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1 e STAT5-regulated milk genes casein beta and whey acidic protein.
2 land and regulated similar to the endogenous whey acidic protein.
3                            The expression of whey acidic protein and beta-casein and the amount of ac
4 ferentiation (eg, beta-casein, kappa-casein, whey acidic protein) and induced morphological different
5  decreased expression of alpha-lactoalbumin, whey acidic protein, and beta-casein, possibly because o
6 ression of the milk proteins beta-casein and whey acidic protein, and deficient lactation.
7 helial cells expressed abundant beta-casein, whey acidic protein, and WDNM1 mRNA, indicating a relati
8 , phosphorylation of STAT5 and expression of whey acidic protein are significantly reduced in the mam
9  protease inhibition activity in a number of whey acidic protein domain-containing proteins.
10 ecreased postlactational apoptosis, elevated whey acidic protein expression and aberrant pErk2 activa
11 87 amino acids) is an atypical member of the whey acidic protein family (WFDC12).
12 ility, neither EPPIN nor any closely related whey acidic protein four-disulfide core (WFDC) gene have
13                              WFDC1/ps20 is a whey acidic protein four-disulfide core member that exhi
14 recombinase under regulatory elements of the whey acidic protein gene (Wap).
15 owever, targeted expression of maspin by the whey acidic protein gene promoter inhibits the developme
16 MMP-3/stromelysin-1 under the control of the whey acidic protein gene promoter.
17  using the regulatory sequences of the mouse whey acidic protein gene.
18  expression of transmembrane ErbB-2 from the whey acidic protein-Her-2 cassette and its up-regulation
19 rminal, cysteine-rich (Cys-box) domain and a whey acidic protein-like (WAP) domain, followed by four
20 terminus of anosmin-1 (cysteine-rich region, whey acidic protein-like domain and the first fibronecti
21                                     However, whey acidic protein mRNA was reduced, and alpha-lactalbu
22 d alveolar buds and the induction of casein, whey acidic protein, PgR and C3.
23 cell (PcSC) subset and a more differentiated whey acidic protein-positive (WAP+) cell subset in mamma
24                TGF-beta1 expression from the whey acidic protein promoter (WAP) in triply transgenic
25 ession following pregnancy and involution in whey acidic protein promoter (WAP)-Cre/Rosa26-flox-stop-
26          Using Cre/LoxP technology, with the whey acidic protein promoter driving transgenic expressi
27 jected cells and transient activation of the whey acidic protein promoter-Cre gene during pregnancy a
28                                              Whey acidic protein promoter-driven HA-14-3-3zeta transg
29 mmary cancer were investigated utilizing the whey acidic protein promoter-T antigen transgenic mouse
30 mor virus (MMTV) long terminal repeat or the whey acidic protein promoter.
31 h wild-type human c-ErbB-2 (Her-2) under the whey acidic protein promoter.
32 ther the retinoblastoma gene promoter or the whey acidic protein promoter.
33                                  Analysis of whey acidic protein-transforming growth factor-alpha tra
34                  The ps20 protein contains a whey acidic protein-type four-disulfide core domain, whi
35                                          The whey acidic protein (WAP) domain is a conserved motif, c
36 is of AIDS for three distinct members of the whey acidic protein (WAP) family, secretory leukocyte pr
37                       One such region is the whey acidic protein (WAP) four-disulfide core domain loc
38 e distal region (-830 to -720 bp) of the rat whey acidic protein (WAP) gene contains a composite resp
39  driven by control sequences from the murine whey acidic protein (WAP) gene have been generated.
40 nsgene under control of the mammary-specific whey acidic protein (WAP) gene promoter or the mouse mam
41 e of interleukin-2, under the control of the whey acidic protein (WAP) gene promoter, exhibit aberran
42             Transgene targeting employed the whey acidic protein (WAP) gene promoter, which is expres
43 ium under the transcriptional control of the whey acidic protein (WAP) gene promoter.
44 ss we developed a transgenic model using the whey acidic protein (WAP) gene to direct expression of r
45 lular domain (Int3) under the control of the whey acidic protein (WAP) or mouse mammary tumor virus-l
46  targeted to mammary epithelial cells by the Whey Acidic Protein (WAP) promoter for overexpression.
47 this idea, the CRD-BP was expressed from the whey acidic protein (WAP) promoter in mammary epithelial
48 n CR-1 transgene under the regulation of the whey acidic protein (WAP) promoter in the FVB/N mouse ba
49 e established, in which the transgene is the Whey acidic protein (WAP) promoter linked to h-Int3sh.
50  targeted to the mammary gland by means of a whey acidic protein (WAP) promoter were characterized as
51 dings, truncated Int3 was expressed from the whey acidic protein (WAP) promoter, the activity of whic
52 ession of Bcl-2 in the mammary gland using a whey acidic protein (WAP) promoter-driven Bcl-2 transgen
53 ta-casein protein was inhibited 85%-100% and whey acidic protein (WAP) was undetectable.
54 genous MMTV as well as alpha-lactalbumin and whey acidic protein (WAP) were elevated.
55 d by the expression of abundant beta-casein, whey acidic protein (WAP), and WDNM1 mRNA.
56  its major components, alpha/beta-casein and whey acidic protein (WAP), is significantly reduced due
57  late pregnancy and lactation via use of the whey acidic protein (WAP)-Cre cre-lox system.
58                                          The whey acidic protein (WAP)-Cre-mediated deletion of ERalp
59 ene in the mammary glands of mice expressing whey acidic protein (Wap)-Int3.
60                                              Whey acidic protein (WAP)-transforming growth factor (TG