1 We proposed that 1 first reacts with X in a fast equilib
2 We propose that 53BP1 has evolved to avoid mutagenic rep
3 We propose that a broader framework is needed to account
4 Here,
we propose that a combination therapy of current antigli
5 We propose that a complete understanding of how ageing a
6 We propose that a failure in iron protein maturation lea
7 omain boundaries in ferroelectric materials,
we propose that a ferroelectric polaron localizes to pla
8 We propose that a Global Immunological Observatory and a
9 Here
we propose that a normalization mechanism that operates
10 Thus,
we propose that aberrant expression of fusogens in the n
11 From these results,
we propose that ability of an amine to form multiple int
12 We propose that abundant, RSC-like stem cells exist in o
13 We propose that AC9 dually regulates ERK7 by scaffolding
14 We propose that accounting for polygenic background is l
15 We propose that accumulation of ssDNA in the lagging-str
16 We propose that acetylation is involved in regulating of
17 We propose that additional sex-dependent co-morbidities,
18 Thus,
we propose that adiponectin, or its downstream pathway,
19 To account for these findings
we propose that,
after establishment of primary RdDM as
20 We propose that AHR is likely protective based on these
21 We propose that an action-based developmental account ca
22 We propose that an increase in the incidence of sudden s
23 We propose that applying these key rules of life to cryo
24 We propose that approaches that seek to maintain mitocho
25 We propose that as myonuclear numbers increase, the rang
26 We propose that,
as well as generally acting to ensure f
27 We propose that ASAP1 is a hub protein for dynamic prote
28 We propose that AT-rich centromeres drive karyotype dive
29 We propose that BAIAP2L2 is a new key protein required f
30 We propose that BAIAP2L2 is key to maintenance of the no
31 We propose that basin-impact-generated hurricane-force w
32 omputational properties of the motor system,
we propose that beat anticipation relies on action-like
33 We propose that behavioral tagging with novel virtual en
34 Hence,
we propose that BMP2K-L and BMP2K-S differentially regul
35 We propose that body weight sensing constitutes a feedba
36 nsertion occurred millions of years ago, and
we propose that both alleles have been maintained in the
37 gg3 proteins are transcriptional repressors,
we propose that both are capable of transcriptional acti
38 On the basis of these findings,
we propose that both pH and redox environments regulate
39 Thus,
we propose that both proteins are necessary for retinal
40 We propose that C-C bond coupling in Y-DeAlBEA proceeds
41 We propose that cargo recruitment into vesicles creates
42 We propose that CaSR-mediated NLRP3 inflammasome activat
43 We propose that Cdkl5 is a stress-responsive kinase that
44 We propose that cells control RNA condensation through A
45 We propose that cellular immune responses reduce the thr
46 We propose that certain molecularly defined pathways wit
47 Together,
we propose that certain tumor damage(s) occurring during
48 We propose that CLas uses D. citri nymphs mainly for pat
49 We propose that Cnr is an unstable gene that is mutated
50 We propose that cohesin occasionally bypasses boundaries
51 We propose that combined with established roles of parie
52 We propose that combining specific SIRT6 amino acid subs
53 We propose that communication between the actin- and nuc
54 To address this issue,
we propose that communities of 'rigor champions' be esta
55 Therefore,
we propose that complex III is central for MRC maturatio
56 We propose that crystalline guanine is the elusive N dep
57 Herein,
we propose that CTGF is a promising cancer therapeutic t
58 We propose that cytoplasmic cilia assembly requires the
59 We propose that DCL5 and 24-nt phasiRNAs are critical fo
60 We propose that DDX11 is a DNA helicase protecting again
61 We propose that delta-catenin regulates the dendritic ar
62 We propose that developmental cognitive science should i
63 We propose that dietary influences on protein synthesis
64 Here
we propose that displacement of carbon-enriched Tanzania
65 pression patterns, not protein sequence, and
