ライフサイエンスコーパス: wheezing

1 follow-up at 3 years, and 31% had recurrent wheezing.

2 d with a higher risk for allergy, asthma and wheezing.

3 to promote airway obstruction and recurrent wheezing.

4 l eczema and rhinoconjunctivitis, but not to wheezing.

5 of breath, chest tightness, coughing, and/or wheezing.

6 shortness of breath, chest pain, cough, and wheezing.

7 crobiota abundances to the risk of recurrent wheezing.

8 diagnostic tests for recurrent or persistent wheezing.

9 n and jaundice, itching, flushing, cough and wheezing.

10 he prevention of postbronchiolitis recurrent wheezing.

11 he occurrence of postbronchiolitis recurrent wheezing.

12 ng is an important risk factor for recurrent wheezing.

13 valence or incidence of atopy, AD, asthma or wheezing.

14 due to concerns over its potential to induce wheezing.

15 group is generally referred to as preschool wheezing.

16 quelae of persistent airway inflammation and wheezing.

17 stress during pregnancy with early childhood wheezing.

18 sults and were not associated with childhood wheezing.

19 e heterogeneity of both paediatric and adult wheezing.

20 itis who are at risk of developing recurrent wheezing.

21 cell debris, and increased risk of recurrent wheezing.

22 to identify (latent) phenotypes of childhood wheezing.

23 fined as >=3 episodes of physician-diagnosed wheezing.

24 en at increased risk of developing recurrent wheezing.

25 cts the airways and presents as coughing and wheezing.

26 abolite of di-isodecyl phthalate (DIDP), and wheezing.

27 ore likely to experience frequent attacks of wheezing, 3.44-fold (1.19-9.94) more likely to experienc

28 rs; 77% phlegm; 70% shortness of breath; 47% wheezing; 46% chest pain; 42% abnormal peak flow), 334 (

29 n with asthma and 54 preschool children with wheezing [68.2% of whom were atopic]) were included in t

30 Asthma/wheezing accounted for 10%-18% of total 5-year physician

31 rt through age 7 years, reflecting symptoms (wheezing), aeroallergen sensitization, pulmonary functio

32 Patterns of wheezing, allergic sensitization, and lung function iden

33 lass mixed models identified trajectories of wheezing, allergic sensitization, and pulmonary function

34 Disease phenotypes included asthma, atopy, wheezing, altered lung function, and bronchial reactivit

35 , NVAS was associated with increased risk of wheezing among females (RR 1.80 [1.03-3.17], but not amo

36 and early life are associated with recurrent wheezing and aeroallergen sensitivity and altered cytoki

37 ied and mainly differentiated by patterns of wheezing and allergic sensitization (low wheeze/low atop

38 t of physician-diagnosed asthma or recurrent wheezing and allergic sensitization to food or environme

39 atory morbidity among children with frequent wheezing and allergic sensitization.

40 asurements, and assessed physician-diagnosed wheezing and asthma by questionnaires.

41 es predictors of remission or persistence of wheezing and asthma from early childhood through adultho

42 ay play a role in the inception of recurrent wheezing and asthma in childhood.

43 n 3 expression and correlated inversely with wheezing and asthma in nonatopic subjects.

44 al exhaled nitric oxide (Feno), and risks of wheezing and asthma in school-aged children.

45 Wheezing and asthma information up to 8/10 years was col

46 y microbiome in the development of recurrent wheezing and asthma remains uncertain, particularly in t

47 thma risk factors and the natural history of wheezing and asthma through childhood and beyond.

48 erventions that could decrease virus-induced wheezing and asthma.

49 infancy is a major risk factor for recurrent wheezing and asthma.

50 lationship with the development of recurrent wheezing and asthma.

51 associated with the risk of transient early wheezing and atopic dermatitis by the age of 5 years, bu

52 with emergency department visits for asthma/wheezing and congestive heart failure than PM2.5.

53 episodic shortness of breath with expiratory wheezing and cough, is a serious health concern affectin

54 man was admitted to our hospital because of wheezing and dyspnea.

55 respectively); rs3918396 was associated with wheezing and eczema comorbidity (p = 3.41*10(-4)).

56 Due to its association with wheezing and eczema comorbidity, ADAM33 may also be invo

57 blings, reported childcare attendance, early wheezing and eczema in the first 3 years of life.

