ライフサイエンスコーパス: water intake

1 on in the NAc shell did not alter sucrose or water intake.

2 ypotensive drug that typically produces only water intake.

3 allow those prone to it to liberalize their water intake.

4 dilute urine, allowing for a broad range of water intake.

5 ts of ANG II on cardiovascular functions and water intake.

6 n homozygous individuals and decreased their water intake.

7 les every 6 h for 68.75 days over a drinking water intake.

8 months, which was paralleled by an increased water intake.

9 T knockdown mutant mice have higher food and water intake.

10 rtension, cardiac hypertrophy, and increased water intake.

11 % (+/-69%) increase in NAcc DA compared with water intake.

12 tered feeding behavior with reduced food and water intake.

13 ssible sites were ineffective at suppressing water intake.

14 Successful NIL-D was confirmed by increased water intake.

15 the non-NMDA receptor antagonists to induce water intake.

16 tense, dose-related feeding without altering water intake.

17 ted with lower urine osmolality and elevated water intake.

18 t encompasses key elements needed to improve water intake.

19 tion of pIC-BLA projection neurons decreased water intake.

20 the gut rapidly inhibits thirst neurons upon water intake.

21 he detection of thirst and the regulation of water intake.

22 oval of CB(1) receptors in the pIC decreases water intake.

23 rther structural instabilities and increased water intake.

24 along the southern shoreline past the Toledo water intake.

25 ntribute to long term adaptation to variable water intake.

26 tributed 12% and 10%, respectively, to total water intake.

27 t suppressed sucrose intake had no effect on water intake.

28 and sucrose solutions, but do not potentiate water intake.

29 er drinking, whereas raclopride also reduced water intake.

30 higher beverage and food moisture and total water intakes.

31 nhibited isoproterenol-, but not PEG-induced water intakes.

32 dy weight (7-17 g), food intake (8-13 g) and water intake (11-23 ml), while the vehicle or MS1 condit

33 ynaptic dopamine on (1) spontaneous food and water intake, (2) incentive motivation and learning to o

34 significantly increased deprivation-induced water intake (256%) over a 60 min time course.

35 bidity and COVID-19 had significantly higher water intake (37.3% vs. 2.2%, p < 0.001) and poorer slee

36 n sham feeding rats, and deprivation-induced water intake (51%) in sham drinking rats.

37 corresponding controls in sucrose, chow, or water intake across a range (0.0001-20%) of sucrose conc

38 ng at baseline and a significant decrease in water intake after administration of AdCre/adenovirus en

39 creases in home-cage ambulatory activity and water intake after exposure to shock than did swim-test

40 y (PRA) and found that weight loss decreased water intake after Iso, but had no effect on PRA.

41 tremic rats exhibited a triphasic pattern of water intake after SC, with peak intakes 3 times higher

42 whereas lesions of the MnPO or LPO increased water intake after the treatment.

43 fection risk as a function of grass, soil or water intake, age of carcass sites, and the exposure req

44 responded well with the measured decrease in water intake and an increase in urine volume with surplu

45 m neurons that induce a parallel increase in water intake and arginine vasopressin (AVP) secretion to

46 Lipopolysaccharide (LPS) decreased food/water intake and body weight in ethanol-naive and ethano

47 ection of physostigmine into the SFO induced water intake and c-fos expression in the AV3V area as we

48 into the LHA on the stimulation of food and water intake and c-Fos expression.

49 ANP was found to inhibit the water intake and corticosterone release induced by i.c.v

50 ortex cells participate in the regulation of water intake and deconstructed the circuit mechanisms of

51 on of xerostomia was determined by increased water intake and decreased salivary flow rate.

52 level of obesity, presence of comorbidities, water intake and food consumption variables.

53 or antagonist, losartan, to reverse both the water intake and Fos-immunoreactivity (Fos-ir) induced i

54 rtan to assess the contribution of Ang II to water intake and Fos-ir responses to peripheral injectio

55 weight status modifies the relation between water intake and hydration status.

56 el, obesity modifies the association between water intake and hydration status.

57 ing was associated with substantially higher water intake and increased renal reactive oxygen species

58 ut vitamin C did not affect maternal food or water intake and led to a significant increase in matern

59 influenza-induced decreases in food intake, water intake and mobility during early-stage infection a

60 for measuring breast milk intake and nonmilk water intake and our understanding of nutritional needs

61 Daily water intake and plasma glucose and insulin were not cha

62 ned the effect of weight loss on Iso-induced water intake and plasma renin activity (PRA) and found t

63 Daily drinking water intake and rice consumption rate distributions wer

64 R-null mice, leading to a marked increase in water intake and salt appetite.

65 (SFO) has been suggested to be important for water intake and secretion of vasopressin (AVP).