we propose that distinct LIN-29 dose sensitivities of th
66 We propose that DNA damage-elicited double-strand DNA br
67 We propose that Dna2 fulfils its essential function by p
68 We propose that DNMT1 RFTS mutations deregulate metaboli
69 On the basis of these findings,
we propose that DRs functions like a bracket holding the
70 We propose that dual-initiation on shared promoters repr
71 We propose that during TRAP, assembled ribosomes associa
72 We propose that dynamic regulation of ion transport and
73 Thus,
we propose that DYRK1A controls cyclin L2 expression, le
74 lifespan on pathogen spread in a population,
we propose that epidemics drive lifespan setpoints' evol
75 As an alternative to ideological diversity,
we propose that epidemiologists take up an existing fram
76 Focusing on a group of ABCG transporters,
we propose that exaptation by regulatory divergence cont
77 We propose that Exo1 serves as a central coordinator in
78 Collectively,
we propose that EXOSC10 promotes normal growth-to-matura
79 We propose that exposure to decreased pH combined with h
80 We propose that exposure to microbial amyloids in the ga
81 We propose that FOXO1 inhibitors may have potential as c
82 We propose that functional perturbation of male microgli
83 We propose that further research be undertaken to determ
84 We propose that G protein-biased mu opioid receptor agon
85 Here
we propose that Gestalt theory may explain why rodent is
86 s genome-wide would obligate H2A.Z turnover,
we propose that global transcription at yeast promoters
87 We propose that glutamate activates TRESK through NMDA a
88 pathways can function in multiple modes and
we propose that GRN changes that lead to switches betwee
89 We propose that GTPBP5 primarily fuels proper mtLSU matu
90 We propose that guanidinium reactivates a latent septin
91 We propose that H1N1 virus replication in neuronal cells
92 We propose that hand control occupies a higher dimension
93 We propose that harnessing mast cells' host defense and
94 In this opinion article,
we propose that highly conserved DNA methyltransferases
95 We propose that hPSCs represent a powerful tool to model
96 To explain our data,
we propose that,
in each cycle of ATP turnover, forward
97 We propose that increased tumor microenvironment stiffne
98 Taken together,
we propose that individual intestinal IF polypeptides co
99 We propose that indole spatially segregates cells based
100 We propose that injecting at the intersection point of a
101 We propose that Inpp5e attenuates Shh signaling in the n
102 We propose that inside-out regulation of protein exchang
103 er lifetime when CLIP-170 is phosphorylated,
we propose that instead of acting at the time of rescue
104 We proposed that insufficient melatonin levels impair mi
105 Here,
we propose that interactions with other people can influ
106 We propose that interfering with these pathways would mo
107 We propose that interrupting the POOR-get-POORer progres
108 We propose that interventions aimed at modifying the str
109 We propose that intrinsic properties of the filament-bin
110 We propose that INVs are a generic class of transport ve
111 istry reconstructed from the FeNi(Cu) alloy,
we propose that it formed by decomposition of a complex
112 Finally,
we propose that it is possible to titrate the amount of
113 te shuttling occurs at a high frequency, and
we propose that it serves as a proofreading mechanism to
114 We propose that KIN-29/SIK acts in nuclei of sensory neu
115 We propose that L4 restricts disinhibition and gates OD
116 We propose that Lis1 binding releases dynein from its au
117 We propose that local ROS production can activate astroc
118 We propose that maize Rtn1 and Rtn2 act as receptors for
119 We propose that manipulating CEP signalling strength or
120 We propose that mechano-responsive transcription factors
121 Accordingly,
we propose that membrane budding, rather than proplatele
122 We propose that metabolic subphenotypes exist between CR
123 We propose that modulation of CMU protein levels and mic
124 We propose that motifs of the scaffold protein IKKgamma/
125 We propose that mouse nighttime sleep, analogous to the
126 urces of these eruptions remain unknown, but
we propose that Mt.
