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1 e STAT5-regulated milk genes casein beta and whey acidic protein.
2 land and regulated similar to the endogenous whey acidic protein.
4 ferentiation (eg, beta-casein, kappa-casein, whey acidic protein) and induced morphological different
5 decreased expression of alpha-lactoalbumin, whey acidic protein, and beta-casein, possibly because o
7 helial cells expressed abundant beta-casein, whey acidic protein, and WDNM1 mRNA, indicating a relati
8 , phosphorylation of STAT5 and expression of whey acidic protein are significantly reduced in the mam
10 ecreased postlactational apoptosis, elevated whey acidic protein expression and aberrant pErk2 activa
12 ility, neither EPPIN nor any closely related whey acidic protein four-disulfide core (WFDC) gene have
15 owever, targeted expression of maspin by the whey acidic protein gene promoter inhibits the developme
18 expression of transmembrane ErbB-2 from the whey acidic protein-Her-2 cassette and its up-regulation
19 rminal, cysteine-rich (Cys-box) domain and a whey acidic protein-like (WAP) domain, followed by four
20 terminus of anosmin-1 (cysteine-rich region, whey acidic protein-like domain and the first fibronecti
23 cell (PcSC) subset and a more differentiated whey acidic protein-positive (WAP+) cell subset in mamma
25 ession following pregnancy and involution in whey acidic protein promoter (WAP)-Cre/Rosa26-flox-stop-
27 jected cells and transient activation of the whey acidic protein promoter-Cre gene during pregnancy a
29 mmary cancer were investigated utilizing the whey acidic protein promoter-T antigen transgenic mouse
36 is of AIDS for three distinct members of the whey acidic protein (WAP) family, secretory leukocyte pr
38 e distal region (-830 to -720 bp) of the rat whey acidic protein (WAP) gene contains a composite resp
40 nsgene under control of the mammary-specific whey acidic protein (WAP) gene promoter or the mouse mam
41 e of interleukin-2, under the control of the whey acidic protein (WAP) gene promoter, exhibit aberran
44 ss we developed a transgenic model using the whey acidic protein (WAP) gene to direct expression of r
45 lular domain (Int3) under the control of the whey acidic protein (WAP) or mouse mammary tumor virus-l
46 targeted to mammary epithelial cells by the Whey Acidic Protein (WAP) promoter for overexpression.
47 this idea, the CRD-BP was expressed from the whey acidic protein (WAP) promoter in mammary epithelial
48 n CR-1 transgene under the regulation of the whey acidic protein (WAP) promoter in the FVB/N mouse ba
49 e established, in which the transgene is the Whey acidic protein (WAP) promoter linked to h-Int3sh.
50 targeted to the mammary gland by means of a whey acidic protein (WAP) promoter were characterized as
51 dings, truncated Int3 was expressed from the whey acidic protein (WAP) promoter, the activity of whic
52 ession of Bcl-2 in the mammary gland using a whey acidic protein (WAP) promoter-driven Bcl-2 transgen
56 its major components, alpha/beta-casein and whey acidic protein (WAP), is significantly reduced due