66 rtant role in the hypothalamic regulation of water intake and the endocrine axis, in the regulation o

67 We evaluated the relation of plain-water intake and the substitution of plain water for SSB

68 roduced a marked increase in food intake and water intake and this effect was completely reversed by

69 hypohydration between subjects with adequate water intake and those with low water intake was 0.56 (9

70 VDR-null mice still maintained the increased water intake and urinary output.

71 We examined how the association between water intake and urine osmolality, which is a hydration

72 altered renal water handling, with decreased water intake and urine volume, alongside higher urine os

73 This was accompanied by reduced daily water intake and urine volume, as well as increased urin

74 , we investigated the regulation of food and water intake and weight loss following BNST PACAP infusi

75 In the brain AM inhibits water intake and, in a physiologically relevant manner,

76 ribution of iAs exposure rates from drinking water intakes and rice consumption in the U.S. populatio

77 4-h spontaneous ambulatory activity and food/water intake) and several aspects of brain catecholamine

78 BDNF significantly decreased food and water intake, and body weight gain.

79 Food, water intake, and body weight were measured daily.

80 Food intake, water intake, and body weight were monitored daily.

81 es in GLP-1-associated gene expression after water intake, and compared the effects of fluid intake t

82 uding decreased activity, decreased food and water intake, and decreased interest in social interacti

83 s, such as reduced caloric intake, increased water intake, and decreased reproduction and growth.

84 act (commNTS) on body weight, daily food and water intake, and plasma glucose and insulin in rats.

85 e fluctuation, metabolism, food consumption, water intake, and renal salt and water excretion.

86 excitation robustly elevated blood pressure, water intake, and sodium intake, while optogenetic inhib

87 Ang II-induced increases in blood pressure, water intake, and sympathoadrenal catecholamine release;

88 The detection of water and the regulation of water intake are essential for animals to maintain prope

89 Self-reported change in water intake at 6 months was greater in the water group

90 Second, using international data, water intake at the national, state, and county-levels b

91 g a method for estimating U.S. manufacturing water intake at the necessary spatial and sectoral resol

92 hly diluted urine) and polydipsia (increased water intake), both features of diabetes insipidus.

93 ly in the highest quartile of total or plain water intake but did not approach the Bonferroni-correct

94 nist, losartan (1 microgram/200 nl), reduced water intake but not 0.3 M NaCl intake induced by subcut

95 II into the lateral ventricle (LV) increases water intake, but a similar response is not observed aft

96 is critical for limiting excessive food and water intake, but the underlying gut-brain communication

97 recommendation to alter the daily amount of water intake by a specific amount for a predefined perio

98 ions, and practices needed to ensure optimal water intake by all in the United States and elsewhere.

99 odel where CB(1) receptor signaling promotes water intake by inhibiting the pIC-BLA pathway, thereby

100 ogen reduces angiotensin II (Ang II)-induced water intake by ovariectomized rats.

101 Similarly enhanced water intake by rats with APX also was observed when mar

102 be responsible for the increases in salt and water intake caused by hypovolaemia.

103 Instead, a pattern of reduced food and water intake, combined with feces replete with lipid and

104 We used public-domain, mortality-linked water intake data from the NHANES conducted in 1988-1994

105 Although water intake decreased there was no reduction in total f

106 Non-breast milk oral water intake did not differ by group (P = 0.39) and was

107 Various contributors of total water intake differed in their association with dietary

108 d approach to identify Pennsylvania drinking water intakes downstream of wet FGD discharges and to as

109 Rats reduced water intake during 24 h HU in all conditions.

110 g sham feeding or deprivation (24 h)-induced water intake during sham drinking in rats with gastric f

111 Food and water intake, feeding microstructure, and general motor

112 ventral median preoptic nucleus) suppressed water intake following 24 h water deprivation.

113 ns involved in blood pressure regulation and water intake following dehydration.

114 Here, we show that water intake, food consumption, stool weight, urine volu

115 ACE did not alter body weight, water intake, food intake, or treadmill performance (p >

116 ke Erie, including a station proximal to the water intake for the city of Toledo.

117 higher estimates of breast milk and nonmilk water intake; for example, in infants aged 6-12 mo, diff

118 Values of breast milk intake and nonmilk water intake from 130 DTM calculation spreadsheets were

119 The percentage of total water intake from plain water increased with age.

120 al actions, including inhibition of food and water intake, gastric emptying, and stimulation of neuro

121 e human body in response to the individual's water intake habit.

122 Subsequent experiments showed that water intake had a selective effect on central GLP-1-rel

123 reases in bromide concentrations at drinking water intakes; however, similar low flow conditions in s

124 can be accomplished with increased salt and water intake in conjunction with fludrocortisone.

125 ge in association with higher total or plain water intake in men or women in this national cohort.

