ライフサイエンスコーパス: volume index

1 every 1 mL/m(2) increase in LV end systolic volume index.

2 -diastolic volume index, and LV end-systolic volume index.

3 riation monitoring, and global end-diastolic volume index.

4 y resistance index and an increase in stroke volume index.

5 istance, while increasing cardiac and stroke volume index.

6 of arterial afterload were related to stroke volume index.

7 ncrease in the left ventricular end-systolic volume index.

8 ing was defined as change in LV end-systolic volume index.

9 -diastolic volume index, and LV end-systolic volume index.

10 study provides reference values of normal LA volume index.

11 ic dysfunction (E/e') and 5 with left atrial volume index.

12 itus and smoking were not associated with LA volume index.

13 e, and larger left ventricular end-diastolic volume index.

14 P<0.05) ethnicities were associated with LA volume index.

15 x, and larger left ventricular end-diastolic volume index.

16 oint was change in left ventricular systolic volume index.

17 ments in both LV end-systolic and -diastolic volume indexes.

18 end-diastolic pressure and left ventricular volume indexes.

19 ; 95% CI, -0.008-0.014; p = .571) and stroke volume index (0.17 mL/m(2); 95% CI, -0.03-0.37; p = .094

20 iated with higher mean pulse pressure/stroke volume index (1.24 and 1.15 versus 1.02 mm Hg/mL x m2) a

21 54 +/- 9%, p < 0.0001) and LV end-diastolic volume index (108 +/- 28 ml/m(2) to 78 +/- 24 ml/m(2), p

22 ection fraction (11% versus 3%), left atrial volume index (-11.9 versus -4.7 mL/m(2)), and right vent

23 e (79 pg/mL), left ventricular end-diastolic volume index (110 mL/m2), and left ventricular ejection

24 jection fraction, 23+/-9%; mean end-systolic volume index, 113+/-48 mL; mean total myocardial scar %,

25 The decrease in end-diastolic volume index (14+/-3) was different from that seen at ba

26 mendan increased cardiac index (22%), stroke volume index (15%), and heart rate (7%) and decreased sy

27 6.7+/-6.9 mL/m(2); P<0.001) and right atrial volume index (15.66+/-3.09 versus 20.47+/-4.82 mL/m(2);

28 Left atrial volume index (24.0+/-3.6 versus 26.7+/-6.9 mL/m(2); P<0.

29 mprovement in left ventricular end-diastolic volume index (-26.2 versus -7.4 mL/m(2)), left ventricul

30 mm Hg vs. 10 +/- 5 mm Hg, p < 0.001) and LA volume index (28 +/- 12 ml/m(2) vs. 21 +/- 14 ml/m(2), p

31 -7.4 mL/m(2)), left ventricular end-systolic volume index (-28.7 versus -9.1 mL/m(2)), left ventricul

32 MR patients had larger left atria (volume index: 32 cm(3)/m(2) vs. 26 cm(3)/m(2), p = 0.008

33 itus (25% versus 41%, P=0.009), lower stroke volume index (36.4+/-8.4 versus 34.4+/-8.7 mL/m2, P=0.02

34 lar velocity greater than 10 and left atrial volume index 40 mL/m2 or more were associated with a 3.4

35 ; P<0.001) and had increased RV end-systolic volume index (43 versus 35 mL/m2; P=0.03), decreased RV

36 The median E/e' (16), left atrial volume index (44 mL/m2), and pulmonary artery systolic p

37 d dilated right-sided chambers (right atrial volume index, 44 +/- 19 mL/m(2); RV end-diastolic area,

38 th LF compared with those with normal stroke volume index (47% versus 34%; hazard ratio, 1.5; 95% con

39 minute; P<0.001) and to reduced peak stroke volume index (47+/-10 mL/min per m(2) versus 54+/-15 mL/

40 2), as did the left ventricular end-systolic volume index (48.4 +/- 19.7 ml/m(2) vs. 43.1 +/- 16.2 ml

41 -62 ms; P<0.0001), and increased left atrial volume index (49+/-18 versus 42+/-15 mL/m2; P=0.02).

