ライフサイエンスコーパス: wilt

1 liae, a fungal pathogen causing Verticillium wilt.

2 cular pathogens use many mechanisms to cause wilt.

3 the stems of tomato plants just beginning to wilt.

4 cally has not resulted in control of spotted wilt.

5 ional tool for management programs of laurel wilt.

6 elatively unrelated causal agent of Fusarium wilt.

7 ne that experienced a recent outbreak of oak wilt.

8 g cotton cultivars resistant to Verticillium wilt.

9 ENC, as they persist until the 7-day flowers wilt.

10 tegies to improve resistance to Verticillium wilt.

11 decreases cotton resistance to Verticillium wilt.

12 ronmentally responsible control of bacterial wilt.

13 Ceratocystis fagacearum, causal agent of oak wilt.

14 na rustica) stomata did not until the leaves wilted.

15 raulic failure and carbon starvation in tree wilting.

16 ite isoprene emission decreasing before leaf wilting.

17 pecies a visible sign of petal senescence is wilting.

18 ses to facilitate management of Verticillium wilts across a broad range of crops.

19 ed drought tolerance demonstrated by delayed wilting after watering was ceased and quicker and better

20 ng C. nebraskensis (Cn), which causes Goss's wilt and blight of maize.

21 er content, and cellular damage detected oak wilt and drought 12 d before visual symptoms appeared.

22 predicted ecophysiological indicators of oak wilt and drought decline in both potted and field experi

23 Both oak wilt and drought resulted in blocked water transport thr

24 orne levels to enable differentiation of oak wilt and drought, and detection prior to visible symptom

25 differentiate plant stress types such as oak wilt and drought.

26 e factors in the cause of Stewart's vascular wilt and fire blight.

27 road-spectrum resistance to potato bacterial wilt and late blight diseases.

28 of cotton genotypes tolerant to Verticillium wilt and was induced early and strongly by inoculation w

29 ense and SlFRK3-RNAi lines exhibited similar wilting and anatomical effects, confirming that these ef

30 aliana delays germination and leads to rapid wilting and chlorosis in mature plants.

31 stem of susceptible cotton, leading to rapid wilting and death.

32 sis and alter plant water relations to delay wilting and maintain productivity during water limiting

33 severe tissue damage during drought, such as wilting and substantial stem embolism.

34 Dutch elm disease, sudden oak death, laurel wilt, and others have resulted in large economic losses

35 with those identified by human assessment of wilting, and could detect QTLs earlier than human assess

36 hich it requires for efficient colonization, wilting, and killing of plants.

37 matal conductance, leaf temperature, reduced wilting, and maintenance of photosynthesis.

38 examined further to determine the cause for wilting, and thus better understand how the anionic pero

39 Most new occurrences of Fusarium wilt appear to be the result of a recent introduction ra

40 esistance to the fungal disease Verticillium wilt are top priorities for the mint industry.

41 the plant survival with no symptoms of leaf wilting as compared to untreated Catharanthus growing in

42 ater among species whose leaves lost turgor (wilted) at more negative water potentials and experience

43 A cDNA, PvNCED1, was cloned from wilted bean (Phaseolus vulgaris L.) leaves.

44 lect melon cultivars to avoid melon Fusarium wilt, but also to monitor how quickly a Fom population c

45 (FOV4), a soil-borne fungus causing Fusarium wilt by infecting the roots and vascular system of susce

46 auvignon wines made with grapes infected and wilted by brown rot (Plasmopara viticola).

47 from previous studies associated with canopy wilting, canopy temperature, water use efficiency, and o

48 Fusarium wilt caused by the ascomycete fungus Fusarium oxysporum

49 Tomato spotted wilt caused by thrips-vectored tomato spotted wilt virus

50 Spotted wilt caused by tomato spotted wilt virus (TSWV) is one o

51 Fusarium wilt, caused by Fusarium oxysporum f.

52 Verticillium wilt, caused by Verticillium dahliae, is a serious vascu

53 Bacterial wilt, caused by Xanthomonas translucens pv. graminis (Xt

54 nic acid and Abscisic acid), salt (NaCl) and wilt causing pathogen (Fusarium oxysporum).