127 We propose that MTH1 acts in concert with adenosine deam
128 We propose that multiple PCNA-interaction motifs embedde
129 Thus,
we propose that mutations in centrosome genes cause micr
130 We propose that Myc-regulated fatty acid synthesis is a
131 We propose that Myo is a myokine that helps mediate an a
132 We propose that nestin changes growth cone behavior by r
133 We propose that nitrile rubber gloves should be produced
134 Here
we propose that nonnative plants introduced to Great Bri
135 We proposed that normalization of TME using antiangiogen
136 We propose that nSH3/cSH3 binding peptides, which effect
137 We propose that NUP153 links the nuclear pore complex (N
138 We propose that oncohistones inhibit the H3K27 methyltra
139 We propose that one concrete way to monitor and redress
140 We propose that other repair and tolerance mechanisms op
141 We propose that P. falciparum virulence in areas of seas
142 Thus,
we propose that parasite rhythms are generated by the pa
143 We propose that PbYOP1's disruption may lead to defects
144 We propose that persistence of the overlying epithelial
145 Here,
we propose that phagocytosis is not merely passive corps
146 Although challenges remain,
we propose that phase 0 approaches be at least considere
147 We propose that PKD2-mastigoneme arrays, on opposing sid
148 We propose that Pop-mediated stabilization of Est1 bindi
149 We propose that PRC2-mediated silencing of enhancers inv
150 Finally,
we propose that previous conservation units should be mo
151 We propose that prophase cohesin removal is a key step i
152 We propose that pulse-chase SILAC labeling is a useful t
153 We propose that RAGE is involved in modulating blood coa
154 We propose that rapid local structural fluctuations caus
155 Here,
we propose that redox shifts can also arise from small p
156 We propose that redox vulnerabilities could be exploited
157 We propose that regeneration of a mucociliated epitheliu
158 We proposed that reinstatement of respiratory sinus arrh
159 We propose that renal accumulation of pro-oxidant elemen
160 Thus,
we propose that repetitive IgH switch regions represent
161 We propose that representations with common geometric st
162 We propose that research on the specific receptors for s
163 We propose that reversal of SERCA-PLB inhibition is achi
164 Together,
we propose that ribosome hibernation is a specific and c
165 We propose that ring currents in organic semiconductors,
166 Finally,
we propose that RNA ligand discovery can benefit from us
167 We propose that SAMHD1-mediated dNTP balance regulates d
168 We propose that secondary structure of miRNA precursors
169 We propose that sex differences in immunopathogenesis wi
170 Based on these observations,
we propose that shade avoidance is regulated by a three-
171 We propose that similar mechanisms are likely to be used
172 As a result of these findings,
we propose that SinI utilizes a residue replacement mech
173 We propose that SlGBP1 acts as an inhibitor of cell divi
174 Thus, from our study,
we propose that SM/Cer and SMPD1 are new potential targe
175 We propose that social resistance can act as an agent of
176 We propose that SPEN acts as a molecular integrator for
177 We propose that SPO11-RI forms because chromatin-bound P
178 We propose that striated muscle force is generated by a
179 We propose that substantial UDB contributes to the obser
180 We propose that such sequential electron transfer in oli
181 We propose that sugar-binding pockets spatially closer t
182 Furthermore,
we propose that T-channel blockers may be further explor
183 We propose that tandem LIM domains recognize an F-actin
184 We propose that task-relevant neurons strengthen while t
185 We propose that TERT-alt analysis should be implemented
186 We propose that TGFbeta/SMAD3 inhibition may represent a
187 In this manuscript,
we propose that the 99-aa C-terminal fragment of APP (C9
188 Here
we propose that the acclimation of R(d) follows an optim
189 We propose that the allosteric mechanism of nickel-activ
190 We propose that the ancestral pterosaur diet was dominat
191 In this perspective,
we propose that the application of smarter, more actiona
192 Using mathematical modelling,
we propose that the BotC network organization and its in
193 We propose that the brain has an identifiable set of sta
194 We propose that the C8D1A cell line could be used to dec
195 Therefore,
we propose that the cGAS-STING pathway senses unnatural
196 Thus,
we propose that the CIP participates in a checkpoint, ca
197 We propose that the combined loss-of-function of both re
198 We propose that the complexity of cortical circuits is g
199 We propose that the cross-editing activity of ThrRS is e
200 Here,
we propose that the crosstalk between neurotransmission
201 s, and high levels of sequence conservation,
we propose that the delta-HXTXs were repurposed from an
202 We propose that the dendritic architecture of pyramidal
203 Taken together,
we propose that the DHCR7 inhibitors and 7-DHC are poten
204 From these results,
we propose that the differential abilities of posterior
205 In particular,
we propose that the endosome may be more important than
206 We propose that the engagement of these homeostatic feed
207 We propose that the EPFL2 signaling module evolved to co
208 We propose that the ESR serves two purposes in aneuploid
209 We propose that the extension domain acts as a conformat
210 We propose that the growth rate of GA area is directly p
211 We propose that the immunophenotype and molecular compos
212 We propose that the induction of ER constrictions contri
213 We propose that the initial genetic polymers were random
214 We propose that the interaction between misfolded SOD1 a