42 ence interval, 0.2-0.5; P=0.0001) and stroke volume index (5.2 mL.m(-2); 95% confidence interval, 2.0

43 icant reduction of left ventricular systolic volume index (-5.8%, P=0.017), and noninfarct myocardial

44 9), HFrEF patients had larger LA volumes (LA volume index 50 versus 41 mL/m(2); P<0.001), whereas HFp

45 ose with persistent AF had larger maximum LA volume index (56+/-17 versus 49+/-13 mL/m(2); P=0.036),

46 nts with transplants had lower end-diastolic volume index (59.3+/-15.2 ml/m(2) vs. 71.4+/-15.9 ml/m(2

47 nd significant reductions in LV end systolic volume index (-6.7 +/- 21.1 versus 2.1 +/- 17.6 mL/m(2);

48 mposite score, left ventricular end systolic volume index, 6-minute walk time, and quality of life in

49 59% specificity), change in pulmonary blood volume index (77% sensitivity, 82% specificity), and cha

50 45%; P<0.001) vascular compliance and stroke volume index (8%; P=0.01).

51 +/- 33 g/m2; P < 0.001) and LV end-diastolic volume index (82 +/- 35 versus 34 +/- 16 ml/m2; P < 0.00

52 tivity, 98% specificity) and pulmonary blood volume index (92% sensitivity, 68% specificity), and by

53 ients with a right ventricular end-diastolic volume index above 150 ml/m(2) and peak exercise systoli

54 ients with a right ventricular end-diastolic volume index above 150 ml/m(2) and peak exercise systoli

55 h (P < 0.01): right ventricular end-systolic volume index adjusted for age and sex, and the relative

56 essure, and left ventricular and left atrial volume index (all P<0.001) were more benign in mid-late

57 aller baseline left ventricular end systolic volume index also were also associated with better survi

58 in the MMF-withdrawal group, the left atrial volume index (an indicator of chronic LV diastolic dysfu

59 alysis showed that left ventricular systolic volume index and %MDE(periphery) were the strongest pred

60 rom baseline right ventricular end-diastolic volume index and a 429 ml (P < 0.001) reduction in resid

61 ase (least square mean+/-SE) in end-systolic volume index and a 6+/-1% increase in left ventricular e

62 osis stages correlated with both left atrial volume index and AF duration.

63 and left ventricular function, end-systolic volume index and B-type natriuretic peptide were most st

64 t the patients with LGSAS had reduced stroke volume index and cardiac index (P=0.003 for both).

65 renal dysfunction, yet smaller end-diastolic volume index and cardiac output and increased EDP compar

66 We measured stroke volume index and collapsibility index of the inferior ve

67 LA volume index and E/e' predicted exercise PCWP>30 mm Hg w

68 Left ventricle end-diastolic volume index and end-systolic volume index were reduced

69 After adjustment for LV volume index and functional class, patients with LGE had

70 ression model, left ventricular end-systolic volume index and left atrial volume index were independe

71 Right ventricular end-systolic volume index and left ventricular strain-rate e'-wave pr

72 is associated with reduced LV end-diastolic volume index and LV end-diastolic mass index in a large

73 oups in baseline values for LV end-diastolic volume index and LV end-systolic volume index were negli

74 eater reduction of LV systolic and diastolic volume index and LV mass index as compared with placebo.

75 LV end-diastolic volume index, end-systolic volume index and LVEF between diabetic patients and heal

76 eft ventricular mass index, left ventricular volume index and maximal left atrial anterior-posterior

77 In the proximal stent segment, stent volume index and minimum stent area were larger after PO

78 combined with right ventricular end-systolic volume index and strain-rate e'-wave in the multivariate

79 nship between left ventricular end-diastolic volume index and stroke volume index was confirmed in gr

80 2+/-2% and 38+/-5% versus 21+/-2% for stroke volume index and stroke volume, respectively, both P<0.0

81 , and waveform analysis of changes in stroke volume index and systemic vascular resistance index were

82 . 37.7 +/- 15.4; p = .03), plus lower stroke volume index and worse cardiac function with higher left

83 duction in LV end-diastolic and end-systolic volume indexes and a 3-fold greater increase in LV eject

84 dial function, as suggested by higher stroke volume indexes and left ventricular stroke work indexes

85 pressure, 0.56 for the global end-diastolic volume index, and 0.64 for the left ventricular end-dias

86 ociated with lower ejection fraction, stroke volume index, and aortic valve mean gradient up to 3 yea

87 in cardiac index, change in pulmonary blood volume index, and change in PaO2/FIO2 ratio individually

88 in cardiac index, change in pulmonary blood volume index, and change in PaO2/FIO2 ratio were lower i

89 index, relative wall thickness, left atrial volume index, and deceleration time were still associate

90 ution such as cardiac index, pulmonary blood volume index, and extravascular lung water.

