ライフサイエンスコーパス: white blood cell

1 rase (LE), an enzyme produced by leukocytes (white blood cells).

2 evere disability and high levels of infected white blood cells.

3 ion of disseminated cells with platelets and white blood cells.

4 gnetophoresis to remove magnetically-labeled white blood cells.

5 an in adjacent unaffected adrenal tissue and white blood cells.

6 e suggested that lipids increase activity of white blood cells.

7 of illegitimate transcripts from peripheral white blood cells.

8 e3 and H3K27me3 target sites in single human white blood cells.

9 ed tissue), and injection of (111)In-labeled white blood cells 24 h earlier.

10 macrophages engulf large numbers of red and white blood cells, a process called hemophagocytosis.

11 tudy binding of human platelets and platelet-white blood cell aggregation.

12 een reduction in platelet count and platelet-white blood cell aggregation.

13 e size, we observed significant reduction of white blood cell and absolute lymphocyte count up to 1 y

14 White blood cell and absolute lymphocyte count were sign

15 ads to substantial increases in the clinical white blood cell and granulocyte count and is a well-doc

16 rval, fibrinogen level, factor VII level and white blood cell and platelet counts in 15 755 individua

17 CSF in immunocompetent hosts with normal CSF white blood cell and protein levels (</=5 cells/mm(3) an

18 inine clearance, calcium level, below-normal white blood cell and/or platelet count, polychemotherapy

19 leukocytes, we achieve 3.8-log depletion of white blood cells and a 97% yield of rare cells with a s

20 find more than 4,000 protein-coding mRNAs in white blood cells and adipose tissue to have seasonal ex

21 c images of patient blood samples containing white blood cells and CTCs.

22 ur new method in the classification of OT-II white blood cells and SW-480 epithelial cancer cells wit

23 Transcriptome analysis of white blood cells and T cells reveals strong signatures

24 r aim was to determine the minimal number of white blood cells and the specific abilities of mononucl

25 pathogenesis; for example, they lyse red and white blood cells and trigger inflammatory responses.

26 otropy, being also associated with platelet, white blood cell, and lipid traits.

27 detected at 5 years of age, at least 20% of white blood cells, and 44% at 8 years of age.

28 bundance cell types such as red blood cells, white blood cells, and platelets.

29 for triglyceride level, total protein level, white blood cells, and red blood cells.

30 hod to count red blood cells, platelets, and white blood cells, as well as to determine hemoglobin in

31 eads to a progressive increase in peripheral white blood cells, associated with increasing splenomega

32 e on average a reduced number of circulating white blood cells, because of the Duffy-null (CC) genoty

33 rmone (PTH), 25(OH)D, calcium and peripheral white blood cells broad gene expression were evaluated.

34 lineages by Pitx1, Pitx2, Barhl2, and Lmx1a; white blood cells by Myb, Etv2, and Tbx6, and ovary by P

35 ted a vertical flow platform that quantifies white blood cells by trapping them in the paper.

36 g dd-cfDNA to the proportion of donor DNA in white blood cells can differentiate between relapse and

37 ived from red blood cells (RBCs), platelets, white blood cells, cancer cells, and bacteria, exhibit p

38 ecovery of spiked cancer cells with very low white blood cell contamination (<1000).

39 cell count <13 x 10(9)/L) and proliferative (white blood cell count >/=13 x 10(9)/L) CMML.

40 te >90 bpm, mean arterial pressure <60 mmHg, white blood cell count >/=15 000 cells/mL, age >60 years

41 onvert to 109 per liter, multiply by 0.001); white blood cell count >/=15000/microL, 27% (95% CI, 18%

42 High-risk patients (those presenting with a white blood cell count >10 x 10(9) cells per L) could re

43 aboratory abnormality, commonly defined by a white blood cell count >100,000/microL, caused by leukem

44 oups: temperature >=39 degrees C, peripheral white blood cell count >=20 000/mm3, C-reactive protein

45 liferative chronic myelomonocytic leukaemia (white blood cell count <13 000/muL), and had anaemia wit

46 ed, CMML is stratified into myelodysplastic (white blood cell count <13 x 10(9)/L) and proliferative

47 Optimal thresholds for white blood cell count (11600/microL), absolute neutroph

48 ia (26 [29%]), anaemia (26 [29%]), decreased white blood cell count (17 [19%]), and decreased lymphoc

49 iarrhoea (12 [5%] vs 17 [7%]), and decreased white blood cell count (18 [7%] vs nine [4%]).

