ライフサイエンスコーパス: walker

1 y (Crump) or reduce confounding (Sturmer and Walker).

2 d displacement (OSD) reaction to release the walker.

3 modified procedure described by Robinson and Walker.

4 ith substrate (or "track") for the molecular walker.

5 neously be induced for forwards and reversed walkers.

6 facilitate the development of functional DNA walkers.

7 inetics and processivity of DNA enzyme-based walkers.

8 design of small-molecule synthetic molecular walkers.

9 hose that come closest to being optimal Levy walkers.

10 s are known to enhance exploration of random walkers.

11 gnitude more efficiency than Brownian random walkers.

12 ar events in a cohort of older long-distance walkers.

13 tions and clinical outcomes in long-distance walkers.

14 ant implications for the design of molecular walkers.

15 to real-life race performance in elite race walkers.

16 st walkers (<0.5 m/s) constituting 28%, slow walkers (0.5-0.83 m/s) making up 48%, and normal walkers

17 ling was attenuated in slow compared to fast walkers (2.8%; 95% confidence interval [CI], -0.9 to 6.6

18 istar rats were inoculated with 2 x 10(7) of Walker 256 tumor cells [tumor bearing (TB) rats].

19 ines that lie close to the LSGGQ (P517C) and Walker A (I1050C) sites of NBD1 and NBD2, respectively,

20 we have mutated conserved residues in Fml1's Walker A (K99R) and Walker B (D196N) motifs to determine

21 r mutagenesis analyses indicated that Rrp2's Walker A and B motifs are required for borrelial growth;

22 and hydrolysis activities engendered by the Walker A and B motifs respectively.

23 These motors have conserved Walker A and B motifs that bind Mg2+-ATP and a catalytic

24 Here, we use Walker A and B mutants to demonstrate that D1 is capable

25 and NMS-873) have differential responses to Walker A and B mutations, to disease-causing IBMPFD muta

26 Alanine substitution mutants in the deviant Walker A and Signature motifs retain significant, albeit

27 The combined data suggest that the Walker A and Walker B motifs of Rrp2 are involved in the

28 ndent on ATP and have identified ATP-binding Walker A and Walker B motifs that are present in Escheri

29 including the G239 residue and the putative Walker A and Walker B motifs.

30 ins contain the nucleotide-binding sequences Walker A and Walker B, respectively.

31 ported by experiments, wherein the conserved Walker A arginine 'toggles' between interacting with a g

32 D-loop aspartate functions to stabilize the Walker A asparagine in a position favorable for catalysi

33 tation of either the D-loop aspartate or the Walker A asparagine results in dramatic reductions in AT

34 This interaction depends on the Walker A ATP binding motif of SpoIVA and the LysM domain

35 ation of the conserved Lys-669 to Met in the Walker A box of the first nucleotide-binding domain (Ycf

36 e cis-variant FlrC(C)-R349A drastic shift of Walker A encroached ATP-binding site, whereas the site r

37 Further, a divergent Walker A glutamic acid residue acts synergistically with

38 partate and an asparagine residue located in Walker A loop of the opposing subunit.

39 ries of point mutants, each lacking a single Walker A Lys residue, was generated to study the effects

40 glycine (G6) and lysine (K7) residues of the Walker A motif (-GPAGTG(6)K(7)S-) were found to be criti

41 including a conserved Lys located within the Walker A motif (or P-loop).

42 e conserved lysine to alanine (K629A) in the Walker A motif abolished ATP hydrolysis and substrate tr

43 ing affinity (K(d) = 1.81 x 10(-7) M) of the Walker A motif beyond the motif's nucleotide-binding cha

44 It is an ATPase in the MinD/ParA/Mrp deviant Walker A motif family which is within the P loop GTPase

45 Interestingly, the Walker A motif is located in the C-terminal region of RN

46 Confirming this, a FleQ Walker A motif mutant failed to bind c-di-GMP.

47 The Walker A motif of FleQ is perfectly conserved, opening u

48 -di-GMP competes with ATP for binding to the Walker A motif of FleQ.