91 on, including severe tachycardia, low stroke volume index, and high inferior vena cava collapsibility

92 id therapy was directed by maximizing stroke volume index, and in the study group, stroke volume inde

93 age, elevated creatinine, larger left atrial volume index, and larger left ventricular end-diastolic

94 entral venous pressure, global end-diastolic volume index, and left ventricular end-diastolic area in

95 ts in LV ejection fraction, LV end-diastolic volume index, and LV end-systolic volume index.

96 lume, LV ejection fraction, LV end-diastolic volume index, and LV end-systolic volume index.

97 ntricular mechanical delay, the end-systolic volume index, and the area of the mitral regurgitant jet

98 ume indexes (LVESVI and LVEDVI), left atrial volume index, and ventricular-vascular coupling ratio.

99 s of E/e' ratio, and decrease in left atrial volume index as compared with placebo.

100 volume index, and in the study group, stroke volume index assessed prior to severe acute pancreatitis

101 h a regime using individual values of stroke volume index assessed prior to severe acute pancreatitis

102 natriuretic peptide, and larger left atrial volume index at HFpEF diagnosis compared with sinus rhyt

103 sus 7.0 +/- 3.7 mm, P=0.002), a larger lipid volume index [averaged lipid arcxlipid length] (1605.5 +

104 g (B=-0.1; P=0.0001), lower LV end-diastolic volume index (B=0.6; P=0.0001), and lower LV end-diastol

105 ty (beta=1.3 mL/m(2), P<0.01), end-diastolic volume index (beta=0.4 mL/m(2), P<0.0001), Chinese Ameri

106 ular reconstruction reduced the end-systolic volume index by 19%, as compared with a reduction of 6%

107 acebo, empagliflozin reduced LV end-systolic volume index by 6.0 (95% CI, -10.8 to -1.2) mL/m(2) (P=0

108 Empagliflozin reduced LV end-diastolic volume index by 8.2 (95% CI, -13.7 to -2.6) mL/m(2) (P=0

109 3DE LA volumes indexed by body surface area were similar in men

110 age-predicted right ventricular end-systolic volume index can identify a high percentage of patients

111 central venous pressure and change in stroke volume index/cardiac index and the percentage of fluid r

112 central venous pressure and change in stroke volume index/cardiac index was 0.18 (95% CI, 0.1-0.25),

113 This new total right/left-volume index correlated with almost all clinically used

114 denervation, left ventricular end-diastolic volume index, creatinine, and no angiotensin inhibition.

115 1 to 12.0+/-3.3 mm/mm, P=0.01), as did lumen volume index (DA+, 13.2+/-3.1 to 10.5+/-2.7 mm/mm, P<0.0

116 a cross-sectional analysis of Google search volume index data and US cancer incidences and mortaliti

117 The left ventricular end-diastolic volume index decreased (90.1 +/- 28.2 ml/m(2) at baselin

118 Left ventricular mass index and left atrial volume index decreased from 122.6 +/- 42.6 to 98.5 +/- 3

119 In both groups vessel volume index decreased significantly (DA+, 16.3+/-3.6 to

120 LV end-diastolic and end-systolic volume indexes decreased in patients with CRT turned on

121 50-mg group, end-systolic and end-diastolic volume indexes decreased relative to baseline but were n

122 triuretic peptide, ejection fraction, stroke volume index, E/E', and left ventricular mass index (haz

123 ratio and preceding changes in end-diastolic volume indexed (EDVi) to body surface area and the eject

124 iovascular responses (cardiac output, stroke volume index, ejection fraction, peak systolic pressure/

125 ificant differences in mean LV end-diastolic volume index, end-systolic volume index and LVEF between

126 , extravascular lung water index, and stroke volume index, especially when double-lumen 5F peripheral

127 EF below the median (27%), and end-systolic volume index (ESVI) above the median (79 ml/m(2)).

128 3D wall motion tracking for RV end-systolic volume index (ESVi), RV ejection fraction (EF), and RV g

129 outcomes could be modulated by end-systolic volume index (ESVi).