126 tions also significantly stimulated chow and water intake in nonfood-deprived rats.

127 tor subtype mediation of deprivation-induced water intake in real drinking and sham drinking conditio

128 Deprivation-induced water intake in sham drinking rats was significantly red

129 e that weight loss decreases Ang II-elicited water intake in the female rat by down-regulating the ex

130 y decreased body weight, as well as food and water intake in the first 24 h following infusion.

131 ve systematically examined the correlates of water intake in the US population.

132 ons neither altered micturition patterns nor water intake in the young adult rat.

133 l water shortages based on concentrations of water intake in water-stressed regions.

134 We review water intakes in the United States relative to requireme

135 The water intake induced by DNQX was also blocked by pretrea

136 rption but rather is the result of increased water intake induced by the increase in systemic and bra

137 atrial junction (SVC-RAJ) reduces sodium or water intake induced by various experimental procedures

138 Water intake induces chemical shift changes up to 2 and

139 thods were developed to study or to quantify water intake into starch-based matrices.

140 Adequate water intake is critical to physiologic and cognitive fu

141 studies provide evidence that Ang II-induced water intake is mediated via the classical G protein cou

142 How food and water intake is reciprocally regulated to maintain homeo

143 ecoupling design and the low-cost renewal of water-intake layer.

144 romide concentrations at downstream drinking water intakes, leading to increased formation of toxic d

145 icKO and MS differentially affected food and water intake, locomotor activity as well as panic-like e

146 y noted in women with higher total and plain water intakes may be spurious and requires further inves

147 carbohydrates as well as deprivation-induced water intake (mu) under real-feeding and real-drinking c

148 ta (i.e., location, quantity, and purpose of water intake) needed to determine this impact does not e

149 are involved in behaviors including food and water intake, nociceptive responses, breathing regulatio

150 In men, neither total water intake nor each of the individual water source var

151 Intra-vmPFC DAMGO affected neither water intake nor nonspecific oral behavior.

152 In both conditions, the increased water intake occurred within the first 15 min of the dri

153 lacked free access to water, 74% had enteral water intake of less than 1 L/d, and 94% received less t

154 e plasma concentrations corresponding to tap water intake of PFAS.

155 ge of Cpd1324 dose-dependently increased the water intake of wild-type (WT) C57BL/6J mice only and wa

156 ontrast, hyponatremic SC rats exhibited peak water intakes of 600 ml/24 hr, approximately 9-10 times

157 f 600 ml/24 hr, approximately 9-10 times the water intakes of sham-operated normonatremic rats.

158 mall number of studies suggested benefits of water intake on weight loss and nephrolithiasis, while s

159 ted rats with 100 mg/kg of captopril reduced water intake only during the initial 15 min after a gava

160 a fat-rich diet, with no changes in chow and water intake or body weight.

161 osses and impaired thirst or restricted free water intake or both.

162 rttin palmitoylation upon increased salt and water intake or water deprivation, indicating that this

163 ial contamination of water sources with MAP, water intake, or water treatment.

164 d significant increases in Br/Cl at drinking water intakes over the first year of the study (2009-201

165 in ethanol-dependent rats without affecting water intake overall or basal ethanol intake in control,

166 Total water intake (P = 0.002), urine osmolality (P < 0.001),

167 Water intake partially decreased ANG I-induced Fos-ir in

168 a slight but not significant suppression of water intake, particularly after the higher dose.

169 s indicate that the O1 channel is the likely water intake pathway, and the Cl1 channel is the likely

170 Plain-water intake, per se, was not significantly associated w

171 allow a quantitative characterization of the water intake process inside PEFC catalysts with nanoscal

172 W% at drinking water treatment plant surface water intakes ranged from <0.01 to 2.0% under mean-annua

173 ld cause adverse effects due to a high daily water intake rate over the long term.

174 SFO lesions reduced water intake regardless of hydration condition.

175 erature assessing the benefits of increasing water intake related to a large variety of health outcom

176 ight, and an increase in nicotine, food, and water intake relative to controls.

177 Water intake requirements largely reflect water turnover

178 that receptor number is more critical to the water intake response than the saline intake response, o

179 e repeated injections, however, decrease the water intake response to Ang II without affecting saline

180 the G protein mediate desensitization of the water intake response.

181 cise in the heat, sweat output often exceeds water intake, resulting in a body water deficit (hypohyd

182 de facto reuse is analyzed for 2056 surface water intakes serving 1210 DWTPs across the U.S.A. that

183 despite normal activity levels and food and water intake, Smn deficiency caused constipation, delaye

184 onsumption was accompanied by an increase in water intake, so that the total volume of liquid consume

185 I in samples collected at WWTPs and drinking water intakes (source water) during one year were quanti

186 Interactions between weight status and water intake status were significant in linear (P = 0.00

187 plants as the dry season progressed, dietary water intake still declined.