55 zed the published data for stomatal closure, wilting, declines in hydraulic conductivity in the leave

56 es with omics approaches to unravel Fusarium wilt defense mechanisms in garden pea, aiming to acceler

57 c viruses, and on the incidence of bacterial wilt disease (a fatal disease vectored by cucumber beetl

58 tomato (Solanum lycopersicum) suffering from wilt disease (caused by Ralstonia solanacearum) as sourc

59 Pine wilt disease (PWD) is perhaps the most serious threat to

60 Erwinia tracheiphila - which causes a fatal wilt disease - alters the foliar and floral volatile emi

61 Plant pathogenic forms cause a wilt disease and are grouped into formae speciales based

62 ccurrence, severity, and symptoms of spotted wilt disease are highly variable from season to season,

63 g a suite of 10 traits to quantify bacterial wilt disease based on plant shape and size.

64 ance to cucumber beetles or the incidence of wilt disease before the spread of the virus.

65 resistance response in chickpea to Fusarium wilt disease by modulating the transcription of defense

66 while mitigating development of the Fusarium wilt disease caused by Fusarium oxysporum f.

67 st century, the repeated emergence of coffee wilt disease caused by the fungal pathogen Fusarium xyla

68 protects tomato plants against Verticillium wilt disease caused by the soilborne pathogen Verticilli

69 he arabica and robusta populations of coffee wilt disease correlates with temperature.

70 in plant xylem vessels and causes bacterial wilt disease despite the low nutrient content of xylem s

71 solanacearum although its individual role in wilt disease development may be minor.

72 or the aer1/aer2 mutant had slightly delayed wilt disease development.

73 expression of EPS is critical for Stewart's wilt disease development.

74 many of which probably facilitate bacterial wilt disease development.

75 8 y the number of plants dying from a sudden wilt disease has increased, leading to crop failure.

76 Verticillium dahliae, is a serious vascular wilt disease in cotton (Gossypium spp.).

77 the first to report that F. kuroshium causes wilt disease in mango trees and that it is a primary fun

78 l-borne fungal pathogen that causes vascular wilt disease in many economically important crops worldw

79 Xylella fastidiosa (Xf) causes wilt disease in plants and is responsible for major econ

80 p. nicotianae is a causal agent for vascular wilt disease in tobacco.

81 borne fungal pathogen that causes Sclerotium wilt disease in tomato and several other economically im

82 previous observation of dramatically reduced wilt disease incidence in ZYMV-infected plants.

83 Pine wilt disease is a lethal tree disease caused by nematode

84 lstonia solanacearum, which causes bacterial wilt disease of many plant species, produces several ext

85 quitous fungal pathogen that causes vascular wilt disease on a wide range of plant species and can pr

86 fungus Verticillium dahliae causes vascular wilt disease on hundreds of plant species.

87 l-borne plant pathogen that causes bacterial wilt disease on many plant species.

88 fungus Verticillium dahliae causes vascular wilt disease on more than 200 plant species worldwide.

89 h increased exposure to and the incidence of wilt disease on the transgenic plants.

90 he idea that SlyFRG4 is essential for tomato wilt disease resistance.

91 e fungus Fusarium xylarioides, is a vascular wilt disease that has affected coffee production in sub-

92 chus xylophilus, is the causal agent of pine wilt disease that has devastated pine forests in Asia.

93 Coffee wilt disease, caused by the fungus Fusarium xylarioides,

94 Banana Xanthomonas wilt disease, caused by Xanthomonas campestris pv. musac

95 ycopersici (Fol), the causal agent of tomato wilt disease, produces effector protein Avr2.

96 earum (Smith), the causal agent of bacterial wilt disease, secretes > 70 different effectors inside p

97 chus xylophilus, the causative agent of pine wilt disease, which is a major threat to pine forests wo

98 od nematode and its vector beetle cause pine wilt disease, which threatens forest ecosystems world-wi

99 e the defense of watermelon against Fusarium wilt disease.

100 ea lauricola, the causal agent of the laurel wilt disease.

101 causing a lethal wilting known as bacterial wilt disease.

102 tributed to the repeated emergence of coffee wilt disease.

103 , a soilborne pathogen that causes bacterial wilt disease.

104 e hosts, and consequently the spread of pine wilt disease.

105 key role in the survival and spread of pine wilt disease.

106 is a soil-borne phytopathogen that causes a wilting disease of many important crops.