50 ients), leucopenia (27 [10%]), and decreased white blood cell count (21 [8%]).

51 count (31 [10%] vs 41 [13%]), and decreased white blood cell count (39 [13%] vs 33 [11%]).

52 (4.9 [4.0-5.8] vs 4.5 [3.7-5.5] mg/dL), and white blood cell count (7000 [5900-8200] vs 6600 [5600-7

53 eutropenia (31 [42%] vs 28 [39%]), decreased white blood cell count (8 [11%] vs 5 [7%]), and hypophos

54 ant was associated with a 29% lower baseline white blood cell count (95% confidence interval [CI], -7

55 prior skin infection (aOR 1.14), recent high white blood cell count (aOR 1.08), and genitourinary dis

56 02), bruising (aOR, 3.17; P=.0059), abnormal white blood cell count (aOR, 0.52; P=.0100), and prior a

57 s significantly higher in patients with high white blood cell count (HR 2.45, p 0.011), raised serum

58 s: age (hazard ratio [HR], 3.299; P < .001), white blood cell count (HR, 1.910; P = .017), platelet c

59 04 [95% CI, .006-.23], P < .0001); and lower white blood cell count (OR = 0.93 [95% CI, .89-.97], P <

60 ent of Model for End-Stage Liver Disease and white blood cell count (OR, 4.68; 95% CI, 1.80-12.17; P

61 1), whereas it was inversely associated with white blood cell count (P < 0.0001).

62 ower body mass index (BMI; P = .003), higher white blood cell count (P = .005), and higher D-dimer le

63 cose level, hypertension (each P < .01), and white blood cell count (P = .04).

64 eal inflammation, fluid, appendicoliths, and white blood cell count (WBC) were significantly correlat

65 White blood cell count also provides predictive informat

66 White blood cell count analysis after alpha-radioimmunot

67 ose episodes, 213 had data allowing complete white blood cell count analysis and were included in the

68 White blood cell count and C-reactive protein level were

69 the mean value of several laboratory tests (white blood cell count and hepatic and lipid panels), ye

70 ence, waist-hip ratio, alanine transaminase, white blood cell count and lower high-density lipoprotei

71 l fluid examination revealed mildly elevated white blood cell count and protein levels.

72 ses by urbanicity and effect modification by white blood cell count as an inflammatory marker were al

73 bin (MCHC) and platelet number at day 10 and white blood cell count at day 60.

74 te respiratory, cardiac, and liver function, white blood cell count at least 3 x 10(9) cells per L, p

75 36) and OS (HR, 0.64; P = .02), with initial white blood cell count being the only factor significant

76 In this large cohort of APL patients, high white blood cell count emerged as an independent predict

77 ven percent of patients with CDI had a serum white blood cell count greater than 12 000 cells per muL

78 Conclusions and Relevance: White blood cell count greater than 20000 cells/microL a

79 al bilirubin level greater than 10 mg/dL and white blood cell count greater than 20000 cells/microL.

80 The proposed platform enabled rapid white blood cell count in low resource settings with a s

81 far there has been no rapid test that allows white blood cell count in low-resource settings.

82 cholesterol in men, and with higher BMI and white blood cell count in women (differences 0.03-0.06 s

83 vein occlusion, n=1 each; placebo: vomiting, white blood cell count increased, n=1 each).