130 etween examinations 1 and 5) in end-systolic volume indexed (ESVi) to body surface area.

131 ed LV strain rates and LVES wall stress/LVES volume index following MV repair indicate contractile dy

132 difference in the change in LV end-diastolic volume index from days 0 to 90 with PG-116800 versus pla

133 in/m(2); P = .001), and global end-diastolic volume index (GEDVI) (726 vs 775 mL/m(2); P = .003) incr

134 measurements included cardiac index, stroke volume index, global and regional contractile function (

135 rest and during exercise in patients with LA volume index >/=35 mL/m(2) (P<0.01), despite similar car

136 into 2 groups according to left atrial (LA) volume index >/=35 mL/m(2).

137 , RAP >10 mm Hg, sPAP >40 mm Hg, left atrial volume index >33 ml/m(2), ratio of mitral inflow early d

138 vided into those with DD (MI+DD; left atrial volume index >34 mL/m(2) and diastolic E/e' ratio>8; n=3

139 lar in 125 patients with normal flow (stroke volume index >35 mL/m2; P=0.22).

140 e of preload dependence (increases in stroke volume index >= 15%).

141 ntricular ejection fraction >=50% and stroke volume index >=35 mL/m(2)) and LG.

142 re defined in CG-AS with normal flow (stroke-volume-index >35 ml/m(2)).

143 aortic valve area (<1 cm(2)), normal stroke volume index (>/=35 mL/m(2)), and either high mean gradi

144 respectively), LV end-systolic dimension and volume index, >= moderate tricuspid regurgitation (TR) (

145 on (increased right ventricular end-systolic volume index), high Acute Physiology and Chronic Health

146 ximetry index (COx and wCOx), and hemoglobin volume index (HVx and wHVx).

147 a 13% reduction in left ventricular systolic volume index in comparison with the lowest quartile.

148 related with fluid-induced changes in stroke volume index in preload-dependent cases (r = 0.618; p =

149 olume expansion, a relevant (>/= 10%) stroke volume index increase was recorded in 56% patients.

150 Resting left ventricular end-diastolic volume index increased (P=0.001) within the sildenafil g

151 Stroke volume index increased after first fluid bolus in 80% of

152 Left atrium volume index increased during CNI (46.73+/-16.3 5-54.20+

153 During the first year after Tx, plaque volume index increased significantly in DA+ group, but d

154 age-predicted right ventricular end-systolic volume index independently predicted outcome and, when u

155 LV end systolic volume index is a significant predictor of mortality/mor

156 sex (both P< or =0.01), were LV end-systolic volume index, LA volume, atrial fibrillation, and sympto

157 The left atrial end-diastolic volume index (LAEDVI), representing the minimum or resid

158 The left atrial end-systolic volume index (LAESVI) is a predictor of cardiovascular o

159 tients were divided according to left atrial volume index (LAVi) >/=32 ml/m(2).

160 al longitudinal strain (GLS) and left atrial volume index (LAVI) have been recently proposed as novel

161 e was to evaluate whether normal left atrial volume index (LAVI) is a predictor of a normal stress ec

162 this study was to assess whether left atrial volume index (LAVI) measured in routine clinical practic

163 -systolic volume index (LVESVI), left atrial volume index (LAVI), and ratio of early transmitral Dopp

164 al annular velocity (S, E'), and left atrial volume index (LAVI).

