ライフサイエンスコーパス: wind

1 and the Yellow Sea sector (through westerly winds).

2 cently ionized, and "picked up" by the solar wind.

3 f the sulfide by energetic ions of the solar wind.

4 st to the natural diversity in stratospheric winds.

5 ices, their debit, and the dimensions of the wound.

6 he spillage of intestinal effluvium from the wound.

7 um chloride in the immediate vicinity of the wound.

8 ergy from the disk in the form of a galactic wind(2).

9 This wind-albedo-wind feedback also leads to an increase in t

10 However, the interaction between wind and sex has not been comprehensively tested.

11 in coastal lowlands that are exposed to both wind and storm surge hazards.

12 n oil slick origin, deflection of which with wind and surface current leads to the formation of an oi

13 riability in microclimate driven by terrain, wind and vegetation, and ultimately bear little resembla

14 This reduces surface winds and decreases evaporative cooling and wind-driven

15 High winds and precipitation associated with storms can affec

16 ive stars are ejected into space via stellar winds and supernova explosions.

17 lus end-binding protein EB1/EBP-2 around the wound and actin ring formation, dependent on ARP2/3 bran

18 t previous instrumental signs of penetrating wound and complete visual restoration after surgery.

19 m flies, C. macellaria, that invade necrotic wound and feed on dead tissue.

20 luenced by the land sector (through easterly winds) and the Yellow Sea sector (through westerly winds

21 nnections generate oppositely directed trade-wind anomalies in the Central and Eastern Pacific during

22 eter wound (posterior) and one 4 mm diameter wound (anterior), each 1-1.5 mm deep, were created on bo

23 ts necessary to achieve 20% electricity from wind are analyzed using high resolution numerical simula

24 hase gradient, is distinguished by a current winding around the optical beam axis with a magnitude pr

25 rompted researchers to re-examine the normal wound bed physiology, resulting in new approaches to MSC

26 rative capacity of resident cells within the wound bed to overcome stalled wound healing.

27 R/SFRP4 and CD31 in the regenerated diabetic wound bed with TWIST1 overexpression or silencing (piLen

28 Compared with that of DeltasarA infection, wound biofilm burden was significantly higher in respons

29 an established preclinical porcine model of wound biofilm infection.

30 cluding acute irradiation injury and ectopic wound biopsies.

31 nkton in major areas of oceans and deposited wind-blown desert dust is a primary Fe source to these r

32 the costs of barrier crossings in prevailing winds can disrupt migratory routes towards slightly diff

33 Appropriate wound care is also essential.

34 Wound care professionals rely heavily on images and imag

35 a steadfast but time-consuming component of wound care with limited technical advancements to date.

36 and Tolvaptan, or V2R gene silencing reduced wound closure and cell viability of 786-O and Caki-1 hum

37 esis in the wound tissues results in delayed wound closure and healing.

38 TSG-6 plays an important role in regulating wound closure and inflammation during cutaneous wound re

39 We investigated how loss of TSG-6 affects wound closure and skin inflammation.

40 TSG-6-null wounds rescues both the delay in wound closure and the aberrant neutrophil phenotype.

41 0 from no suture group (nSG) showed complete wound closure at day 14 (P >0.05) and at 30 days, comple

42 ound edges enhanced inflammation and delayed wound closure in mice.

43 rmis is decreased in mouse models of delayed wound closure intended to mimic old age, obesity, and al

44 Wound closure rates are significantly delayed in TSG-6-n

45 ng model, i.p. MV administration accelerated wound closure through recruitment of PD-L1-expressing my

46 tial advantages over sutures and staples for wound closure, hemostasis, and integration of implantabl

47 0.1%, while the cooked honey had incomplete wound closure, the vacuum-treated honey trended towards

48 vacuum-treated honey trended towards faster wound closure.

49 Wound complication was present in 27.3% of patients, wit

50 4-6.35), biologic mesh (3.1, 1.67-5.75), and wound complications (3.01, 1.69-5.39) were predictors of

51 significantly reduced risk for perioperative wound complications (Odds Ratio 0.400 [95% confidence in

52 d models were used to identify predictors of wound complications and hernia recurrence, respectively.