84 White blood cell count is an important indicator of each

85 Long-term monitoring of white blood cell count is compulsory in patients taking

86 Currently, white blood cell count is primarily conducted in central

87 roup (ECOG) performance status of 3 or less, white blood cell count less than 10 x 10(9) per L, and a

88 plete blood cell count parameter thresholds: white blood cell count less than 5000/microL, 10% (95% C

89 4 vs 0-2 and 5.20 (95% CI, 2.70-10.02) for a white blood cell count of >/=20 000/muL vs <20 000/muL.

90 esenting features (age >= 1 and < 10 years), white blood cell count of <50 x109/L, lack of extramedul

91 of 133 mg/L (normal range, 0.2-0.9 mg/L), a white blood cell count of 11.69 x10(9)/L (normal range,

92 He was found to have an elevated white blood cell count of 12.2 x 10(9)/L (reference rang

93 inent blood test results showed an increased white blood cell count of 13 000/muL (13 x10(9)/L) (norm

94 inent blood test results showed an increased white blood cell count of 13,000/uL (13 x 109/L) (normal

95 ratory studies revealed leukocytosis, with a white blood cell count of 15.1 x 10(3)/uL (15.1 x 10(9)/

96 ratory studies revealed leukocytosis, with a white blood cell count of 15.1 x 103/muL (15.1 x 109/L)

97 oratory analyses were notable for a complete white blood cell count of 17000/muL (31% blast cells), a

98 vel of 206 mg/L (normal range, 0-10 mg/L), a white blood cell count of 24.5 x 10(9)/L (normal range,

99 (n = 11), median age of 12 years, and median white blood cell count of 48.8 x 10(9)/L.

100 tologic laboratory investigations revealed a white blood cell count of 6.7 x 10(9), a C-reactive prot

101 ere within normal limits, including a normal white blood cell count of 6400/muL.

102 analysis of the validation cohort confirmed white blood cell count of more than 20000 cells/microL (

103 Therefore, monitoring white blood cell count on a regular basis can potentiall

104 globulins vs. late immunotherapy), and a low white blood cell count on the first cerebrospinal examin

105 significant interaction between PM(2.5) and white blood cell count only in the model of lung functio

106 factor positivity, and inflammatory markers (white blood cell count or cytokine level).

107 halopathy, longer length of stay, and higher white blood cell count or MELD score at discharge.

108 r both for clinical examination findings and white blood cell count parameters compared with a valid

109 uppression, defined as a decline in absolute white blood cell count to 2.5 x 109/L or less or a decli

110 An abnormal white blood cell count usually results from an infection

111 The patient's white blood cell count was 6.8 x 10(9)/L.

112 The highest quartile of white blood cell count was associated with lower lung fu

113 The white blood cell count was determined by measuring the c

114 White blood cell count was higher in the n-3 PUFA group

115 The median initial white blood cell count was significantly higher in patie

116 No significant differences in sex and white blood cell count were found.

117 Her coagulation profile and white blood cell count were within normal limits.

118 elevated total protein and a mildly elevated white blood cell count with lymphocytic predominance.

119 atients with eosinophil counts (out of total white blood cell count) of 2% or greater (rate ratio 1.2

120 2 laboratory markers (C-reactive protein and white blood cell count).

121 /m(2) on day 1) added to high-risk patients (white blood cell count, >10 x 10(9)/L), as well as low-r

122 Laboratory evaluation revealed leukocytosis (white blood cell count, 15.4 x 10(9)/L; normal range, [3

123 atase, 88.35 U/L (58.94-117.76 U/L); and for white blood cell count, 6890/microL (5700/microL-8030/mi

124 Lower levels of C-reactive protein, white blood cell count, absolute neutrophil count, and p

125 We tested the accuracy of the white blood cell count, absolute neutrophil count, and p

126 Lower levels of C-reactive protein, white blood cell count, absolute neutrophil count, and p

127 subset of patients with FLT3-ITD, only age, white blood cell count, and < 4-log reduction in PB-MRD,

128 Toxicity analysis included body weight, white blood cell count, and hematocrit.

129 core, red blood cell transfusion dependency, white blood cell count, and marrow blasts retained indep

130 cases had normal, the other two had elevated white blood cell count, but all of them had elevated CRP

131 lycerides, waist circumference), and immune (white blood cell count, C-reactive protein) markers.

