1 We demonstrate that 15-PGDH shows areas of increased exp
2 We demonstrated that (
18)F-JK-PSMA-7 was not inferior wh
3 We demonstrate that 2'-azidouridine is only incorporated
4 In this study,
we demonstrated that 3T3L1 adipocytes differentiation in
5 Finally,
we demonstrate that a significant fraction of PSVs in se
6 We demonstrate that a wide array of substituents is tole
7 We demonstrate that AAA4 controls the priming stroke of
8 ing of Htt was recapitulated in the MEFs and
we demonstrated that ablation of SRSF6 did not modulate
9 Functionally,
we demonstrate that ACh contributes to sustaining high d
10 In addition to confirming the role of MDD,
we demonstrate that ADHD and schizophrenia likely play a
11 sensitive dyes, Di-4-ANEPPDHQ and C-laurdan,
we demonstrated that AdipoRon decreased the rigidity of
12 We demonstrate that all four PARP inhibitors have a uniq
13 We demonstrate that alphaCD3 alone induced substantial T
14 Here,
we demonstrate that although high-level PA (HPA) indeed
15 Here,
we demonstrate that among the many motile cilia of a mul
16 Finally,
we demonstrate that AMPK affects cellular physiology by
17 of molybdenum disulfide and graphene oxide,
we demonstrate that an accurate position can be obtained
18 In conclusion,
we demonstrate that an endogenous healing response can b
19 Here,
we demonstrate that an essential component of the ESCRT-
20 Here
we demonstrate that an extension of the level-crossing m
21 We demonstrate that an increase in culture temperature t
22 We demonstrate that Anr and Mhr contribute to LasR- stra
23 For the first time,
we demonstrate that anthropophagy was better explained b
24 Here,
we demonstrate that Arabidopsis (Arabidopsis thaliana) E
25 In this work
we demonstrate that aromatic compounds, dissolved in the
26 Here
we demonstrate that ATG16L1 and other ATG proteins media
27 Finally,
we demonstrate that autism risk genes are enriched in pa
28 Taken together,
we demonstrated that autophagy and Lkb1 work synergistic
29 Here
we demonstrate that autoSTOMP can efficiently label, pur
30 We demonstrate that BAIAP2L2 localization to stereocilia
31 Further,
we demonstrate that basal gene expression patterns are p
32 methodologies and virus-specific reporters,
we demonstrate that beta-coronaviruses utilize lysosomal
33 We demonstrate that bioactive lipid profiles can be read
34 Using single-molecule imaging,
we demonstrate that both condensin I and II exhibit ATP-
35 Here,
we demonstrate that BRI1 binds directly to the medium AP
36 In summary,
we demonstrate that bupropion inhibits heteromeric 5-HT(
37 Moreover,
we demonstrate that by inhibiting the interaction betwee
38 Using a reporter gene assay,
we demonstrated that C/P efficiency of CSTF2D50A was low
39 Here,
we demonstrate that carcinogens, such as aflatoxin B(1)
40 teered molecular dynamics (SMD) simulations,
we demonstrate that cbEGF domain calcium binding decreas
41 We demonstrate that CBFbeta is essential to maintain the
42 We demonstrate that cell shape in V. cholerae is regulat
43 Here,
we demonstrate that cells treated with pharmaceutical-gr
44 We demonstrate that CERBERUS has auto-ubiquitination act
45 Additionally,
we demonstrate that chABC increases TRKB phosphorylation
46 Using a custom developed force apparatus,
we demonstrate that chick dorsal root ganglion axons exh
47 We demonstrate that click-ExM is applicable in cell cult
48 On three real datasets,
we demonstrate that cLV recapitulates interactions betwe
49 We demonstrate that complex ETPs equipped with a strateg
50 Here,
we demonstrate that constitutive AKT activation in intes
51 Here,
we demonstrate that copy number variation of TdDof, a ge
52 Here,
we demonstrate that corticotrophin-releasing factor (CRF
53 We demonstrate that cTRP nanoparticles can self-assemble
54 nsmigration through endocervical epithelium,
we demonstrated that CVF samples with greater concentrat
55 We demonstrate that CYP24A1 expression is cell cycle-dep
56 Therefore,
we demonstrate that cytokinesis is implemented in a spec
57 GH variant engineered to bind mouse CD11b-I,
we demonstrate that cytolytic activity does not only req
58 We demonstrate that DarT(Mtb) -DarG(Mtb) form a function
59 Here,
we demonstrate that DCLK1-overexpressing primary human h
60 Here,
we demonstrate that deletion of beta-klotho in glutamate
61 We demonstrate that deposition both under white light il
62 Here,
we demonstrate that detection of Borrelia burgdorferi (B