53 posite of surgical-site infections and other wound complications, and adverse skin reactions.

54 Secondary outcomes included other wound complications, composite of surgical-site infectio

55 s requiring biologic mesh were predictors of wound complications, whereas recurrent hernia repair (2.

56 llergic contact dermatitis, and incidence of wound complications.

57 omentoplasty for the prevention of perineal wound complications.

58 ted to adjust foraging behaviour to variable wind conditions to minimize movement costs.

59 over the 55-y study period, improvements in wind conditions, especially in the Maghreb and Mediterra

60 getically expensive behaviour, to favourable wind conditions.

61 Training consisted of wind consistently preceding light from either the same o

62 The strength of the alongshore wind controls the thickness of the inflowing warm water

63 that basin-impact-generated hurricane-force winds created sediment-laden atmospheric conditions, and

64 biases inherent in peripheral mechanics, and wind cues are brought into the same circular coordinate

65 Surgical wound cultures and resistance data were obtained within

66 licly accessible tool, this study presents a wind damage model that is built around publicly availabl

67 matoma, seroma, surgical site infection, and wound dehiscence), abdominal eventration, and hernia rec

68 ndicates that iNPWT also reduces the risk of wound dehiscence, skin necrosis, and seroma.

69 vidence suggests that iNPWT may also prevent wound dehiscence, skin necrosis, seroma, and hematoma.

70 which a climatic driver, the Atlantic trade winds, determines the viability of a bird population.

71 During S phase, Hmo1 protects under-wound DNA from Top2, while Top2 confines Pol II and Top1

72 Zonal (latitudinal) winds dominate the bulk flow of planetary atmospheres.

73 f Enterococcus, which was also cultured from wound drainage.

74 ical incision (n = 816), or receive standard wound dressing (n = 808).

75 candidates for controlled drug release based wound dressing applications.

76 indicated that the prepared mat is a proper wound dressing for DFU management and treatment.

77 n 6.68% (50 of 749 patients) of the standard wound dressing group (odds ratio, 0.87 [95% CI, 0.57 to

78 s 13.2% [78 of 590 patients] in the standard wound dressing group; odds ratio, 0.84 [95% CI, 0.59 to

79 egative pressure over the wound, vs standard wound dressing not involving negative pressure (n = 763)

80 Here we describe the use of discarded wound dressings as a novel, cost effective, accessible,

81 om textiles to composites, and waveguides to wound dressings.

82 winds and decreases evaporative cooling and wind-driven upper ocean mixing.

83 during the Northeast monsoon is triggered by wind-driven upwelling, during the Southeast monsoon, it

84 l three modes of change in Southern Westerly Wind-driven upwelling, each affecting atmospheric carbon

85 This trend is independent of the solar wind driving, suggesting that it is an effect associated

86 nsity at geosynchronous orbit, and the solar wind dynamic pressure and IMF flux density at L1.

87 in their exposure to radiation, moisture and wind (e.g. topography, radiative forcing or cold-air poo

88 mulation of podoplanin-positive cells at the wound edge.

89 In this study, we show that in human wound-edge keratinocytes, the expressions of microRNA (m

90 -19a/b and miR-20a antisense inhibitors into wound edges enhanced inflammation and delayed wound clos

91 n chemical storage of intermittent solar and wind energy(1,2).

92 echniques for mapping land susceptibility to wind erosion hazards.

93 Thirteen factors controlling wind erosion were mapped, and multicollinearity among th

94 expertise can lead to improper diagnosis of wound etiology and inaccurate wound management and docum

95 millimeter Array (ALMA) and found that their winds exhibit distinct nonspherical geometries with morp

96 t state of knowledge on interactions between wind farms and bats in Europe, and compares it with the

97 This wind-albedo-wind feedback also leads to an increase in the frequency

98 esterly winds (SWW), changes in the westerly wind forcing could significantly affect the circulation

99 tes is pathogenic and contributes to chronic wound formation.