49 ParA that are altered in a key lysine in the Walker A motif of the ATP binding site.

50 Amino acid substitutions in the Walker A motif of TssM caused reduced ATP binding and hy

51 l microbalance, both the consensus motif and Walker A motif showed strong affinities to PI5P-containi

52 position is conserved in four of the deviant Walker A motif subfamilies (MinD, chromosomal ParAs, Get

53 maS adopts an extended conformation, and the Walker A motif undergoes substantial conformational chan

54 Site-directed mutagenesis showed that a Walker A motif was vital to the function of GguA.

55 Here we show that the putative Zn finger, Walker A motif, KNRXG motif and Lon protease homology do

56 huttling and function depend on a functional Walker A motif.

57 80% of the AAA+ ATPase domain, including the Walker A motif.

58 onstrate that mutations in Cdc6 sensor-2 and Walker A motifs, which are predicted to affect ATP bindi

59 t greatly reduced ACOT activity, whereas the Walker A mutant K487A had greatly reduced ATPase and no

60 hat differences in the binding affinities of Walker A mutants stem from differences in stabilization

61 Walker A mutations had a smaller effect on DNA binding t

62 Despite the conservation of its Walker A nucleotide-binding motif, the NTPase activity o

63 re distinguished most sensitively by the two Walker A nucleotide-binding segments.

64 Hsp104(A503V) variants bearing inactivating Walker A or Walker B mutations in both NBDs are inoperat

65 ing bonds between adenine, triphosphate, and Walker A P-loop (Y142, Q143, and R140).

66 analog, ATPgammaS, tightly restructures the Walker A segments and drives the global open-to-closed/e

67 nsus motif (IVGPAGTGKSTLF) that contains the Walker A sequence, a well-known nucleotide-binding motif

68 A unique "closed to open" movement of Walker A, assisted by trans-acting "Glu switch" Glu-286,

69 The NBD is defined by six motifs: the Walker A, Q-loop, Signature, Walker-B, D-loop, and H-loo

70 with sterol transport, whereas swapping the Walker A, Walker B, and signature motifs together result

71 istic, deviant ATP-binding site with altered Walker A, Walker B, Signature (C-loop), and Q-loop resid

72 Arg(349) to maintain proper conformation of Walker A.

73 )@Ir nanorods (Ce@IrNRs) and enzyme-free DNA walker, a novel electrochemical aptasensor was developed

74 A Walker-A "P-loop" motif is proposed to coordinate ATP bi

75 We identified putative ATP-binding motifs (Walker-A and Walker-B) in each of the ATPases and genera

76 aired translocation phenotypes and show that Walker-A residues play important roles in determining mo

77 had 35% higher 30-day mortality than normal walkers (adjusted odds ratio, 1.35; 95% confidence inter

78 e show that the movement of a small-molecule walker along a five-foothold track can be monitored cont

79 hydrolysis of the rear foot of macrocyclized walkers (an information ratchet mechanism), the rear foo

80 We applied the Walker and Avant method to identify the antecedents, att

81 The methods of Walker and Avant were used to identify antecedents, attr

82 gest instead that different responses of the Walker and Hadley circulations to the increasing CO(2) c

83 the Inter-tropical Convergence Zone (ITCZ), Walker and Hadley circulations, and Rossby wave propagat

84 Walker and Pusey show that dispersal decisions in chimpa

85 ote formation of the ester bonds between the walker and the track.

86 e their exquisite programmability, DNA-based walkers are challenging to work with because of their lo

87 The fittest composite correlated random walkers are found to be those that come closest to being

88 DNA based synthetic molecular walkers are reminiscent of biological protein motors.

89 ales, but not females, toward perceiving the walkers as more feminine.

90 emales, but not males, toward perceiving the walkers as more masculine.

91 ibution significantly biased in favor of the walker at that site.

92 erved residues in Fml1's Walker A (K99R) and Walker B (D196N) motifs to determine whether its activit

93 t, unlike other AAA+ proteins, the catalytic Walker B aspartate is required for oligomerisation.

94 loading and Cdt1 release, similar as a Cdc6 Walker B ATPase mutant.

95 ecause they can alternately contact ptDNA or Walker B glutamate in the ATPase site and lie close to t

96 e third, D290N mutation in the conserved NS3 Walker B motif appeared >/=16 days post-transfection.

97 he C-terminal region of RNase R, whereas the Walker B motif is in its N-terminal region implying that

98 A loop region near the catalytic Walker B motif of Orc1 directly contacts DNA, allosteric

99 Mutation of the Walker B motif of RPT6 prevented XopJ-mediated degradati

100 Importantly, we discovered the Walker B motif of TssM and demonstrated that it is criti

101 otein F (PspF), by specifically altering the Walker B motif sequence required in catalyzing ATP hydro

102 ite with a non-catalytic residue next to the Walker B motif.

