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1 er, maintains VF by generating new reentrant wave front.
2  was activated predominantly by the superior wave front.
3  the concave shape of the Ca2+ fertilization wave front.
4 sary to propagate a mechanically coordinated wave front.
5  mapped area displacing the entire reentrant wave front.
6 lided with the leading edge of the reentrant wave front.
7 sed current load experienced by the pivoting wave front.
8 e border or by collision with an outside new wave front.
9  of these 3 patterns could occur in the same wave front.
10  magnetic susceptibility gradient across the wave front.
11  due to higher rates of genetic drift on the wave front.
12 lion cell genesis behind the differentiation wave front.
13  fronts and multiple simultaneous-activation wave fronts.
14 t role in re-formation of unstable reentrant wave fronts.
15 erns, or varying orientations of circulating wave fronts.
16  functional conduction block and circulating wave fronts.
17  cycle length, or core size of the reentrant wave fronts.
18  speed equal to the propagation speed of the wave fronts.
19 common tangent surfaces, the new propagating wave fronts.
20 ained by both focal and incomplete reentrant wave fronts.
21 quency of deleterious mutations on expanding wave fronts.
22 but broad spectra in AF, reflecting multiple wave fronts.
23 greater negative curvature of the activation wave front (-1.86+/-0.40 mm in chimeric mice versus -0.8
24 low waves, propagated without decrement as a wave front (2-5 mm/s) through the ICC-SM network lying a
25  second wave front met the tail of the first wave front 69 +/- 11 ms (range, 40 to 90 ms) after its l
26 dy aimed to validate the accuracy of a novel wave-front aberrometry-based auto refractometer, Instare
27 inear relationship exists between activating wave-front acceleration and deceleration in the SCZ and,
28 lation showed neither reentrant circuits nor wave fronts activating the right atrium via BB.
29                          The analysis of the wave-front activation patterns is crucial for the compre
30                                       In 342 wave fronts, activation was mapped arising focally.
31                                       In 142 wave fronts, activation was mapped propagating from PF t
32                                       In 534 wave fronts, activation was mapped propagating from work
33            The length of the pathway of each wave front along the catheter was 6.5 +/- 4.5 cm.
34           Activation times were grouped into wave fronts along the catheter, and the lengths of the w
35            New technologies reported include wave-front analysis for amblyogenic factors and a visual
36 ions were determined using the Topcon KR-1 W wave-front analyzer.
37 odes were associated with a single reentrant wave front anchored to the PM.
38 n the origin and direction of the excitation wave front and are responsible for polymorphic arrhythmi
39 tex, which means that it possesses a helical wave front and carries orbital angular momentum.
40 side new wave front merged with the original wave front and excited the core.
41      A fraction of mutations travel with the wave front and generate mutant populations that are on a
42 ose ancestors lived mostly on the colonizing wave front and individuals whose ancestors remained in t
43                We experimentally observe the wave front and the distortion of modulated wave packets
44 progression of ganglion cell differentiation wave front and to induce its own expression.
45 cities leading to the self-steepening of the wave front and to the formation of a shock.
46                                The number of wave fronts and approximate entropy were significantly (
47 ve velocity can strongly distort propagating wave fronts and degrade image quality.
48 s independent of the rate of invasion of new wave fronts and epicardial breakthroughs.
49 ation were seen, including single-activation wave fronts and multiple simultaneous-activation wave fr
50 sing evidence that both functional reentrant wave fronts and multiple wavelets are present during ven
51 l arrhythmia dynamics showed that arrhythmia wave fronts and phase singularities (rotors) were relati
52  exceptional abilities to dynamically tailor wave fronts and polarization states, while maintaining s
53 on between epicardial-endocardial arrhythmic wave fronts and rotors that are relatively more organize
54 etween epicardial and endocardial arrhythmic wave fronts and rotors.
55  human hearts with DCM, epicardial reentrant wave fronts and transmural scroll waves were present dur
56 aps showed incessantly changing beat-to-beat wave fronts and varying spatiotemporal behavior of drive
57 frequency region, reverse rotation of spiral wave front, and side-band modes generation during propag
58 inal phonon cooling effect on the molten COD wave front, and the formation of patterns due to laser l
59 excitation caused disruption of the original wave front, and the newly formed wave front(s) vanished
60 ent activated first by the paced orthodromic wave front, and were mainly due to local pacing latency,
61  globally synchronized activity, propagating wave fronts, and spiral waves that have been previously
62 quations are used only near action potential wave fronts, and the macroscopic equations are used ever
63 , both the amplitude and phase of an optical wave front are acquired with a dual-plane construct.