132 Six biomarkers (white blood cell count, fibrinogen, D-dimer, troponin T,

133 is of hematological cancer requires complete white blood cell count, followed by flow cytometry with

134 th poor outcome (CSF culture positivity, CSF white blood cell count, hemoglobin, Glasgow Coma Scale,

135 performance status of two or more, increased white blood cell count, high-risk IPSS score, and higher

136 tors using tricuspid regurgitation velocity, white blood cell count, history of acute chest syndrome,

137 or 3 AKI included initial respiratory rate, white blood cell count, neutrophil/lymphocyte ratio, and

138 and >60 years and for high vs lower initial white blood cell count, no significant differences betwe

139 hypertension, C-reactive protein level, and white blood cell count, this association remained signif

140 ion, serum levels of albumin and sodium, and white blood cell count, to identify metabolites that dif

141 iles (C-reactive protein, plasma fibrinogen, white blood cell count, vitamin D, high-density lipoprot

142 ted prediction highlighted by our tool: that white blood cell count--a quantitative trait of the immu

143 pokalemia, decreased appetite, and decreased white blood cell count.

144 tween gene fusions, immunophenotypic groups, white blood cells count, gender or age.

145 ore (age, creatinine clearance, haemoglobin, white-blood-cell count, and previous spontaneous bleedin

146 Seventeen patients (42.5%) had CSF white blood cell counts >20/muL (mean, 57/muL), and 27 (

147 >=1 day (43.9% vs 36.6%; P < .0001), and had white blood cell counts >=15 000 cells/uL (31.4% vs 21.4

148 atients with severe COVID-19 had higher peak white blood cell counts (15.8 vs 7 x 10(3) /uL, P = .019

149 ep fragmentation raises inflammatory-related white blood cell counts (neutrophils and monocytes), the

150 This C. difficile variant elicited higher white blood cell counts and caused disease in younger pa

151 A content is also influenced by platelet and white blood cell counts and estroprogestogen intake.

152 AST and ALT, and negatively correlated with white blood cell counts and fibrinogen in free-ranging d

153 emonstrated severe thrombocytopenia, reduced white blood cell counts and high fever with 93% mortalit

154 ents, from oncology to psychiatry, can lower white blood cell counts and thus access to these treatme

155 Raised white blood cell counts as well as peaks of serum levels

156 high-dose group had significantly lower mean white blood cell counts at months 5 and 6; however, prem

157 was no significant difference in any of the white blood cell counts by diet group.

158 Red and white blood cell counts can also be performed on human b

159 lenged mice, UFP-exposed offspring had lower white blood cell counts in bronchoalveolar lavage fluid

160 The lower white blood cell counts observed in low and non-meat eat

161 e protected against viremia, fever, elevated white blood cell counts, and CHIKF-associated cytokine c

162 d aldosterone), hepatobiliary enzyme levels, white blood cell counts, and iron homeostasis.

163 nical information, complete and differential white blood cell counts, and lymphocyte subsets for 301

164 We examined hemoglobin, red and white blood cell counts, and platelet counts and volume

165 al use of G-CSF in these patients to support white blood cell counts, and suggest that direct targeti

166 telomere length increased red blood cell and white blood cell counts, decreased mean corpuscular hemo

167 etry, blood samples analyzed for hemoglobin, white blood cell counts, eosinophil counts and total ser

168 lmonary disease and had significantly higher white blood cell counts, lower lymphocyte counts, and in

169 etes and lower C-reactive protein levels and white blood cell counts.

170 suppressant FK506 (tacrolimus) decreases CSF white blood cell counts.

