ライフサイエンスコーパス: voluntary movement

1 sts modestly improved the range and speed of voluntary movement.

2 motor commands from the motor cortex during voluntary movement.

3 ty of the nervous system to plan and control voluntary movement.

4 igra, where it regulates DA biosynthesis and voluntary movement.

5 not gate the central mechanism that produces voluntary movement.

6 and another that was linked to onset of the voluntary movement.

7 cantly influencing locomotion and control of voluntary movement.

8 neural bases of agency and ownership during voluntary movement.

9 have linked the hippocampal theta rhythm to voluntary movement.

10 STN beta band power just prior to and during voluntary movement.

11 ng bearing weight on the paretic limb during voluntary movement.

12 e vestibular neural drive during unperturbed voluntary movement.

13 n the cortico-cerebellar motor system during voluntary movement.

14 muscles, which should be inhibited to enable voluntary movement.

15 the encoding of stable muscle synergies for voluntary movement.

16 resentation of hand position is critical for voluntary movement.

17 toneurone (alpha-MN) drive to muscles during voluntary movement.

18 a brain structure involved in the control of voluntary movement.

19 critical for correcting perturbations during voluntary movement.

20 ate a flexible fusimotor ecosystem to enable voluntary movement.

21 actuality most stimulation is the result of voluntary movement.

22 al field potential recordings in mice during voluntary movement.

23 e, excluding its contribution to unperturbed voluntary movement.

24 omuscular junction (NMJ) is critical for all voluntary movement.

25 striatal pathway to the striatum to regulate voluntary movement.

26 ole of KMT2B in the physiological control of voluntary movement.

27 sia and studied them both at rest and during voluntary movement.

28 lated oscillations are minimally affected by voluntary movement.

29 ts in the initiation, speed, and fluidity of voluntary movement.

30 that include cognition, reward learning, and voluntary movement.

31 is associated with and explains a change in voluntary movement.

32 the normal sense of agency that accompanies voluntary movement.

33 ng energy expenditure, in part by increasing voluntary movement.

34 al communication, seizures, and disorders of voluntary movement.

35 spinal cord circuits that are essential for voluntary movement.

36 ctions but is attenuated prior to and during voluntary movement.

37 as of the human medial frontal lobe prior to voluntary movement.

38 models has emerged as a leading paradigm for voluntary movement.

39 in healthy subjects and show that this slows voluntary movement.

40 he involuntary movement and during a matched voluntary movement.

41 ctions and is suppressed prior to and during voluntary movement.

42 tor (M1) cortex that differentiate tics from voluntary movements.

43 tion of postural adjustments associated with voluntary movements.

44 estigate their role in the online control of voluntary movements.

45 insights into the adaptive control of other voluntary movements.

46 n of primary motor cortex (MI) is to control voluntary movements.

47 movements triggered or exacerbated by sudden voluntary movements.

48 spatial representations in conjunction with voluntary movements.

49 ecific role in the planning and execution of voluntary movements.

50 he moment-to-moment vigor and variability of voluntary movements.

51 ials (MEPs) after agonistic and antagonistic voluntary movements.

52 ontal motor areas are central to controlling voluntary movements.

53 ull-spectrum synthesis of EMG signals during voluntary movements.

54 ractions of antagonist muscle groups, during voluntary movements.

55 knowledged for its crucial role in executing voluntary movements.

56 e in the planning, control, and execution of voluntary movements.

57 pecific, goal-directed way characteristic of voluntary movements.

58 mportantly to the muscle activity underlying voluntary movements.

59 es the direction, velocity, and amplitude of voluntary movements.

60 ciated with the preparation and execution of voluntary movements.

61 including locomotion, postural control, and voluntary movements.

62 involuntary tics but was not present during voluntary movements.

63 n Purkinje cells and regions that coordinate voluntary movements.

64 rease in M1 that was present during tics and voluntary movements.

65 ction and with reductions in the velocity of voluntary movements.

