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1 ed disease at a similar rate of onset as the wild type strain.
2 nd sex ratio were comparable to those of the wild type strain.
3 tine at significantly higher levels than the wild type strain.
4  of these matched the stress proteome of the wild type strain.
5 le to increase BAFF at levels similar to the wild type strain.
6  the pat mutant reaching the spleen than the wild type strain.
7 nockout strains and patients infected with a wild-type strain.
8 notypes to a level comparable to that of the wild-type strain.
9  viability upon exposure to NO compared with wild-type strain.
10 y to initiate growth was comparable with the wild-type strain.
11 hyperplasia on day 14 postinfection than the wild-type strain.
12 lipid A moiety compared to that found in the wild-type strain.
13  cleared more efficiently, compared with the wild-type strain.
14  on capsule production when expressed in the wild-type strain.
15 ision point is present and functional in the wild-type strain.
16 pared with the black spore suspension of the wild-type strain.
17  titer when using the RARE strain versus the wild-type strain.
18 e, grows at a rate comparable to that of the wild-type strain.
19 of proteins within the complex compared to a wild-type strain.
20 essful sexual reproduction with the parental wild-type strain.
21 n MP1 and performed competitions against the wild-type strain.
22 mouse model of GAS invasive disease than the wild-type strain.
23 sitive to oxidative stress than the parental wild-type strain.
24 DeltaalgZ mutant was 40% of the level of the wild-type strain.
25 ed, showed a lower electro-activity than the wild-type strain.
26 provided protection against infection by the wild-type strain.
27 kness and biomass than those of the parental wild-type strain.
28 esent in the DeltaartA strain but not in the wild-type strain.
29  mutant strain compared to expression in the wild-type strain.
30 d more resistant to LpxC inhibitors than the wild-type strain.
31  10-fold lower in the hfq mutant than in the wild-type strain.
32 d urethral epithelial cells, relative to the wild-type strain.
33 ic activity and cytotoxicity compared to the wild-type strain.
34  differences between the yfiR mutant and the wild-type strain.
35 umb to infection than mice infected with the wild-type strain.
36 decreased in the ccpE mutant relative to the wild-type strain.
37 kinetics comparable to those observed in the wild-type strain.
38 olonged carriage, exceeding even that of the wild-type strain.
39 on mutant S. exfoliatus ZD27 compared to the wild-type strain.
40 hemolytic activities similar to those of the wild-type strain.
41  cells selected for longer chains within the wild-type strain.
42 strain at nearly the same levels as from the wild-type strain.
43  an increase in chain length relative to the wild-type strain.
44 y to oxidative stress similar to that of the wild-type strain.
45 ar bone resorption than the encapsulated W50 wild-type strain.
46 as defective in colonization compared to the wild-type strain.
47 U, and reduced dissemination compared to the wild-type strain.
48 ificantly higher in mice inoculated with the wild-type strain.
49 ing acute infection when coinoculated with a wild-type strain.
50  lgt mutant were as virulent as those of the wild-type strain.
51 e also probed and compared with the parental wild-type strain.
52 rdF elevated 35-fold relative to that of the wild-type strain.
53 nd aureolysin (Aur) activity relative to the wild-type strain.
54 l biofilm on the implanted catheter than the wild-type strain.
55 f the tested compounds than does an isogenic wild-type strain.
56 reased ethanol levels in comparison with the wild-type strain.
57 DeltaSho1 strain was reduced compared to the wild-type strain.
58 s also showed greater cooperativity than the wild-type strain.
59 d spleens during an acute infection than the wild-type strain.
60 t or low ribosome content, compared with the wild-type strain.
61 and SIN-1 both induced Ohr expression in the wild-type strain.
62 onditions the mutant did not differ from the wild-type strain.
63 sozyme and cold treatments than those of the wild-type strain.
64 fected on plastic surfaces compared with the wild-type strain.
65 tracellular concentration as compared with a wild-type strain.
66 tant produced less pyruvate and H2S than the wild-type strain.
67 al challenge with the B. mallei lux (CSM001) wild-type strain.
68 ration were similar compared to those of the wild-type strain.