100 The evolution of a spherical wind from an asymptotic giant branch (AGB) star into a n

101 to perform a floating ostomy to isolate the wound from the debit enteric.

102 ll as healthy volunteers to study effects on wound healing (NCT04045405; NCT03603431).

103 l characteristics that might reproduce pASCs wound healing ability.

104 persecreting hMSC lines as short-term, local wound healing agents with superior therapeutic efficacy

105 reservoir of bioactive molecules to support wound healing and bone regeneration.

106 Sustained cell migration is essential for wound healing and cancer metastasis.

107 itecture of collagen type I is a hallmark of wound healing and cancer that is commonly attributed to

108 ectrotherapy protocol may accelerate palatal wound healing and decrease patient discomfort after FGG

109 allmarks that might apply to both cancer and wound healing and discuss how wounding, as in biopsy and

110 tory cells, which are active participants in wound healing and fibrogenic processes.

111 r matrix protein that has important roles in wound healing and fibrosis.

112 d, in so doing, to promote barrier function, wound healing and hair growth, while limiting cancer dev

113 ion of ribosomal proteins, and initiation of wound healing and humoral immune responses.

114 epithelial homeostasis, tissue regeneration, wound healing and immune modulation.

115 ial role in organ development and repair, in wound healing and in numerous pathological processes suc

116 s learned from the significant literature on wound healing and macrophage response to implanted bioma

117 Wound healing and Matrigel invasion assay, sphere format

118 Besides applications to wound healing and metastatic cancer, these studies are r

119 significantly reduce both corneal epithelial wound healing and nerve density in diabetic mice.

120 ways underlying angiogenesis, fertilization, wound healing and regeneration.

121 ained popularity in the field of periodontal wound healing and regeneration.

122 w of the angiogenic process during cutaneous wound healing and the regulatory roles played by catecho

123 ory responses, activities that contribute to wound healing and tissue repair.

124 d drug/cell delivery, thrombus ablation, and wound healing are reviewed from these viewpoints.

125 r is introduced for analyzing a standardized wound healing assay by observing cell growth and quantif

126 xperiments of cell proliferation, apoptosis, wound healing assay, as well as reverse-phase protein ar

127 Using an in vitro wound healing assay, cell migration was evaluated 2, 4,

128 is evaluated on sparse real-world data from wound healing assays with varying initial cell densities

129 tissue and its up-regulation is required for wound healing but is also involved in fibrosis.

130 After surgery or traumatic injury, corneal wound healing can cause a scarring response that stiffen

131 culture and mouse models of angiogenesis and wound healing confirmed these predicted deficiencies and

132 The wound healing efficacy of the fabricated dressings was e

133 of reducing bacterial load and accelerating wound healing in an excisional wound model.

134 egulatory function of WDR26 in FPR1-mediated wound healing in intestinal epithelial cells.

135 In an excisional wound healing model, i.p. MV administration accelerated

136 Using an endoscopic biopsy-based wound healing model, we report that RvE1 is locally prod

137 ype 1 and 2 diabetic full-thickness splinted wound healing murine model enhanced the microcirculatory

138 ion of this mechanism in studies in vivo, in wound healing or angiogenesis, in which fibrin is contra

139 ective tissue shares features with mammalian wound healing or fibrosis.

140 and non-sutured sites display similar early wound healing outcomes and patient-reported outcomes.

141 to chemoattraction of cells critical to the wound healing process, eCRT induces abundant neo-dermal

142 ted for their potential to contribute to the wound healing process.

143 Reduced wound healing ratios and nerve densities were not fully

144 scaffolds have great potential for improving wound healing treatments by providing controlled drug de

145 may help to uncover and develop fat-related wound healing treatments.

146 wounds were created using an Algerbrush, and wound healing was monitored over time.

147 ved in the LCD1 patients, corneal epithelial wound healing was significantly delayed in TGFBI-R124C m

148 oteins associated with skin blood supply and wound healing were altered.

