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1 ly used variables (i.e. mixed venous oxygen, wedge pressure).
2 rdiac filling pressures (pulmonary capillary wedge pressure).
3 patient age, and varying pulmonary capillary wedge pressure.
4 pressure, heart rate, or pulmonary capillary wedge pressure.
5 orse symptoms and higher pulmonary capillary wedge pressure.
6 and severity of emphysema, and directly with wedge pressure.
7 ar resistance as well as pulmonary capillary wedge pressure.
8 ar systolic pressure and pulmonary capillary wedge pressure.
9 nary artery pressure and pulmonary capillary wedge pressure.
10 elocities for estimating pulmonary capillary wedge pressure.
11 nary artery pressure and pulmonary capillary wedge pressure.
12 ic filling, despite high pulmonary capillary wedge pressure.
13 lmonary artery pressure and pulmonary artery wedge pressure.
14 ctional flow reserve and increasing coronary wedge pressure.
15 ltaneous measurements of pulmonary capillary wedge pressure.
16 ction in mean pulmonary artery and pulmonary wedge pressures.
17 nge systemic arterial or pulmonary capillary wedge pressures.
18 42; median RAP, 4 mm Hg; pulmonary capillary wedge pressure, 11 mm Hg), those with a low JV distensib
20 e >240 dyn-sec/cm(-5) , and pulmonary artery wedge pressure 15 mm Hg without another cause of pulmona
21 ong them, 26 (6.5%) had a pulmonary arterial wedge pressure 15 mm Hg, and 18 (4.5%) had a pulmonary a
22 was defined according to pulmonary capillary wedge pressure ( 15 mm Hg at rest or 25 mm Hg during exe
23 in, p < 0.01), increased pulmonary capillary wedge pressure (17 +/- 7 vs. 14 +/- 6 mm Hg, p < 0.002)
24 g; P=0.0002), and higher pulmonary capillary wedge pressure (17+/-5 versus 9+/-3 mm Hg; P=0.0001).
25 m Hg to 98 +/- 13 mm Hg; pulmonary capillary wedge pressure: 17 +/- 6 mm Hg to 21 +/- 7 mm Hg; cardia
26 s. 108 +/- 14 mm Hg) and pulmonary capillary wedge pressure (18 +/- 2 vs. 12 +/- 4 mm Hg) versus prei
27 -4 mm Hg; P=0.0001), and pulmonary capillary wedge pressure (18+/-5 versus 10+/-4 mm Hg; P<0.0001) co
28 mg significantly reduced pulmonary capillary wedge pressure (-2.6+/-0.7, -5.4+/-0.7, and -4.6+/-0.7 m
29 tric LVH and estimated mean pulmonary artery wedge pressure (20 mm Hg vs. 16 mm Hg) and shorter isovo
30 p < 0.01 by ANOVA) and mean pulmonary artery wedge pressure (21+/-3 to 16+/-2 mm Hg, p < 0.05 by ANOV
31 58; median RAP, 8 mm Hg; pulmonary capillary wedge pressure, 22 mm Hg; P<0.0001 for both) were more l
32 arly reductions were sustained for pulmonary wedge pressure (24+/-9 to 15+/-5 mm Hg early; 12+/-6 mm
34 produced significant reductions in pulmonary wedge pressure (27% to 39% decrease by 6 h), mean right
35 s. 16 +/- 8 mm Hg, p = 0.007, mean pulmonary wedge pressure: 27 +/- 8 mm Hg vs. 20 +/- 7 mm Hg, p = 0
36 s. 13 mm Hg; p = 0.001), pulmonary capillary wedge pressure (29 vs. 24 mm Hg; p = 0.001), but similar
37 with lower mean exercise pulmonary capillary wedge pressure (-3 mm Hg [95% CI, -5 to -1] ) and higher
38 pressure (18+/-2 to 7+/-1 mm Hg), pulmonary wedge pressure (32+/-3 to 15+/-2 mm Hg), and systemic va
39 vascular resistance 25%, pulmonary capillary wedge pressure 33%, and central venous pressure 27% whil
40 nitrite reduced resting pulmonary capillary wedge pressure (-4+/-3 versus -1+/-2 mm Hg; P=0.002), im
41 uctions in 0-hour trough pulmonary capillary wedge pressure (-4.3 mm Hg; P=0.16), pulmonary artery di
42 sulted in an increase in pulmonary capillary wedge pressure (54 +/- 78% vs. 32 +/- 26%, p = NS), card
43 es significantly reduced pulmonary capillary wedge pressure (-6.4 +/- 4.1 mm Hg, -5.7 +/- 4.6 mm Hg,
44 icacy end point included pulmonary capillary wedge pressure (72 to 96 hours) and days alive out of ho
45 iameter, 3.53+/-0.51 cm; pulmonary capillary wedge pressure, 8.1+/-3.1 mm Hg; pulmonary artery satura
47 of ADMA correlated with pulmonary capillary wedge pressure and both systolic and diastolic pulmonary
49 e actual R(micro) and IMR after the coronary wedge pressure and collateral flow were incorporated int
50 tment markedly decreased pulmonary capillary wedge pressure and improved pressure-flow relationship.