103 tly connected to the catalytically important Walker B motif.

104 combined data suggest that the Walker A and Walker B motifs of Rrp2 are involved in the control of a

105 and have identified ATP-binding Walker A and Walker B motifs that are present in Escherichia coli RNa

106 riant (ClpC(trap)) with mutations within the Walker B motifs to identify the direct substrates of Clp

107 e G239 residue and the putative Walker A and Walker B motifs.

108 V) variants bearing inactivating Walker A or Walker B mutations in both NBDs are inoperative.

109 of the noncanonical methionine residue M584 (Walker B sequence of nucleotide binding site 1) by gluta

110 somerization of a conserved cis peptide near Walker B to the trans configuration, which appears to pr

111 ol transport, whereas swapping the Walker A, Walker B, and signature motifs together resulted in fail

112 he nucleotide-binding sequences Walker A and Walker B, respectively.

113 iant ATP-binding site with altered Walker A, Walker B, Signature (C-loop), and Q-loop residues.

114 molecule techniques to confirm the predicted Walker-B ATP-binding motif in the phage lambda motor and

115 ies thus illuminate the mechanistic roles of Walker-B residues in ATP binding, hydrolysis, and DNA tr

116 ed putative ATP-binding motifs (Walker-A and Walker-B) in each of the ATPases and generated mutant st

117 six motifs: the Walker A, Q-loop, Signature, Walker-B, D-loop, and H-loop.

118 approximately 0.7 s per step) than previous walkers based on covalent chemistry and is weakly proces

119 entirely to autonomous decisions made by the walker, behaviour analogous to amorphous chemical reacti

120 g protein ParG, the parH centromere, and the Walker box ATPase ParF.

121 jP protein (Soj from pXO1) contains putative Walker box motifs and belongs to the ParA family of ATPa

122 Each of the Walker box mutant strains exhibited properties identical

123 Walker box mutations in RFS-1, which abolish filament re

124 e insight into a unique mechanism by which a Walker box protein forms polymers that involves the gene

125 Moreover, it is unknown how any Walker box protein polymerizes in an ATP-dependent manne

126 , and an assembling protein that is either a Walker-box ATPase (type I) or an actin-like ATPase (type

127 Well-studied plasmid Walker-box partition modules require ParA, centromere-DN

128 Walker-box partition systems are ubiquitous in nature an

129 tion, carboxysome maintenance systems employ Walker-box proteins as DNA-binding motors while McdB pro

130 McdA is similar to partition ParA Walker-box proteins, but lacks the P-loop signature lysi

131 induced propagation both below and above the Walker breakdown field it is shown that all experimental

132 It is also noted that Walker breakdown occurs at lower fields and peak DW velo

133 th dopants such as holmium, acts to suppress Walker breakdown phenomena.

134 rvation and the piezoelectric control of the Walker breakdown separating two regimes with different m

135 ith increasing applied field associated with Walker breakdown.

136 aids the velocity enhancement by preventing Walker breakdown.

137 pplications of this SNA-based stochastic DNA walker by exploiting movement-triggered cascade signal a

138 el microparticles and the detection of those walkers by substrate-coated microparticles.

139 on reactions, the nanoscale movements of the walker can lead to the generation of a single-stranded p

140 The walking behaviour is robust and the walker can take more than 30 continuous steps.

141 ticle, we show that the presence of multiple walkers can be beneficial for a procedure to estimate th

142 show that the translocation kinetics of DNA walkers can be effectively controlled by external light

143 In addition, DNA walkers can be used to create CRNs with controlled diffu

144 onvergence Zone and weakening of the Pacific Walker circulation (PWC) between approximately 1000 and

145 The Pacific Walker Circulation (PWC) fluctuates on interannual and m

146 of ENSO in October-December by weakening the Walker circulation and enhancing eastward-propagating oc

147 In addition to the anomalous SST, the Walker circulation anomalies at different ENSO phases al

148 ve orbitally-driven weakening of the Pacific Walker Circulation as contributing to this change.