63 We demonstrated that deviance-related activity is observ
64 In this work,
we demonstrate that,
due to the nonlinear ESI response,
65 Here,
we demonstrate that DXO also catalyzes the hydrolysis of
66 Further,
we demonstrated that EBOV can induce satellite cell and
67 In prior studies,
we demonstrated that electrically stimulating the residu
68 Here
we demonstrate that electron ptychography can recover th
69 Further,
we demonstrate that ELK1 is required by the BRAF-ERK1/2
70 Finally,
we demonstrate that embedding in Matrigel induces gastru
71 In this study,
we demonstrate that endogenous fumarate accumulation upr
72 structural modeling, and molecular docking,
we demonstrate that Endosidin20 (ES20) targets the catal
73 adult mice, as well as embryonic zebrafish,
we demonstrate that endothelial-specific gain of functio
74 sis of M. tuberculosis In our earlier study,
we demonstrated that ESAT-6 protein interacts with beta-
75 We demonstrate that even under extreme conditions, SARS-
76 Taken together,
we demonstrate that even without physical purification,
77 We demonstrate that exogenously applied sphingosine susp
78 Here,
we demonstrate that exonuclease 1 (EXO1) plays a key rol
79 Using genetic and chemical tools,
we demonstrated that FACT is needed to overcome replicat
80 Drosophila hematopoietic organ: lymph gland,
we demonstrate that Fatty Acid Oxidation (FAO) is essent
81 We demonstrate that FDL1 directly binds to CCAACC core m
82 We demonstrate that feedback from the autocorrelation fu
83 Furthermore,
we demonstrate that FGF signalling controls the phase an
84 We demonstrated that for both H3N8 and H3N2 viruses, tru
85 We demonstrate that force suppresses depolymerization at
86 We demonstrate that FPs specifically bind amyloid fibril
87 We demonstrate that fusion between chemoattractive neuro
88 Using p14 mutants,
we demonstrate that fusion is abrogated when binding of
89 We demonstrate that future observational and theoretical
90 We demonstrate that GAPVD1 is a substrate of CK1delta/ep
91 We demonstrate that GBF1 is present in mouse spinal cord
92 We demonstrate that genes controlling endodermal functio
93 Last,
we demonstrated that genetic induction of lipid cacostas
94 Mechanistically,
we demonstrate that glutamine deficiency regulates EMT t
95 Here
we demonstrate that glutaraldehyde cross-linking of PEGy
96 We demonstrated that Grh remains bound to mitotic chromo
97 Using a reaction-diffusion model,
we demonstrate that growth rates are inextricably couple
98 Overall,
we demonstrate that H3.3K27M and mIDH1 hijack a conserve
99 rhinal cortex-hippocampal (EC-HIPP) network,
we demonstrate that hAPP overexpression aggravates EC-Ta
100 with an a.c. supply at household frequency,
we demonstrate that hBN-SSWC is able to support an ultra
101 We demonstrate that heat-induced DV destabilization in P
102 Here,
we demonstrate that helminth-induced group 2 innate lymp
103 We demonstrate that heterogeneous/biphasic chemical reac
104 li cell function during nitrogen starvation,
we demonstrate that Hfq foci have a role in the adaptive
105 Here,
we demonstrate that high-throughput in vitro DNA binding
106 We demonstrate that HiNT outperforms existing methods on
107 We demonstrate that hippocampal ensembles encode space a
108 Here,
we demonstrate that HIV-1 Vpu downregulates Tim-3 from t
109 We demonstrate that holographic particle characterizatio
110 Here
we demonstrate that human Sox2 interacts directly with o
111 Indeed, herein
we demonstrate that I(h) is fundamental for the recruitm
112 Using real-world datasets,
we demonstrate that iDrug achieves superior performance
113 EC-specific overexpression of Phb1 (Phb1Tg),
we demonstrate that IEC-specific PHB1 combats intestinal
114 sing different subtypes of B cell neoplasms,
we demonstrate that IgCaller identifies both heavy and l
115 We demonstrate that IL-2 activates binding of AP-1 and S
116 We demonstrate that IL-4/13A and IL-4/13B are required f
117 Second,
we demonstrate that illusory responses are orientation-s
118 Finally,
we demonstrate that important covalent, i.e., spin pairi
119 ty matrix renormalization group calculations
we demonstrate that in one dimension this transition is
120 We demonstrate that in the lysolecithin demyelination mo
121 We demonstrated that in these T-ALLs and in distinct pop
122 We demonstrated that in vitro transduction of normal hum
123 Here
