ライフサイエンスコーパス: we think

1 lfactory results, however, may not mean what we think.

2 may not always be as benign or beneficial as we think.

3 ract by appealing to basic principles of how we think.

4 more common in plants and also animals than we thought.

5 ave even more far-reaching consequences than we thought.

6 Or so we thought.

7 etastable liquid may be more ubiquitous than we thought.

8 In naming population groups, we think a chief aim is to use terms that the group memb

9 lting structures have revolutionized the way we think about aberrant protein aggregation and propagat

10 eism and as a catalyst for change in the way we think about aging per se and late-life mental disorde

11 le forms suggests that we need to adjust how we think about animal mating systems and the evolution o

12 Falkow's many contributions remade the way we think about bacterial pathogens, antibiotic resistanc

13 ft interfaces could profoundly influence how we think about biomaterials.

14 ecognizing this invites us to change the way we think about both the threats and promises of the comi

15 inhibitors (ICIs) is revolutionizing the way we think about cancer treatment.

16 ancestry has the potential to shape the way we think about developmental systems and oncology.

17 of medicine and has forever changed the way we think about disease.

18 gy have begun to dramatically change the way we think about evolution, development, health and diseas

19 What determines what we think about from one moment to the next?

20 These findings expand on the way we think about gene regulation and introduce new candida

21 died in real time, dramatically changing how we think about gene regulation on chromatin templates.

22 ealth law has transcending importance in how we think about government, politics, and policy.

23 This sensory-based inference reshapes how we think about hierarchy learning, stability, and behavi

24 gating, and pharmacology has changed the way we think about iGluR function.

25 this new understanding could add to the way we think about immunologic memory, vaccine development,

26 ction call for some modifications in the way we think about innate immunity strategies.

27 Thus, our studies, revise how we think about iron import and export from the retina.

28 the efferocyte is markedly changing the way we think about macrophage immunometabolism.

29 genetic versus microbial) may change the way we think about neurological disorders and how to treat t

30 How should we think about offering payment to participants in these

31 nd metamaterials have revolutionized the way we think about optical space (varepsilon,mu), enabling u

32 s, if any, the brain spontaneously uses when we think about others.

33 ural reuse has profound implications for how we think about our continuity with other species, for ho

34 eeping imposed on us probably influences how we think about patients.

35 tudies, there has been a major change in how we think about perioperative management of anticoagulati

36 This revolution in how we think about phenotypes will require a radical change

37 to highlight some of the advances in the way we think about rhabdomyosarcoma (RMS).

38 the ways these techniques have reshaped how we think about RNA structure.

39 ese data add critical constraints to the way we think about saccade generation in the brain.

40 he potential to revise significantly the way we think about seed plant evolution, especially with reg

41 f sexual selection fundamentally changed how we think about sex and evolution.

42 ention, argues for a major change in the way we think about suicide and its prevention.

43 d techniques that challenges notions for how we think about T-cell receptor signaling.

44 um complex, which profoundly changes the way we think about the assembly, maintenance, and remodeling

45 ve represents a significant shift in the way we think about the cellular bases of perception.

46 uss the implications of our findings for how we think about the current communication environment, ex

47 nd type 2 diabetes is fundamental to the way we think about the disease.

48 al, it could mark a turning point in the way we think about the evolution of the genome.

49 ics of eukaryotic microbial life changes how we think about the flow of genetic information and the e

50 e influences how we interpret the world, how we think about the future, and how we perceive ourselves

51 r intended arm movements has changed the way we think about the order of operations in the sensory to

52 to a wealth of information that changes how we think about the pathogenesis of immune-mediated disea

53 onal autophagic mechanisms have reframed how we think about the role of local regulation of autophagy

54 behaving animals are revolutionizing the way we think about the role of Purkinje cells in sensori-mot

55 there has been a dramatic change in the way we think about the role of vascular smooth muscle cells

56 g studies have fundamentally changed the way we think about the vegetative and minimally conscious st

57 dea that prokaryotes may not form species as we think about them for plants and animals.

58 how this new information is changing the way we think about this fatal disorder.

59 How do we think about time?

60 slational failures that have changed the way we think about tissue damage after stroke.

61 cellular and system level and challenges how we think about vaccine protection against these infectio

62 uencing technologies have revolutionized how we think about viruses.

63 s are fundamental to the way that, nowadays, we think about vision, somato-sensory function, the spin

64 What we think actually happens is that there is an effective

65 However, we think additional mechanisms are important when people

66 es when trying to understand and predict how we think and behave.