51 -confirmed PH with elevated pulmonary artery wedge pressure and low pulmonary vascular resistance, we
52 ight atrial pressure and pulmonary capillary wedge pressure and lower cardiac index (CI) but not Paco
54 ssure-volume curves from pulmonary capillary wedge pressure and LV end-diastolic volume and Starling
56 d normative responses of pulmonary capillary wedge pressure and mean pulmonary arterial pressure to v
57 the largest increases in pulmonary capillary wedge pressure and mean pulmonary arterial pressure.
58 the presence of a normal pulmonary capillary wedge pressure and portal hypertension, is a known compl
59 dose-dependently reduced pulmonary capillary wedge pressure and pulmonary and systemic vascular resis
60 ulmonary exercise tests, pulmonary capillary wedge pressure and serum sodium were strong predictors o
61 hemodynamics in AMI-VSD, pulmonary capillary wedge pressure and shunting were worsened by extracorpor
62 ane oxygenation worsened pulmonary capillary wedge pressure and shunting, which could be improved by
63 and Starling curves from pulmonary capillary wedge pressure and SV during lower body negative pressur
65 ary artery catheter use, pulmonary capillary wedge pressure and the incidence of diabetes mellitus an
67 placement therapy, lower pulmonary capillary wedge pressure and vasoactive-inotropic score, and highe
70 re, pulmonary vascular resistance, capillary wedge pressure, and cardiac index were also obtained at
72 d right atrial pressure, pulmonary capillary wedge pressure, and pulmonary hypertension, respectively
73 cardiac output, decrease pulmonary capillary wedge pressure, and reduce pulmonary and systemic vascul
74 cardiac index, elevated pulmonary capillary wedge pressure, and renal impairment or substantial diur
75 ons include: (1) liver biopsy, portal venous wedge pressure, and reversal of portal venous flow on Do
76 Primary end point was pulmonary capillary wedge pressure, and secondary end points comprised cardi
77 diac output, heart rate, pulmonary capillary wedge pressure, and systemic vascular resistance were me
78 alculated values such as pulmonary capillary wedge pressure, aortic pulsatility index, and cardiac in
80 s that pulmonary artery and pulmonary artery wedge pressures are higher in SIPE-susceptible individua
81 tly greater reduction in pulmonary capillary wedge pressure at 3, 4, and 8 hours and during the presp
82 was no effect of age on pulmonary capillary wedge pressure at any point throughout the cardiac cycle
83 r than 40%, and a raised pulmonary capillary wedge pressure at rest (>15 mm Hg) or during exercise (>
84 e were no differences in pulmonary capillary wedge pressure at rest (13+/-4 versus 13+/-3 mm Hg, P=0.
