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1 18.1% for bacterial vaginosis, and 8.6% for vulvovaginal candidiasis.
2 o colonization and immunopathogenesis during vulvovaginal candidiasis.
3 protection in oral infection but exacerbate vulvovaginal candidiasis.
4 ed to fluid from healthy women or women with vulvovaginal candidiasis.
5 March 2001 from 429 patients with suspected vulvovaginal candidiasis.
6 reduce the rate of recurrence of symptomatic vulvovaginal candidiasis.
7 zole was effective in preventing symptomatic vulvovaginal candidiasis.
8 be effective for the management of recurrent vulvovaginal candidiasis.
9 acy to fluconazole in the treatment of acute vulvovaginal candidiasis.
10 lbicans, in mice models of oropharyngeal and vulvovaginal candidiasis.
11 conazole, are now available for treatment of vulvovaginal candidiasis.
12 trial, NDV-3A protected women from recurrent vulvovaginal candidiasis.
13 of oral ibrexafungerp in patients with acute vulvovaginal candidiasis.
15 anslocation, nor do we implicate the gene in vulvovaginal candidiasis among mice in pseudoestrus.
16 oth clinical studies of women with recurrent vulvovaginal candidiasis and a murine model of experimen
17 24 premenopausal women with acute recurrent vulvovaginal candidiasis and from 21 healthy asymptomati
19 infection, Trichomonas vaginalis infection, vulvovaginal candidiasis, and bacterial vaginosis) in HI
24 ), gonorrhea (HR, 1.6; 95% CI, 1.1-2.2), and vulvovaginal candidiasis (HR, 1.5; 95% CI, 1.3-1.8).
28 chlamydia, bacterial vaginosis, trichomonas, vulvovaginal candidiasis, pelvic inflammatory disease, g
34 is sensitivity 61.6%, specificity 46.0%; and vulvovaginal candidiasis sensitivity 74.6%, specificity
35 and moderate sensitivity and specificity for vulvovaginal candidiasis (sensitivity 64.4%, specificity
36 e antifungal susceptibility of yeast causing vulvovaginal candidiasis, since cultures are rarely perf
37 women with non-antibiotic-induced recurrent vulvovaginal candidiasis suffering from acute Candida va
38 ypeptides in both bacterial vaginosis and in vulvovaginal candidiasis, suggesting that the abnormalit
40 e randomly assigned 387 women with recurrent vulvovaginal candidiasis to receive treatment with fluco
48 sseminated candidiasis (HDC) and episodes of vulvovaginal candidiasis (VVC) in humans, we found evide
56 Candida test using a reference standard for vulvovaginal candidiasis (VVC) of yeast culture plus exc
59 bacterial vaginosis (BV) and/or symptomatic vulvovaginal candidiasis (VVC), 195 (18.5%) had one or m
60 s the causative agent of acute and recurrent vulvovaginal candidiasis (VVC), a common mucosal infecti
61 rial vaginosis (BV), 25 acquired symptomatic vulvovaginal candidiasis (VVC), and 7 acquired vaginal t
62 re assessed for bacterial vaginosis (BV) and vulvovaginal candidiasis (VVC), and tested for M. homini
63 s vaginitis due to bacterial vaginosis (BV), vulvovaginal candidiasis (VVC), and Trichomonas vaginali
64 mmon, diagnosis of bacterial vaginosis (BV), vulvovaginal candidiasis (VVC), and Trichomonas vaginali
65 ion with lactobacilli may reduce the risk of vulvovaginal candidiasis (VVC), but supporting data are
69 opharyngeal candidiasis (OPC), as opposed to vulvovaginal candidiasis (VVC), is a common opportunisti
71 ections, including bacterial vaginosis (BV), vulvovaginal candidiasis (VVC), or Trichomonas vaginalis
72 ctions, notably bacterial vaginosis (BV) and vulvovaginal candidiasis (VVC), particularly in the sett
73 t common infection caused by Candida spp. is vulvovaginal candidiasis (VVC), which affects >70% of wo
76 b-), the relative risk of chronic recurring vulvovaginal candidiasis was 2.41-4.39, depending on the
78 hoeae, T vaginalis, bacterial vaginosis, and vulvovaginal candidiasis were the gold standard, and all