64                            (2) New reentrant wave fronts are generated when one wave front interacts
65    Also there are two tangential surfaces so wave fronts are propagated in both the forward and backw
66 tablishing segmentation, including clock and wave front, are not perturbed.
67 d to rotate as a single reentrant activation wave front around the TA, and the role of LLR in those p
68 ls of Shh expression are detected behind the wave front as ganglion cells accumulate, while the Patch
69 f naturally occurring unshocked fibrillation wave fronts, at comparable CIs and Vms.
70 el we numerically illustrated how excitation wave-fronts behave in a mycelium network colonising an i
71 tained VT always occurred when the reentrant wave front blocked in the central common pathway in reen
72 ion always occurred when the two circulating wave fronts blocked in the central common pathway (CCP).
73            The distally initiated activation wave front blocks near the S2 site because the same S2-i
74  AFL, reflecting its single macro-re-entrant wave front, but broad spectra in AF, reflecting multiple
75     A single meandering functional reentrant wave front can be induced in human atrial tissues and pr
76 hat a single meandering functional reentrant wave front can result in rapid and irregular electrogram
77 tion was caused by two successive activation wave fronts circulating in the same direction along the
78 ing in a direction opposite to the reentrant wave front collided with the leading edge of the reentra
79 ested by early breakthrough in the lower RA, wave-front collision in the high lateral RA or septum, a
80 ise orientation with early annular break and wave-front collision over the isthmus.
81 ns of wavelets that exist outside of the new wave front combine to form a wake.
82                Intercycle interval (ICI) and wave front conduction time (WCT) were determined for the
83 he heart', Mines proposed that an activation wave front could propagate repeatedly in a circle, initi
84                                   Density of wave fronts decreased during both global ischemia (P<0.0
85  As tissue mass was decreased, the number of wave fronts decreased, the life-span of reentrant wave f
86 eterogeneities of repolarization may lead to wave-front destabilizations and initiation of ventricula
87  out of focus light, minimal aberrations and wave-front distortions.
88   The distortion of the shape of the surface wave fronts due to the curvature is corrected with a sui
89 d when one wave front interacts with another wave front during its vulnerable period.
90 We sought to evaluate the characteristics of wave fronts during ventricular fibrillation (VF) in huma
91 The effects of PA on cellular properties and wave front dynamics were reversed during 60 minutes of d
92  areas in which the cathode-break excitation wave fronts easily invaded the spatial excitable gap via
93                                          All wave fronts emanated from foci and breakthrough sites, a
94 During persistent and LSP AF in 12 patients, wave fronts emanating from foci and breakthrough sites m
95 ventricles at such high frequencies that the wave fronts emanating from them breakup at varying dista
96 inside the medium with the distortion that a wave front, emitted from that point, experiences due to
97                                           If wave fronts ever arise focally during VF, it should be p
98                            (1) The reentrant wave front for each morphology rotated around the same l
99 it and difficulty to preserve a high-quality wave front for focus.
100  spontaneous wave break and by new reentrant wave front formation.
101 that occur before the arrival of the seismic wave front from the mainshock, which violates causality.
102 s such as rigid gas permeable lenses, custom wave front-guided soft contact lenses, hybrid lenses and
103                                        These wave fronts had a mean cycle length of 229+/-45 ms (160
104 The roles of Purkinje fibers (PFs) and focal wave fronts, if any, in the maintenance of ventricular f
105 conduction was equal to the native reentrant wave front in completely excitable regions and slower th
106 ented settlement of pioneers directly on the wave front in North America has contributed to low level
107 itable regions and slower than the reentrant wave front in partially excitable regions.
108         (2) In 5 VT episodes, the activation wave front in the CCP abruptly broke across a stable arc
109 onal maps revealed the presence of reentrant wave fronts in 17 of 33 runs of VF in ablated ventricles
110      Knowledge of the pathway common to both wave fronts in figure-8 reentrant circuits (ie, the isth
111 tylcholinoceptors activates propagating Ca2+ wave fronts in oligodendrocytes and that the characteris
112 ) induces nonstationary meandering reentrant wave fronts in the atrium.
113 uent intra-PV conduction blocks and multiple wave fronts in the PVs were recorded during 10 AF episod
114                          Interaction between wave fronts in the right and left atrium may be importan
115 opment of dynamic components for controlling wave fronts in the sub-terahertz region of the electroma
116   In conclusion, a decrease in the number of wave fronts in ventricular fibrillation by tissue mass r
117 lesion (n = 8), the induced single reentrant wave front, in the form of a spiral wave, meandered irre
118 fronts decreased, the life-span of reentrant wave fronts increased, and the cycle length, the diastol
119  showed that the PV was activated by a focal wave front independent of left atrial (LA) activation in
120               Postshock activation times and wave-front interaction patterns were determined with an
121 reentrant wave fronts are generated when one wave front interacts with another wave front during its
122 ocated by the site of entry of the premature wave front into the circuit.