165 .04-1.43; P=0.015) and increased left atrial volume index (LAVi; adjusted hazard ratio/unit increase,

166 or Doppler jet/left atrial area, left atrial volume index, left ventricular end-diastolic volume inde

167 es on ECG, and left ventricular end-systolic volume index, LGE maintained a >4-fold hazards increase

168 body mass index, diabetes, LV end-diastolic volume index, LGE, EF) (hazard ratio = 2.051 per mm decr

169 mportant subsets of patients with low stroke volume index (low flow) and low-gradient with reduced (c

170 evaluable echocardiograms (92%), LF (stroke volume index </=35 mL/m(2)) was observed in 530 (55%); L

171 d gradient: low flow was defined as a stroke volume index </=35 mL/m(2), low gradient as a mean gradi

172 ears, 90% for left ventricular end-diastolic volume index <100 mL/m(2) versus 48% for >/=100 mL/m(2))

173 ined by Doppler echocardiography as a stroke volume index <30 mL/m(2) (n=131) or a stroke volume <55

174 ted HR 2.17 [1.51-3.13]; P<0.0001 for stroke volume index <30 mL/m(2) and adjusted HR 1.86 [1.29-2.68

175 35) and those without DD (MI-DD; left atrial volume index <34 mL/m(2) and E/e' ratio<8; n=11).

176 Flow status was defined according to stroke volume index <35 mL/m(2) (low flow, LF) or >=35 mL/m(2)

177 ntricular ejection fraction >=50% but stroke volume index <35 mL/m(2)) and LG; and normal-flow (left

178 including paradoxical low-flow (i.e., stroke volume index <35 ml/m(2)), low-gradient (LF-LG) and norm

179 r ejection fraction [LVEF] >/=50% but stroke volume index <35 ml/m(2)), low-gradient (mean gradient [

180 hort identified preoperative RV end-systolic volume index <90 mL/m(2) and QRS duration <140 ms to be

181 Patients were stratified by stroke volume index (<35 mL/m(2) [low flow, LF] versus >/=35 mL

182 g water (<10 mL/kg) and global end-diastolic volume index (<850 mL/m) in the transpulmonary thermodil

183 ients were hypovolemic (global end-diastolic volume index<680 mL/m) on enrollment.

184 agnetic resonance measures (LV end-diastolic volume index, LV ejection fraction), diuretic intensific

185 subsample of 200 patients with HF, LV stroke volume index, LV filling pressure estimation, tricuspid

186 ean change from baseline in LV end-diastolic volume index (LVEDVI) at 6 months.

187 d left ventricular (LV) EF, LV end-diastolic volume index (LVEDVI), LV end-systolic volume index (LVE

188 LV mass index, aortic valve area, and stroke volume index, LVEF was independently predictive of morta

189 point was the left ventricular end-systolic volume index (LVESVI) at 12 months, as assessed with the

190 The left ventricular end-systolic volume index (LVESVI) decreased from 80.4 +/- 51.4 ml/m(

191 crease in left ventricular (LV) end-systolic volume index (LVESVI) of more than 10% from 1 week to 6

192 ference in the left ventricular end-systolic volume index (LVESVI) or survival after 1 year.

193 atients with a left ventricular end-systolic volume index (LVESVI) reduction of at least 15% were con

194 ta analysis of left ventricular end-systolic volume index (LVESVi) was performed to adjust for the co

195 l changes in MR severity and LV end-systolic volume index (LVESVi) were evaluated by linear mixed-mod

196 point was the left ventricular end-systolic volume index (LVESVI), a measure of left ventricular rem

197 tolic volume index (LVEDVI), LV end-systolic volume index (LVESVI), left atrial volume index (LAVI),

198 ference in the left ventricular end-systolic volume index (LVESVI), survival, or adverse events at 1

199 t ventricular end-systolic and end-diastolic volume indexes (LVESVI and LVEDVI), left atrial volume i

200 Right ventricular end-diastolic volume index measured by means of cardiovascular magneti

201 Left atrial volume index might be a simple means of identifying pati

202 ting for clinical risk factors, end-systolic volume index, mitral regurgitation, incomplete revascula

203 carotid intima-media thickness, left atrial volume index, monocyte count and serum YKL-40 levels.

204 he CABG group: left ventricular end-systolic volume index, MR volume, and plasma B-type natriuretic p

205 an increase in left ventricular end-systolic volume index of >15% at 24 months.

206 of 227% or a left ventricular end-diastolic volume index of 58 ml/m(2) identified patients at low (<

207 stemic vascular resistance index, and stroke volume index on days 3-7.

208 nts with increased preoperative end-systolic volume index or B-type natriuretic peptide.

209 ostic value beyond left ventricular systolic volume index or ejection fraction.

210 te with changes in end-diastolic ventricular volume indexes or cardiac performance.