69 higher lipid production from phenol than the wild-type strain.
70 ficantly more biomass than those formed by a wild-type strain.
71 ppressor strains tested as compared with the wild-type strain.
72 ced fewer, smaller lesions compared with the wild-type strain.
73 Caenorhabditis elegans, whereas it is not in wild type strains.
74 he production of new metabolites not seen in wild type strains.
75 minants associated with the pathogenicity of wild-type strains.
76 aracterize the latent infection of two HSV-1 wild-type strains.
77 nsformants were similar to the corresponding wild-type strains.
78  survival rates comparable to those found in wild-type strains.
79 fective cheats in vivo and cannot outcompete wild-type strains.
80 d colonization of BALB/cByJ mice compared to wild-type strains.
81 n of host cells compared with their isogenic wild-type strains.
82 ntly more pyruvate in the growth medium than wild-type strains.
83  and higher Chl a/b ratio than corresponding wild-type strains.
84 el of C1q recognition when compared with the wild-type strains.
85  and higher Chl a/b ratio than corresponding wild-type strains.
86 ompared the lipidome of loa1Delta mutant and wild-type strains.
87  more trehalose under stress conditions than wild-type strains.
88 e less tolerant of deep ocean pressures than wild-type strains.
89 t do not accumulate in detectable amounts in wild-type strains.
90  intestinal epithelial cells compared to the wild-type strain 01/51.
91 n the Stargardt model Abca4(-/-) than in the wild type strains 129/sv and C57Bl/6.
92 ation-essential ICP4 and ICP8 genes of HSV-1 wild-type strain 17syn+ were brought under the control o
93 ia in human skin grafted SCID mice using the wild-type strain 2C4.3.
94 ase over the colocalization exhibited by the wild-type strain (8% +/- 6%).
95                                            A wild type strain, A/Hong Kong/1/68 (H3N2) developed resi
96                              Compared to the wild-type strain, a H201R isogenic mutant strain caused
97 ess susceptible to early host clearance than wild-type strains after intravenous infection, but impai
98                 Titan cell production by the wild-type strain also resulted in increased eosinophil a
99 with mif(-/-) L. major, when compared to the wild-type strain, also showed a 3-fold reduction in para
100 mately 10-fold higher when compared with the wild-type strain, although the expression levels of gene
101 dual ribosome-bound nascent chains between a wild type strain and a mutant defective in CTPD.
102  of proteins that could confer protection to wild type strain and help establish infection.
103 mes of two Caenorhabditis elegans strains, a wild-type strain and a strain containing two complex rea
104 scriptome-sequencing (RNA-seq) analysis of a wild-type strain and an isogenic fabT deletion mutant st
105 ecoded strain grows at a similar rate to the wild-type strain and does not exhibit any major growth d
106  altered expression compared to the isogenic wild-type strain and included transcriptional regulators
107  toxin (CDT) were evaluated first by using a wild-type strain and its corresponding cdtB isogenic mut
108 concentrations that gave derepression of the wild-type strain and retained sufficient ligation activi
109 ld improvement in FA titer compared with the wild-type strain and the strain carrying the uncontrolle
110 erleukin 8 expression was evaluated by using wild-type strains and their corresponding CdtB isogenic
111 46a1(-/-), Cyp27a1(-/-)Cyp46a1(-/-), and two wild type strains) and human brain specimens.
112       FtsH2 also interacts with FtsH3 in the wild-type strain, and a mutant depleted in FtsH3, like f
113 eater bactericidal activity than against the wild-type strain, and the IgG1 MAbs had similar or great
114 ower for the T2SS mutant than it was for the wild-type strain, and the mutant's defect was maintained
115 altered biofilm architecture relative to the wild-type strain, and these phenotypes were partially co
116 activation of macrophages, compared with the wild-type strain, and with delayed inflammatory stimuli
117                                              Wild-type strains are polymorphic for the npr-1 gene, an
118 d a higher accumulation of the drug than the wild-type strain as predicted.