66 viors ranging from basic reflexes to complex voluntary movements.

67 oadband power in primary motor cortex during voluntary movements (200-300Hz: Pearson's r = 0.51, p =

68 gamma power in primary sensory cortex during voluntary movements (70-200Hz: r = 0.44, p = 0.035, 95%

69 ng performance of stereotypic locomotion and voluntary movement: adaptive locomotion over obstacles a

70 nsory incomplete spinal cord injury regained voluntary movement after 7 months of epidural stimulatio

71 Voluntary movement also caused increased gamma band acti

72 the basic assumption that spasticity impairs voluntary movement and a review of the methodology of th

73 eus (STN) neurons are observed during normal voluntary movement and abnormal movement in Parkinson's

74 rone syndrome, reduce spasticity and improve voluntary movement and active function.

75 f a wide range of brain functions, including voluntary movement and behavioral processes.

76 regulate diverse brain functions, including voluntary movement and cognitive and emotive behaviors.

77 njugated fatty acids on non-exercise form of voluntary movement and lipid and glucose metabolisms for

78 The effects of involuntary, passive and voluntary movement and muscle-tendon vibration on freque

79 neurons could contribute to the decrease in voluntary movement and other dopamine-mediated behaviors

80 elucidated their contribution to generating voluntary movement and revealed their involvement in mov

81 esodiencephalic dopaminergic neurons control voluntary movement and reward based behaviours.

82 Voluntary movement and the heart beat require this calci

83 the sensations generated as a consequence of voluntary movement and those arising from events in the

84 by transcranial magnetic stimulation, rapid voluntary movements and a fatiguing exercise test of a s

85 ng motor restlessness and chorea, slowing of voluntary movements and cognitive impairment.

86 lays a fundamental role in the generation of voluntary movements and motor learning in adults.

87 ansmission in climbing fibre pathways during voluntary movements and motor learning.

88 the effects of ipsilateral misprojections on voluntary movements and stereotypic locomotion.

89 S patterns, evoked by the interplay of their voluntary movements and the virtual textures of each obj

90 e learning, through repeated cooccurrence of voluntary movements and their outcomes.

91 y tasks, such as reward-related behavior and voluntary movement, and excessive loss of these neurons

92 ons play a central role in the regulation of voluntary movement, and their degeneration is associated

93 of substantia nigra in the midbrain control voluntary movement, and their degeneration is the cause

94 onsible for the initiation and modulation of voluntary movement, and their degeneration is the hallma

95 the CM thalamus that differentiated tic from voluntary movement, and this physiological feature could

96 TEMENT Variability is an inherent feature of voluntary movement, and traditionally more variability i

97 o which unmodulated stretch reflexes disrupt voluntary movement, and whether and how they are inhibit

98 Voluntary movements are frequently composed of several a

99 or functional restoration after stroke, when voluntary movements are no longer possible.

100 or functional restoration after stroke, when voluntary movements are no longer possible.SIGNIFICANCE

101 ow that the motor commands needed to produce voluntary movements are preferentially released from the

102 Voluntary movements are widely considered to be planned

103 Ns) play a central role in the regulation of voluntary movement as well as other complex behaviors, a

104 scious awareness, so that subjects perceived voluntary movements as occurring later and their sensory

105 nergic neurons are essential for appropriate voluntary movement, as epitomized by the cardinal motor

106 ated during the preparation and execution of voluntary movements at both cortical and subcortical lev

107 cal modulator of brain circuits that control voluntary movements, but our understanding of its influe

108 cord can be modulated presynaptically during voluntary movement by mechanisms that depolarize afferen

109 emisphere) in the process of generation of a voluntary movement by the paretic hand in patients with

110 emisphere) in the process of generation of a voluntary movement by the paretic hand.