67 They provide a link between how we think and feel and the function of the adrenal medull

68 signals interact centrally to influence how we think and feel, generating our sense of the internal

69 thesis about early-emerging conception - how we think and reason about the world - here we present an

70 ian hypothesis, that language influences how we think; and that the "language of thought" maps to spo

71 and so here we restrict our focus to matters we think are of broadest interest.

72 We also pose questions whose answers we think are pivotal to understanding this path, and we

73 ill not be the solution to the problems that we thought", but "5G is still developing".

74 our surprise, the PDZ and FAB domains, which we thought connected junctions and F-actin, are not requ

75 We highlight 3 key reasons why we think epidemiologists should be engaging with these m

76 nts with systemic lupus erythematosus (SLE), we thought F4+ IgG antibodies might constitute a useful

77 We think future research will require consistent approac

78 e opine on topics and lines of investigation we think hold promise for expanding our mechanistic unde

79 be decreased to a remarkable 2 mol % in what we think is a general approach.

80 of a min slmA double deletion mutant, which we think is due to the elimination of polar Z rings (or

81 By reviewing what we have learned and what we think is going on during development, we hope to lure

82 swimming are similar processes, a comparison we think is helpful in understanding how cells migrate.

83 echanisms of INH resistance and propose what we think is the means by which INH kills M. tuberculosis

84 thorough details on the subject at hand, and we think it can serve as a guide for further investigati

85 Specifically, we think it is important to challenge assumptions that c

86 Because of these, we think it is premature to strongly advocate for such a

87 lower limbs associated with weight loss, and we think it is under-recognized; (ii) recovery from the

88 Thus, we think it likely that arrest in both mre11-1 and the c

89 Instead, we think it likely that both pathways contribute to esta

90 As unlikely as we think it might be as the result of natural selection,

91 ations in outer membrane protein biogenesis, we think it possible that Spy may be involved in this pr

92 ciated specific standards for the process as we think it should be practiced.

93 of AVATAR therapy and, if proven effective, we think it should become an option in the psychological

94 ce on Broca's area as a major speech centre, we thought it important to re-inspect these brains to de

95 to show significant levels of neurogenesis, we thought it likely that BLBP expression would also be

96 course emphasized real-world data analysis, we thought it would be an appropriate setting to carry o

97 s functionally independent from what and how we think, know, desire, act, and so forth".

98 We think Lemon will become a one-stop-shop to quickly mi

99 delivery as the field we know the best; but we think many of the points are relevant in many fields.

100 ncept of rejuvenation or regeneration, which we think may impact future development in the field.

101 We put forth a series of questions that we think need to be investigated if the conservation res

102 nvironment works, as distinguished from what we think of as (e) imitation (the copying of the demonst

103 When we think of extremophiles, organisms adapted to extreme

104 points to the power of language to shape how we think of gender in the professional domain.

105 or FeCl(3) that may qualitatively change how we think of Lewis acid activation.

106 While we think of neurons as having a fixed identity, many sho

107 Typically, we think of policymakers looking to scientists for advic

108 We think of transcription factors as being confined to t

109 ty to separate private states of mind - what we think or believe - from public actions - what we say

110 We explore meta-perceptions (i.e., what we think others think about reality), their impact on in

111 We think our findings could potentially aid therapeutic

112 This is presumably not because we think parents view their children as worthy cooperati

113 We end with an example of how we think plasticity may play out in stickleback life his

114 ther, we wrote this perspective to share how we think predictive models can be integrated into medici

115 results obtained from studying humans, whom we think provide an underutilized, yet critical, animal

116 imilation and highlight several aspects that we think require further quantitative studies, including

117 irus disease 2019 (COVID-19) and discuss why we think that a clinical trial of a drug in this class c

118 O2 uptake and photosynthetic plant activity, we think that a minimum CO2 mixing ratio might be needed

119 We think that a subset of patients with asymptomatic bra

120 Making conservative assumptions, we think that about 100000 excess deaths, or more have h

121 We think that all patients with advanced-stage carcinoid

122 We think that an abrupt change in the adapted state of t

123 We think that an algorithmic approach will result in a l

124 We think that DWMR have important role due to quantitati

125 We think that exposures to hazardous substances migratin

126 on of rationalization as a "useful fiction," we think that Fiery Cushman's claim remains ambiguous in

127 We think that Flex-M coil can easily be used in routine

128 fforts to lower cholesterol should continue, we think that important biology may be reflected in the

129 We think that improvements in technology and workflow st

130 ncertainty that we have about these effects, we think that it is helpful for the NIH to engage in ope

131 ind a unified evolutionary theory promising, we think that long-term and large-scale, scientifically