58 stion, he experienced coughing, dyspnea, and wheezing and had to be treated by anti-histamine and ste

59 with atopy, atopic dermatitis (AD), asthma, wheezing and impaired lung function in a prospective stu

60 n of maternal stress/depression to recurrent wheezing and peripheral blood mononuclear cell cytokine

61 n, including the increased risk of childhood wheezing and subsequent asthma.

62 qPCRs, was inversely associated with risk of wheezing and was significantly (inverted-U shape) associ

63 were more likely to have chest retractions, wheezing, and a history of underlying asthma/reactive ai

64 infants developing bronchiolitis, recurrent wheezing, and asthma following infection.

65 also observed between 2,5-dichlorophenol and wheezing, and between monocarboxynonyl phthalate, a meta

66 ronic respiratory conditions such as asthma, wheezing, and bronchitis.

67 ma including maternal atopy, early childhood wheezing, and bronchodilator response.

68 rhea, restlessness/sleep disturbances, minor wheezing, and cold extremities).

69 respiratory symptoms (difficulty breathing, wheezing, and cough) lasting >/= 2 days or requiring pre

70 yfish product (100g), he experienced nausea, wheezing, and erythema and had visited our hospital.

71 rs), symptoms of cough, nasal congestion and wheezing, and longer interval from illness onset to clin

72 Five-year cumulative all-cause, asthma/wheezing, and respiratory event-related hospitalization

73 In addition to parental asthma, eczema, and wheezing apart from colds, variables that predicted asth

74 Although respiratory symptoms, including wheezing, are common in preterm-born subjects, the natur

75 ntibody is available; (b) rhinovirus-induced wheezing, associated with atopic predisposition of the p

76 Early-life wheezing-associated respiratory infection with human rhi

77 Early-life wheezing-associated respiratory tract infection by rhino

78 Wheezing, asthma and AD were assessed from questionnaire

79 -term lung health, with potential effects on wheezing, asthma, and chronic lung disease.

80 cally significant change in the incidence of wheezing/asthma after introduction of smoke-free legisla

81 extracted monthly counts of new diagnoses of wheezing/asthma and RTIs among children aged 0-12 years

82 366,642 new wheezing/asthma diagnoses and 4,324,789 RTIs were observ

83 le reductions in the incidence of paediatric wheezing/asthma or RTIs following introduction of smoke-

84 of eczema; 14, allergic sensitization; nine, wheezing/asthma; six, food allergy; three, allergic rhin

85 hagia at 1 month, nausea at 3 and 12 months, wheezing at 6 months; and inability to belch at 12 month

86 oking, higher maternal education levels, and wheezing at age 36-72 months.

87 ness at baseline and the onset of asthma and wheezing at the age of 7.

88 asthmatic symptoms, as well as virus-induced wheezing, at any time before biomarker assessment at age

89 atopy (OR = 0.35, 95% CI = 0.13-0.90), fewer wheezing attacks (OR = 0.40, 95% CI = 0.17-0.97; >3 vs <

90 Asthma and wheezing begin early in life, and prenatal vitamin D def

91 siveness that leads to recurrent episodes of wheezing, breathlessness, chest tightness, and coughing.

92 gitudinal prevalence of fever, coughing, and wheezing but increased incidence and longitudinal preval

93 were significantly associated with recurrent wheezing but not increased atopy or reduced antiviral re

94 ssociated with increased risk for persistent wheezing, but only long-term exposure to high levels of

95 with a greater risk of developing recurrent wheezing, but with currently available tools, it is impo

96 associated with increased risk of recurrent wheezing by age 3 years and asthma that persisted throug

97 Recurrent wheezing by age 3 years was defined based on parental re

98 ospitalization was associated with recurrent wheezing by age 3 years, possibly providing new avenues

99 eal microbiota, and development of recurrent wheezing by age 3 years.

100 and 3 later points to the risk of recurrent wheezing by age 3 years.

101 s (P = .03), as well as male sex (P = .025), wheezing causing shortness of breath (P = .002), and ACS

102 ory symptoms [apnea, stridor, nasal flaring, wheezing, chest indrawing, and/or central cyanosis]) wer

103 no fevers, chills, night sweats, hemoptysis, wheezing, chest pain, palpitations, orthopnea, paroxysma

104 Fifty-seven percent of wheezing children and 56% with acute rhinitis had nasal

105 rhinovirus (RV) is most commonly detected in wheezing children thereafter.

