), vitreous hemorrhage (n = 2), and retinal tear (n = 1).
132 ina (n = 1); and had vitreous seeds (n = 3), vitreous hemorrhage (n = 2), retinal hemorrhage (n = 4),
133 stent fetal vasculature (PFV; n = 158; 28%), vitreous hemorrhage (n = 27; 5%), ocular toxocariasis (n
134 edema (n = 14 [8.6%]); retinal detachment or vitreous hemorrhage (n = 3 [1.9%]); posterior uveitis, r
135 etic tractional retinal detachment (n = 49), vitreous hemorrhage (n = 40), full-thickness macular hol
137 ude misplaced gas injection, subretinal gas, vitreous hemorrhage, new retinal breaks, failure to reat
138 ect a significant difference in incidence of vitreous hemorrhage, NVI, NVG, or need for vitrectomy.
141 phics, visual acuity, cause of "spontaneous" vitreous hemorrhage, ocular and systemic findings at pre
142 regression analysis revealed the presence of vitreous hemorrhage (odds ratio [OR], 7.29; P < 0.001),
143 p, which was associated with RVO (P = .005), vitreous hemorrhage on presentation (P = .001), and no p
144 p, which was associated with RVO (P = .005), vitreous hemorrhage on presentation (P = .001), and no p
145 of retinal tears, extent of RRD, presence of vitreous hemorrhage or choroidal detachment, date and le
146 P < .001), pseudophakia (OR 1.25, P < .001), vitreous hemorrhage (OR 1.22, P = .001), and worse syste
147 III vs. I OR, 2.29 [1.53-3.41]; P < 0.0001), vitreous hemorrhage (OR, 2.29 [1.54-3.1]; P < 0.0001), a
148 vitreous pigment (OR, 57.0; CI, 39.7-81.7), vitreous hemorrhage (OR, 5.9; CI, 4.6-7.5), lattice dege
149 anretinal photocoagulation, (4) experiencing vitreous hemorrhage, or (5) undergoing vitrectomy for th
158 real aflibercept-based strategy for diabetic vitreous hemorrhage presents a 76% increased profit over
160 he plan, history of nondiabetic retinopathy, vitreous hemorrhage, previous RD, or any other surgicall
161 retinal neovascularization, with subsequent vitreous hemorrhage, putatively through inflammatory mec
162 rgical procedure optimization, postoperative vitreous hemorrhage rate was 4.5% (7/157; 1 event classi
163 tion completion rates, and reduced recurrent vitreous hemorrhage rates suggest biologic activity of r
165 and development of fellow eye complications (vitreous hemorrhage, retinal break, and retinal detachme
166 curately coded (k = 0.61, 0.48, and 0.52 for vitreous hemorrhage, retinal detachment, and neovascular
167 tely coded (kappa = 0.61, 0.48, and 0.52 for vitreous hemorrhage, retinal detachment, and neovascular
168 (DR) and DR-related complications (including vitreous hemorrhage, retinal detachment, and neovascular
169 g outcome defined as the first occurrence of vitreous hemorrhage, retinal detachment, anterior segmen
170 ly was performed in patients with persistent vitreous hemorrhage, retinal detachment, lens dislocatio
171 gressive bullous retinoschisis, non-clearing vitreous hemorrhage, rhegmatogenous retinal detachment (
172 wound leaks, hypotony, choroidal detachment, vitreous hemorrhage, rhegmatogenous retinal detachment,
173 examination: preoperative PVR grade A or B, vitreous hemorrhage, RRD involving >= 50% of retinal are
175 panretinal photocoagulation for nonclearing vitreous hemorrhage secondary to proliferative diabetic
177 reases in local growth factor expression, or vitreous hemorrhage, suggesting that other mechanisms ar
178 ed and refined to help prevent postoperative vitreous hemorrhage using cryotherapy around sclerotomy
179 .006) in addition to presenting acuity, age, vitreous hemorrhage, uveal prolapse, and afferent pupill
180 ork Protocol S suggested that vitrectomy for vitreous hemorrhage (VH) or tractional retinal detachmen
181 dicting eyes that developed new or recurrent vitreous hemorrhage (VH) or tractional retinal detachmen
182 eal bevacizumab (IVB) use in patients with a vitreous hemorrhage (VH) secondary to proliferative diab
183 ) undergoing pars plana vitrectomy (PPV) for vitreous hemorrhage (VH) with IVB pretreatment were pros
184 underwent pars plana vitrectomy (PPV): 6 for vitreous hemorrhage (VH), 1 for epiretinal membrane (ERM
185 resence of fibrovascular proliferation (FP), vitreous hemorrhage (VH), and tractional retinal detachm
186 secondary outcome included incidence of PPV, vitreous hemorrhage (VH), or tractional retinal detachme
187 ng vitreoretinal surgeon for the presence of vitreous hemorrhage (VH), retinal tear (RT), retinal det
188 adverse visual outcomes including blindness, vitreous hemorrhage (VH), tractional retinal detachment
189 .79 versus 0.77 versus 1.20 (P < 0.0001) for vitreous hemorrhage (VH), vitreomacular interface abnorm
193 ons after first-eye PVD within 1 year (24.3% vitreous hemorrhage [VH], 24.1% retinal break [RB], 5.2%
195 rate of any complication was 25.0%, isolated vitreous hemorrhage was 13.1%, retinal breaks without de
196 the median time to clearance of the initial vitreous hemorrhage was 36 (interquartile range [IQR], 2
201 detachment during the follow-up period, and vitreous hemorrhage was observed in 96.5% of cases (n =
202 ery and vitrectomy resulting from persistent vitreous hemorrhage was performed in 3.5% (n = 3) and 5.
205 with proliferative diabetic retinopathy and vitreous hemorrhage, where the highest concentrations of
207 ow a close association of a retinal tear and vitreous hemorrhage with whole-body vibration training.