211 1, P=0.31) or end-diastolic (r=0.10, P=0.38) volume indexes or LV ejection fraction (r=0.07, P=0.72).

212 p = 0.035), and smaller baseline left atrial volume index (OR: 1.47; p < 0.001).

213 x (p < 0.001; 95% CI, 0.97-0.99), and stroke volume index (p < 0.01; 95% CI, 0.96-0.99) in predicting

214 ce index (P < .01), right ventricular stroke volume index (P </= .01), and pulmonary artery capacitan

215 th changes in left ventricular end-diastolic volume index (p = 0.26), LVESVI (p = 0.41), or left vent

216 0.001 and P=0.0007, respectively), RV stroke volume index (P<0.0001), and left ventricular end-diasto

217 n reduction in left ventricular end-systolic volume index (P<0.0001), whereas non-LBBB patients had s

218 reduction in left ventricular end-diastolic volume index (P<0.001), with a concomitant small stable

219 rease in the right ventricular end-diastolic volume index (P<0.001).

220 ween noninvasive and invasive AVA was stroke volume index (P<0.01).

221 eased heart rate (P(group)=0.01), and stroke volume index (P(group)=0.004) compared with TTM36.

222 ejection fraction (P=0.001), LV end-systolic volume index (P=0.0006), or segmental WMA (P=0.002).

223 oke volume index (P=0.015) and end-diastolic volume index (P=0.001) decreased during exercise.

224 <0.0001), and left ventricular end-diastolic volume index (P=0.0015).

225 ncreased cardiac index (P<0.0001) and stroke volume index (P=0.003), especially at high-intensity exe

226 Stroke volume index (P=0.015) and end-diastolic volume index (P

227 lar velocity (P=0.02), increased left atrial volume index (P=0.05), and lower mean arterial pressure

228 7 mL/m(2) at 3-year follow-up (end-diastolic volume index, P=0.0056; end-systolic volume index, P=0.4

229 astolic volume index, P=0.0056; end-systolic volume index, P=0.4719).

230 the cardiac traits such as left ventricular volume index, parasternal long axis interventricular sep

231 volume index, left ventricular end-diastolic volume index, peak E wave, and the presence of pulmonary

232 tion fraction, LV end-systolic dimension and volume index, presence of FMR was independently associat

233 The coronary plaque volume index (PVI) was determined by dividing the wall v

234 = 0.17; p = .001), baseline pulmonary blood volume index (r = 0.15; p = .001), change in pulmonary b

235 = 0.15; p = .001), change in pulmonary blood volume index (r = 0.16; p < .001), and change in PaO2/FI

236 c volume index (r=0.62, P<0.01), left atrial volume index (r=0.41, P<0.05) but lower left ventricular

237 with increased left ventricular end-systolic volume index (r=0.62, P<0.01), left atrial volume index

238 ocardial 3-nitrotyrosine content with stroke volume indexes (r = -.701; p = .004).

239 ndex: r = 0.35, p < 0.0001; LV end-diastolic volume index: r = 0.43, p < 0.0001) and LVEF (r = -0.66,

240 volume measurements in CMR: total right/left-volume index=(RA+aRV+fRV)/(LA+LV).

241 R interval, p < 0.0001), as LVES stress/LVES volume index ratio was depressed at baseline and followi

242 End-systolic volume index remained unchanged (P=0.8).

243 Automated vessel wall volume index remained unchanged from baseline to follow-

244 he MRI-derived left ventricular end-systolic volume index, RV, and OMR category (severe versus modera

245 Our data suggest that the total right/left-volume index should be used as a new and simplified CMR

246 A measure of flow (stroke volume index) should be included in the evaluation and t

247 CMR-derived end-diastolic volume index showed a weaker association with outcome (p

248 nary artery diastolic pressure (PAD), stroke volume index (SV index), systolic blood pressure (sBP) a

249 tional class, 6-minute walk distance, stroke volume index (SVI), and right atrial pressure were indep

250 -one subjects (25 with low flow [LF], stroke volume index [SVI] </=35 ml/m(2), 16 with normal flow [N