149 eview provides a brief overview of cutaneous wound healing with discussion on how extracellular matri

150 ferences were found for complicated perineal wound healing within 30 days (RR 1.30; 95% CI 0.92-1.82)

151 sed two pathway-focused RT-PCR gene arrays ("wound healing" and "neurogenesis") to evaluate tissue sa

152 iseases (e.g., cancer, heart attack, stroke, wound healing).

153 nning, fragility, wrinkles, laxity, impaired wound healing, and a microenvironment conducive to cance

154 ding on the involvement of adipose tissue in wound healing, and may help to uncover and develop fat-r

155 for normal processes such as development and wound healing, but can go awry, as in oncogenesis and fi

156 gh cell plasticity underlying embryogenesis, wound healing, cancer metastasis and drug resistance.

157 fferent roles of fibroblasts in ECM biology, wound healing, diseases, and aging.

158 Striking similarities between wound healing, epimorphic regeneration and the progressi

159 gene expression in three settings: in vitro wound healing, live lymph node sections and a live tumor

160 rom bone marrow cells is required for normal wound healing, revealing a physiological role for this g

161 proliferation, colony formation, migration, wound healing, tumor growth, and lung metastasis.

162 and it is an essential step during cutaneous wound healing, which supports cells at the wound site wi

163 lteration resembling the remodeling phase of wound healing, with increased matrix metalloproteinase e

164 ion, cell dispersion, neuronal survival, and wound healing.

165 ined in the animal studies indicate a proper wound healing.

166 ers that coalesce into clots which assist in wound healing.

167 tiation and phenotypic changes in cancer and wound healing.

168 could be reactivated and play roles in adult wound healing.

169 s medically and/or electronically facilitate wound healing.

170 es of microcurrent electrotherapy on palatal wound healing.

171 /f)Lyz2(Cre+)) resulted in improved diabetic wound healing.

172 in vitro slows keratinocyte migration during wound healing.

173 ion of inflammation, cell proliferation, and wound healing.

174 L28 and IL-6 levels associated with improved wound healing.

175 the transcriptional landscape that influence wound healing.

176 glycoprotein 1 (LRG1) in normal and diabetic wound healing.

177 EMT is crucial to embryonic development and wound healing.

178 actor-beta (TGFbeta) play a critical role in wound healing.

179 lls within the wound bed to overcome stalled wound healing.

180 MoS(2) on integrins, cellular migration, and wound healing.

181 with the NPWT system in order to accelerate wound healing.

182 tial changes in collagen organization during wound healing.

183 FU, indicating a potential important role in wound healing.

184 the immune system is inextricably linked to wound healing/remodeling in the ischemically injured hea

185 stem cells (ASCs) have potential to improve wound healing; however, their equivalents from domestic

186 epared derivatives showed promising in vitro wound-healing activity.

187 We report on in vitro wound-healing and cell-growth studies under the influenc

188 l wounding in Hydra, suggesting that Hydra's wound-healing and self-organization capabilities may emp

189 The wound-healing assay demonstrated that, at concentrations

190 Subsequent in vitro wound-healing assays also confirmed that M2 and M13 acce

191 starting point for the development of novel wound-healing promoters.

192 y is thus due to an acute inflammatory-based wound-healing response that rejuvenates the infarcted ar

193 W amplitude, resulting in an increase of the wound-healing speed of up to 135 +/- 85% as compared to

194 Wound-healing was also impaired in PSW compared to contr

195 balance between external forcing (radiation, winds, heat and freshwater fluxes) and the emergent turb

196 nergy infrastructure associated with onshore wind, hydropower and solar photovoltaic generation, with

197 lated from a normally sterile site or from a wound in patients with necrotizing fasciitis or streptoc

198 n screw configuration, further increased the wound-induced accumulation of total free (296.6%) and bo

199 This localized auxin increase balances wound-induced cell expansion and restorative division ra

200 m cell persistence after injury, implicating wound-induced ERK activity in this response.

201 ells in the notochord bead and for promoting wound-induced proliferation required for efficient regen

202 lenged in vivo with the polybacterial bovine wound infection 'digital dermatitis', Zn/Cu-shellac adhe

203 ponse syndrome, sepsis, acute kidney injury, wound infection (superficial and deep), rate of intraope

204 yse both host and pathogen biomarkers during wound infection in near real-time.