85 d with directly measured pulmonary capillary wedge pressure at rest (r=0.63, P<0.0001) and during exe
86 our period and decreased pulmonary capillary wedge pressure at rest by 1 mm Hg (95% CI, -2 to 0) and
87 with HFpEF (n=34; median pulmonary capillary wedge pressure at rest, 13 mm Hg; at stress, 27 mm Hg) h
88 ients had a reduction in pulmonary capillary wedge pressure at rest, 34 (58%) of 59 had a lower pulmo
89 y be changed by elevated pulmonary capillary wedge pressure, augmenting right ventricular pulsatile l
90 ose-related decreases in pulmonary capillary wedge pressure (average change -5.9+/-0.9 mm Hg and -5.3
92 d pressure, and baseline pulmonary capillary wedge pressure between groups (eg, pulmonary capillary w
93 ulmonary artery pressure or pulmonary artery wedge pressure between SIPE-susceptible subjects and con
95 roving cardiac index and pulmonary capillary wedge pressure, but statistical significance for the pri
96 ram per minute decreased pulmonary-capillary wedge pressure by 6.0 and 9.6 mm Hg, respectively (as co
97 systemic blood pressure, pulmonary capillary wedge pressure, cardiac index, and estimated glomerular
98 mean arterial pressure, pulmonary capillary wedge pressure, cardiac index, or systemic vascular resi
99 gnificant differences in pulmonary capillary wedge pressure, cardiac output, pulmonary vascular resis
100 artery, right atrial and pulmonary capillary wedge pressures, cardiac index, systemic and pulmonary v
101 al, pulmonary artery, or pulmonary capillary wedge pressures; cardiac index; respiratory rate; or hea
102 11; LVAD, 18+/-4 mm Hg) and pulmonary artery wedge pressure (CHF, 16+/-10; LVAD 5+/-3 mm Hg) were red
103 , 30+/-5 mm Hg) and mean pulmonary capillary wedge pressure (CHF, 31+/-11; LVAD, 14+/-6 mm Hg) were l
104 ated LV end-diastolic or pulmonary capillary wedge pressure, consistent with diastolic dysfunction, w
105 as the inverse was true for pulmonary artery wedge pressure (corresponding muPAWP were 21.5, 16.5, an
106 d using rest or exercise pulmonary capillary wedge pressure criteria (>=15 mm Hg or Deltapulmonary ca
107 (-2)]; P<0.001) and mean pulmonary capillary wedge pressure decreased (from 15 mm Hg [IQR, 12-20 mm H
108 48 hours after implant, pulmonary capillary wedge pressure decreased 44%, systemic vascular resistan
109 decreased 19% (P=0.03), pulmonary capillary wedge pressure decreased 46% (P=0.002), and mean arteria
110 iac index increased 43%, pulmonary capillary wedge pressure decreased 52%, systemic vascular resistan
111 +/-5 mm Hg, P<0.001) and pulmonary capillary wedge pressure decreased by 27% (31+/-8 versus 22+/-9 mm
113 25.9 +/- 1.7 mm Hg; mean pulmonary capillary wedge pressure decreased from 25.1 +/- 1.1 to 13.2 +/- 1
117 flow relationship (Delta pulmonary capillary wedge pressure/Delta cardiac output) significantly durin
118 eria (>=15 mm Hg or Deltapulmonary capillary wedge pressure/Deltacardiac output slope, >2.0 mm Hg.L(-
119 sion (n=1009) and normal pulmonary capillary wedge pressure displayed a consistent R(PA)-C(PA) hyperb
122 ion of at least 40%, and pulmonary capillary wedge pressure during exercise of at least 25 mm Hg whil
124 (58%) of 59 had a lower pulmonary capillary wedge pressure during exertion, and 23 (39%) of 59 fulfi
126 Exercise elicits greater pulmonary capillary wedge pressure elevation compared with saline in HFpEF b
128 p < 0.01) and increased pulmonary capillary wedge pressure (from 25 +/- 2 to 29 +/- 3 mm Hg, p < 0.0
129 is predictive of a mean pulmonary capillary wedge pressure greater than 15 mm Hg with a 92% sensitiv
130 ction 21+/-1%) who had a pulmonary capillary wedge pressure >/=15 mm Hg and a cardiac index </=2.5 L