107 Root-infecting vascular fungi cause wilt diseases and provoke devastating losses in hundreds

108 trains responsible for the various bacterial wilt diseases has in recent years led to the concept of

109 fungal pathogen responsible for devastating wilt diseases in many crops) cotton plants increase prod

110 s a multitude of strains that cause vascular wilt diseases of economically important crops throughout

111 s differ in their susceptibility to Fusarium wilt diseases.

112 biotics to help control soil-borne bacterial wilt diseases.

113 -hypersensitive stomatal closing and reduced wilting during drought.

114 d the hypothesis that oxalate induces foliar wilting during fungal infection by manipulating guard ce

115 ces, each of which helps to suppress spotted wilt epidemics.

116 d cucurbit beetles residing within the humid wilted floral chambers.

117 of male squash bees and cucurbit beetles in wilted flowers compared with controls.

118 rious concentrations in controlling Fusarium wilt for tomato crop improvement under laboratory, green

119 The absence of chr(Y-frag) in wilt-fragariae suggests that multiple, distinct pathogen

120 'yellows-fragariae') and the other did not ('wilt-fragariae').

121 chr(Y-frag) was not present in wilt-fragariae; isolates causing this syndrome evolved p

122 rstanding of the cross talk between vascular wilt fungi and the host plant, which eventually leads to

123 Vascular wilt fungi are a group of hemibiotrophic phytopathogens

124 tively, these findings suggest that vascular wilt fungi employ conserved infection strategies on nonv

125 to race 1 strains of the soil-borne vascular wilt fungi Verticillium dahliae and Verticillium albo-at

126 ymorpha (Mp) and the root-infecting vascular wilt fungus Fusarium oxysporum (Fo).

127 sequencing of a set of strains of the melon wilt fungus Fusarium oxysporum f. sp. melonis (Fom), bio

128 Resistance to wilt fungus Fusarium oxysporum f.sp. matthioli (FOM) is

129 sequencing of a set of strains of the melon wilt fungus Fusarium oxysporum f.sp. melonis (Fom), bioi

130 on homeostasis and virulence in the vascular wilt fungus Fusarium oxysporum.

131 Among these, Fusarium wilt (FW) caused by pathogen Fusarium oxysporum f.

132 he plant-infecting bunyavirus Tomato spotted wilt, Gc localizes at endoplasmic reticulum (ER) membran

133 Control of Verticillium wilt has relied on a panoply of chemical and nonchemical

134 However, oak wilt impaired conduits in localized regions of the xylem

135 leaf abscission zones when the leaves become wilted in response to limited water and HAE continues to

136 of cer9 are associated with delayed onset of wilting in plants experiencing water deficit, lower tran

137 stress hormone ABA and increased whole-plant wilting in response to drought and ABA.

138 n of the Verticillium genus, causes vascular wilts in a wide variety of economically important crops.

139 en Fusarium oxysporum (Fo) provokes vascular wilts in more than a hundred different crops.

140 oad host-range pathogen that causes vascular wilts in plants.

141 s in diverse species of bacterial and fungal wilt-inducing pathogens suggests that microbial expansin

142 bacterial plant pathogen that causes a fatal wilt infection in some cucurbit crop plants.

143 Though watermelon Fusarium wilt is a severe soil-borne disease, the effect of wheat

144 genetic basis of resistance to Verticillium wilt is unknown in most crops, as are the subcellular si

145 than in control plants, thus indicating that wilting is a consequence of peroxidase expression in the

146 olonizes the xylem vessels, causing a lethal wilting known as bacterial wilt disease.

147 ed with glycinebetaine accumulation, include wilting, loss of chlorophyll, and increase in thiobarbit

148 that coincide with flowering result in leaf wilting, necrosis, tassel browning, and sterility, a str

149 Fusarium wilt of banana, caused by Fusarium oxysporum f.

150 potent biocontrol activity against Fusarium wilt of banana.

151 s a soilborne pathogen that causes bacterial wilt of diverse plant species.

152 olanacearum is the causal agent of bacterial wilt of many agriculturally important crops.

153 Fusarium wilt of spinach, caused by Fusarium oxysporum f.

154 sp. fragariae, the cause of Fusarium wilt of strawberry, with isolates from four continents.

155 f potato, gray leaf spot of maize, bacterial wilt of tomato, and the soybean cyst nematode.