132 Thus, we think that mechanism-based novel strategies should be

133 We think that our approach will lead to better character

134 We think that our data provide insight into expression r

135 We think that our data provide thrombosis-relevant infor

136 We think that PTVI is a good alternative to repeated sur

137 We think that RACK1 is an important Src substrate that s

138 We think that recipient BM-HSC-derived hepatocyte repopu

139 tional approach to the analysis of teaching, we think that she ignores important features of the soci

140 Thus, we think that TCRs are structurally able to recognize a

141 We think that the actin cytoskeleton, in particular the

142 We think that the data we are presenting is important be

143 We think that the early intermediates with peaks at 155

144 and that osteocytes produce paracrine DKK1, we think that the G171V mutation may cause an increase i

145 However, we think that the growing number of oral anticoagulant c

146 Thus, we think that the hydrophobic effect and hydrogen bondin

147 We think that the integration of thread electrodes withi

148 We think that the negative cooperativity occurs when sat

149 We think that the prevalence of its use will be further

150 We think that the probability index provides an immediat

151 Therefore, we think that the process of working through religious l

152 We think that the proposed anticooperative binding mecha

153 We think that the recognition and killing of target cell

154 We think that the unbiased reader will follow our argume

155 We think that these results could serve as valuable indi

156 We think that these results propose a new mechanism by w

157 We think that this approach to electrocatalytic reductio

158 We think that this difference underscores the potential

159 We think that this hierarchy of control explains many of

160 We think that this integrated and tiered approach will l

161 In view of the data obtained, we think that this is a significant and important step t

162 We think that this kind of quantitative analysis can be

163 We think that this method will provide the most benefit

164 We think that this protocol can be successfully applied

165 We think that this should be linked to the concept of so

166 We think that this study presents a novel approach for (

167 We think that tyrosine phosphorylation of these proteins

168 We think that US and clinical findings may sometimes mis

169 We think that working together, using appropriate scienc

170 At this time, we think that, for most conditions, surgical procedures,

171 t for efficient charging of methionine tRNA, we thought that 1.72 distortion is probably effected by

172 As we thought that endogenous somatostatin might control th

173 refore, require multiple angiogenic factors, we thought that injection of BM, which contains cells th

174 We thought that polymeric matrices, which release growth

175 We thought that terminal homology would promote bidirect

176 y selective for microtubule-targeted agents, we thought that the active compounds might inhibit cell

177 Due to structural similarity, we thought that their metabolites might extend anticance

178 eletion events and multiple point mutations, we thought that these multiple-mutated loci might repres

179 ces contain determinants for aminoacylation, we thought that they might also play a role in editing t

180 Just as we thought that we know everything about superantigens,

181 We think the genomic resource and the candidate genes re

182 nd web systems are invaluable resources, and we think the scientific community will benefit for this

183 We think the term monoclonal gammopathy of renal signifi

184 We thought the attack caused from rhinitis by Staphyloco

185 We think there is now compelling evidence that a full un

186 We think these muscle-synergy patterns can be used as ph

187 We think these novel revascularization strategies are li

188 ype structures with considerable Si content, we thought these metastables would be only of the diamon

189 hted sums of risk alleles are measuring what we think they are in the various scenarios in which PRSs

190 We think they should be included within all evidence-bas

191 raction for the purpose of GPCR purification-we think this approach holds excellent potential to isol

192 entations and musculoskeletal manifestations.We think this article will heighten awareness of these d

193 We think this benefit is largely due to these drugs' abi

194 We think this effect may reasonably be attributed to pre

195 ic progress and provide a perspective on why we think this is only the beginning of a new era in scie

196 We think this is possible if funding agencies around the

197 tivariable model, and the large sample size, we think this is the most definitive analysis of the pre

198 We think this mechanism has potentially significant impl

199 We think this pattern should change.

200 's proposed explanation of what goes on when we think through other minds.

201 We thought to determine the underlying molecular causes

202 Therefore, we thought to evaluate avian paramyxovirus serotype 3 (A

203 We thought to tackle this problem by taking a systems bi

204 We think very little is needed.

205 Evidence that what we thought was a single species based on morphology is,

206 Aristotle explains how we think we have knowledge of a thing only when we have

207 We conclude by describing what we think we have learned about the conditions that suppo

208 ensory modalities can interact, shaping what we think we have seen, heard, or otherwise perceived.

209 nores this topic and because so much of what we think we know about faith sharing is based on US Chri

210 phosphate chemistry, as well as much of what we think we know about metabolism, will need rewriting.

211 see, what we see, and how we interpret what we think we saw and remembered are offered by a surprisi

212 developed the human beta cell lines and why we think we succeeded where others, despite major effort

213 While a resident, we thought we had found a treatment for multiple scleros

214 hrenia and related psychotic disorders that, we think, will be of interest to psychiatric clinicians

215 We then selected variables we thought would be associated with increased rates of 1