149 o such as the rising branch of the anomalous Walker circulation being shifted to the central Pacific

150 ing the Green Sahara termination shifted the Walker circulation eastward and cooled the Indian Ocean,

151 ey do show that the observed slowdown in the Walker circulation is presumably driven by oceanic rathe

152 patterns are the main cause of the weakened Walker circulation over the past six decades (1950-2009)

153 AMO state, there is an anomalously weakened Walker circulation over the tropical Pacific that transp

154 simulate the observed changes, including the Walker circulation slowdown and the eastward shift of at

155 lation above the tropical Pacific Ocean (the Walker circulation) over the twentieth century.

156 ics of the hiatus, including the intensified Walker circulation, the winter cooling in northwestern N

157 n rainfall variability by altering the local Walker circulation, whereas the influence of the Pacific

158 Zone (ITCZ) and intensification/slowdown of Walker circulation.

159 ia global reorganizations of the atmospheric Walker Circulation.

160 ing across the tropical Pacific basin, i.e., Walker circulation.

161 to insolation forcing through changes in the Walker circulation.

162 risingly, the NHSM as well as the Hadley and Walker circulations have all shown substantial intensifi

163 Dynamic DNA enzyme-based walkers complete their stepwise movements along the pres

164 follows from this that vast numbers of Levy walkers could be hiding in plain sight.

165 per day (-813 steps, 95%CI,-1043,-582) than walkers/cyclists.

166 lic transport had similar activity levels to walkers/cyclists.

167 In this issue of Blood, Walker et al investigate the preclinical potential of KP

168 of Blood, Keats et al,(1) Egan et al,(2) and Walker et al(3) provide a genome-wide snapshot of the cl

169 2010;172(7):843-854), and Walker et al. (Comp Eff Res.

170 ncorrectly placed to tribe and that Sophonia Walker, Evacanthus Le Peletier &Serville, Bundera Distan

171 ease, neighbourhood-based methods and random walkers exploit the interactome allowing the prediction

172 The "walker" features a palladium(II) complex "foot" that can

173 nditions in the Friedmann-Lemaitre-Robertson-Walker (FLRW) spacetime are discussed for the generic fu

174 We report the release of catalytic DNA walkers from hydrogel microparticles and the detection o

175 ers (0.5-0.83 m/s) making up 48%, and normal walkers (>0.83 m/s) constituting 24% of the population.

176 The slowest walkers had 35% higher 30-day mortality than normal walk

177 The bipedal walker has identical carboxylic acid feet, and "steps" a

178 n, a variety of DNA-based and small-molecule walkers have been created, but observing the translation

179 al Runners' (17,201 male, 16,173 female) and Walkers' Health Studies (3,434 male, 12,384 female).

180 olled three isoenergetic diets in elite race walkers: high CHO availability (g kg(-1) day(-1) : 8.6 C

181 e characterized the function of the Q and I (Walker I) motifs in hBRG1 (SMARCA4).

182 of finite length and the protein as a random walker in the free energy landscape.

183 Twenty-two fast and 20 slow walkers in the lowest quartile of cerebral vasoreactivit

184 of the intrinsic quantum nature of multiple walkers, in order to achieve the full computational powe

185 as the locomotion speed or the weight of the walker increases.

186 aining and mild energy deficit in elite race walkers increases peak aerobic capacity independent of d

187 ssibility of gathering information about the walkers indistinguishability from the observation of bun

188 The walker is an organoarsenic(III) molecule with exchangeab

189 The random walker is designed to make random moves to adjacent node

190 The navel orangeworm, Amyelois transitella (Walker), is an agricultural insect pest that can be cont

191 ., Miller, S., Ho, S-Y., Wang, W., Chen, Q., Walker, K., Wypych, J., Narhi, L., and Gunasekaran, K.

192 Espinoza Suarez NR, Walker LE, Jeffery MM, et al.

193 hen the alanine residue (A6) in the atypical Walker-like A box of AaTadZ was converted to lysine, the

194 at position 6 (K6) present in the canonical Walker-like A box.

195 at all TadZ proteins are predicted to have a Walker-like A box.

196 W displaces under a magnetic field above the Walker limit, the oscillatory motion of the DW is observ

197 percentile, 0.47-0.79 m/s), with the slowest walkers (<0.5 m/s) constituting 28%, slow walkers (0.5-0

198 ng the anti-inflammatory actions of statins.-Walker, M.

199 frequency of the transitions that the random walker makes - between nodes in different PPI networks,

200 sent a large-exome sequencing study of Dandy-Walker malformation (DWM) and cerebellar hypoplasia (CBL

201 FOXC1 loss contributes to Dandy-Walker malformation (DWM), a common human cerebellar mal

202 ith the common cerebellar birth defect Dandy-Walker malformation (DWM).