156 ant growth-inhibition effects, including the wilting of mature leaves.

157 phytopathogenic species that cause vascular wilts of plants.

158 The fungus Verticillium dahliae causes wilts of several hundred plant species, including potato

159 is a soil-borne fungus that causes vascular wilt on numerous plants worldwide.

160 ctural protein NSs encoded by tomato spotted wilt orthotospovirus (TSWV), contains an unusually large

161 var 7 strains cause ongoing tomato bacterial wilt outbreaks in the southeastern United States.

162 ne and death due to both drought and the oak wilt pathogen (Bretziella fagacearum).

163 ed resistance to the root-infecting vascular wilt pathogen Fusarium oxysporum.

164 n effector protein secreted by the bacterial wilt pathogen Ralstonia solanacearum, undergoes phosphor

165 oil-borne, fungal, xylem-colonizing vascular wilt pathogen Verticillium dahliae exploits effector pro

166 The oak wilt pathogen, Ceratocystis fagacearum, may be another e

167 icating that gene(s) important for bacterial wilt pathogenesis were interrupted by the IVET insertion

168 s have been studied mainly in model vascular wilt pathogens Fusarium oxysporum and Verticillium dahli

169 ewer root branches, which contributes to the wilting phenomenon seen in these plants.

170 gene interaction, and lacked an accompanying wilting phenotype.

171 Leaf water potential at turgor loss, or wilting (pi(tlp) ), is classically recognised as a major

172 used to assess plant available water (PAW), wilting point (WP), and water holding capacity (WHC).

173 er retention at pressure potentials near the wilting point of plants.

174 n, we were able to narrow down the permanent wilting point of the seedlings.

175 d leaf water potential at turgor loss (i.e. 'wilting point'; n(tlp) ), wood density (WD) and leaf mas

176 i.e., water content lower than the permanent wilting point).

177 g the leaf and stem hydraulic traits and the wilting point, or turgor loss point, beyond those expect

178 l moisture, with the highest increase at the wilting point.

179 tolerance traits, including turgor loss or 'wilting' point (pitlp ).

180 cific suppression to four diseases, Fusarium wilts, potato scab, apple replant disease, and take-all,

181 se activity also decrease the rate of flower wilting, promote the rooting of cuttings, and facilitate

182 been used to provide probes for screening a wilt-related tomato cDNA library.

183 ed reduced rates of water uptake and delayed wilting relative to wild-type plants.

184 Molecular markers linked to spotted wilt resistance could overcome this problem and allow se

185 were associated significantly with Fusarium wilt resistance in 10 QTL regions located on chromosomes

186 A major QTL controlling the spotted wilt resistance in Florida-EP(TM) '113' was identified.

187 titative trait loci (QTLs) linked to spotted wilt resistance in Florida-EP(TM) '113'.

188 d a substantial role in shaping two Fusarium wilt resistance loci with known causal genes.

189 6_38110665 can be used to introduce Fusarium wilt resistance QTL into cultivated spinach (S. oleracea

190 , GbCGF2/3 positively regulates Verticillium wilt resistance through promoting suberin biosynthesis,

191 pe line, introduced from Mexico with spotted wilt resistance.

192 Florida-EP(TM) '113' is a spotted wilt resistant cultivar with a significantly lower infec

193 de novo genome and plastome assemblies for a wilt-resistant South African accession of Mentha longifo

194 The development of a spotted wilt risk index has aided greatly in relaying informatio

195 butions, notably tropical race 4 of Fusarium wilt, rival its impact.

196 nly of broad host-range pathogens that cause wilt, rot, and blackleg diseases on a wide range of plan

197 ell as marginally significantly greater mean wilting score, for the entire severe drought period afte

198 isease progress curve (AUDPC) calculated for wilt severity 28, 35, and 42 days after planting (DAP) r

199 somes D03 and A02 linked to reduced Fusarium wilt severity.

200 effect (-2.48 to -2.79) at reducing Fusarium wilt severity.

201 y, irNaMPK4 plants transpired more water and wilted sooner than did wild-type plants when they were d

202 ted for 3 to 9 weeks to reach various visual wilting stages, from slightly wilted to dead.

203 2 to 4 weeks, the seedlings were scored for wilt state and survivorship.