119 ter membrane proteins, TraK and TraB, in the wild-type strain as well as in overexpression strains an
120 povesiculation phenotype of DeltanlpA in the wild-type strain as well as in the degP deletion strain
121 eater biomass productivity than the parental wild-type strains as well as near wild-type ability to c
122  and differentiation in THP-1 cells than the wild type strain, as determined by carboxyfluorescein di
123 icantly attenuated virulence relative to the wild-type strain, as manifested by prolonged survival, r
124 mutant compared with those infected with the wild-type strain, as well as significantly greater expre
125 , which differs by a point mutation from the wild-type strain, assembles into straight filaments in w
126               A mutation in the batA gene of wild-type strain ATCC 23344 was found to be particularly
127  increases in intracellular C. jejuni 11168H wild-type strain bacteria were observed after 24-h cocul
128 t were very similar to those of the parental wild-type strains both in vitro and in vivo.
129  three alkaline-stable lipids present in the wild-type strain but absent from the mutant.
130 tely attenuated virulence as compared to the wild-type strain but did not significantly affect bacter
131 significantly up-regulated in the irradiated wild-type strain but not in the irradiated wdpks1 mutant
132 ed intact into the surrounding medium in the wild type strain, but not in the PG0026 mutant strains.
133  mutants were not attenuated compared to the wild-type strain, but multiple plc mutants showed reduce
134 verexpressing PR1 enhanced resistance to the wild-type strain, but not to the Sscp1 knockout strain o
135 M mutant was less efficient than that by the wild-type strain, but only upon HBMEC infection.
136 strains were more sensitive to acid than the wild-type strain, but the Deltahyc strains were like the
137 d hha mutant strains compared to that in the wild-type strain, but the secretion of virulence protein
138      Removal of GPI-anchored proteins in the wild-type strain by hydrofluoric acid (HF) pyridine trea
139 arasite mitochondrion, even outcrossing with wild-type strains cannot facilitate spread of resistance
140 duced by ER stress, and their knockdown in a wild-type strain caused ER stress.
141           Pulmonary spore challenge with the wild-type strain caused high mortality, intra-alveolar h
142 , for both classes of promoters, peak KOA in wild-type strains coincided late in the circadian cycle
143 t (Triticum aestivum) spikelets, whereas the wild-type strain colonized the whole wheat spike.
144 re more immune-stimulatory than the parental wild-type strains, consistent with an immune-suppressive
145 nt and its isogenic choline-treated parental wild-type strain D39 degrade extracellular DNA readily,
146                                Compared with wild-type strain, DeltacolA mutant displayed much-attenu
147 n of the mazF gene does not eliminate PCD in wild-type strain DK1622 as originally seen in DZF1.
148 cells 3 to 5 times more extensively than the wild-type strain does.
149 unts of cytochromes c(1) and c(2) than did a wild-type strain due to their restricted Ccm capabilitie
150                      Particularly, we used a wild type strain (E1162) and a mutant (E1162Deltaesp) to
151  report, we demonstrate that the majority of wild-type (strain EGDe) and mouse-adapted (InlA(m)-expre
152                                         In a wild-type strain, elevated levels of Hda conferred sensi
153 ant lacking all three genes behaved like the wild-type strain for all phenotypes mentioned above, but
154                                Compared with wild-type strains for FKS1, echinocandin-resistant C. al
155                                    Different wild-type strains form social interaction networks with
156 ceptible to phagocytosis by THP-1 cells than wild type strain (G37).
157                    Agrobacterium tumefaciens wild-type strain (GW4) was studied to determine how the
158 istopathologic examination revealed that the wild-type strain had a greater ability to colonize tissu
159                                 However, the wild-type strain had a greater advantage over the SdiA d
160 genome per generation; (ii) mutations in the wild-type strain have the expected mutational bias for G
161 lonized at a level comparable to that by the wild-type strain; however, in cocolonization experiments
162  mutants in defined competitions against the wild-type strain identified nine mutants that exhibited
163 mune responses was compared with that of the wild-type strain in a bacteremic mouse model.
164 nt mutant is less virulent than its parental wild-type strain in BALB/C mice.
165 onization, and is unable to compete with the wild-type strain in co-colonization assays.