203 chromosome 3q25.1 are associated with Dandy-Walker malformation of the cerebellum, and loss of the o

204 posterior fossa defects (including the Dandy-Walker malformation), and renal cystic dysplasia.

205 rain abnormalities (e.g., heterotopia, Dandy-Walker malformation), pituitary insufficiency, and/or sy

206 Loss of Foxc1 is associated with Dandy-Walker malformation, the most common human cerebellar ma

207 erior cerebellar vermis hypoplasia and Dandy-Walker malformation.

208 (350-450 g; n = 61) and the recently updated Walker-Mason rat scald burn model, we demonstrated that

209 Habitual runners and walkers may quantify exercise in terms of distance (km/d

210 : First, we develop a fast, automatic random walker method for cell segmentation.

211 tation is faster than the traditional random walker method or the level set method, and performs bett

212 The dynamics of the walker migration is well described by the random walk of

213 In 2009, Walker, Miles, and Davis proposed a model of amygdala-BN

214 idence interval [0.19, 0.78]) under the Eyre-Walker model.

215 e ParA motor, which contains a bacteria-like Walker motif.

216 alytic activity of the motor by bridging the Walker motifs and precisely contributing its charged gro

217 RNase R due to mutations in its ATP-binding Walker motifs exhibit growth defects at low temperatures

218 rporate these characteristics, the idea that walker motion is encoded directly, such that viewpoint a

219 to look for the co-encoding of viewpoint and walker motion, a hallmark of motion template analysis.

220 enzyme, leading to high processivity of the walker movement along the track.

221 esign parameters enables us to demonstrate a walker movement near 5 mum at an average speed of approx

222 The walker moves considerably faster ( approximately 0.7 s p

223 predictions derived from this strategy: (1) Walkers must have information about upcoming footholds d

224 ution of crosslinks and consumes neither the walker nor the track irreversibly.

225 ling microscopy shows that the motion of the walker occurs along the [110] direction of the Cu surfac

226 On the longest track (n = 9) the fraction of walkers on each end-foothold can be quantified with resp

227 ikely to require assistive devices such as a walker or wheelchair for mobility (OR=23.00; p=0.007).

228 e-body fat oxidation during exercise in race walkers over a range of exercise intensities.

229 rrent, atmospheric wind, a preference of the walker owing to prior experience, or a general bias in a

230 ) for males and P=0.02 for females) and male walkers (P=0.01 for males and P=0.08 for females) and fo

231 P<10(-5) for males and P=0.003 for females; walkers: P=0.03 for males and P<10(-4) for females), hig

232 We encode the space of walker positions in particular multi-qubit states and pr

233 Herein we review recent progress on DNA walker principles and characterization methods, and eval

234 field effect is found to change sign in the Walker regime.

235 e track via reversible protonation while the walker remains attached to the track throughout by means

236 , and 5.4% for the slowest, slow, and normal walkers, respectively (P<0.001).

237 and 13, or the medial OFC (mOFC), targeting Walker's area 14, to a group of unoperated controls on b

238 r's areas 11 and 13 and the other centers on Walker's area 14.

239 One subregion encompasses Walker's areas 11 and 13 and the other centers on Walker

240 to either the lateral OFC (lOFC), targeting Walker's areas 11 and 13, or the medial OFC (mOFC), targ

241 sensitive to lesions of the PFo as a whole (Walker's areas 11, 13, and 14).

242 tricted excitotoxic lesions targeting either Walker's areas 11/13 or area 14.

243 so the resulting ballistic trajectory of the walker's center-of-mass will facilitate stepping on targ

244 trotter" was sufficient to bias the bistable walker's direction, as were full-body adaptors.

245 ile varying the presentation duration of the walker selectively affected body motion discriminations.

246 mple model for a chemically driven molecular walker shows that the elastic energy stored by the molec

247 demonstrate that previously developed random walkers-so-called molecular spiders that comprise a stre

248 ryos had cerebellar aplasia similar to Dandy-Walker spectrum malformations observed in human patients

249 aligning the ellipses that made up a dynamic walker stimulus selectively disrupted body form discrimi

250 3), distal arthrogryposis 5 (DA5), or Marden-Walker syndrome (MWKS), which encompass contractures of

251 istal arthrogryposis type 5 (DA5) and Marden-Walker syndrome (MWS).