106 ive polymerase chain reaction in washes from wheezing children was 2.8-fold lower, but did not differ

107 se bronchodilators to manage childhood acute wheezing conditions in the emergency department (ED), an

108 Asthma, wheezing, cough, and atopic dermatitis were assessed usi

109 ariable airflow obstruction and intermittent wheezing, cough, and dyspnea.

110 case status, especially among children with wheezing disease.

111 association of parental migrant status with wheezing disorders among children born in Hong Kong, a d

112 contribute to the development of early life wheezing disorders and asthma, and discuss the external

113 higher risk of hospitalization for childhood wheezing disorders compared to the native population, pa

114 tematic review investigating risks of asthma/wheezing disorders in children born preterm, including t

115 delivery by Caesarean section with childhood wheezing disorders may vary with setting and may not be

116 d with hospitalizations for asthma and other wheezing disorders to 12 years [hazard ratio (HR) 1.11,

117 ibutable risk of preterm birth for childhood wheezing disorders was >/=3.1%.

118 risk of hospitalization for asthma and other wheezing disorders, compared to both parents being Hong

119 uting factor to the increasing prevalence of wheezing disorders.

120 the aetiology of asthma and other childhood wheezing disorders.

121 s (g/dL) in pregnancy with hayfever, eczema, wheezing, doctor-diagnosed asthma, allergic sensitisatio

122 eczema, allergen sensitization, or recurrent wheezing during the first three years of life.

123 erized by episodic or persistent symptoms of wheezing, dyspnea, and cough.

124 nal period and (ii) documentation of asthma, wheezing, eczema, or other atopic disease in the offspri

125 steroid-naive children with the first severe wheezing episode (90% hospitalized/10% emergency departm

126 piratory syncytial virus/rhinovirus-negative wheezing episode (adjusted OR, 8.0; P = .001), first whe

127 ment resulted in prolonged time to the third wheezing episode (P = .048) and in fewer days with respi

128 istics during the first severe virus-induced wheezing episode are associated with pulmonary function

129 irate by using PCR, 79 children with a first wheezing episode at age 3 to 23 months were randomized t

130 episode (adjusted OR, 8.0; P = .001), first wheezing episode at age less than 12 months (adjusted OR

131 d of 76 children presenting with their first wheezing episode at the ages of 3 to 23 months.

132 to identify risk factors at the first severe wheezing episode for current asthma 7 years later and se

133 cute, moderate-to-severe, rhinovirus-induced wheezing episode in young children.

134 ensitization at the time of the first severe wheezing episode is an important early risk factor for i

135 topic sensitization at the time of the first wheezing episode were more often likely to develop bronc

136 omes were long term: new physician-confirmed wheezing episode within 2 months, number of physician-co

137 uffering from severe bronchiolitis (or first wheezing episode): (a) respiratory syncytial virus (RSV)

138 IL-8 levels, prolonged the time to the third wheezing episode, and reduced overall respiratory morbid

139 ical markers at the time of the first severe wheezing episode.

140 cute, moderate-to-severe, rhinovirus-induced wheezing episode.

141 -treated participants experienced at least 3 wheezing episodes compared with 50% of participants in t

142 c subjects are uniquely susceptible to acute wheezing episodes provoked by rhinovirus.

143 The occurrence of wheezing episodes was assessed monthly over the ensuing

144 Maternal stress, depression, and childhood wheezing episodes were assessed by quarterly questionnai

145 placebo, unscheduled medical attendances for wheezing episodes were reduced in children given montelu

146 thin 2 months, number of physician-confirmed wheezing episodes within 12 months, and initiation of re

147 lar positive associations with the number of wheezing episodes, wheezing patterns, and physician-diag

148 umber of unscheduled medical attendances for wheezing episodes.

149 rapeutic options to decrease the severity of wheezing exacerbations caused by respiratory viral infec

150 ue of the use of oral corticosteroids during wheezing exacerbations in preschool-aged children by dem

151 he patient presented with swollen eyelid and wheezing following combined intake of orange and aspirin

152 Participants with asthma and/or wheezing from 4 independent cohorts were included; Breat

153 A quarter of infants had recurrent wheezing (>/=3 episodes) and more frequent in the presen

154 Early life aeroallergen sensitization and RV wheezing had additive effects on asthma risk at adolesce

155 ffspring measured at 7 years of age (asthma, wheezing, hay fever, eczema, atopy, and total IgE).