251 Low flow (i.e., reduced stroke volume index [SVi]) can occur with both reduced and pres

252 We compared LF (stroke volume index, SVI < 35 ml/m(2)) and NF severe AS.

253 extent of LGE and a higher LV end-diastolic volume index than other groups, but levels of inflammato

254 line cardiac index, baseline pulmonary blood volume index, the change in cardiac index, change in pul

255 For stroke volume index, the median AUC for esmolol was 4 mL/m2 (IQ

256 age-predicted right ventricular end-systolic volume index threshold of 227% or a left ventricular end

257 Left atrial volume indexed to body surface area (LA index) was 55 +/

258 n between diastolic function and left atrial volume indexed to body surface area (LAVi) in a populati

259 PLF as defined by LVEF of >/=50% but stroke volume indexed to body surface area (SVi) of </=35 ml m(

260 from baseline to 36 weeks in LV end-systolic volume indexed to body surface area and LV global longit

261 METHODS AND LA volume indexed to body surface area was measured by card

262 Left ventricular EDV and end-systolic volume indexed to body surface area were smaller in wome

263 raction (LVEF) and left atrial end-diastolic volume indexed to body surface area, were assessed using

264 r left ventricular (LV) mass and left atrial volume indexed to height(2.7) in both men and women (P<0

265 th changes in LV end-diastolic and -systolic volumes indexed to body surface area, ejection fraction,

266 reduced aortic valve area and normal stroke volume index undergoing AVR underwent echocardiography,

267 baseline in right ventricular end-diastolic volume index versus placebo.

268 lumetric data were standardized by length as volume index (VI).

269 In 25 patients (64%) LA volume index was >/=35 mL/m(2).

270 Mean total right/left-volume index was 2.6+/-1.7 (normal values: 1.1+/-0.1).

271 The mean LA volume index was 36.5+/-11.4 mL/m(2) in the entire cohor

272 whereas left ventricular end-systolic (LVES) volume index was 60% above normal pre- and post-MV repai

273 nflow velocities, RAP, sPAP, and left atrial volume index was 90% accurate in distinguishing normal f

274 LV end systolic volume index was also evaluated as a predictor of mortal

275 Left atrial volume index was categorized as < or =28 ml/m2 (normal),

276 icular end-diastolic volume index and stroke volume index was confirmed in group 2 subjects using mat

277 A total right/left-volume index was defined from end-diastolic volume measu

278 End-diastolic volume index was determined by echo Doppler.

279 Left atrial volume index was measured retrospectively with the bipla

280 p < 0.05), but left ventricular end-systolic volume index was not different (p = 0.49).

281 Stroke volume index was obtained by transpulmonary thermodiluti

282 LV end systolic volume index was predictive of mortality/morbidity; the

283 The difference in left atrium (Delta LA) volume index was significant (P=0.002) and was not assoc

284 , extravascular lung water index, and stroke volume index were also overestimated (853 +/- 240 vs 688

285 argest increases in stroke volume and stroke volume index were during zero-resistance cycling.

286 Baseline cardiac index and pulmonary blood volume index were higher, whereas change in cardiac inde

287 ar end-systolic volume index and left atrial volume index were independent predictors of extracellula

288 jection fraction and left ventricular stroke volume index were most strongly predictive of survival o

289 d-diastolic volume index and LV end-systolic volume index were negligible (g<0.10).

290 Optimal thresholds of stroke volume index were obtained for men (40 ml/m(2)) and wome

291 end-diastolic volume index and end-systolic volume index were reduced from 128.4+/-22.1 and 94.9+/-2

292 Changes in the left atrial volume index were significantly associated with treatmen

293 ic blood pressure, cardiac index, and stroke volume index were significantly greater during ITD-assis

294 Plaque, lumen, and vessel volume indexes were calculated by volume/measured length

295 During Ex plus expiratory load, stroke volume indexes were reduced to baseline, non-exercise le

296 function (based on e', E/e', and left atrial volume index) were each independently and additively ass

297 ic blood pressure, cardiac index, and stroke volume index, when the ITD alone and ITD plus positive e

298 ts of such consequent maximization of stroke volume index with a regime using individual values of st

299 r of change in left ventricular end-systolic volume index with monotonic increases as QRS duration pr

300 Doxycycline improved cardiac and stroke volume index with no chronotropic effect in doxycycline-