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1 during the dark in both the Crx(-/-) and the wild type mouse.
2 c, NPS 2143, inhibited secretion only in the wild-type mouse.
3 hereas a blunted response is observed in the wild-type mouse.
4 en fluorescent protein (GFP) (delta neo) and wild-type mouse.
5 gp91(phox-/-)) mice as much as it was in the wild-type mouse.
6 that are between 9 and 40% of the Gclm(+/+) wild-type mouse.
7 hages decreased with age at 18 months in the wild-type mouse.
8 t mouse was approximately double that of the wild-type mouse.
9 conditions in which it had no effect in the wild-type mouse.
10 0-fold lower in the mutant mouse than in the wild-type mouse.
11 the ADG to the levels found in the HG of the wild-type mouse.
12 from Osx-expressing cells isolated from the wild-type mouse.
13 m-operated site and the sites in the injured wild-type mouse.
14 ansplantation of skin from a transgenic to a wild-type mouse.
16 GAD67-GFP (Deltaneo) mouse compared with the wild type mouse after prolonged exposure to kainate.
17 e and barrier function compared with that of wild-type mouse, after tape stripping, S1pr2(-/-) mouse
19 eral lines of transgenic mice overexpressing wild-type mouse amyloid precursor protein (moAPP) either
20 eous Ca(2+) leak both in cardiomyocytes from wild-type mouse and from a gene-targeted mouse model of
22 tiserum to the C-terminus (CT-2) labels both wild-type mouse and human Purkinje cell nuclei, but not
24 mical detection of endogenous alpha-syn in a wild-type mouse and transgenic SNCA-A30P alpha-syn prote
25 G1alpha disulfide formation was increased in wild-type mouse aortas by in vivo nitroglycerin treatmen
27 cess, we determined the crystal structure of wild-type mouse apoE, which, like apoE4, forms a four-he
28 dy used a beta(3) integrin (-/-) mouse and a wild-type mouse as a control for in vivo PO by transvers
31 the causative bacterial species in different wild-type mouse backgrounds as well as in knockout, tran
33 0.5 microg/ml human G117H BChE, 2 microg/ml wild-type mouse BChE, and 0.06 microg/ml wild-type mouse
35 duce the formation of PrP(Sc) molecules from wild type mouse brain homogenate substrate in serial pro
37 protein profiles between KIBRA knockout and wild-type mouse brain showed significantly decreased Rab
39 nerating regions of pcd mouse brain, but not wild-type mouse brain, show elevated autophagy, which ca
40 these peptides, designated WE-15, exists in wild-type mouse brain, thus validating the approach to i
44 mceph mouse brains are enlarged and - unlike wild-type mouse brains - they keep growing throughout ad
45 significantly decreased in BACE1-null versus wild-type mouse brains, remaining unchanged in BACE1-het
49 mensional structure of myosin filaments from wild-type mouse cardiac muscle and from a MyBP-C knockou
52 yo fibroblasts from Fbxw7-deficient mice, or wild-type mouse cells expressing Fbxw7 small interfering
53 d3(-/-) mouse cells migrate more slowly than wild-type mouse cells, a change in cell behaviour that p
55 roblasts with kinetics equivalent to that of wild-type mouse CMV, did not negatively affect replicati
57 ation of Angptl4 and Adrp mRNA expression in wild-type mouse colon but not in Pparbeta/delta-null mou
58 expression patterns for Cdx-2 and PTEN along wild-type mouse colon, as well as in colon tumors occurr