171 (30%) patients were afebrile and had normal white blood cell counts.

172 nogen, increasing platelet counts, and lower white blood cell counts.

173 e were no significant changes in hemoglobin, white blood cells, creatinine, or tubular extraction rat

174 White blood cell cystine concentration is the current go

175 to be significant independent predictors for white blood cell cystine levels in patients of all ages

176 extrarenal complications and was superior to white blood cell cystine levels in predicting the presen

177 om poor therapeutic control (on the basis of white blood cell cystine levels of <2 nmol 1/2 cystine/m

178 decreased (ten [16%] vs four [6%]; p=0.09), white blood cell decreased (15 [24%] vs seven [11%]; p=0

179 r depressed flows, endothelial inflammation, white blood cell-derived tissue factor, and ample red bl

180 Assessment of red blood cell integrity, white blood cell differential counts, and plasma biochem

181 approach yields 1) a quantitative five-part white blood cell differential, 2) quantitative red blood

182 normal progenitor self-renewal and defective white blood cell differentiation.

183 One white blood cell displayed the average esterolytic activ

184 ensity sequencing assay of cfDNA and matched white blood cell DNA covering a large genomic region (50

185 enic exposure and gene-specific differential white blood cell DNA methylation, suggesting that epigen

186 l arsenic concentrations, and epigenome-wide white blood cell DNA methylation.

187 ed and fixed cells, such as red blood cells, white blood cells, DU-145 prostate cancer cells, MCF-7 b

188 e ratio of Gr1(+) granulocytes in peripheral white blood cells following bacteremia.

189 or future investigations when alterations in white blood cell fractions are unavailable.

190 reduced ubiquitination activity in vitro and white blood cells from affected individuals exhibited si

191 nstrated improved enrichment and recovery of white blood cells from human blood.

192 at increases mobilization and trafficking of white blood cells from the bone marrow.

193 visually differentiate the abnormal count of white blood cells from the normal count.

194 ted sequencing analyses of matched cfDNA and white blood cells from the same patient.

195 ed by a simultaneous increase in circulating white blood cells, granulocytes, and interleukin 17A (IL

196 major blood cells (i.e., red blood cells and white blood cells) have a size distribution of 3-5 and 6

197 atures derived from genome-wide profiling in white blood cells, identifying 26 expression probes and

198 also demonstrate that accurate estimates of white blood cell images can be recovered from extremely

199 Neutrophils are the predominant circulating white blood cell in humans, and contain an arsenal of to

200 device proved effective to retain >99.9% of white blood cells in 100 mul of WB without affecting pla

201 sing this platform, we were able to quantify white blood cells in 15 muL of blood, and visually diffe

202 ormed a genome-wide DNA methylation study of white blood cells in a population-based study (N = 717).

203 For proof of principle, enumeration of the white blood cells in human blood samples on the RDM prov

204 fection marks neutrophils (the most abundant white blood cells in humans) as vital immune defenders.

205 n patterns of gene expression in circulating white blood cells in response to infection.

206 le cancer cell lines and 99.99% depletion of white blood cells in whole blood.

207 al fluid opening pressure of 28 mm H2O and 8 white blood cells, including 1 atypical plasma cell.

208 Observations noting the presence of white blood cell infiltrates within tumors date back mor

209 the model were: prothrombin activity, urea, white blood cell, interleukin-2 receptor, indirect bilir

210 nduced increase in lactate:pyruvate ratio in white blood cells is alleviated.

