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1 each aphids) and Bemisia tabaci (silver leaf whitefly).
2 resulting in elevated cAMP levels within the whitefly.
3 ons in CMD-infected cassava and viruliferous whiteflies.
4 ent acquired and subsequently transmitted by whiteflies.
5 )' for this new endosymbiont associated with whiteflies.
6 y exacerbate MYMIV impact by attracting more whiteflies.
7 y susceptible tomato and by local and exotic whiteflies.
8 storical tritrophic relationships in Bemisia whiteflies.
9 GS-2 episomes were also found in virions and whiteflies.
10 nto virions and transmitted as an episome by whiteflies.
11 ed structural constituent of cuticle in male whiteflies.
12 primary endoparasitoids of scale insects and whiteflies.
13 ptera, such as scale insects, mealybugs, and whiteflies.
14 plant resistance against begomoviruses, high whitefly abundance, and whitefly's ability to develop in
18 phidomorpha), scale insects (Coccidomorpha), whiteflies (Aleyrodomorpha) and psyllids (Psylloidea).
19 arental cassava varieties, ECU72 tolerant to whiteflies and COL2246 a susceptible line, have been use
22 Hemiptera), including aphids, planthoppers, whiteflies and stink bugs, present one of the greatest c
23 r respective probabilities of acquisition by whiteflies and to the titers of each segment acquired an
24 t population values of 26.3 and 39.2 mug per whitefly and 23.1 and 35.2 mug per aphid for BGL-1 and u
25 s are that cassava genotypes possessing both whitefly and disease resistances are needed urgently.
26 emonstrates how begomovirus retention within whitefly and its transmission can be modulated by alteri
28 ther species, females are endoparasitoids of whiteflies, and males are primary endoparasitoids of egg
32 d performance and sex-ratio bias of infected whiteflies are sufficient to explain the spread of Ricke
33 )-specific phosphodiesterase-4 (PDE4) of the whitefly as an interacting partner with capsid proteins
36 a PGRP gene, BtPGRP, encodes a PGRP from the whitefly Bemisia tabaci (MEAM1) that binds and kills bac
38 dy, the gender difference in the sweetpotato whitefly Bemisia tabaci was investigated using Illumina-
40 um of whiteflies, including the sweet potato whitefly Bemisia tabaci, and provides essential nutrient
41 tivated during feeding by a pest insect, the whitefly Bemisia tabaci, and the resulting hydrogen cyan
42 pathogen of tomato crops, is vectored by the whitefly Bemisia tabaci, yet the mechanisms underlying T
45 that influence the defense mechanism against whitefly (Bemisia tabaci Asia II 7) and leafminer (Phtho
46 egomoviruses, transmitted by the sweetpotato whitefly (Bemisia tabaci Gennadius), are the causal agen
48 development of the phloem-feeding silverleaf whitefly (Bemisia tabaci type B; SLWF) on Arabidopsis (A
50 roxyfen in 84 populations of the sweetpotato whitefly (Bemisia tabaci) from cotton fields in central
54 ly driven by two so-called supervectors: the whitefly, Bemisia tabaci, and the Western flower thrips,
55 transmitted specifically by the sweetpotato whitefly, Bemisia tabaci, in a semipersistent manner.
59 the management of begomoviruses by targeting whitefly cAMP using chemicals, botanicals, or RNAi-based
60 n dynamics and genetic patterns by comparing whiteflies collected on cassava in 1997, during the firs
61 7, a putative species of the Bemisia tabaci whitefly complex (Hemiptera: Aleyrodidae), and occasiona
65 of individual fields with varying levels of whitefly density crops under low and high disease pressu
66 formation indicating an involvement of SA in whitefly-derived plant defence against Agrobacterium.