131 Patients with a baseline pulmonary capillary wedge pressure >/=15 mm Hg and a cardiac index </=2.5 L.
132 ventricular [RV] DD) or pulmonary capillary wedge pressure >/=18 mm Hg (left ventricular [LV] DD) wi
133 pressure <100 mm Hg and pulmonary capillary wedge pressure >/=24 mm Hg and dependent on >/=2 inotrop
134 ients in MI+DD (29%) had pulmonary capillary wedge pressure >15 (14+/-4 mm Hg), whereas none of the M
135 identified patients with pulmonary capillary wedge pressure >15 mm Hg (area under the curve: 0.73 to
136 ssified as cold and wet (pulmonary capillary wedge pressure >15 mm Hg) and cold and dry (pulmonary ca
138 ubgroups: (1) patients with pulmonary artery wedge pressure >15 mm Hg; (2) pulmonary vascular resista
139 ic balloon pump support, pulmonary capillary wedge pressure >20 mm Hg and serum creatinine >1.5 mg/dl
140 lvement evident at rest (pulmonary capillary wedge pressure >=15 mm Hg; area under the curve, 0.81 ve
141 exercise stress induced pulmonary capillary wedge pressure >=25 mm Hg (area under the curve, 0.79 ve
142 on class II-IV, elevated pulmonary capillary wedge pressure (>/=15 mm Hg at rest or >/=25 mm Hg durin
143 ise, an abnormal rise in pulmonary capillary wedge pressure (>25 mm Hg) was observed in 94% of MI+DD
144 80% of predicted and peak pulmonary arterial wedge pressure>/=20 mm Hg) with 31 age- and sex-matched
146 fter adjusting for exercise pulmonary artery wedge pressure (HR 2.08 per WU.m2, 95% CI 1.19-3.62; P =
147 0.022), diastolic PAP - pulmonary capillary wedge pressure (HR, 2.19; 95% CI, 1.23-3.89 per 10 mm Hg
148 eart failure to decrease pulmonary capillary wedge pressure, improve cardiac output, stimulate natriu
150 In healthy subjects, pulmonary capillary wedge pressure increased from 10+/-2 to 16+/-3 mm Hg aft
152 y arterial pressure, and pulmonary capillary wedge pressure increased similarly with saline and exerc
155 volume overload, a treatment-induced drop in wedge pressure is often accompanied by a rapid drop in N
156 ch as ejection fraction, cardiac output, and wedge pressure, is available, but also in clinical trial
157 d-diastolic pressures and pulmonary arterial wedge pressures, is associated with remarkably increased
159 ndex, ejection fraction, pulmonary capillary wedge pressure, left ventricular dimensions, watts achie
160 power output and higher pulmonary capillary wedge pressure, less likely to receive vasopressors or t
162 pressure >/=25 mm Hg and pulmonary capillary wedge pressure </=15 mm Hg at right heart catheterizatio
165 nous pressure <12 mm Hg, pulmonary capillary wedge pressure <18 mm Hg, and cardiac index >2.2 L/(min.
166 furosemide, mannitol (if pulmonary capillary wedge pressure <20 mm Hg), and low-dose dopamine (n = 43
167 e >240 dyn-sec/cm(-5) , and pulmonary artery wedge pressure <=15 mm Hg without another cause of pulmo
168 mm Hg) and cold and dry (pulmonary capillary wedge pressure <=15 mm Hg) based on pre-LVAD hemodynamic
170 sure between groups (eg, pulmonary capillary wedge pressure: LVH, 13.4+/-2.7 versus control, 11.7+/-1
171 tage have revealed a normal pulmonary artery wedge pressure, marked elevation of pulmonary artery pre
172 hemodynamic performance with lower pulmonary wedge pressure (mean difference, 2.23 mm Hg [95% CI, 0.4
173 the true IMR (IMR(true)), which incorporates wedge pressure measurement to account for collateral flo
174 th Revision procedure codes describing PA or wedge-pressure monitoring, measurement of mixed venous b
176 mic severity is the mean Pulmonary Capillary Wedge Pressure (mPCWP), which is ideally measured invasi
178 owever, elevation of the pulmonary capillary wedge pressure (n=8142) had a larger impact, significant
180 versus 32 +/- 12%) and mean pulmonary artery wedge pressure of < or = 18 versus > 18 mm Hg (84 +/- 6%
181 wo post-CBC variables: mean pulmonary artery wedge pressure of < or = 18 versus > 18 mm Hg (90 +/- 6%
182 dex (2.4 liters/min.m2), pulmonary capillary wedge pressure of 16 +/- 9 mm Hg (mean +/- SD) and maxim
183 ter, 127 patients with a pulmonary-capillary wedge pressure of 18 mm Hg or higher and a cardiac index
184 re of 40.5+/-11.4 mm Hg, pulmonary capillary wedge pressure of 22.6+/-8.9 mm Hg, and pulmonary vascul
