戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (1語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 f melanocytes and the clinical appearance of white spots.
2 ease of the skin that results in disfiguring white spots.
3 atches of depigmentation that are visible as white spots.
4 of me(v)/me(v) mice revealed numerous, small white spots.
5 abs were demineralized to produce artificial white spots.
6 he retinal pigment epithelium (89.3%), small white spots (80.2%), and optic nerve atrophy (76.9%) wer
7                         Saponins appeared as white spots against a pink background.
8 ption, and was marked by the appearance of a white spot and a dark ring, coincident with entry into m
9  we observed highly penetrant hydrocephalus, white spotting and soft tissue syndactyly.
10 f esophagitis including oedema, granularity, white spots, and furrowing, while histology revealed oed
11  mature leaves suffered from burnt edges and white spots as well as a reduction in photosynthetic pig
12 ed a naive vNAR phage display library from a white-spotted bamboo shark (Chiloscyllium plagiosum), wi
13 ith progressive piebaldism, exhibit dominant white spotting but show no evidence of progressive depig
14 ents with TSC develop hypomelanotic macules (white spots), but the molecular mechanisms underlying th
15 the survival of these cells in vivo, because white spotting (c-kit(W/W)) mice, carrying a natural ina
16                   A low-caries group had no "white spot" caries (ws) in the selected dentition area;
17 cassava orthologous gene resulting in yellow-white spots characteristic of the inhibition of su expre
18 mestic cattle, the variants underpinning the white-spotted coat pattern of Holstein-Friesian and rela
19 caries-free subjects (SRS), (3) plaque from "white spot" coronal lesions and sound coronal surfaces o
20          Finally, we present a case study of white spot disease (WSD) in penaeid shrimp with rates of
21 that include chipping, demineralisation, and white spot formation.
22  virus susceptibility gene 2 (Fv2), dominant white spotting gene (W), and Steel gene (Sl), regulate t
23 normal a-wave and b-wave activity, yellowish-white spots, hyperfluorescence, and reduced retinal thic
24      We have used the varied expressivity of white spotting (hypopigmentation) observed in intrasubsp
25 n, which are defined clinically as yellowish white spots in the outer retina, are cardinal features o
26  biochemical evidence that hydrocephalus and white spotting in B3glct mutants resulted from loss of A
27 s generalized hypopigmentation and localized white-spotting in mice, with a lack of pigment on the be
28 es that differentiate this entity from other white spots, including acute placoid multifocal pigment
29  moderate certainty) and in the incidence of white spot lesions (risk ratio, 0.48; 95% CI, 0.35 to 0.
30                                              White spot lesions (WSL) due to enamel demineralization
31                                              White-spot lesions (WSL) associated with orthodontic app
32                                 Using enamel white-spot lesions, we hypothesize that the optical scat
33  appliances carrying an enamel slab cut from white-spot lesions.
34 estigating mice with mutations affecting the white spotting locus (which encodes KIT) or the steel lo
35 e have a naturally occurring mutation in the white spotting locus that causes reduced c-Kit receptor
36           Adamts20 mutations cause the mouse white-spotting mutant belted (bt), whereas Adamts9 is es
37 nlight (mnlt), a second hypopigmentation and white-spotting mutation identified on the C57BL/6J backg
38 que from those described for other classical white-spotting mutations.
39 descriptions of a dilution of coat color and white spotting of the belly and extremities, suggesting
40 vitiligo and persist to maintain disease, as white spots often recur rapidly after discontinuing ther
41  stage caries lesions typically present as a white spot on a white background, resulting in many lesi
42 he silencing phenotype ranged from scattered white spots on the normal purple background to entirely
43 ommon autoimmune conditions characterized by white spots on the skin (vitiligo) and bald spots on the
44 r its chalky lavender wings with conspicuous white spots on the ventral wings.
45 natal lethality, with viable mice exhibiting white spotting on their ventral surface.
46 e report that a point mutation, the dominant white spotting oncogene allele, Kit(W-42J), exacerbates
47 a pronounced forehead blaze, with additional white spots over the cervical region, as well as a very
48 lication to ancient DNA data associated with white spotting patterns in horses.
49    Transgene induction in skin resulted in a white spotting phenotype due to somatic ORFeus-mediated
50 us that were exclusively present in white or white-spotted pigs, carrying the Dominant white, Patch,
51 traits with mendelian inheritance: the major white spotting (S) locus and the hair ridge in Rhodesian
52 nal detachment of the fovea accompanied with white spots surrounding the fovea in both eyes.
53                           Exposure to virus (white spot syndrome baculovirus or M. rosenbergii nodovi
54                                              White Spot syndrome virus (WSSV) causes rapid shrimp mor
55                                              White spot syndrome virus (WSSV) is one of the major and
56        In this study, the complete genome of white spot syndrome virus (WSSV) was reconstructed from
57 mmune defense toward a major viral pathogen, white spot syndrome virus (WSSV).
58  vannamei hemocytes following infection with white spot syndrome virus (WSSV).
59 ons of circular RNAs (circRNAs) derived from white spot syndrome virus (WSSV).
60 tance to infection by two unrelated viruses, white spot syndrome virus and Taura syndrome virus.
61 susceptibility of this nephrocomplex to both white spot syndrome virus and Vibrio infection compared
62                             Viruses, such as white spot syndrome virus, and bacteria, such as Vibrio
63 includes vertebrates, nonvertebrates, shrimp white spot syndrome virus, Streptococcus equi, and Bacil
64 d, accurate, single copy detection assay for White Spot Syndrome Virus, the most devastating virus im
65 retinal vasculitis, a characteristic retinal white spot syndrome, Bartonella retinitis, branch retina
66  diseases of the posterior fundus, including white spot syndromes and autoimmune, hereditary, paraneo
67 ilar presentations using the terms MEWDS and white spot syndromes.
68 estruction of epidermal melanocytes produces white spots that can be repigmented by melanocyte precur
69 varian cancer risk using germ cell-deficient white-spotting variant (Wv) mice, incorporating oncogeni
70 us (IHHNV), a single-stranded DNA virus, and white spot virus (WSV), a double-stranded DNA (dsDNA) vi
71 clones from a hepatopancreas cDNA library of white spot virus (WSV)-infected shrimp provided a partia
72  lacking a SCF-responsive population of HPC [White spotted (W/WV) and Steel (SI/SId)] are unresponsiv
73 are encoded in close proximity at the murine white spotting (W) and patch (Ph) loci.
74 its receptor, c-kit, encoded by the dominant white spotting (W) gene.
75       Mice carrying certain mutations in the white spotting (W) locus (ie, c-kit) exhibit reduced c-k
76        Finally, mice having mutations in the white spotting (W) locus, which encodes the SCF-receptor
77 rtile mouse strains, Steel (Sl) and dominant white spotting (W), to determine if stem cells from an i
78  leading these mutants to be termed dominant White spotting (W).
79 the c-kit receptor tyrosine kinase (dominant white spotting, W).
80 nary night blindness characterized by yellow-white spots, which were classically described as subreti