59 napA strain showed normal (like that of the wild type) mouse colonization efficiency in the conventi
61 alization and enhanced cell proliferation of wild-type mouse corneal epithelial cells in an organ cul
63 lantation and naked MT1-MMP DNA injection in wild-type mouse corneas compared with either bFGF pellet
64 l electrophoresis were present in mutant and wild-type mouse cortex and hypothalamus at comparable le
66 the only two sites basally phosphorylated in wild-type mouse cTnI with full sequence coverage, which
68 ouse-derived brown adipocytes, compared with wild-type mouse-derived brown adipocytes, displayed an i
70 rs analysed DNA binding affinity data of the wild-type mouse EGR1 protein and four variants differing
71 for such cleavage, it is shown that, whereas wild type mouse embryo fibroblasts (MEFs) produce cleave
72 a superfamily) induce p19(Arf) expression in wild type mouse embryo fibroblasts (MEFs), and they enha
73 1,25(OH)(2)D(3) induces p27 accumulation in wild type mouse embryo fibroblasts and arrests wild type
74 essing of endogenous pro-alpha1(V) chains by wild type mouse embryo fibroblasts and by fibroblasts de
75 B1 is similar to that occurring in infected wild-type mouse embryo fibroblast cells but the levels o
76 UV light inhibits translation of IkappaB in wild-type mouse embryo fibroblasts (MEF(S/S)) and that t
79 results in accelerated TRAF2 degradation in wild-type mouse embryo fibroblasts (MEFs), as compared w
80 uces ECM and integrin alpha5 proteins in K41 wild-type mouse embryo fibroblasts (MEFs), CRT null MEFs
83 induced nuclear Nrf2 and HO-1 effectively in wild-type mouse embryo fibroblasts (wt MEFs) and in B-Ra
84 roteins, DNA, and mRNA and virus yields from wild-type mouse embryo fibroblasts and sibling cells lac
86 p53 knockout mice, which was not the case in wild-type mouse embryo fibroblasts, suggesting that p53
91 overexpression of miR-199 or miR-214 in the wild-type mouse embryonic brains was sufficient to distu
93 properties of 26S proteasomes purified from wild-type mouse embryonic fibroblast cells and those lac
95 ssion in tuberous sclerosis complex 2 (TSC2) wild-type mouse embryonic fibroblasts (MEF) but not in T
96 s virus led to higher levels of autophagy in wild-type mouse embryonic fibroblasts (MEF) than in RNas
100 hat veliparib enhanced the effects of CPT in wild-type mouse embryonic fibroblasts (MEFs) but not Par
105 in human umbilical vein endothelial cells or wild-type mouse embryonic fibroblasts because of an inte
106 AP 2, Bcl-x(L), Bfl-1/A1, TRAF1, and FLIP in wild-type mouse embryonic fibroblasts but not in GSK-3be
107 minantly to the nuclear compartment, whereas wild-type mouse embryonic fibroblasts localize survivin
109 ormation and anchorage-independent growth in wild-type mouse embryonic fibroblasts, and that these an
110 paB activation as measured by DNA binding in wild-type mouse embryonic fibroblasts, but deletion of G
112 NF-kappaB and JNK are activated by LIGHT in wild-type mouse embryonic fibroblasts, no activation of
115 re of cystogenesis was assessed in explanted wild-type mouse embryonic metanephroi, using 8-Br-cAMP a
124 to alphaAsp-206-Arg-231, reversibly inhibits wild-type mouse ENaCs expressed in Xenopus oocytes, as w
125 vo, miR-exon4 was most highly amplified from wild-type mouse enamel organs at the secretory stage.