204 wn under standard conditions or subjected to wilt stress.

205 mic movement through xylem, leading to rapid wilt symptom development and higher rates of plant death

206 th putrescine before inoculation accelerated wilt symptom development and R. solanacearum growth and

207 Wilt symptoms are thought to be caused by systemic bacte

208 aexlx-gh5) resulted in decreased severity of wilt symptoms, decreased mortality rate, and impaired sy

209 altered in the degraded soils with bacterial wilt (termed as degraded soils).

210 various visual wilting stages, from slightly wilted to dead.

211 cess NH4+, low pH, salinity, osmotic stress, wilting) to induce substantial increases in Put in plant

212 irulence genes in culture nor the ability to wilt tomato plants.

213 production of several virulence factors and wilted tomato plants several days more slowly than the w

214 Tomato spotted wilt tospovirus (TSWV), one of the most important plant

215 e nucleocapsid (N) protein of tomato spotted wilt (TSWV) tospovirus.

216 spheric microbial networks and caused peanut wilt under high than low soil phosphorus conditions.

217 s gene in rice alleviates leaf chlorosis and wilting under cold stress.

218 ted increased stomatal conductance and rapid wilting under water deficit stress.

219 t overproduce the tobacco anionic peroxidase wilt upon reaching maturity, although having functional

220 icylate and verbenone to two putative laurel wilt vectors in avocado, Xyleborus volvulus (Fabricius)

221 its most important contribution to bacterial wilt virulence in the early stages of host plant invasio

222 nonstructural protein NSs of tomato spotted wilt virus (a plant-infecting bunyavirus), the interfero

223 The plant-pathogenic virus tomato spotted wilt virus (TSWV) encodes a structural glycoprotein (G(N

224 hrips tabaci, which transmits Tomato spotted wilt virus (TSWV) in a persistent and propagative manner

225 es are described not only for Tomato spotted wilt virus (TSWV) in pepper and tomato but also for othe

226 baci is the primary vector of Tomato spotted wilt virus (TSWV) in some areas of the world, it is not

227 Tomato spotted wilt virus (TSWV) is a devastating disease to peanut gro

228 Tomato spotted wilt virus (TSWV) is a generalist pathogen with one of t

229 ilt caused by thrips-vectored tomato spotted wilt virus (TSWV) is a very serious problem in peanut (A

230 Spotted wilt caused by tomato spotted wilt virus (TSWV) is one of the major peanut (Arachis hy

231 Tomato spotted wilt virus (TSWV) is transmitted by Frankliniella occide

232 Tomato spotted wilt virus (TSWV) is transmitted exclusively by thrips i

233 content in populations of the tomato spotted wilt virus (TSWV) using microscopy and tomography.

234 Tomato spotted wilt virus (TSWV), a member of the Tospovirus genus with

235 lycoprotein N (G(N)) from the tomato spotted wilt virus (TSWV), a representative member of the Tospov

236 ng the model plant bunyavirus tomato spotted wilt virus (TSWV), and the most efficient thrips vector,

237 e we show that infection with Tomato spotted wilt virus (TSWV), type member of the only plant-infecti

238 yellow spot virus (IYSV) and Tomato spotted wilt virus (TSWV), were investigated for inter-virus int

239 l Sardinia virus (TYLCSV) and Tomato spotted wilt virus (TSWV).

240 entalis (Pergande), vector of tomato spotted wilt virus (TSWV).

241 The movement protein VP37 of broad bean wilt virus 2 (BBWV 2) forms tubules in the plasmodesmata

242 their efficient vectoring of tomato spotted wilt virus and iris yellow spot virus.

243 for quantitative detection of tomato spotted wilt virus as early as 4 days after inoculation.

244 Tomato spotted wilt virus is a wide-spread plant disease in the world.

245 Y, tobacco mosaic virus, and tomato spotted wilt virus were mapped in two or more genera and did not

246 red strong resistance against tomato spotted wilt virus, an economically important pathogen.

247 braskensis (Cmn), the causal agent of Goss's wilt, were incorporated into polyvinyl polymers with alc

248 These grafted plants never wilted when grown in the greenhouse though shoot peroxid

249 s of the osm1 mutant also were more prone to wilting when grown with limited soil moisture compared w

250 urrently provide adequate control of spotted wilt where severe epidemics occur.