111 hemisphere) in the process of generation of voluntary movements by the paretic hand, a disorder corr

112 Mammalian motor circuits control voluntary movements by transmitting signals from the cen

113 Therefore, each voluntary movement carries a cost because its duration d

114 ALS patients exhibit alterations of voluntary movements caused by degeneration of motor neur

115 The control of voluntary movement changes markedly with age.

116 lei that play essential roles in controlling voluntary movements, cognition and emotion.

117 he KI/KO mice have a significant decrease in voluntary movement compared with wild-type and KI/KI mic

118 Voluntary movement control depends on plasticity in seve

119 bstantia nigra (SN) neurons is essential for voluntary movement control.

120 del parameter averaging we found that during voluntary movements, DBS reversed the effective connecti

121 Planning and execution of voluntary movement depend on the contribution of distinc

122 Voluntary movement difficulties in Parkinson's disease a

123 explored the pathophysiological basis of the voluntary movement disorder, in particular the role of t

124 ) or in arm muscles either at rest or during voluntary movement during GVS in neurotypical adults.

125 to that observed during the performance of a voluntary movement during wakefulness.

126 Myotonia can be triggered by voluntary movement (electrically induced myotonia) or pe

127 pper limb impairments who retain antigravity voluntary movement, enabling them to monitor rehabilitat

128 Distinct striatal subfields are involved in voluntary movement generation and cognitive and emotiona

129 Whereas voluntary movements have long been understood to derive

130 ystonia, generalized athetosis, and impaired voluntary movement in all patients.

131 egy that can dramatically affect recovery of voluntary movement in individuals with complete paralysi

132 nding pathway contributing to the control of voluntary movement in mammals.

133 e neurotransmitter dopamine, which regulates voluntary movement in many organisms, can stimulate move

134 ed spatiotemporal gait parameters, decreased voluntary movement in open field testing, and higher mod

135 basal ganglia are associated with slowing of voluntary movement in patients with Parkinson's disease.

136 A basic EEG feature upon voluntary movements in healthy human subjects is a beta

137 olved in the preparation of externally paced voluntary movements in humans and, secondly, the degree

138 ts that disturbed proprioceptive guidance of voluntary movements in Parkinson's disease is related to

139 ed stage at the onset of the often excessive voluntary movements in postnatal mice.

140 the extent of initial recovery of lower limb voluntary movements in those with clinically motor compl

141 , posture, gait and also coordinates skilled voluntary movements including eye movements.

142 elop normally for 6-18 months, but then lose voluntary movements, including speech and hand skills.

143 rtical regions considered to be essential to voluntary movement initiation and behavioral control.

144 Voluntary movement initiation involves the modulations o

145 n of a cerebellar-thalamocortical pathway to voluntary movement initiation remains poorly understood.

146 Because brain function is central to voluntary movement, interventions that aim to improve mo

147 pinal (CS) circuits, which are essential for voluntary movements, involves both guidance molecule- an

148 s likely reflect requirements for control of voluntary movement involving different body parts.

149 d, to help guide and improve the accuracy of voluntary movements involving motion of the head in spac

150 Voluntary movement is a result of signals transmitted th

151 Voluntary movement is accompanied by changes in the degr

152 phases of ongoing brain rhythms, but whether voluntary movement is constrained by brain rhythm phase

153 Human voluntary movement is controlled by the pyramidal motor

154 trate that a fundamental component of normal voluntary movement is impaired in patients with function

155 synaptic activation from afferent input and voluntary movement is important for production of plasti

156 ement, their involvement in externally paced voluntary movement is less clear.

157 thin the distributed brain network mediating voluntary movement is needed to facilitate recovery, min

158 he motor cortex and spinal cord, its role in voluntary movement is poorly understood.

159 Control of voluntary movement is predicated on integration between

160 Executive control of voluntary movements is a hallmark of the mammalian brain

161 Behavioral adaptation, a central feature of voluntary movement, is known to rely on top-down cogniti

162 l several essential functions, including the voluntary movement, learning, and motivated behavior.