166 8-0643, and the ure mutant was used with the wild-type strain in competition experiments in mouse mod
167  defect of the Deltatkt strain compared with wild-type strain in early intracellular proliferation an
168 ow as well as or better than a corresponding wild-type strain in EVPL.
169               A single gavage of C. glabrata wild-type strain in mice with DSS-induced colitis caused
170  kidneys when tested in competition with the wild-type strain in the murine model of UTI.
171 mug of human fH/ml permitted survival of the wild-type strain in up to 60% infant rat serum, whereas
172  in vivo-selected mutant grew as well as the wild-type strain in vitro.
173 he trehalose production genes to out-compete wild-type strains in mice and macrophages.
174 xpressed VPS colonized less effectively than wild-type strains in more distal intestinal regions.
175        Rapid differentiation of vaccine from wild-type strains in suspect measles cases is a valuable
176 shift in exflagellation EC50 relative to the wild-type strains in the presence of compound 1294, prov
177  to bind fibronectin relative to that of the wild-type strain; in situ reconstitution of fnm in the d
178 at coinoculation of an SPI-1 mutant with the wild-type strain increased the number of mutant organism
179 ced stress, and in response to H(2)O(2), the wild-type strain increases superoxide radical production
180 criptomes of an rppH deletion mutant and the wild-type strain incubated at 20 degrees C and 30 degree
181                    Comparison of the REd and wild-type strains indicates that restriction did not aff
182 uced resistance to quinolone compared to the wild-type strain, indicating that fur functions as a pos
183 rt succumbed to infection with a more recent wild-type strain, indicating that immune responses to th
184 t in cells exposed to the isogenic S. aureus wild-type strain, indicating that PSMs inhibit the produ
185  sulfate was 40-fold slower than that of the wild-type strain, indicating that the efficient uptake o
186 e highly induced under oxidative stress in a wild-type strain, indicating the critical role of Cys-25
187 lic adjustment (MOMA) from the corresponding wild-type strains instead of having maximal growth rates
188 8 deletion mutants were constructed from the wild-type strain K96243.
189 ] 1.64 x 10(4) CFU) compared to the parental wild-type strain LD(50) (2.98 x 10(2) CFU).
190 c changes are reduced in comparison with the wild type strain, likely due to ineffective translation
191 howed a smaller hepcidin increase than their wild-type strain-matched controls, Bmp6(-/-) mice showed
192 ize porcine lungs and was outcompeted by the wild-type strain (median competitive index of 2 x 10(-5)
193 n is not detected in cell-free extracts from wild-type strain methanol- and lanthanum-grown cultures.
194                                       In the wild-type strain, MpkA phosphorylation levels progressiv
195 pression was also noticed in an M. smegmatis wild-type strain (MSWt) induced with cumene hydroperoxid
196 aride (LOS) of a lic2B mutant to that of the wild-type strain NT127 revealed that lic2B is required f
197 se data indicate that murine death caused by wild-type strains occurs by a mechanism different from t
198 rain were purified and used to show that the wild-type strain of Cba. tepidum can grow on biogenic S(
199 anscript in host macrophages infected with a wild-type strain of M. tuberculosis or an attenuated mut
200                              We examined the wild-type strain of Synechocystis sp. PCC 6803 and a ser
201                              We examined the wild-type strain of Synechocystis sp.PCC 6803 and a seri
202  development of reverse genetics systems for wild-type strains of CDV and the use of the resulting re
203                                We found that wild-type strains of fruit flies, Drosophila melanogaste
204  McKrae is highly virulent compared to other wild-type strains of HSV-1.
205 hesis after infection with vaccine than with wild-type strains of MeV.
206 th conidia (asexual spores) of two different wild-type strains of N. fumigata and three different erg
207 er, infection of guinea pigs with nonadapted wild-type strains of the different species resulted in f
208 The number of disease lesions incited by the wild-type strain on bean was also significantly higher t
209 y oral inoculation with the encapsulated W50 wild-type strain or isogenic non-encapsulated DeltaPG011
210 ed with the fully virulent B. anthracis Ames wild-type strain or the isogenic toxin-deficient mutants
211  recruited to the site of infection with the wild-type strain or the mutant strain complemented with
212                                              Wild type strains overexpressing YpdA showed increased t
213                                     When the wild-type strain overexpressing ecm was tested with the
214 -sigH, compared with those infected with the wild-type strain (P < .05).
