ライフサイエンスコーパス: white cell count

1 arthritis), MGA (blood pressure) and COMMD7 (white cell counts).

2 1 are older (median age 9 years), with a low white cell count.

3 itution of chemotherapy and normalization of white cell count.

4 e protein, haemoglobin, platelets, and total white cell count.

5 io of FLT3-ITD to wild-type FLT3 and for the white-cell count.

6 linical examinations, hemoglobin levels, and white cell counts.

7 t 60 h, through comparable CSF bacterial and white cell counts.

8 o, the S. typhi vaccination caused a rise in white cell count (11.1 +/- 0.5 x10(9)/l vs. 7.9 +/- 0.8

9 wice the upper limit of normal (2N) or more, white cell count 150 x 10(9)/L or more, abnormal chromos

10 White cell count (16.2 +/- 10.5 v 6.9 +/- 3.5 (x 109/L);

11 raised platelets (3.48, 3.35 to 3.62), total white cell count (3.01, 2.89 to 3.14), and C reactive pr

12 ducing the time of return to normalcy of the white cell counts after chemotherapy in patients with ac

13 Laboratory studies revealed a normal white-cell count, an international normalized ratio of m

14 inine, haemoglobin, potassium, sodium, urea, white cell count and an index NEWS undertaken within +/-

15 d with steroid withdrawal were reductions in white cell count and haemoglobin and increases in plasma

16 ransporter SPNS2 are associated with reduced white cell count and hearing defects.

17 aminotransferase (ALT), blood pressure, and white cell count and lower HDL cholesterol compared with

18 0.83 (0.73, 0.94) and 0.81 (0.69, 0.93) for white cell count and platelet count respectively.

19 Vaccination resulted in elevations in white cell count and serum levels of interleukin-6 and i

20 model identified age, NEWS, albumin, sodium, white cell count and urea as significant (p<0.001) predi

21 patients present at diagnosis with increased white cell counts and hepatosplenomegaly, and are at an

22 White cell counts and plasma lipoprotein profiles were s

23 cerebrospinal fluid comprising either raised white cell counts and/or raised levels of interferon-alp

24 Elevations in CSF white-cell count and protein were reported as adverse ev

25 e analysis that was adjusted for the initial white-cell count and the day of illness on which stool w

26 ls or plasmacytosis >20% of the differential white cell count) and WHO performance status 0-3.

27 Age, white cell count, and day of illness at study enrollment

28 ate analysis with cytogenetic category, age, white cell count, and French-American-British subtype de

29 Serum fibrinogen, white cell count, and high-sensitivity C-reactive protei

30 status, high fungal burden, high peripheral white cell count, and older age.

31 ing and beta(2)M, hemoglobin, prior therapy, white cell count, and platelet level (P =.005).

32 ncluding treponemal and lipoidal antibodies, white cell count, and protein concentration.

33 type, and Ho-Tr was independent of sex, age, white cell count, and T-cell status among Ph-negative pa

34 covery was associated with older ages, lower white cell counts, and earlier stages of illness at pres

35 transport variables, total and differential white cell counts, and serum concentrations of TNF and I

36 Levels of C-reactive protein, the white-cell count, and fibrinogen levels were strong pred

37 The fetal hemoglobin level, white-cell count, and platelet count and the use of hydr

38 Multivariate analysis incorporating age, white-cell count, and treatment parameters showed that s

39 n modeling defined a minimal model including white cell count at diagnosis, pretreatment cytogenetics

40 CRLF2-d was not associated with age, sex, or white cell count, but IGH@-CRLF2 patients were older tha

41 Preoperative acute phase proteins (white cell count, C-reactive protein, and albumin), cyto

42 elet count and hematocrit in addition to the white cell count during the first 3 months of therapy wi

43 n activator antigen, C-reactive protein, and white cell count, even after adjustment for possible con

44 s, LDL cholesterol, HbA(1c) (A1C), increased white cell count, ever having smoked, and previous retin

45 d glucose >150 mg/dL (8.2 mmol/L), admission white cell count >14,300 cells/mm3 (14.3 x 10(9) cells/L

46 In a murine MPN model, CYT387 normalized white cell counts, hematocrit, spleen size, and restored

47 were older, had higher admission anion-gap, white cell count, hemoglobin (hb), neutrophil/lymphocyte

48 lammation at 8 hours, reflected by increased white cell count in both sexes.