123 the translocation of Ca2+ from the spreading wave front into the SR.
124 gesting that the leading edge of the reentry wave front is not always at the TA.
125  a source of secondary wavelets, and the new wave front is the tangential surface to all the secondar
126 amic strategy treating sites on the epidemic wave front leads to optimal performance.
127 whether excitation of the core by an outside wave front leads to termination of the reentry in the at
128 o provides a natural anchor to the reentrant wave front, lengthening the life span of reentry.
129 odes, reentry terminated when an outside new wave front merged with the original wave front and excit
130                                   The second wave front met the tail of the first wave front 69 +/- 1
131 uring rapid ventricular stimulation by AC, a wave front might encounter the refractory tail of an ear
132 um) observed during AF were reactivated by a wave front most often coming from the atrial septum via
133 as the result of interference by an invading wave front (n = 19 or meandered off the mapped region (n
134 vations because of interference with outside wave fronts (n=5) or spontaneous separation of waves fro
135 posite polarities, we detected a propagating wave front of activity that originated at the cortical l
136 lates the fate of mutations occurring at the wave front of an expanding population.
137                                      A broad wave front of atrial activation was recorded in 17 patie
138  neuroepithelium peripheral to the expanding wave front of differentiation.
139 s from the central to peripheral retina as a wave front of differentiation.
140                   During the third instar, a wave front of enhanced Notch activity progressing over t
141  anterior barrier constraining the reentrant wave front of human counterclockwise atrial flutter.
142 design of artificial meta-atoms to shape the wave front of light by optimal control of both its phase
143 of data that were analyzed, 1018 PF or focal wave fronts of activation were identified.
144 elimination of most, if not all, propagating wave fronts of electrical activation by a shock constitu
145 ing ventricular fibrillation (VF), reentrant wave fronts often transiently anchored to the PM.
146 tation, VF consists primarily of a few large wave fronts on the endocardium.
147  humans, consisting primarily of a few large wave fronts on the order of 6 to 9 cm.
148  front velocity and could be used to predict wave front orientation with respect to the surface.
149 rior to superior regions, whereas 2 opposing wave fronts originated on the left septum in both the su
150                  In acute AF, a single broad wave front originating from the posterior and medial atr
151 eentrant impulse circulated or was caused by wave fronts originating outside the reentrant circuit.
152  apex, V(m) was expected to exhibit circular wave front patterns and B(z) to reflect the circular com
153  and advance for many millimeters as a sharp wave front perpendicular to the pial surface, at speeds
154  (32.8 +/- 6.5 mm, P < .02), indicating that wave-front pivoting was primarily responsible for shorte
155             Abrupt changes in loading during wave-front pivoting, rather than membrane ionic properti
156 quences expected to result in differentiated wave-front populations with low genetic variation and po
157           The increase in [Ca(2+)](i) at the wave front preceded depletion of the SR at each point al
158 , activation of the septum was discordant; 1 wave front propagated rapidly on the right septum from i
159 nus rhythm and superior right septal pacing, wave fronts propagated predominantly from superior to in
160              Although caffeine-evoked Ca(2+) wave fronts propagated throughout the cell, in most cell
161                                   The Ca(2+) wave fronts propagated with constant amplitude; the spre
162 ntry terminated abruptly when an outside new wave front propagating in a direction opposite to the re
163 VF maintenance, it should be possible to map wave fronts propagating from PFs into the working ventri
164 patially distributed reaction can support pH wave fronts propagating with a speed of the order of 0.1
165 ropagating in a negative-index material have wave-front propagation (wavevector, k) opposite in direc
166  orientation in the pulmonary veins (PVs) in wave-front propagation are poorly understood.
167 f shock-depolarized areas (BSDAs) from which wave-front propagation could have arisen were identified
168  We conclude that the incidence of postshock wave-front propagation decreases with increasing refract
169                   Furthermore, incidences of wave-front propagation following shocks were consistentl
170 ired to guarantee the cessation of continued wave-front propagation in defibrillation.
171 w that plasmonic nanoparticles can produce a wave-front reconstruction when they are sampled on a dif
172                                The number of wave fronts recorded by the catheter was 9.2 +/- 2.9 wav
173                                The reentrant wave fronts remained stationary and rotated around these
174                            Coordinating this wave front requires fast and robust signaling mechanisms
175  encounter the refractory tail of an earlier wave front, resulting in the formation of a wave break a
176            The central core around which the wave front rotated became smaller, which caused shorteni
177     The central area (core) around which the wave fronts rotated had a mean surface area of 12 +/- 3
178 by decreasing the core size around which the wave front rotates.