215 We propose that the intrinsic geometric heterogeneity of
216 We propose that the lack of chlorophyll exerts growth co
217 We propose that the length and complexity of many long-r
218 We propose that the LrS attenuates proinflammatory media
219 Specifically,
we propose that the lysosomotropic effects of hydroxychl
220 We propose that the molecular machinery for dopamine sec
221 developmental dynamics of brown adipocytes,
we propose that the murine iBAT has two different growth
222 We propose that the N >= 9 carotenoids found in light-ha
223 coupled with a computational investigation,
we propose that the new pathway entails hydride transfer
224 We propose that the observed epigenetic alterations refl
225 We propose that the opposing FBF-1 and FBF-2 activities
226 We propose that the origin and optimisation of this diff
227 Thus,
we propose that the persistent PARP1 foci are formed by
228 We propose that the pig influenza model will be useful f
229 We propose that the PM-anchored Rsp5/Rcr1 ubiquitin liga
230 icrobial signatures and within this context,
we propose that the protection and offshore nature of Ja
231 We propose that the reduction in GPP/SIF with decreasing
232 We propose that the release of axial respiratory constra
233 very good overall agreement between species,
we propose that the RNA-binding characteristics we obser
234 We propose that the role of land management practices-fr
235 We propose that the SBS-A1 site is unique to alternansuc
236 wined right- and left-handed single gyroids,
we propose that the simultaneous presence of both R- and
237 Here
we propose that the size of an organism' brain could be
238 We propose that the slower and rate-invariant speed of d
239 We propose that the solution to this paradox is the cogn
240 We propose that the song system motor code must compensa
241 We propose that the specific suppression of dynamic micr
242 We propose that the spectrum is due to the evolution fro
243 We propose that the synergistic use of the techniques di
244 We propose that the temporal flexibility of a GRN is a g
245 Based on these findings,
we propose that the threshold to find a particular level
246 As such,
we propose that the use of additives (anionic or cationi
247 We propose that the ventral circuit defines behavioral g
248 If such thymuses could be identified,
we propose that their use would offer a compelling appro
249 We propose that therapeutic and delivery strategies that
250 We propose that therapeutic approaches that target infla
251 We propose that these adaptations of LDs support IMTG st
252 We propose that these behaviours could be due to the inc
253 We propose that these changes in neural synchronization
254 We propose that these communities were resilient to clim
255 We propose that these findings have important implicatio
256 We propose that these mechanisms improve cellular and or
257 We propose that these roles for both Mpp5a and Rab11a op
258 We propose that these strong H-bonds serve to stabilize
259 We propose that these trait combinations should be prior
260 We propose that these two perceptual pathways can be ind
261 We propose that these variants render the eIF2B complex
262 We propose that this actin-based impaired relaxation is
263 We propose that this activity would protect the cell fro
264 We propose that this compensatory mechanism, together wi
265 We propose that this complex functions to detach newly s
266 We propose that this event was central to the evolution
267 We propose that this finding should be taken into accoun
268 We propose that this gp41-cholesterol interaction mediat
269 We propose that this inhibitory mechanism must differ qu
270 We propose that this is due to a local vascular problem
271 We propose that this knowledge can inform studies in hig
272 We propose that this M6PR pathway is most utilized in VZ
273 We propose that this molecular switch acts to drive prog
274 We propose that this oversight may be associated with th
275 We propose that this paradigm will allow researchers to
276 We propose that this phenomenon is a diagnostic signatur
277 We propose that this plaque-independent inflammatory rea
278 We propose that this process of neurodegeneration ensues
279 We propose that this reorganization in descending contro
280 We propose that this replacement is linked to the transi
281 We propose that this run is instrumental for bringing th
282 We propose that this thick crust represents a major part
283 We propose that this use of genetic marking for microana
284 icient for TIMP-1's MMP-inhibitory activity,
we propose that those C-terminal amino acid residues are
285 We propose that to understand causes of sex-biased morta
286 We propose that transcriptional regulation incorporates
287 We propose that transfer functions can be considered as
288 We propose that TTC-B mediates NusG- and NusA-dependent
289 Here,
we propose that turbulent coherent structures, long-last
290 We propose that turbulent mixing of the seamount sheath-
291 Taking together,
we proposed that two tonoplast proton pumps are required
292 We propose that U21 employs the novel mechanism of formi
293 We propose that unintegrated HIV-1 DNA adopts a repressi
294 We propose that,
upon telomere shortening, early apoptos
295 We propose that USP(4207) sequesters KATms from diverse
296 lps explain asymmetries in blame and praise:
we propose that while blame is primarily for punishment
297 Thus,
we propose that while CP-delta likely mediates host cell
298 We propose that working representations of speech catego
299 We propose that WOX5 acts mainly in the QC, where other
300 In view of these new findings,
we propose that ZI serves as an integrative node for glo