211 nce that transcript abundance in circulating white blood cells is associated with fertility in heifer

212 Whether ZIKV infection of white blood cells is required for dissemination of the v

213 ic loss of chromosome Y (LOY) in circulating white blood cells is the most common form of clonal mosa

214 In contrast to CD45(pos) white blood cells isolated and processed by the identica

215 mmune system through activation of a type of white blood cell known as natural killer T cell (NKT cel

216 helial cells/low-power field [lpf] and >/=25 white blood cells/lpf or a quality score [q-score] defin

217 Salsalate treatment decreased total white blood cell, lymphocyte, monocyte, and neutrophil c

218 Cerebrospinal fluid (CSF) pleocytosis (>5 white blood cells/microL) was common (81%).

219 asts could also be distinguished from benign white blood cells (notably these also lacked MDR activit

220 s (Siglecs) are expressed on the majority of white blood cells of the immune system and play critical

221 rine culture) plus pyuria (ie, any number of white blood cells on urinalysis) assessed every 2 months

222 h they showed significantly lower peripheral white blood cell (P = 0.014) and absolute lymphocyte (P

223 tification of an ESBL gene; (3) pyuria (>=10 white blood cells per high powered field in the urine);

224 adhesion between RBCs, and between RBCs and white blood cells, platelets, and the endothelium.

225 White blood cells play diverse roles in innate and adapt

226 Of note, neutrophils are the largest human white blood cell population.

227 reted by Staphylococcus aureus, which target white blood cells preferentially and consist of an S- an

228 tricted to individuals with normal levels of white blood cells, principally in order to minimize risk

229 regulated genes were linked to activation of white blood cells, production of cytokines, and inhibiti

230 There were concomitant increases in CSF white blood cells, protein, interferon-gamma (IFNgamma),

231 protein-coding genes expressed in peripheral white blood cells (PWBCs), and circulating micro RNAs in

232 perm from the original sample while removing white blood cells, replacing the seminal plasma, and red

233 nd 1289 genes up- or down-regulated in their white blood cells, respectively.

234 rrelation between PPP1R11 and TLR2 levels in white blood cell samples isolated from patients with Sta

235 decarboxylase (HDC) gene directly from human white blood cells samples.

236 ues such as positron emission tomography and white blood cell scintigraphy have been shown to reduce

237 o compared with available bone scintigraphy, white blood cell scintigraphy, and (18)F-FDG PET/CT resu

238 emphasizing the importance of matched cfDNA-white blood cell sequencing for accurate variant interpr

239 il (nine [20%]), platelet (eight [18%]), and white blood cell (seven [16%]) counts.

240 Patient bone marrow-derived neutrophils and white blood cells showed a severely impaired chemotactic

241 Furthermore, white blood cells showed defective in vitro killing of S

242 yglucose positron emission tomography/CT and white blood cell single photon emission CT/CT in a time

243 yglucose positron emission tomography/CT and white blood cell single photon emission CT/CT scans were

244 mography/CT and 60%, 100%, 100%, and 85% for white blood cell single photon emission CT/CT.

245 hy/computed tomography (CT) and radiolabeled white blood cells single photon emission CT/CT in a coho

246 Autoimmune diseases mediated by a type of white blood cell-T lymphocytes-are currently treated usi

247 of 115) and decreased neutrophil (16 [14%]), white blood cell (ten [9%]), and platelet (nine [8%]) co

248 Eosinophils are white blood cells that function in innate immunity and p

249 ay produce interacting epigenetic effects in white blood cells that influence immune function and hea

250 s (CACs) are an exercise-inducible subset of white blood cells that maintain vascular integrity.

251 od CD14(+) monocytes are bone marrow-derived white blood cells that sense and respond to pathogens.

252 sity of gold nanoparticles on the surface of white blood cells that were trapped in the paper mesh.

253 After filtering alterations from matched white blood cells, the presence of ctDNA predicts recurr

254 L and monocytes accounting for >/=10% of the white blood cells, this aging-associated disease combine

255 Further development of white blood cell transcriptional biomarkers for inflamed

256 nally interrogating the lipid metabolism and white blood cell transcriptomic markers in healthy, norm

257 We studied the relationship between major white blood cell types and blood pressure in the UK Biob

258 vidual plasma proteins decreased, as did the white blood cell types.

259 s the processing of a semen sample to remove white blood cells, wash away seminal plasma, and reduce

260 White blood cell (WBC) analysis provides rich informatio

261 age at time of cough onset, and higher peak white blood cell (WBC) and lymphocyte counts.

