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1 ith the expected field, temperature and gate voltage dependencies.
2 channels characterized by positively shifted voltage dependencies and very fast deactivation rates.
3 junctional hemichannels that display altered voltage dependency and reduced permeability, and which c
6 a mechanism underlying the diverse BacNa(V) voltage dependencies, and demonstrate that a discrete do
8 C1, which had a similar channel activity and voltage dependency as full-length, was hypoxia-inducible
10 reaks down without electrical coupling; NMDA voltage-dependency doubles the range of synaptic feedbac
12 me constant of inactivation (tauh) derives a voltage dependency from coupling to voltage-dependent ac
14 rents have similar amplitudes, kinetics, and voltage dependencies in male and female myocytes, simula
15 ocytes had smaller Na+ currents with altered voltage dependencies of activation and inactivation and
18 rent using protocols employing the different voltage dependencies of the channel types and their diff
20 alian cells, induced a positive shift in the voltage dependency of K(+) current activation, and slowe
23 nally, the beta3 subunit alters the rate and voltage dependency of relief of the inhibition produced
24 nnel activation and inactivation in that the voltage dependency of tauh is substantially reduced whil
25 ome independent, parallel processes, and any voltage dependency of tauh is then entirely intrinsic to
26 te constant of zero also removes the derived voltage dependency of tauh, but activation and inactivat
28 ICa as manifested by flattened and broadened voltage dependency of the amplitude of cytosolic Ca2+ tr
29 f I(Ca)- induced Ca transients (at 0 mV) but voltage dependency of the Ca transients was markedly wid
31 manner: (1) by providing the driving force (voltage dependency of the transport itself) and (2) by l
33 a depolarizing prepulse and also shifts the voltage dependency of this relief to more hyperpolarized
34 channel CCA-1 and symmetrically re-tune its voltage-dependencies of activation and inactivation towa
36 the second and third resting states, and the voltage-dependency of forward transitions through restin
37 of activation/deactivation and a bell-shaped voltage dependency, reminiscent of the rapid (R)-type an
39 gh Cav1 channels of lymphocytes retain their voltage dependency, T cell receptor stimulation dramatic
42 fold to fourfold by 50 microm NEM, and their voltage dependencies were negatively shifted by 10-20 mV
43 different time-dependent characteristics and voltage dependencies, which interact with each other and