215 tion by HEp-2 cells compared to the parental wild-type strain (P < 0.05).
216 mation significantly compared to that in the wild-type strain PA14 in an abiotic biofilm system.
217 howed that OMVs generated from P. aeruginosa wild-type strain PAO1 were more cytotoxic to alveolar ep
218 ts was significantly reduced compared to the wild-type strain PH-1, while 10 Group 2 mutants grew nor
219 d disease upon subsequent challenge with the wild-type strain PI1428, which appears to be driven by a
220 ection model, 1 week following infection the wild-type strain produced significantly more widespread
221  attenuated in the mouse and equine, whereas wild-type strain RacL11 induces severe inflammation of t
222 thermore, impairment of the Sec pathway in a wild-type strain reduced Ag43 production but did not sig
223 on of Cse4, transient induction of PSH1 in a wild-type strain resulted in phenotypes similar to a pat
224 in-deficient S. aureus and the corresponding wild-type strain reveal that activation of acid sphingom
225      The average genome configuration of the wild-type strain revealed strong intra- and inter-chromo
226 inst a leaky Escherichia coli strain and the wild-type strain reveals the contribution of the formida
227 rs genotypically and phenotypically from the wild-type strain S26/3.
228 inoculated with the formyl peptide-producing wild-type strain showed a significantly increased freque
229  observed in domesticated and undomesticated wild-type strains sporulating in liquid and on solid med
230 e either of these cell lines relative to the wild type strain, suggesting that it is attenuated in vi
231  with efficiency equal to that seen with the wild-type strain, suggesting that competing commensal or
232 d increase in virulence as compared with the wild-type strain, suggesting that LOS-IV plays a role in
233 ant was still less virulent in vivo than the wild-type strain, suggesting that SecA2 function was sti
234 was observed between the luxS mutant and the wild-type strain, suggesting that the defect in virulenc
235 wed an adhesion level similar to that of the wild-type strain, suggesting that the gene does not dire
236 id not spread in tomato stems as well as the wild-type strain, suggesting that these exDNases facilit
237 paired for stable colonization relative to a wild-type strain, suggesting the presence of genotoxic s
238 ficient in eliminating SIC compared with the wild-type strain, terminal half-lives being 6 and 1.5 h,
239 erence to HEp-2 cells were much lower in the wild-type strain than in the hha mutant, but no signific
240 ly engineered strains, but incompatible with wild-type strains that lack resistant alleles.
241                            Compared with the wild-type strain, the esxH-deficient strain induced five
242        Thus, at lower epitope density in the wild-type strain, the IgG3 anti-fHbp MAbs had the greate
243                      Relative to that of the wild-type strain, the infectivity of the mutant was seve
244 ltasll1951 strain was similar to that of the wild-type strain, the viability of the Deltasll1951 stra
245 ted during amino acid stress, whereas in the wild-type strain these levels declined under the same gr
246                                           In wild-type strains, these mismatches are repaired by the
247 can be generated through seeds by crossing a wild-type strain to a transgenic strain with altered cen
248 g1Delta mutant was less susceptible than the wild-type strain to noniron metalloporphyrins, further i
249 tants with altered biofilm formation and the wild-type strain to predict drug targets against P. aeru
250 into gnotobiotic mice together with 11 other wild-type strains to generate a 15-member artificial hum
251 h the molecular clock for VP1 of circulating wild-type strains to infer that the mean time from the i
252                                       In the wild-type strain, transcription of B. henselae hbpC was
253 rmed comparative genome sequence analysis of wild-type strain TX82 and TX6051 and found a single muta
254 rn mice were inoculated intramuscularly with wild-type strain type 3 Dearing (T3D) and T3D-sigma1R202
255 ntaining medium and grew slower than did the wild-type strain under aerobic and anaerobic conditions,
256 d staphyloxanthin production compared to the wild-type strain under aerobic culturing conditions.