156 sociated with an increased risk of recurrent wheezing (hazard ratio [HR] of 1.38 and 95% high-density

157 models including age, asthma diagnosis, and wheezing histories.

158 tory tract infection history during infancy, wheezing history to 5 age years, and ensuing maturation

159 associated with a greater risk of recurrent wheezing (HR, 1.76; 95% HDI, 1.15-3.27).

160 s into asthma ever, recent asthma, or recent wheezing illness (recent asthma or recent wheeze).

161 n were further validated using virus-induced wheezing illness and asthma phenotypes in an independent

162 Wheezing illnesses among young children are common and a

163 Early life rhinovirus (RV) wheezing illnesses and aeroallergen sensitization increa

164 ses that affect the development of recurrent wheezing illnesses and allergic sensitization.

165 us C (RV-C) species are more likely to cause wheezing illnesses and asthma exacerbations compared wit

166 n of CDHR3 protein, and an increased risk of wheezing illnesses and hospitalizations for childhood as

167 o new strategies for the prevention of viral wheezing illnesses and perhaps reduce the subsequent ris

168 Sex influences the risk of wheezing illnesses and the prevalence of asthma througho

169 (eg, airway microbiome) promote more severe wheezing illnesses and the risk for progression to asthm

170 ds and the predominant microbes during acute wheezing illnesses are both associated with the subseque

171 Early life aeroallergen sensitization and wheezing illnesses associated with virus and bacterial i

172 Wheezing illnesses cause major morbidity in infants and

173 RSV-induced wheezing illnesses during infancy influence respiratory

174 The etiology and timing of viral wheezing illnesses during the first 3 years of life were

175 isease in childhood and is often preceded by wheezing illnesses during the preschool years.

176 Viral respiratory infections can cause acute wheezing illnesses in children and exacerbations of asth

177 Viral infections are closely linked to wheezing illnesses in children of all ages.

178 mage and yet is a significant contributor to wheezing illnesses in young children and in the context

179 was most strongly associated with outpatient wheezing illnesses with RV and aeroallergen sensitizatio

180 During wheezing illnesses, detection of rhinoviruses and predom

181 nd early life stress may influence childhood wheezing illnesses, potentially through effects on immun

182 The associations between viral wheezing illnesses, presence and pattern of aeroallergen

183 330 mutation could be a risk factor for RV-C wheezing illnesses.

184 MSP increases risk of asthma and wheezing in adolescence; mechanisms go beyond reducing l

185 rnal asthma was a stronger predictor of ever wheezing in boys (odds ratio [OR], 2.15; 95% CI, 1.74-2.

186 was no effect on rates of medically attended wheezing in children aged 1-3 years (190 [14.9%] of part

187 d-dose prenatal vitamin D supplementation on wheezing in children at the age of 3 years extends the f

188 ed significantly increased risk of recurrent wheezing in children with profile A (hazard ratio, 2.64;

189 detect atopy in individuals with asthma and wheezing in cohorts with different age groups and could

190 breastfeeding on respiratory infections and wheezing in early infancy.

191 linked to the pathogenesis of viral-induced wheezing in early life.

192 Wheezing in infancy has been associated with subsequent

193 causes respiratory diseases, including acute wheezing in infants, of which life-threatening cases hav

194 ironmental factors associated with recurrent wheezing in inner-city environments.

195 lementation in pregnancy on asthma/recurrent wheezing in offspring (P for interaction = .77).

196 ory tract infection and subsequent long-term wheezing in term infants.

197 ciated with an increased risk for persistent wheezing in the child until the age of 5.

198 Wheezing in the children was annually examined by using

199 tions and has been associated with decreased wheezing in the first years of life.

200 Wheezing in the last 12 months was reported by 10.7% of

201 intake during pregnancy may protect against wheezing in the offspring, but the preventive effect of

202 rgic rhinitis increased, whereas the rate of wheezing in the past 12 months decreased from 2006 to 20

203 ple per household, US region, and history of wheezing in the past year), household endotoxin level wa

204 The incidence of asthma and recurrent wheezing in their children at age 3 years was lower by 6

205 egnancy is associated with increased odds of wheezing in their children during the first 6 years of l

206 associated with increased risk of atopy and wheezing, in this study only among females.

207 risk factors facilitating severe asthma and wheezing, including airborne viruses, smoke, indoor damp

208 ontroversies in the diagnostic evaluation of wheezing infants.