49 We determined differential white cell counts in peripheral blood of 189 adults who

50 clinical signs of encephalitis had elevated white cell counts in the blood caused mostly by increase

51 d in the highest and most sustained systemic white cell count increase.

52 ong patients with AML, independently of age, white-cell count, induction dose, and post-remission the

53 he prognostic significance of the presenting white cell count is weaker and the rate of decline in mi

54 arge were body mass index less than 28 kg/m, white cell count less than 15,000/mL, C-reactive protein

55 ate >90/minute, respiratory rate >20/minute, white cell count <4 x 10(9)/L or >/= 20 x 10(9)/L, album

56 APL classified as low-to-intermediate risk (white-cell count, </=10x10(9) per liter).

57 (median 9 years vs 5 years), and had a lower white cell count (median 3.9 vs 12.4) compared with chil

58 C-reactive protein (n = 2), albumin (n = 2), white cell count (n = 3), neutrophils (n = 2), and plate

59 and symptoms together with laboratory tests (white cell count, neutrophil count and C-reactive protei

60 ormal value, <8.0 mg/L [76.2 nmol/L]), and a white cell count of 7 x 10(9)/L (normal range, [4-11] x

61 dverse event in the entire cohort included a white-cell count of 200,000 per cubic millimeter or high

62 that fever, a virus-specific rash, and a CSF white-cell count of 5/microL or more were independent pr

63 ts one to nine years of age, all of whom had white-cell counts of at least 50,000 per cubic millimete

64 ALL who were either 1 to 9 years of age with white-cell counts of at least 50,000 per cubic millimete

65 The median cerebrospinal fluid white-cell count on the first lumbar puncture among pati

66 outcome, but either high cerebrospinal fluid white-cell counts or severe hyponatremia did.

67 specialty (medical versus surgical), raised white cell count, or co-morbidity.

68 There was a significantly higher white cell count (P = 0.014) and CRP (P = 0.004) on admi

69 Mutations were associated with a high white cell count (P =.006) and patients with inv(16) (P

70 to age, neutrophil JAK2 V617F allele burden, white-cell count, platelet count, or clonal dominance.

71 Decreases in white cell count, platelets and C-reactive protein were

72 aboratory data including CD4 cell count, CSF white cell count, protein, glucose, and quantitative cry

73 olytic-uremic syndrome were a higher initial white-cell count (relative risk, 1.3; 95 percent confide

74 specific effects on mortality and neutrophil/white cell counts (rho = 0.48), C-reactive-protein (rho

75 al antibodies) was 67.9%, whereas normal CSF white cell counts ruled out Lyme neuroborreliosis with a

76 of C-reactive protein (CRP), fibrinogen, and white cell count to components of IRS in the nondiabetic

77 was consistent with meningitis or if the CSF white cell count was >100 cells/mm(3) (>50% neutrophils)

78 Increased circulating white cell count was associated with increased neutrophi

79 rticipants with early ART initiation had CSF white cell count (WCC) >/=5/microL at day 14 (58% vs 40%

80 ts with culture-negative meningitis with CSF white cell count (WCC) above 20 cells per muL were inclu

81 Mean C-reactive protein (CRP), and white cell count (WCC) were significantly higher in the

82 Primary outcomes were CSF white cell count (WCC), CSF-to-serum albumin ratio, CSF

83 sedimentation rate (ESR), hemoglobin, total white cell count (WCC), estimated glomerular filtration

84 analysis adjusted for age, sex, haemoglobin, white cell count (WCC), platelet count, creatinine, and

85 re older or presented earlier and with lower white cell counts were more likely to have poor platelet

86 eactive protein, platelet aggregability, and white cell counts were not modified by losartan.

87 tive protein, and fibrinogen levels, and the white-cell count were measured at base line, along with

88 ACLF grade and white cell count, were independent predictors of mortali

89 ic utility of quantifying the synovial fluid white cell count, with two recent systematic reviews rea