257 hile the Deltaspx strain grew as well as the wild-type strain under anaerobic conditions, the mutant
258  development by a developmentally proficient wild-type strain under high-nutrient conditions.
259 sted that the same transcript appears in the wild-type strain under nutrient-limiting conditions.
260  resulted in the inability to compete with a wild-type strain under selective conditions that require
261 of two, ylxY and ylyA, were outcompeted by a wild-type strain under sporulation-inducing conditions,
262 ed in MoHMT1 deletion mutants, compared with wild-type strains under normal growth conditions.
263 IPV production (sIPV), rather than the usual wild-type strains used for conventional IPV (cIPV).
264 h based on the V-nitrogenase on acetate, the wild-type strain uses exclusively the Mo-nitrogenase on
265 nt did not attach to tomato roots as well as wild-type strain UW551.
266                 Deletion of the rtfA gene in wild-type strains (veA+) resulted in a slight decrease i
267 rated in different genetic backgrounds (e.g. wild-type strain versus knockout strain).
268 lymphoid tissues and reveals the protective (wild-type strain) versus harmful (yopP-deficient strain)
269 e miRNA-mRNA interactions between Drosophila wild-type strain W(1118) and a mutant of the key circadi
270                      OMVs were isolated from wild-type strain W83 and the gingipain-deficient strain
271 s able to tolerate 100 mM serine whereas the wild type strain was already inhibited by 1 mM of the am
272                                          The wild type strain was sensitive to BTZ043; however, C. gl
273 ia, diet did not affect the outcome that the wild-type strain was better able to persist and colonize
274 ted in static conditions, the fitness of the wild-type strain was higher under fluctuating humidity c
275 nt also failed to induce abortion, while the wild-type strain was highly abortifacient.
276 per(Bd-Kah) element isolated from the Kahuku wild-type strain was highly degenerate and appeared to h
277                                          The wild-type strain was twice as likely to survive passage
278                The clinically important M1T1 wild-type strain was used in this study, and isogenic mu
279 acid-dependent growth arrest, and unlike the wild-type strain, was susceptible to fatty acid synthesi
280 data from five single gene knockouts and the wild type strain, we decrease the mean squared error of
281 ed by the Bvg(+) phase-locked mutant and the wild-type strain were indistinguishable.
282       Edmonston vaccine/laboratory and IC323 wild-type strains were equally affected by the inhibitor
283                                              Wild-type strains were subjected to VP1 gene sequencing
284 in this Tn library, and in various C. jejuni wild type strains, were compared and correlated to detec
285  mutant was significantly outcompeted by the wild-type strain when birds were inoculated with a low d
286 he mutant was the same as that of the parent wild-type strain when cultured in nutrient-rich media wi
287 oteins that disrupt function in an otherwise wild-type strain when overexpressed.
288 de a substantial fraction of isoleucine in a wild-type strain when propionate is available in the med
289 n mice compared to the levels induced by the wild-type strain, whereas an msbB pagP mutant induced le
290 er level in the DeltaytoI strain than in the wild-type strain, whereas two genes (lmo1917 and lmo2103
291 di-GMP as well as stimulates swarming in the wild-type strain, while overexpression of MotA from a pl
292                           Treatment of yeast wild-type strain with 10 mM SSA and 10 mM GHB didn't aff
293 al activity of M. pulcherrima, we compared a wild-type strain with a spontaneously occurring, pigment
294                           Cochallenge of the wild-type strain with an MMR-defective strain showed a s
295  Similarly, bombardment of a morphologically wild-type strain with the Cas9 plasmid and sgRNA plasmid
296 s putida and P. aeruginosa, and compared the wild-type strains with deletion mutants for crc.
297 ed in more-severe pulmonary infection by the wild-type strain (with a 30-fold increase in the number
298 ed EmaA was reduced by 50% compared with the wild-type strain, without changes in mRNA levels.
299  mice using a virulent C. perfringens type D wild-type strain (WT), an isogenic ETX null mutant (etx
300                     Clostridium thermocellum wild-type strain YS is an anaerobic, thermophilic, cellu

 
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