209 s predicting the development of asthma among wheezing infants.

210 ild cohort LINA for whom maternal stress and wheezing information was available (n = 443).

211 Infantile wheezing is a common problem, but there are no guideline

212 Rhinovirus-induced wheezing is an important risk factor for recurrent wheez

213 Wheezing is common in childhood.

214 Childhood wheezing is common particularly in children under the ag

215 Early childhood wheezing is common, but predicting who will remit or hav

216 Although infantile wheezing is common, there is a paucity of evidence to gu

217 ciation of breastfeeding with reduced asthma/wheezing is supported by the combined evidence of existi

218 = 1.2; 95% CI: 1.0, 1.5, respectively), and wheezing lasting >/= 2 days, resulting in a doctor visit

219 tically deregulated and could be linked with wheezing later in children's life.

220 more likely to experience severe attacks of wheezing limiting speech, 10.14-fold (1.27-81.21) more l

221 Variable patterns of childhood wheezing might indicate differences in the cause and pro

222 noea (n=513 [84%]), cough (n=500 [81%]), and wheezing (n=427 [70%]); 294 (48%) patients had fever.

223 f boiled jellyfish, he experienced erythema, wheezing, nausea, and abdominal pain.

224 CI], 0.06 [0.01-0.12]) and increased risk of wheezing (odds ratio [95% CI], 1.07 [1.00-1.14], per Z s

225 gative ARI, were more likely to present with wheezing (odds ratio [OR], 1.7; 95% CI, 1.23-2.35; P < .

226 predicted asthma in the PARS included early wheezing (odds ratio [OR], 2.88; 95% CI, 1.52-5.37), sen

227 sociations seen for PM2.5 with prevalence of wheezing {odds ratio (OR)=1.16 per 5mug/m(3) [95% confid

228 toms, children infected with RV enrolled for wheezing or acute rhinitis had similar viral loads in th

229 demonstrable change in self-reported current wheezing or asthma (adjusted odds ratio 0.81, 95% CI 0.6

230 of the prevalence and severity of recurrent wheezing or asthma in children aged up to 6 years.

231 utcome of interest was self-reported current wheezing or asthma, defined as having medicines prescrib

232 associated with offspring hayfever, eczema, wheezing or asthma.

233 After confirming that there was no wheezing or respiratory symptoms, the lung sound spectru

234 etween-group differences in the incidence of wheezing or shortness of breath.

235 chronic bronchitis or COPD, and a history of wheezing or use of respiratory inhalers in the last 12 m

236 pite this, they had similar rates of current wheezing (OR = 0.93, 95% CI = 0.65-1.32) and were less l

237 ure were significantly more likely to report wheezing (OR = 1.92; 95% CI: 1.32, 2.79); headaches (OR

238 zziness (OR = 4.21; 95% CI: 2.69, 6.58), and wheezing (OR = 4.20; 95% CI: 2.86, 6.17).

239 1.1-1.9), and 26% versus 17% reported recent wheezing (OR, 1.7; 95% CI, 1.3-2.4).

240 a (OR, 1.06 [95% CI, 0.99-1.14]; n = 14438), wheezing, or allergy.

241 with asthma symptoms; and days of coughing, wheezing, or chest tightness) across 6, 9, and 12 months

242 ever (OR, 1.4; 95% CI, 1.05-1.8) and recent wheezing over the last 12 months than full-term control

243 was significantly associated with recurrent wheezing (P </= 0.01).

244 aire symptom scores (p=0.037), and increased wheezing (p=0.018), but no evidence of an association wi

245 ations with the number of wheezing episodes, wheezing patterns, and physician-diagnosed asthma at 6 y

246 Childhood wheezing phenotype was related to 1989, 1995, 2001, and

247 ssociated with both transient and persistent wheezing phenotypes (adjusted odds ratio [aOR] = 1.64; 9

248 g was assessed at each age, and longitudinal wheezing phenotypes (early-transient, late-onset, persis

249 nship between TRAP exposure and longitudinal wheezing phenotypes and asthma at age 7 years.