185 x of 1.7 L/min per m(2), pulmonary capillary wedge pressure of 25.6 mm Hg, and left ventricular eject
186 c filling volume was the pulmonary capillary wedge pressure of the seniors lower than that of the you
187 839]; P < 0.001), estimated pulmonary artery wedge pressure (OR, 1.437; 95% CI [1.131-2.274]; P < 0.0
189 0.02, r = 0.83), higher pulmonary capillary wedge pressure (p = 0.01, r = 0.58) and lower cardiac in
193 y reduced pulmonary arterial (p < 0.001) and wedge pressures (p < 0.01) and pulmonary vascular resist
194 dynamic profiles, including higher pulmonary wedge pressures (P=0.002) and lower cardiac indexes (P<0
195 dilatation, increase of pulmonary capillary wedge pressure, PAP and RAP were more pronounced in AF t
196 fraction was defined as a pulmonary arterial wedge pressure (PAWP) >=15 mmHg (rest) or >=25 mmHg (exe
198 ly correlated with supine pulmonary arterial wedge pressure (PAWP; r=0.36; P<0.001) and demonstrated
201 intraarterial catheter, pulmonary capillary wedge pressure (Pcw), continuous cardiac output (Q), and
202 =2.5 l/min per m(2) and pulmonary capillary wedge pressure (PCWP) > or =15 mm Hg) who were admitted
204 nine (2.6 to 1.5 mg/dL), pulmonary capillary wedge pressure (PCWP) (32 to 14 mm Hg), and right atrial
205 by baseline measures of pulmonary capillary wedge pressure (PCWP) and cardiac index (CI), and by cha
206 ficant increases in both pulmonary capillary wedge pressure (PCWP) and central venous pressure (CVP).
207 sitive relationship with pulmonary capillary wedge pressure (PCWP) and left ventricular end-diastolic
208 al capacity, and dynamic pulmonary capillary wedge pressure (PCWP) and right atrial pressure response
209 ns were observed between pulmonary capillary wedge pressure (PCWP) and sole parameters of mitral flow
210 subjects for measures of pulmonary capillary wedge pressure (PCWP) and SV (thermodilution derived car
211 trial pressure (RAP) and pulmonary capillary wedge pressure (PCWP) are correlated in heart failure, i
212 n the prospective group, pulmonary capillary wedge pressure (PCWP) derived as: PCWP(Doppler) = LV(end
215 ere, we examined whether pulmonary capillary wedge pressure (PCWP) during a passive leg raise (PLR) c
217 has been correlated with pulmonary capillary wedge pressure (PCWP) in a wide variety of cardiac condi
218 s noninvasive markers of pulmonary capillary wedge pressure (PCWP) in the setting of critical illness
220 y measured peak exercise pulmonary capillary wedge pressure (PCWP) of 25 mm Hg or greater were includ
221 ity of life, and dynamic pulmonary capillary wedge pressure (PCWP) responses were compared between th
222 invasive measurement of pulmonary capillary wedge pressure (PCWP) simultaneous with Doppler echocard
223 d ePVH groups had higher pulmonary capillary wedge pressure (PCWP) than the ePH group (P < 0.05).
224 ary outcome was ratio of pulmonary capillary wedge pressure (PCWP) to cardiac index (CI) at peak exer
227 27% (n=161) had elevated pulmonary capillary wedge pressure (PCWP) with postcapillary pulmonary hyper
228 tery pressure>=32 mm Hg, pulmonary capillary wedge pressure (PCWP)>=20 mm Hg, and pulmonary vascular
229 ion fraction, 22 +/- 9%; pulmonary capillary wedge pressure (PCWP), 16 +/- 10 mm Hg; cardiac index (C
230 c nerve activity (RSNA), pulmonary capillary wedge pressure (PCWP), and mean arterial pressure (MAP)
231 in eight males from whom pulmonary capillary wedge pressure (PCWP), central venous pressure and SV (v
232 ressure, as expressed by pulmonary capillary wedge pressure (PCWP), during lower-body negative pressu
233 as associated with lower pulmonary capillary wedge pressure (PCWP), fewer symptoms, and greater quali
235 es of istaroxime lowered pulmonary capillary wedge pressure (PCWP), the primary end point (mean +/- S
237 etween the reductions in pulmonary capillary wedge pressure (PCWP; 25.4, 24.6, 24.0, 23.5, 23.4, 21.5
239 Hg and LV congestion if pulmonary capillary wedge pressure [PCWP] 22 mm Hg) categorizations was the
240 concomitant increase in the pulmonary artery wedge pressure per increase in cardiac output >2 mm Hg/L
241 th a higher increase in the pulmonary artery wedge pressure per increase in cardiac output ratio.