we demonstrate that,
in regions of high exchange energy
124 We demonstrate that increased ICP can contribute to meni
125 Specifically,
we demonstrate that increasing extracellular amino acids
126 Herein,
we demonstrate that inhibition of tissue factor (TF) and
127 Previously,
we demonstrated that integrin alpha5beta1 and Fn1 (fibro
128 Moreover,
we demonstrated that IPO4 and CEBPD knockdown improved C
129 Furthermore,
we demonstrate that IRS4(PVH) neurons lie within a compl
130 We demonstrate that Kpi contributes positively to the ab
131 We demonstrated that LFP powers in the tectofugal pathwa
132 We demonstrate that light-induced transient gene express
133 We demonstrated that light attenuation by RPE melanin ca
134 Furthermore,
we demonstrated that LOB was correlated with low-frequen
135 We demonstrate that local adaptation can alter everythin
136 Finally,
we demonstrate that loss of heterozygosity and temporal
137 Here,
we demonstrate that LSH is enriched at meiotic kinetocho
138 gen-specific" from "bystander" reactivation,
we demonstrate that lung CD8+ TRM cells are reactivated
139 We demonstrate that lytic MHV68 infection of B cells, ma
140 ing human metapneumovirus (HMPV) as a model,
we demonstrate that m(6)A serves as a molecular marker f
141 Using a murine model of nHSV,
we demonstrated that maternal immunization with the triv
142 In this study,
we demonstrate that MDA5/MAVS signaling was essential fo
143 In this Article,
we demonstrate that mid-infrared spectroscopic imaging m
144 We demonstrated that MKP5 was required for TGF-beta1 sig
145 tudy, and the analyses of 64 drug responses,
we demonstrate that MKpLMM consistently outperforms comp
146 We demonstrate that MTR4 is frequently overexpressed in
147 We demonstrate that multiomics data analysis is a powerf
148 Here,
we demonstrate that Myoglianin (Myo), an Activin family
149 r endothelial and alveolar epithelial cells,
we demonstrated that N-WASP downregulation attenuated P
150 n a canine, rapid atrial pacing model of AF,
we demonstrate that NADPH oxidase 2 (NOX2) generated oxi
151 We demonstrated that negative regulation of CCL2 product
152 Here,
we demonstrate that NK cells (haNKs) engineered to expre
153 Finally,
we demonstrated that Nodal and Wnt/beta-catenin signalin
154 We demonstrate that Notch activation in human tonsil-der
155 ntroducing O-glycosylation sites to ST6GAL1,
we demonstrated that O-glycan's effect on Golgi export i
156 sis of non-fixed pancreatic islet microscopy
we demonstrated that ODND probes may be used to distingu
157 We demonstrate that of the 19 currently available biomar
158 We demonstrated that only a fraction of expanded gamma9d
159 body drug substance with low levels of HCPs,
we demonstrated that our method could detect HCP with lo
160 We demonstrated that ovariectomized female FBN-ARO-KO mi
161 We demonstrate that overexpression of SEMA4C promotes pr
162 We demonstrate that P8 affects the proteolytic processin
163 Here,
we demonstrate that Parkin has functions in the nucleus
164 ced data from the Amazon Kindle application,
we demonstrate that passages that people highlighted-col
165 eraction between basal ganglia and thalamus,
we demonstrated that patients with current history prese
166 Additionally,
we demonstrate that PDGFRbeta-initiated phosphorylation
167 Using cell-specific markers,
we demonstrate that pericytes rather than endothelial ce
168 In this study,
we demonstrate that pharmacological manipulations that i
169 Using this information,
we demonstrate that pharmacologically inhibiting necropt
170 Here
we demonstrate that phenformin suppresses tumor growth a
171 We demonstrate that phosphine oxides are highly polar fu
172 We demonstrate that plant traits can predict mean annual
173 We demonstrate that plasmanylethanolamine desaturase def
174 Here,
we demonstrate that PM induces Acod1 and itaconate, whic
175 We demonstrate that PMX is a master modulator of merozoi
176 Using inducible cMYC overexpression,
we demonstrate that post-pregnancy MECs are resistant to
177 ands for the nuclear receptor PPARalpha, and
we demonstrate that Ppara (-/-) mice are less susceptibl
178 Here
we demonstrate that precise control of the heteroepitaxy
179 Here
we demonstrate that protein arginine methyltransferase 5
180 Here
we demonstrate that PUFA analogues vary in their selecti
181 We demonstrate that quantification based on microcystin-
182 We demonstrate that reconstitution of VRCs on GUVs with