188 e showed age-dependent increases in food and water intake, stomach and body weights, and decreases in

189 Here, we describe a water-intake strategy of Pachycrepoideus vindemmiae, a p

190 ter and that it host-feeds on SWD pupae as a water-intake strategy.

191 f brain sites implicated in hunger, salt and water intake, stress, arousal, and reward.

192 r chemistry sensors embedded in a high-speed water intake system to document spatial variability.

193 In addition, the body mass, food, and water intake, systolic blood pressure, biochemical and o

194 elopment of hyponatremia even in the face of water intake that can approach 20 L/d.

195 nterventions, differentiated by advice about water intake (the water group received advice to increas

196 forebrain systems mediating other aspects of water intake, the authors examined the effects of lesion

197 acturing constitutes an estimated 6% of U.S. water intake, the data (i.e., location, quantity, and pu

198 ally, individuals who meet their recommended water intake through only bottled sources may be ingesti

199 sertion, it minimized peptide clustering and water intake through stabilization of the bilayer struct

200 and behavioral support to increase habitual water intake to 8 cups per day.

201 (the water group received advice to increase water intake to 8 cups per day; the control group did no

202 ver in wild populations, as was the ratio of water intake to dietary energy intake (~2.8 mL/kcal).

203 Restriction of water intake to increase vasopressin levels also signifi

204 Additionally, we investigated total water intake (TWI), which comprises drinking water, pref

205 sin II type 1 receptor antagonist normalized water intake, urinary volume, and c-Fos expression in VD

206 Body weight, water intake, urine output, solute and urea excretion, s

207 sured, including arterial pressure, food and water intake, urine volume, and sodium and potassium exc

208 n on measurements of breast milk and nonmilk water intake using the DTM technique.

209 inergic receptor agonist, carbachol, induces water intake, vasopressin (VP) release and an acute incr

210 ith adequate water intake and those with low water intake was 0.56 (95% CI: 0.43, 0.73) in adults who

211 previous 24 h (in 1999-2004, estimated total water intake was 3.35 L), with plain water and beverages

212 Total water intake was determined with the use of a 24-h dieta

213 Plain water intake was inversely associated with the intake of

214 Total water intake was inversely related to energy from fat an

215 Furthermore, water intake was markedly reduced, ameliorating the symp

216 vels of ethanol in dialysates from the NAcc; water intake was negligible.

217 Plain-water intake was not associated with T2D risk in the mul

218 , a significant increase in urine volume and water intake was observed; urine volume rose from 9.5+/-

219 Plain water intake was unrelated to the intake of energy and b

220 , which is known to associate with increased water intake, was increased in the hypothalamic paravent

221 ptor function as measured by agonist-induced water intake, was significantly attenuated in these rats

222 Ad libitum water consumption, total water intake, water output through urine, total water ou

223 astewater discharges and downstream drinking water intakes, we estimate that the sources of drinking

224 No differences in food or water intake were found between groups; however, male Fm

225 Alcohol intake, body weight, and water intake were measured at 24 h post-injection interv

226 Body weight and spontaneous food and water intake were monitored daily.

227 Animal weight and water intake were not altered during 10 days of EPEC inf

228 cial defeat protocol, body weights, food and water intake were recorded, depression and anxiety-like

229 rotarod or calorie consumption, and food and water intake were reduced in the Bmal1(-/-) mice after M

230 Urine volume and water intake were unchanged in all other groups.

231 highest levels of genetic markers at the raw water intakes were associated with human fecal sources (

232 Body weight, kidney weight, and food and water intakes were not different from WT littermates.

233 Water intakes were unaffected by the higher dose of losa

234 Baclofen had no effect on water or sugar water intake when administered to anterior or posterior

235 rPP and NPY significantly increased food and water intake when they were administered into the LHA, a

236 AngIII produced a dose-dependent increase in water intake, whereas saline intake was equivalently inc

237 ed architecture predetermines the pattern of water intake, which sets the stage for the orchestrated

238 Yet, the extent to which increasing dietary water intake while decreasing water loss enables animals

239 rotensin (Nts) neurons preferentially induce water intake while suppressing feeding.

240 udies have examined the association of total water intake with all-cause mortality.

241 out the association of contributors of total water intake with dietary characteristics in US children

242 o examine the association of contributors of water intake with dietary characteristics, meal consumpt

243 ilar rate of increase in locomotion and food/water intake with time.

244 it, and molecular mechanisms for controlling water intake within the insula remains parcellated.

245 receptor (Ptprd) deletion results in reduced water intake without affecting food intake and abolishes

246 terior insula (pIC) increases in response to water intake, yet only the specific removal of CB(1) rec

247 ndations exist regarding the amount of daily water intake, yet the supporting evidence is not clear,