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1 stroboscopy, showed immobility of the right vocal cord.
2 armonics (or overtones) emanating from their vocal cords.
3 thirds of laryngeal cancers originate at the vocal cords.
4 related spectral peaks, e.g., as produced by vocal cords.
5 The lowest densities were seen in the vocal cords.
6 sociated with brief partial adduction of the vocal cords.
8 n apparent exacerbation of the airway edema, vocal cord adduction that hinders glottic evaluation, an
9 , with multiple affected individuals in whom vocal cord and pharyngeal weakness may accompany the dis
10 goscopy revealed paradoxical movement of the vocal cords, and a diagnosis of vocal cord dysfunction (
11 During the attack, bronchoscopy revealed the vocal cord closing with stridor during the inspiratory p
12 ng both necessary and sufficient for driving vocal cord closure and eliciting mouse ultrasonic vocali
14 the age of eleven during an attack revealed vocal cord closure on inhalation and the patient was dia
16 typically involves abnormal adduction of the vocal cords during inspiration, mimics the symptoms of a
22 hy with the added features of pharyngeal and vocal cord dysfunction (VCPDM) that has not been previou
23 Chylothorax occurred in 28 patients (5.4%), vocal cord dysfunction in 22 patients (4.3%), and diaphr
24 of deficient oropharyngeal motor skills and vocal cord dysfunction is crucial to establish enteral n
25 had previous thyroid or neck surgery, known vocal cord dysfunction on laryngoscopy, hearing or voice
26 cribes seven elite athletes with psychogenic vocal cord dysfunction who presented with acute dyspnea
27 esophageal reflux, obstructive sleep apnoea, vocal cord dysfunction, obesity, dysfunctional breathing
28 disorder, panic attacks, globus hystericus, vocal cord dysfunction, scombroid poisoning, vasoactive
32 eflux disease, laryngopharyngeal reflux, and vocal cord dysfunction/inducible laryngeal obstruction).
37 communication in daily life for people with vocal cord lesions and laryngeal and lingual injuries wi
38 At baseline, an inclusion visit assessed vocal cord mobility via nasofibroscopy and voice quality
44 liminate the risk of postoperative bilateral vocal cord palsy (VCP) by indicating staged surgery in c
48 ew found that tracheoesophageal fistulae and vocal cord paralyses were the 2 most common airway injur
51 complications such as hypoparathyroidism and vocal cord paralysis in a small proportion of patients.
54 ion, while 1 minor skin burn and 1 permanent vocal cord paralysis occurred in the RFA treatment group
55 roup, tracheal surgery, hypotonic airway, or vocal cord paralysis or if they died before extubation.
56 e mean (SD) duration of ingestion leading to vocal cord paralysis was shorter than that of the genera
57 ultiple organ system failure, age of <6 mos, vocal cord paralysis, and noncooperation with nasal mask
58 l dysmotility (mid esophageal hematoma), and vocal cord paralysis, resulting in hoarse voice (upper e
59 ntellectual disabilities and, in some cases, vocal cord paralysis, tracheomalacia and cyclic vomiting
64 er radiotherapy as a function of whether the vocal cord regained mobility or remained fixed during or
66 est that oral corticosteroids do not enhance vocal cord remobilization or improve voice quality in pa
68 ity of the speaker is carried largely in the vocal cord source and the message is shaped by the ever-
71 he removal of enteral content from below the vocal cords, usually during endotracheal tube placement.
72 oxicity include congenital onset of disease, vocal cord weakness and motor-predominant disease, where
74 ected involving the whole length of the left vocal cord, with abnormal mucosa also seen in the right
75 ance of liquid or puree bolus below the true vocal cords without coughing during a FEES examination.