179 ed by an interaction between two propagating wave fronts roughly perpendicular to each other.
180 he original wave front, and the newly formed wave front(s) vanished at the tissue border within 77 +/
181 nts recorded by the catheter was 9.2 +/- 2.9 wave fronts/s.
182 inciple (1678) implies that every point on a wave front serves as a source of secondary wavelets, and
183 eously fulfill the requirements of nonlinear wave-front shaping and quasi-phase-matching, the convers
184                   When clearly seen, the AFL wave front split (n = 3) or turned in the region of the
185 eralization of this strategy to more complex wave-fronts, such as vortex beams that carry orbital ang
186                           The integration of wave-front technology and lens-based surgery represents
187                            (3) The reentrant wave fronts terminate spontaneously or as the result of
188                                The reentrant wave fronts terminated spontaneously (n = 7), as the res
189 n analysis of an expanding population with a wave front that advances at a constant slow rate.
190 ansported in the form of a rapidly advancing wave front that progressed along axons, in a microtubule
191 ion, followed 22+/-15 ms later by propagated wave fronts that arose from the same site.
192 arily by propagation of the stimulus-induced wave fronts that closed up the excitable gap.
193  coordinated fashion, generating contractile wave fronts that propagate through the heart with each b
194 te the propagation of a number of activation wave fronts that reenter to maintain the arrhythmia.
195 ithin the measured range of the speed of the wave fronts, the length of breaking fronts per unit area
196                                 The "broken" wave front then circulates around both sides of the bloc
197 ering by causing attachment of the reentrant wave front tip to the obstacle.
198  attachment, both conduction velocity of the wave-front tip and wavelength near the obstacle adapted
199 onverts a nonstationary meandering reentrant wave front to a stationary one.
200 e attachment and detachment of the reentrant wave front to and from the ridge determine "flutter-like
201 egative regulator behind the differentiation wave front to control ganglion cell genesis from the com
202 ct excitation of the core that displaces the wave front to the tissue border or by collision with an
203                                              Wave fronts traveling from one atrium to the other and/o
204 el shows glycolytic oscillations, leading to wave fronts traveling through the monolayered population
205  accelerates local cytosolic Ca decay as the wave front travels to the next cluster, which limits wav
206                                        Tumor wave front velocities determined via a marginal stabilit
207 hly sensitive to the transmural component of wave front velocity and could be used to predict wave fr
208 between the interfacial morphology and tumor wave front velocity provides an explicit, testable, clin
209 aps of reentry were constructed to determine wave-front velocity, and piecewise linear adaptive templ
210 n all samples, a single meandering reentrant wave front was induced, causing irregular and rapid bipo
211 t (78.6%) terminated after one of the double wave fronts was blocked in the TA-ER isthmus.
212                             By analyzing the wave fronts, we found that the cycle length, refractory
213                   Although the circular V(m) wave fronts were detected, the B(z) maps were not as sim
214 s along the catheter, and the lengths of the wave fronts were estimated.
215 ted sinusoidal packets with non-superluminal wave fronts were observed.
216  had their subendocardium ablated, reentrant wave fronts were present in 6 of the 108 runs of VF at b
217               In all episodes, the reentrant wave fronts were spontaneously initiated by an interacti
218                                     The ECL "wave fronts" were visualized and imaged in space and tim
219 ationary, but it propagates only the forward wave front when it is advancing with a speed equal to th
220  be observed among mutations arising in this wave front where extreme population bottlenecks arise an
221 of a larger number of invader alleles at the wave front, where effective population size is thus incr
222 distance between the head of the circulating wave front, which could be located on the activation map
223 peaks and oscillations were found to be Ca2+ wave fronts, which propagate via distinct amplification
224 set was defined as SVE advanced by the paced wave fronts while atrial signal continued at the tachyca
225 on of the SR at each point along the calcium wave front, while during this latency period a transient
226 ime asymptotic behaviour, where a travelling wave front with a constant speed has been established.
227 correlates with position along the advancing wave front with more divergent lineages near the origin
228 ely caused by the interaction of propagating wave fronts with surface topography.
229 and spectral analysis to identify sequential wave fronts with temporal periodicity and similar spatia
230 thus the conduction velocity (CV) of the AFL wave front within the tricuspid annulus-inferior vena ca
231 ons in the PVs and the VOM, and with complex wave fronts within the PVs.

 
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