68 urce plant and the ratios transmitted by the whitefly did not differ from one - the ratio at which th
69 both viruses, DNA-A:DNA-B ratios acquired by whiteflies differed from those in the source plant and t
70 a mosaic begomovirus (CMB) in Bemisia tabaci whitefly, diminished the perceived importance of whitefl
72 mutant CPm genotypes were not transmitted by whiteflies, even though virion concentrations were above
75 ns or recombinant virus capsid components to whiteflies, followed by feeding them antibodies to the v
76 ing RNA interference (RNAi) to down regulate whitefly genes by expressing their homologous double str
79 RiTBi may be a relatively recent intruder in whiteflies given its low abundance in the field and rela
81 resistance and the transmission of SEGS-2 by whiteflies has major implications for the long-term dura
82 eptional horizontal gene transfer event, the whitefly has acquired the plant-derived phenolic glucosi
84 y immunity and provides insight into how the whitefly immunity acts in complex mechanisms of Begomovi
85 ork addresses a visible gap in understanding whitefly immunity and provides insight into how the whit
88 obligate primary endosymbiotic bacterium of whiteflies, including the sweet potato whitefly Bemisia
90 358 genome-wide SNPs from 62 Bemisia cassava whitefly individuals belonging to sub-Saharan African sp
95 ana leaves were exposed to a sucking insect (whitefly) infestation and benzothiadiazole (BTH) for 7 d
98 acterium-mediated transformation of roots of whitefly-infested plants was clearly inhibited when comp
101 , Hemiptera (cicadas, aphids, scale insects, whiteflies, leafhoppers, and bugs), Coleoptera (beetles)
103 ing of virus transmission by aphids, thrips, whiteflies, leafhoppers, planthoppers, treehoppers, mite
104 Disruption of genes/pathways critical for whitefly-mediated transmission can be effective for the
105 The results of this study demonstrate that whitefly-mediated transmission of begomoviruses is regul
108 by real time quantitative PCR indicated that whitefly mortality was attributable to the down-regulati
116 used worldwide for the biological control of whiteflies on vegetables and ornamental plants grown in
117 immature E. formosa, population dynamics of whitefly-parasitoid interactions, and commercial use in
118 enables a wide range of hypotheses regarding whitefly phytopathology without the expense, facilities,
121 , has potential as a bio-pesticide to reduce whitefly population size and thereby decrease virus spre
122 rtite begomoviruses was facilitated by local whitefly populations and the highly susceptible tomato h
127 h uninfected whiteflies, Rickettsia-infected whiteflies produced more offspring, had higher survival
132 The F1 generation has been screened for whitefly resistance, and progeny identified displaying e
133 ures generated clustering of accessions into whitefly resistant and susceptible groups, and different
134 fed to whitefly vectors, significantly more whiteflies retained the recombinant minor coat protein (
136 begomoviruses, high whitefly abundance, and whitefly's ability to develop insecticide resistance rap
139 uses in cassava linked to high abundances of whitefly species within the Bemisia tabaci complex.
140 ED and NW1) were assessed on the four lines, whitefly species-specific responses to R1, R2 and R3 pla
141 merged well before the introduction of MEAM1 whiteflies, suggesting that the evolution of NW monopart
144 to Rickettsia_bellii_MEAM1 found earlier in whiteflies, the new Rickettsia species has more gene fam
146 the hijacking of microbial genes has allowed whiteflies to develop a highly economic and stable nitro
150 e generation of a bi-parental population for whitefly tolerance/susceptibility enabled the identifica
152 on the biology of the virus (host range and whitefly transmission) will be useful in developing and
155 ava mosaic disease is caused by a complex of whitefly-transmitted begomoviruses, which often occur in
156 ANCE Worldwide, diseases of tomato caused by whitefly-transmitted geminiviruses (begomoviruses) cause
160 at controlling pests such as the greenhouse whitefly Trialeurodes vaporariorum (Westwood) which are
162 llected on cassava in 1997, during the first whitefly upsurges in Uganda, with collections made in 20
163 y: Geminiviridae, Genus: Begomovirus) by the whitefly vector Bemisia tabaci MEAM1 (Gennadius) in toma
164 zed in the anterior foregut or cibarium of a whitefly vector biotype but not within those of a whitef
168 strated previously and in the present study, whitefly vectors failed to transmit virions preincubated
169 g host range, semipersistent transmission by whitefly vectors, and impact on diverse cropping systems
170 V capsid components were individually fed to whitefly vectors, significantly more whiteflies retained
173 it on samples from fission yeast, mouse and whitefly, whose reference genome is not yet available.
174 t multigenic and multi-faceted resistance to whiteflies with each B. tabaci species displaying distin