262 PCR rejection criteria, based on white blood cell (WBC) and platelet (PLT) counts, were d

263 An abnormal CSF diagnosis was defined as a white blood cell (WBC) count >20/uL, a CSF protein readi

264 White blood cell (WBC) count appears to predict total mo

265 e negative predictive values of a normal CSF white blood cell (WBC) count for ME panel targets were 1

266 Elevated white blood cell (WBC) count is associated with increase

267 avy marijuana use and HIV disease markers or white blood cell (WBC) count were examined using mixed-e

268 say, C-reactive protein (CRP) concentration, white blood cell (WBC) count, and absolute neutrophil ce

269 reactive protein (hsCRP), lipid profile, and white blood cell (WBC) count, at baseline and 1, 3, and

270 es were the oncogenetic classifier, MRD, and white blood cell (WBC) count.

271 L) patients with >30% bone marrow blasts and white blood cell (WBC) counts </=15 x 109/L (AZA-AML-001

272 ting, though CT-toxin(+) patients had higher white blood cell (WBC) counts (12.5 x 10(3) versus 9.3 x

273 Circulating white blood cell (WBC) counts (neutrophils, monocytes, l

274 lyses of phenotypic variation in circulating white blood cell (WBC) counts from large samples of othe

275 ariability (HRV) with electrocardiogram, and white blood cell (WBC) counts with hematology analyzer.

276 r 2 induction cycles were ELN risk group and white blood cell (WBC) counts; treatment with midostauri

277 Pleocytosis with white blood cell (WBC) levels of >=5 cells/mm(3) and >=1

278 ps between periodontitis, hours of sleep and white blood cell (WBC) markers among a nationally repres

279 brary consisting of over one quarter-million white blood cell (WBC) nuclei together with CD15/CD16 pr

280 ematocrit, hemoglobin, red blood cell (RBC), white blood cell (WBC), and platelet counts with an accu

281 Abnormal CSF was defined as white blood cells (WBC) >20/uL, CSF protein >50 mg/dL, o

282 higher levels of SPMs-RvD3, RvD4 and PD1-and white blood cells (WBC) and platelets.

283 ous titer, the intravenous administration of white blood cells (WBC) resulted in efficient disease tr

284 r confounders, in particular distribution of white blood cells (WBC), as well as random effects.

285 from mammalian cells, more specifically from white blood cells (WBC).

286 Conventional inflammatory markers (white blood cell [WBC] count, erythrocyte sedimentation

287 of Treponema pallidum 16S RNA in CSF or CSF white blood cells (WBCs) >20/uL or a reactive CSF-Venere

288 -associated antigen-1 (LFA-1; CD11a/CD18) on white blood cells (WBCs) and causing cell death.

289 Proper circulation of white blood cells (WBCs) in the pulmonary vascular bed i

290 White blood cells (WBCs) play an important role in host

291 l blood mononuclear cells (PBMCs) and of CSF white blood cells (WBCs) that were activated monocytes (

292 DNA methylation in white blood cells (WBCs) was associated with total IgE l

293 ed high iron intensities, whereas individual white blood cells were characterized by their high phosp

294 White blood cells were tagged with gold nanoparticles, a

295 ndividuals reflected nucleosomal patterns of white blood cells, whereas patients with cancer had alte

296 sidual disease negativity with CLL <1/10 000 white blood cells, which persisted even after ibrutinib

297 0% with a contamination of 0.6 +/- 0.1% from white blood cells with a 23.8 +/- 1.3-fold concentration

298 +/- 2.1% with 0.2 +/- 0.04% contamination of white blood cells with a 9.6 +/- 0.4-fold concentration

299 Eosinophils are phagocytic white blood cells with a variety of roles in the immune

300 , and produce an ultrapure buffy coat (96.6% white blood cell yield, 0.0059% red blood cell carryover