183 Here,
we demonstrate that recruitment of the NF-kappaB factor
184 Using a novel experimental approach,
we demonstrate that reducing pulmonary arterial pressure
185 Using a model of binge-eating,
we demonstrated that relaxin-3/RXFP3 signaling in the hy
186 We demonstrate that Rep and Rep-X (an enhanced version o
187 Here
we demonstrate that reproductive potential does not caus
188 Here,
we demonstrate that RNA labeling with the modified, nont
189 For the first time,
we demonstrate that RocA directly interacts with itself
190 We demonstrate that sealed UV-C flow reactors operating
191 Here
we demonstrate that sequential hypothesis testing on the
192 We demonstrate that signatures from discrete subpopulati
193 As a proof of principle,
we demonstrated that SimCells can be used as a safe agen
194 Here,
we demonstrate that similar approaches can be used to fo
195 We demonstrate that single high dose TAI-FLASH produced
196 We demonstrate that SMARCB1 regulates both replication a
197 Additionally,
we demonstrate that some coincidental and lagging indica
198 We demonstrate that some conjugates act as dNTP analogue
199 Further,
we demonstrate that Sox2 3'UTR AREs are necessary for RB
200 We demonstrated that species inhabiting dynamic systems
201 We demonstrate that spindle checkpoint genes act upstrea
202 We demonstrated that substitutions I38L/M/S/T not only h
203 We demonstrate that SurA samples an array of conformatio
204 Finally, using intermolecular FRET analysis,
we demonstrate that SWG directly binds to the lipid-bind
205 Using inducible genetic switches,
we demonstrate that swimming motility can be manipulated
206 Furthermore,
we demonstrated that SYIPSAEKI-specific CD8(+) T cell re
207 Here
we demonstrate that systemic autophagy inhibition induce
208 We demonstrate that TAD boundaries shared among multiple
209 We demonstrate that targeted delivery of the LRRC31 gene
210 Finally,
we demonstrate that targeted mutations can be introduced
211 We demonstrate that TE-derived CTCF binding divergence m
212 We demonstrate that TETRAC promotes PPARgamma/RXR signal
213 We demonstrate that the 33 amino acids of chicken ANP32A
214 Here,
we demonstrate that the A-site substituent in yttrium ru
215 We demonstrate that the active isomer of JQ1 but not its
216 We demonstrate that the ADR1 and NRG1 families act in an
217 ough the use of in vitro biochemical assays,
we demonstrate that the amide backbone of CTA is assembl
218 Here,
we demonstrate that the analysis of the resulting FLIM o
219 We demonstrate that the approach allows drastic reductio
220 Here,
we demonstrate that the autocrine FGF/FGFR axis is essen
221 eukaryotes is linked with larger genomes and
we demonstrate that the benefit of LGT declines rapidly
222 We demonstrate that the biomarkers do not show significa
223 In this report,
we demonstrate that the CCCTC-binding factor (CTCF), a c
224 Using Cbf1p and MAX,
we demonstrate that the CCRA method is able to detect sm
225 genetic, cellular and molecular approaches,
we demonstrate that the crosstalk between Target of Rapa
226 For the first time,
we demonstrate that the CV of the outer loop, rather tha
227 d using a PTPN14 knockout rescue experiment,
we demonstrate that the degradation of PTPN14 is require
228 Here,
we demonstrate that the deubiquitinase Cyld prevents exc
229 We demonstrate that the direct-space strategy for struct
230 Second,
we demonstrate that the direction and amplitude of chang
231 of the rabbit infective endocarditis model,
we demonstrate that the disulfide bond is a critical reg
232 Here,
we demonstrate that the dominant mechanism for H(2) prod
233 to theories of bottlebrush polymer behavior,
we demonstrate that the experimental values of internal
234 We demonstrate that the gate conformation of the two opp
235 Here,
we demonstrate that the GDF15 receptor, GFRAL, is locate
236 By analyzing the male germline of mice,
we demonstrate that the genome-wide reorganization of su
237 Here,
we demonstrate that the hemagglutinins of LPAIV strains
238 ith reversed, pseudo-palindromic extensions,
we demonstrate that the high stability of FimA polymers
239 We demonstrate that the highly identifiable shape of dim
240 Finally,
we demonstrate that the in vivo function of human-specif
241 We demonstrate that the information provided by HDX-MS e
242 Furthermore,
we demonstrate that the integrity of the CCHC motifs in