126 Interestingly, increasing cytosolic GSH in wild-type mouse endothelial cells decreased constitutive
127 CR1 on their erythrocytes to the results for wild-type mouse erythrocytes that do not express CR1.
129 Thus, 5-aza-dC induces Dnmt1 degradation in wild-type mouse ES cells, but not in Dnmt [3a(-/-), 3b(-
132 y in situ hybridization using wholemounts of wild-type mouse eyes and by immunofluorescence staining
133 eal injection of TRPM1-positive MAR IgG into wild-type mouse eyes, and the appearance of the IgG in t
134 Using cultured neurospheres from PPT1-KO and wild-type mouse fetuses, we further demonstrate that the
136 was observed in p53-deficient but not in p53 wild-type mouse fibroblasts treated with the DNA demethy
139 rrelated with FLVCR1 expression, we examined wild-type mouse Flvcr1 mRNA levels in the posterior colu
143 l of IV VLA4+NPCs was assessed for nGD using wild-type mouse green fluorescent protein (GFP)-positive
144 t, we generated RNA-sequencing profiles from wild-type mouse guts at E10.5, E12.5, and E14.5 from bot
145 rticle tracking to study the mobility of the wild-type mouse H-2L(d) class I MHC molecule and of seve
146 derwent transplantation heterotopically with wild-type mouse heart expressing alpha-gal epitopes and
149 -ND hearts was at a level similar to that of wild type mouse hearts under beta-adrenergic stimulation
150 omyocyte telomere length decrease sharply in wild-type mouse hearts after birth, resulting in cardiom
151 e performed RNA Seq analysis of AE3-null and wild-type mouse hearts and evaluated the data with respe
153 iac damage and impaired function relative to wild-type mouse hearts following ischemia reperfusion.
157 oaches, we show that Il7r-deficient, but not wild-type, mouse hematopoietic progenitors transduced wi
158 Furthermore, human apoE was resecreted from wild-type mouse hepatocytes after a pulse with human VLD
159 mma in vitro inhibited cell proliferation of wild-type mouse hepatocytes, but not STAT1(-/-) hepatocy
161 alpha (PGC1alpha), to the Adam10 promoter in wild-type mouse hippocampal neurons and shifted APP proc
164 gh dose of recombinant human ADAMTS13 into a wild-type mouse immediately before reperfusion reduces i
165 transplantation of a D(5)(-/-) kidney into a wild-type mouse increased the expression of both, sugges
171 Glucose normally does not produce TDP in wild-type mouse islets except under forced intracellular
172 ibited and compared with the TDP response in wild-type mouse islets with and without forced intracell
178 CS expression were analyzed in transgenic or wild-type mouse lens and lens epithelial cells stimulate
179 Appropriate controls were used, including wild-type mouse lens, scrambled oligonucleotides, and a
182 use hepatocytes were incubated (pulsed) with wild-type mouse lipoproteins, and cells and media were c
184 as well as in Smad4, and their reduction to wild-type mouse liver levels in AHR -/- mice fed the ret
185 ibody inhibited RA 4-hydroxylase activity of wild-type mouse liver microsomes to the levels of AHR-nu
187 Livers of SPTBN1(+/-) mice, compared to wild-type mouse livers, display a significant increase i
189 hallenge, Siglec-F ligands were increased in wild-type mouse lungs but less so in St3gal3 mutants, wh
193 affects mammary tumorigenesis, we generated wild-type mouse mammary tumor virus/polyoma middle-T (WT
194 e found that a primary stable cell line from wild-type mouse marrow cells expressing BCR-ABL caused s
196 rotein-PACSIN2 were concentrated in midstage wild-type mouse MKs in a well-defined invagination of th
197 ATP-facilitated sIPSCs were also recorded in wild-type mouse MNTB neurones, but were absent in the MN
198 ing oligomers and the mThy-1 alpha-synuclein wild-type mouse model (Line 61), which accumulates vario
199 duced gastric intraepithelial neoplasia in a wild-type mouse model and show no additive effect of hig
207 We found that prior AICAR stimulation of wild-type mouse muscle increases insulin sensitivity to
208 of mutated human MYOC prevented secretion of wild-type mouse Myoc but did not dramatically affect sec
211 study, Calhm1 knockout, Panx1 knockout, and wild-type mouse nasal septal epithelial cells were grown
213 of CAR on maternally exposed Dhcr7(+/-) and wild-type mouse offspring, and tested the biochemical ef