250 l milk fatty acid composition with childhood wheezing phenotypes and asthma up to age 13 years using

251 The relationship between TRAP exposure and wheezing phenotypes and asthma was examined.

252 Adjusted risk ratios with parent-reported wheezing phenotypes and doctor-diagnosed asthma were com

253 rm-born subjects, the natural history of the wheezing phenotypes and the influence of early-life fact

254 new insights into the physiology underlying wheezing phenotypes based on age of onset and duration o

255 We characterized childhood wheezing phenotypes from infancy to adolescence and thei

256 teristics were similarly associated with the wheezing phenotypes in both groups, the preterm-born gro

257 We used data-driven methods to define wheezing phenotypes in preterm-born children and investi

258 COPSAC study or with asthma or virus-induced wheezing phenotypes in the COAST study.

259 early-life factors and characteristics with wheezing phenotypes was similar between preterm- and ter

260 Four wheezing phenotypes were defined for both groups: no/inf

261 Six wheezing phenotypes were identified: never/infrequent, p

262 isms are associated with asthma and specific wheezing phenotypes; that is, most SNPs are associated w

263 sociated with an increased risk of preschool wheezing (pooled odds ratio [pOR], 1.34; 95% CI, 1.25-1.

264 A reliable asthma diagnosis is difficult in wheezing preschool children.

265 ed to derive phenotypes based on patterns of wheezing recorded at up to 14 time points from birth to

266 All-cause, respiratory-related, and asthma/wheezing-related 5-year average cumulative costs were me

267 t setting and respiratory-related and asthma/wheezing-related costs.

268 he development of allergic sensitization and wheezing respiratory tract illnesses caused by viruses a

269 Intensive MDA had no effect on wheezing (risk ratio [RR] 1.11, 95% confidence interval

270 hma (RR 0.99 [95% CI: 0.77-1.27], P = 0.95), wheezing (RR 1.02 [95% CI: 0.89-1.17], P = 0.76) or rhin

271 flow obstruction characterised clinically by wheezing, shortness of breath, and coughing.

272 isodic nonproductive cough with intermittent wheezing since birth.

273 Primary outcomes were recent wheezing, skin prick test positivity (SPT), and allergen

274 ed history of diagnosis by a physician or by wheezing symptoms in the preceding 12 months.

275 ripheral eosinophil counts (P = .03) but not wheezing symptoms, baseline spirometric indices, or resp

276 ere not associated with an asthma diagnosis, wheezing symptoms, lung function measures, or prior sick

277 tion of infants with recurrent or persistent wheezing that is not relieved or prevented by standard t

278 Early childhood-onset wheezing that persists into adolescence represents the c

279 nfant biomarkers to the history of recurrent wheezing, the Asthma Predictive Index and its subsequent

280 Among children with wheezing, those in the complete mite sensitization traje

281 t of physician-diagnosed asthma or recurrent wheezing through 3 years of age and (2) third trimester

282 zed to vitamin C had significantly decreased wheezing through age 1 year (15/70 [21%] vs 31/77 [40%];

283 Secondary outcomes included incidence of wheezing through age 1 year and PFT results at age 1 yea

284 Asthma and recurrent wheezing until age 3 years were recorded.

285 ants were followed up for medically attended wheezing until they reached age 3 years.

286 Parental-reported wheezing was assessed at each age, and longitudinal whee

287 TSLP to allergic sensitization and recurrent wheezing was conducted in the birth cohort from the Urba

288 A physician's diagnosis of asthma/recurrent wheezing was noted in 67%, and 51% were receiving regula

289 d serum samples from 121 children with acute wheezing were analyzed by means of serology.

290 The odds for wheezing were increased if children who tested positive

291 it inflammation have efficacy for RV-induced wheezing, whereas the anti-RSV mAb palivizumab decreases

292 ren have high rates of allergic diseases and wheezing, which are diseases associated with type 2-bias

293 infection is a common trigger for childhood wheezing, which is a risk factor for subsequent asthma d

294 , aged 1 to 16 years, admitted for asthma or wheezing who identified as African American (n = 441) or

295 1), eczema (adjusted OR, 4.8; P = .014), and wheezing with rhinovirus (adjusted OR, 5.0; P = .035).

296 One vaccinee had grade 2 wheezing with rhinovirus but without concurrent LID/Delt

297 When adjusted for all viral etiologies, wheezing with RV (odds ratio = 3.3; 95% CI, 1.5-7.1), bu

298 hing was reported nearly 46% more often than wheezing, with 42.5% (17/40) coughing until the point of

299 on (defined as COPD exacerbation, tachypnea, wheezing, worsening bronchitis, worsening dyspnea, influ

300 ht be useful in disease modification for the wheezing young child at risk of persistent asthma.