205 ls, (ii) intradermal infection models, (iii) wound infection models, and (iv) epicutaneous infection

206 myositis and a clinically relevant S. aureus wound infection murine model.

207 opathy, cerebral radionecrosis) and surgery (wound infections, flap necrosis, fistulas,...).

208 ted because three patients developed delayed wound infections.

209 9a/b and miR-20a being crucial regulators of wound inflammation, the lack thereof may contribute to s

210 Finally, in simulations of wound invasion, efficient collective migration is achiev

211 ultiple logistic regression model the factor wound irrigation with polyhexanide [odds ratio (OR) 0.44

212 r diagnosis of wound etiology and inaccurate wound management and documentation.

213 Digital education on chronic wound management appears to be less effective than blend

214 is complicated by forecast errors and sparse wind measurements.

215 MSCs face a variety of challenges within the wound micro-environment that curtail their survival afte

216 rotein-rich environment of a purulent animal wound model infected with drug-resistant bacteria.

217 restore saliva flow rate in a salivary gland wound model of C57BL/6 mice.

218 accelerating wound healing in an excisional wound model.

219 invariant (HTI), which consists of distinct winding numbers associated with Berry phases accumulated

220 upercoils associated with the over- or under-winding of duplex DNA.

221 bjected to an electrical current, the chiral winding of the spin texture leads to a deflection of the

222 ly, by its influence on air temperatures and winds, or directly, mostly through its effects on ice sh

223 A cluster analysis of daily wind patterns shows more frequent circulation regimes th

224 owed us to gain mechanistic insight into the wound perception and coordination of wound responses aft

225 Intravital biosensor imaging showed that wound peroxide and arachidonic acid converged on half-mi

226 ogenous FXIII was topically applied into the wound pocket of rats, eleven adhesive failures occurred

227 spatial synchrony-is a pervasive strategy in wind-pollinated trees that is hypothesized to be vulnera

228 sociated with both adaptive benefits of CVp (wind pollination and seed dispersal) and climatic variab

229 One 6 mm diameter wound (posterior) and one 4 mm diameter wound (anterior)

230 Therefore, more research on wind power and bats is needed in this region, as well as

231 meters were analyzed inside tooth-extraction wound: proportion of newly formed bone (bone healing/BH)

232 ice and/or biliary stent only, regardless of wound protector use (odds ratio = 0.69-0.70, P < 0.001).

233 Solar wind provides an example of a weakly collisional plasma

234 our tropical cyclone hazards: peak sustained wind, rainfall, flooding, and tornadoes.

235 lso confirmed that M2 and M13 accelerate the wound recovery process of Caco-2 cells at the concentrat

236 the sexes to exploit regions with different wind regimes.

237 and connected component labelling to segment wound regions from natural images.

238 we show that Pavarotti also functions during wound repair and confirm that while Pavarotti, Tumblewee

239 rly relevant in biological processes such as wound repair and embryonic development where cell spread

240 Resolution of intestinal inflammation and wound repair are active processes that mediate epithelia

241 gator tails identifies a distinct pattern of wound repair in mammals while exhibiting features in com

242 Efficient wound repair is critical to reestablish the mucosal barr

243 We show that WDR26-mediated inhibition of wound repair is mediated through the inhibition of Rac f

244 ctors inducing greater bone regeneration and wound repair than wild-type growth factors, as well as r

245 ellular matrix during embryonic development, wound repair, and tumor invasion.

246 ole of adult skin stem cells in homeostasis, wound repair, inflammation, and cancer.

247 nd closure and inflammation during cutaneous wound repair.

248 he miR-17~92 cluster, are upregulated during wound repair.

249 ration in plants and distinguish between the wound-repair ability of the tissue and its formation dur

250 tion years and may reflect saturation of the wind resource in some areas.

251 and (iv) in vivo temporal monitoring of the wound response in living zebrafish embryos.