163 For voluntary movements like reaching, the motor cortex is a

164 rtex to generate appropriate commands when a voluntary movement must be integrated with an ongoing, s

165 raction of agonist/antagonist muscles during voluntary movement observed in subjects with spastic cer

166 ver, we now report in three more individuals voluntary movement occurred with epidural stimulation im

167 CBF were seen in the contralateral M1 during voluntary movement of either hand; a small ipsilateral M

168 We compared IMC at Rest and unperturbed voluntary movement of the arm in neurotypical adults.

169 r information plays a role in the control of voluntary movement of the upper body.

170 s pallidus interna (GPi) or scalp EEG during voluntary movements of a hand-held joystick in six awake

171 ge lesions of the primary motor cortex (M1), voluntary movements of affected body parts are weak and

172 brain activity to vibratory stimulation and voluntary movements of body parts above and below the le

173 The model represents goals for voluntary movements of the swing leg on the task level o

174 As in voluntary movements, only a single rhythm may be easily

175 ost scES implant and prior to any locomotor, voluntary movement or cardiovascular training.

176 functional brain activity associated with 12 voluntary movement patterns in females versus males.

177 Tics are sometimes described as voluntary movements performed in an automatic or habitua

178 ce, but not of ongoing activation related to voluntary movement planning.

179 0) achieved, to some extent, lower extremity voluntary movements post scES implant and prior to any l

180 Voluntary movement preparation and execution have been l

181 We conclude that voluntary movement preparation/generation and volitional

182 The application of TMS(CT) prior to control voluntary movements produced a significant delay in move

183 cause this tissue plays an essential role in voluntary movement, protecting its integrity is crucial

184 , involuntary movements and abnormalities of voluntary movement, psychiatric symptoms, weight loss, d

185 th healthy subjects even when suppression of voluntary movement rather than tics was required during

186 During normal voluntary movements, re-afferent sensory input continuou

187 In contrast, the more pronounced voluntary movement reductions in non-commercial areas, d

188 Production of voluntary movements relies critically on the functional

189 Voluntary movement requires communication from cortex to

190 Voluntary movement requires motor commands to be release

191 neurons play central physiological roles in voluntary movement, reward learning, and motivated behav

192 behaviors important for survival, including voluntary movement, reward processing, and detection of

193 To test our idea, we considered elementary voluntary movements: saccades of the eye.

194 During voluntary movement, sensory activity may be globally sup

195 The voluntary movement showed activation of the putamen wher

196 of cortically evoked physiological biases to voluntary movements.SIGNIFICANCE STATEMENT Human motor p

197 sh in oil-exposed groups also showed reduced voluntary movement speed.

198 movements are worsened by anxiety but not by voluntary movement, startle, caffeine, or alcohol.

199 the corticospinal tract (CST) in controlling voluntary movements, successful regeneration of large nu

200 al for maintaining postural stability during voluntary movements such as gait initiation.

201 The control of voluntary movements such as skillful reaching and graspi

202 ies in monkeys have shown that parameters of voluntary movement (such as direction) may be specified

203 When the stimulus began 2 s before the voluntary movement the response consisted of two distinc

204 In a voluntary movement, the nervous system specifies not onl

205 cal-basal ganglia network after cessation of voluntary movement: the post-movement beta synchronizati

206 fficiently for the planning and execution of voluntary movements, thereby resulting in direction-depe

207 the conditional knock-outs showed bilateral voluntary movements under conditions when single limb mo

208 The motor commands required to control voluntary movements under various environmental conditio

209 Given the significance of the cerebellum in voluntary movement, we then built a more complete model

210 ditioning protocol in which stimuli gated by voluntary movements were used to produce coactivation of

211 elationship between these two aspects during voluntary movement when such experiences naturally combi

212 lay between agency and body ownership during voluntary movement, which has implications for the devel

213 ons (CSN) are centrally required for skilled voluntary movement, which necessitates that they establi

214 ortex plays a central role in the control of voluntary movements, which are typically guided by senso