243 We demonstrate that the intestinal microbiome contribute
244 Using site-directed mutagenesis,
we demonstrate that the M159I substitution extends the t
245 Finally,
we demonstrate that the mammalian Expi293F amino acid la
246 Here,
we demonstrate that the MAPK signaling pathway is a gene
247 Here,
we demonstrate that the Mcl-1 TMD forms homooligomers in
248 We demonstrate that the N terminus of CTCF interacts wit
249 Here,
we demonstrate that the NRF2 antioxidant gene expression
250 Here,
we demonstrate that the octopamine-Octbeta1R-cAMP pathwa
251 We demonstrate that the order of driver events in colore
252 -ray absorption near-edge structure (XANES),
we demonstrate that the photocycle of ferric Cyt c is en
253 We demonstrate that the plasticity rules depend highly o
254 ntal stage-specific and, for the first time,
we demonstrate that the premature termination of host ge
255 Along with spectroscopic characterization,
we demonstrate that the probe improves quality and condi
256 In this work,
we demonstrate that the product, in fact, is formed via
257 xt, with current data submission statistics,
we demonstrate that the proteomics field is now actively
258 Here,
we demonstrate that the RarA protein makes a major enzym
259 Here,
we demonstrate that the Ras->Raf->rho kinase (ROCK) path
260 igh-end spectroscopic and microscopic tools,
we demonstrate that the reduction proceeds first through
261 Importantly,
we demonstrate that the RNA mutation-based APOBEC3A assa
262 Here
we demonstrate that the roles of the Saccharomyces cerev
263 Next,
we demonstrate that the same pessimistic model but with
264 using structure-based computational models,
we demonstrate that the SARS-CoV-2 spike (S) glycoprotei
265 We demonstrate that the scoliosis in dstyk mutants is re
266 In this study,
we demonstrate that the shedding of syndecan-1 by MMP-3
267 l settling from surface accumulations; here,
we demonstrate that the spatial distribution and ultimat
268 t replicates and both inversion breakpoints,
we demonstrate that the technique is highly accurate and
269 ctron microscopic, and in silico techniques,
we demonstrate that the two peptides, even if applied in
270 Presently
we demonstrate that the V2-specific Ab response from thi
271 In this study,
we demonstrate that the viral protein pUL47 is an essent
272 Here,
we demonstrated that the cytosolic DNA sensor cGAS recog
273 In this study,
we demonstrated that the developed CPC method fractionat
274 a murine model of elastase-induced emphysema
we demonstrated that the most potent agents exhibited a
275 We demonstrated that the precision of normalized MC sign
276 We demonstrated that the T367K mutation attenuates TMUV
277 data from patients with focal brain damage,
we demonstrate that there is a strong psychometric corre
278 We demonstrated that there is a significant relationship
279 We demonstrate that these structural changes have a cons
280 We demonstrated that these patterns enable the construct
281 Most importantly,
we demonstrate that this biphonation is a phenomenon ari
282 We demonstrate that this configuration is a structural s
283 We demonstrate that this mapping fundamentally shapes th
284 We demonstrate that this method is suited to both 3D str
285 We demonstrate that this molecular design principle, i.e
286 on uncertainty and change-of-mind behaviour,
we demonstrate that this phenomenon is associated with t
287 Here,
we demonstrate that three Drosophila opsins, Rh1, Rh4, a
288 We demonstrate that,
through ATP-dependent RNA binding,
289 Using fluorescence vesicle leakage assays,
we demonstrate that TMD2 forms stable holes with an esti
290 Here,
we demonstrate that transcription factors GLK1 and GLK2
291 We demonstrate that transfer of blastocysts naturally co
292 We demonstrate that ULM microbubble data processing meth
293 We demonstrate that under conditions of increased chromo
294 We demonstrate that using cGMP-specific antibody, sGC or
295 We demonstrate that vPAR-CL can readily and reliably ide
296 In the present study,
we demonstrate that VtlR is involved in the ability of A
297 We demonstrated that WDR77 regulated the translation of
298 We demonstrate that Wnt3 spreads extracellularly and int
299 el and selection for G418 resistant genomes,
we demonstrated that Y138 is a critical residue for Brd4
300 NA immunoprecipitation and RNA decay assays,
we demonstrate that ZAP directly and specifically binds