245 aphic severity correlated significantly with wedge pressure (r = 0.93, P < .001) and pulmonary arteri
246 correlated with invasive pulmonary arterial wedge pressure (r(2)=0.57, root mean square error 5.0 mm
247 rong inverse relation with PVR (r=-0.64) and wedge pressure (r=-0.73), and provides stronger predicti
249 VR) ratio had the best correlation with mean wedge pressure (r=0.79, P<0.001), as well as in 24 prosp
251 01), higher right atrium:pulmonary capillary wedge pressure ratio (beta, 0.25; SE, 0.01; P<0.001), an
252 nd right atrial pressure/pulmonary capillary wedge pressure ratio were most associated with worsened
254 d the greatest degree of pulmonary capillary wedge pressure reductions and decreased left-to-right sh
255 aturation percentage and pulmonary capillary wedge pressure relate to cardiac output and congestion,
256 ed a steeper increase in pulmonary capillary wedge pressure relative to infused volume (25+/-12 mm Hg
257 ed a steeper increase in pulmonary capillary wedge pressure relative to volume infused (16+/-4 mm Hg.
258 n mean pulmonary artery and pulmonary artery wedge pressures, respectively, but no change in transpul
259 smural filling pressure (pulmonary capillary wedge pressure - right atrial pressure), LV myocardial s
260 and it had no effect on pulmonary-capillary wedge pressure, right atrial pressure, heart rate, or ca
261 LV transmural pressure (pulmonary capillary wedge pressure-right atrial pressure), which reflects LV
262 trans-septal gradient (=pulmonary capillary wedge pressure-right atrial pressure; r=0.67; P=0.003),
264 in pulmonary artery and pulmonary capillary wedge pressures, suggesting abnormal compliance, with ma
265 c output, stroke volume, pulmonary capillary wedge pressure, systemic and pulmonary vascular resistan
266 g pulmonary arterial and pulmonary capillary wedge pressures than the remaining heart-failure patient
267 trated elevated exercise pulmonary capillary wedge pressure to cardiac output slope (P<0.0001) with n
268 d pulmonary arterial and pulmonary capillary wedge pressures to a greater level than OMA alone or ACE
271 k 12, the change in 25-W pulmonary capillary wedge pressure was -2.8 (6.8) mm Hg in the exercise grou
272 004), and the corresponding pulmonary artery wedge pressure was 11.0 mm Hg versus 18.8 mm Hg (P=0.028
275 L . min(-)(1) . m(-)(2), pulmonary capillary wedge pressure was 31.5 +/- 5.7 mm Hg, and heart failure
276 . kg(-)(1) . min(-)(1), pulmonary capillary wedge pressure was 5.9 +/- 4.6 mm Hg, and cardiac index
279 vascular resistance and pulmonary capillary wedge pressure was evident at 3 min after infusion in 70
282 In upright position, pulmonary arterial wedge pressure was lower in discordant HFpEF both at res
284 r for the two groups but pulmonary capillary wedge pressure was slightly lower for Group C (22 vs. 24
285 administration, exercise pulmonary capillary wedge pressure was substantially improved by nitrite as
288 tion with measurement of pulmonary capillary wedge pressure waveform during 5 different loading condi
289 detailed analysis of the pulmonary capillary wedge pressure waveform obtained by right-heart catheter
290 ar resistance and normal pulmonary capillary wedge pressure, we make a weak recommendation for either
291 vascular resistance and pulmonary capillary wedge pressure were decreased, as compared with untreate
292 The cardiac index and pulmonary-capillary wedge pressure were elevated in the six patients in whom
293 (r=0.947), while PVP and pulmonary capillary wedge pressure were found to be moderately correlated (r
295 rtery pressure (PAP) and pulmonary capillary wedge pressure were obtained by standard techniques.
296 m, pulmonary artery, and pulmonary capillary wedge pressure were obtained in 85 patients during a tot
298 displayed an increase in pulmonary capillary wedge pressure with exercise from 20+/-6 to 34+/-7 mm Hg
299 ubjects displayed higher pulmonary capillary wedge pressure with exercise, but this was solely becaus
300 diac output, and reduces pulmonary capillary wedge pressure without causing deleterious increases in