252 Wound-response plant growth restriction requires the syn

253 nto the wound perception and coordination of wound responses after laser-based wounding in Arabidopsi

254 Shifting the wind rightward rotates the compass as if the fly were tu

255 y be limited to topical applications such as wound rinses and dressings.

256 This involves averaging wind-rotated observations from 14 to 24 overpasses to ac

257 egory 4 or 5 on the Saffir-Simpson Hurricane Wind Scale) hurricane landfalls, prompting questions abo

258 Our observations suggest that the wound signaling involves the sensing of collapse of dama

259 nvolved in physiological processes including wound signaling, stomatal regulation, and pollen tube gr

260 solic Ca(2+) increases to propagate systemic wound signaling.

261 s wound healing, which supports cells at the wound site with nutrition and oxygen.

262 ent of PD-L1-expressing myeloid cells to the wound site.

263 ocations for replacing power plants with new wind, solar, or natural gas to meet a CO(2) reduction ta

264 enewable electricity generated from solar or wind sources.

265 transitioning between states in response to wind speed and relative direction, and sex.

266 integration of a multitude of cues, such as wind speed and solar elevation, and the process is compl

267 in the facility was driven predominantly by wind speed and the formation of the deciduous canopy.

268 a role of ambient temperature, humidity and wind speed in affecting RSV transmission that could be b

269 hold, we regressed the drag coefficients for wind speed scope from 10 ms(-1) to 28 ms(-1).

270 To estimate the saturated wind speed threshold, we regressed the drag coefficients

271 d to the planet's interior, to determine the wind speed.

272 est to flight) in stronger headwinds, and as wind speeds increased, to be in directed flight rather t

273 We apply this method to measure the wind speeds on the brown dwarf 2MASS J10475385+2124234.

274 se in the frequency of hours spent at higher wind speeds, which has implications for dust emission po

275 l-Nino/La-Nina events, the Southeast monsoon wind strength over the south tropical Indian Ocean is th

276 acts in opposition to decreasing basin-scale wind stress and has a potentially important warming impa

277 th initially increases due to changes in the wind stress, then undergoes a slowdown, followed by a re

278 in "small steps" of 5 mm, in an approximate wound-suture ratio of 1:10.

279 fluenced by the Southern Hemisphere westerly winds (SWW), changes in the westerly wind forcing could

280 South America with the circumpolar westerly wind system.

281 light, was influenced to a greater degree by wind than females.

282 Incisional negative pressure wound therapy (n = 785), which involved a specialized dr

283 g 3D printing for use with negative pressure wound therapy (NPWT) in the management of enteroatmosphe

284 atients) of the incisional negative pressure wound therapy group and in 6.68% (50 of 749 patients) of

285 atients] in the incisional negative pressure wound therapy group vs 13.2% [78 of 590 patients] in the

286 port the use of incisional negative pressure wound therapy in this setting, although the event rate a

287 ither undergo prophylactic negative pressure wound therapy, with application of the negative pressure

288 ivated Draper then guides macrophages to the wound through the detection of an as-yet unidentified ch

289 ation tissue collagen leading to compromised wound tissue biomechanics.

290 gh amounts of interleukin 31 (IL-31) in skin wound tissue during the peak of itch responses.

291 induce neovascularization-mediated diabetic wound tissue regeneration.

292 rocirculatory blood flow and accelerated the wound tissue regeneration.

293 Impaired angiogenesis in the wound tissues results in delayed wound closure and heali

294 on that the ripples propagate from the solar wind to the F-region, and that they are a related, persi

295 addition, this fast module does not control wound-triggered JA accumulation in Arabidopsis (Arabidop

296 RCV) was nebulized in a custom-built, 3.86 m wind tunnel housed in a biosafety level class II facilit

297 Impacts from current and future wind turbine (WT) deployments necessary to achieve 20% e

298 m transcriptional responses in the immediate wound vicinity.

299 ng used to create negative pressure over the wound, vs standard wound dressing not involving negative

300 nd attribution analysis of atmospheric zonal wind, we show that the pause in circulation trends is fo