215 to the kinetochores of polar chromosomes of wild-type mouse oocytes, in which polar chromosomes are
216 ely restored after the addition of exogenous wild-type mouse or human alpha-synuclein, but not by A30
217 34-deficient platelets, ex vivo treatment of wild-type mouse or human platelets with the Vps34-specif
219 rative transcriptomic analysis of mutant and wild-type mouse organs revealed lipid metabolic changes
221 hepatocytes, we transplanted suspensions of wild-type mouse pancreatic cells into syngeneic recipien
222 rhodopsin in native membranes obtained from wild-type mouse photoreceptors and opsin isolated from p
223 he 50 known targets from the knockout versus wild-type mouse plasma, class-specific precursor/fragmen
225 LTC4 induced surface expression of CD62P by wild-type mouse platelets in platelet-rich plasma (PRP)
227 Deposition patterns of untreated human and wild-type mouse platelets were consistent with random di
234 iPS-RPE cells compared with that of isolated wild-type mouse primary RPE (mpRPE) cells in vitro and i
238 tion (mouse- equivalent) abolished FRET with wild-type mouse PrP, whereas mutant PrP-P101L displayed
242 zol-induced seizures in postnatal day 7 (P7) wild-type mouse pups and that transgenic knock-in mice h
243 cles containing the mouse opsin promoter and wild-type mouse Rds gene were injected subretinally into
244 mmary gland explants into cleared fat pad of wild type mouse recipients indicates that the observed a
247 oft agar can be reverted by re-expression of wild-type mouse RelA, but not by expression of RelA muta
253 of the total photoreceptor population in the wild-type mouse retina, we generated mouse lines with CN
254 of the total photoreceptor population in the wild-type mouse retina, we used Cngb3(-/-)/Nrl(-/-) mice
256 ARPE-19 and primary RPE cells isolated from wild type mouse retinas from H(2)O(2)-induced cell death
257 e pik3r1 KO mice was slower than that in the wild-type mouse retinas at 5 minutes of exposure to ligh
258 sgene in a line that was hemizygous null for wild-type mouse rhodopsin (mrho(+/-)), and the eyes of t
259 emission, 457 or 540 nm) appears in frog and wild-type mouse rod outer segments reaching a maximum in
262 th a time constant of approximately 40 ms in wild-type mouse rods, much faster than previous estimate
263 RyR2 cDNA were constructed, one encoding the wild type mouse RyR2 (RyR2(wt)) and the other encoding m
265 ferocytosis) is bolstered in the presence of wild-type mouse serum, through the C3 deposition on the
267 was suppressed in TNF-alpha(-/-) compared to wild-type mouse skin in response to the tumour promotor
268 (VD3) and/or all-trans retinoic acid (RA) on wild-type mouse skin induces a human atopic dermatitis-l
269 ted PKCalpha and other kinases were lower in wild-type mouse skin treated with 12-O-tetradecanoylphor
270 nflammation in both PPAR-gamma-deficient and wild-type mouse skin, indicating that the inhibition of
271 ditional pairs of highly branched SGs, or in wild-type mouse skin, K6a expression consistently coinci
277 rimary cortical neurons from mice expressing wild-type mouse SNCA, wild-type human SNCA, or mutant A5
279 ntiation of monocyte-derived fibrocytes from wild-type mouse spleen cells, but not from SIGN-R1(-/-)
280 he vast majority of exons harboring m(6)A in wild-type mouse stem cells is spliced the same in cells
282 Here we report the observed changes for two wild type mouse strains commonly used in transgenic stud
286 ven by a BAC transgene containing the entire wild-type mouse tau locus, including the endogenous prom
288 n of the axons in the corpus callosum from a wild-type mouse to a knockout mouse showed that myelin i
290 nd RT-PCR experiments of NKCC1-deficient and wild type mouse transcriptomes, we confirmed the absence
293 e determine the plasma clearance rate in the wild-type mouse, using time-resolved fluorescence on a s
296 essed in the basolateral membrane of the RPE wild-type mouse, was expressed at very low levels in bot
298 of target transcripts in RNA extracted from wild-type mouse whole eyes or retinas between embryonic
299 ssion profile of the rod-dominated retina of wild type mouse with that of the cone-only retina of Nrl
300 HCN1/HCN2/HCN3/HCN4 = 9:9:1:89 in OC of the wild-type mouse, with HCN4 protein primarily attributabl