252 construct highly processive, autonomous DNA walker systems and to regulate their translocation kinet

253 The functionalities of state-of-the-art DNA walker systems can thus be analyzed for various applicat

254 easures the average number of steps a random walker takes to reach the ith node.

255 tion and hydrolysis reactions lead to 68% of walkers taking two steps directionally along a three-foo

256 uclease III (Exo III)-powered stochastic DNA walker that can autonomously move on a spherical nucleic

257 as been used in a remote-controlled inchworm walker that can directly couple a color-changing skin wi

258 re attached in series to the tile, and a DNA walker that can move on the track from device to device

259 of a model system comprised of a bipedal DNA walker that strides on a DNA origami track powered by in

260 Here, we report a DNA walker that uses hybridization to drive walking on DNA-c

261 been developed including self-assembled DNA walkers that can make stepwise movements on RNA/DNA subs

262 nostructures, such as those reflected in DNA walkers that sort and collect cargo on DNA tracks, DNA r

263 We describe a small-molecule "walker" that uses enzyme catalysis to discriminate betwe

264 des of control work synergistically to allow walkers to negotiate complex terrain with efficiency, st

265 Only macrocyclic walker-track conjugates are efficiently hydrolyzed by th

266 ding molecular muscles, synthesizers, pumps, walkers, transporters and light-driven and electrically

267 Multinomial Sturmer and Walker trimming were more successful in bias reduction w

268 Sackler Foundation, and Sir David and Isobel Walker Trust.

269 ions, we show dynamic chromosome binding and Walker-type ATPase activity are essential for cluster se

270 inding protein, typically called ParB, and a Walker-type ATPase, typically called ParA, which also bi

271 YcjX features a Walker-type nucleotide-binding domain indicating that Yc

272 SegA is an ortholog of bacterial, Walker-type ParA proteins, whereas SegB is an archaea-sp

273 We propose that walkers use visual information to initialize the mechani

274 Auerbach S, Filer D, Reif D, Walker V, Holloway AC, Schlezinger J, Srinivasan S, Svob

275 s more than median walking distance, or slow walker versus fast walker) was also assessed.

276 -35.79, P < 0.001), and amongst recreational walkers walked for longer per week (RR = 1.39, 95% CI =

277 Walker-Warburg syndrome (WWS) is clinically defined as c

278 o identify genetic mutations responsible for Walker-Warburg syndrome (WWS), a genetically heterogeneo

279 congenital muscular dystrophy 1C (MDC1C), to Walker-Warburg Syndrome and Muscle-Eye-Brain disease.

280 stroglycan, which not only causes the severe Walker-Warburg syndrome but is also a common cause of th

281 with brain and eye anomalies and range from Walker-Warburg syndrome to Fukuyama congenital muscular

282 e congenital muscular dystrophies, including Walker-Warburg syndrome, muscle-eye-brain disease, Fukuy

283 fferent clinical manifestations ranging from Walker-Warburg syndrome, the most severe form of dystrog

284 vere of the congenital muscular dystrophies, Walker-Warburg syndrome, to mild forms of adult-onset li

285 ing (ISPD), are a relatively common cause of Walker-Warburg syndrome.

286 nging from limb girdle muscular dystrophy to Walker-Warburg syndrome.

287 uman conditions muscle-eye-brain disease and Walker-Warburg syndrome.

288 The odds ratio of being a slow walker was 6.4 (95% CI, 1.7-24.9; p = 0.007) if there wa

289 Mary Edwards Walker was a gallant woman who stood for women's rights,

290 amplify the signal, and the enzyme-free DNA walker was applied to release more signal probes combine

291 ch suggested that the performance of the DNA walker was critically dependent upon the DNA density and

292 The operation of this Exo III-propelled DNA walker was monitored in real time and at the single-part

293 walking distance, or slow walker versus fast walker) was also assessed.

294 tory conventional metadynamics, and multiple-walkers well-tempered metadynamics calculations.

295 ore reflect the individual steps of a single walker, which require the making and breaking of As-S bo

296 Baseline slow walkers who were able to improve the 6MWT distance prese

297 int-light displays that portray the gaits of walkers whose gender is digitally morphed from male to f

298 l ring of annihilating and coalescing random walkers with deterministic biases due to selection.

299 ques enable direct observation of individual walkers with high temporal and spatial resolution.

300 this problem by modelling animals as random walkers with scent-mediated interaction processes.