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1 iseases (e.g., cancer, heart attack, stroke, wound healing).
2  MSC differentiation and HO formation during wound healing.
3 glycoprotein 1 (LRG1) in normal and diabetic wound healing.
4 ch can stabilize the injury site and promote wound healing.
5 ely promotes epithelial barrier function and wound healing.
6 ls is critical for embryonic development and wound healing.
7 lar matrix (ECM) production, chemotaxis, and wound healing.
8 onses that affected invasion, migration, and wound healing.
9 ia caused by Pseudomonas aeruginosa and poor wound healing.
10 rogression, and the same might be useful for wound healing.
11 diseases, chronic inflammatory disorders and wound healing.
12 muscle actin (alpha-SMA), and cell migration/wound healing.
13 tracellular communication, and attributed to wound healing.
14 he wounded cornea during the early phases of wound healing.
15 ility to invade and migrate during epidermal wound healing.
16 due to their diverse roles in the process of wound healing.
17 esses such as digestion, blood clotting, and wound healing.
18  and resolution of the inflammatory phase of wound healing.
19 mation or its absence may result in impaired wound healing.
20 lium-to the cells and cytokines that mediate wound healing.
21 ered for further cell-based therapy study on wound healing.
22 fection to specific pathogenic mechanisms in wound healing.
23 e treatment of MRSA infections as well as in wound healing.
24 viduals with skin conditions associated with wound healing.
25 is developmental program results in fibrotic wound healing.
26  EMT is crucial to embryonic development and wound healing.
27 actor-beta (TGFbeta) play a critical role in wound healing.
28  acute kidney injury, acute liver injury and wound healing.
29 lls within the wound bed to overcome stalled wound healing.
30 n intestinal epithelial barrier function and wound healing.
31 MoS(2) on integrins, cellular migration, and wound healing.
32 ase growth factors for the promotion of skin wound healing.
33 IAMs with the injury microenvironment during wound healing.
34 tion of tumor angiogenesis without affecting wound healing.
35  with the NPWT system in order to accelerate wound healing.
36 perience impaired tube formation and delayed wound healing.
37 ature dermal adipocytes for hair cycling and wound healing.
38  scaffold functionality during the course of wound healing.
39 nd myeloid cells during the course of normal wound healing.
40 ysiological tissue regeneration and inhibits wound healing.
41 tion acts to suppress infections and support wound healing.
42 or testing of new therapeutics for cutaneous wound healing.
43 ntly been shown to directly affect nonneural wound healing.
44 tages of M1 macrophage activation to promote wound healing.
45 tial changes in collagen organization during wound healing.
46 FU, indicating a potential important role in wound healing.
47 atively affecting tissue regeneration during wound healing.
48 y diseases that are associated with impaired wound healing.
49 determine MLL1 drives Tlr4 expression during wound healing.
50 ion, cell dispersion, neuronal survival, and wound healing.
51 ined in the animal studies indicate a proper wound healing.
52 ers that coalesce into clots which assist in wound healing.
53 tiation and phenotypic changes in cancer and wound healing.
54 could be reactivated and play roles in adult wound healing.
55 s medically and/or electronically facilitate wound healing.
56 es of microcurrent electrotherapy on palatal wound healing.
57 /f)Lyz2(Cre+)) resulted in improved diabetic wound healing.
58 ion of inflammation, cell proliferation, and wound healing.
59 L28 and IL-6 levels associated with improved wound healing.
60 the transcriptional landscape that influence wound healing.
61 in vitro slows keratinocyte migration during wound healing.
62 al to ensure efficient, spatially restricted wound healing.
63 tracellular creatine, barrier formation, and wound healing.
64 associated with tissue injury is followed by wound healing, a complex process by which damaged cellul
65            Their possible role in human oral wound healing, a process that differs from cutaneous wou
66 ny mouse, Acomys cahirinus displays a unique wound healing ability with regeneration of all skin comp
67 l characteristics that might reproduce pASCs wound healing ability.
68 w that VWF deficiency in mice causes delayed wound healing accompanied by decreased angiogenesis and
69 lar mechanism underlying the peptide-induced wound healing activity, these studies have contributed t
70 epared derivatives showed promising in vitro wound-healing activity.
71  on presacral abscess formation and perineal wound healing after APR, while it increases the likeliho
72 cation on the efficacy and safety of various wound healing agents to improve chronic diabetic wounds.
73 persecreting hMSC lines as short-term, local wound healing agents with superior therapeutic efficacy
74  of the immune system in response to trauma, wound healing and anaesthesia.
75 anical mechanism in fibrin, a key element in wound healing and angiogenesis processes.
76   Here, we investigated the differentiation, wound healing and angiogenic capacity of plastic adheren
77  reservoir of bioactive molecules to support wound healing and bone regeneration.
78 rophic and keloid scars result from abnormal wound healing and can have a variable response to a numb
79    Sustained cell migration is essential for wound healing and cancer metastasis.
80 itecture of collagen type I is a hallmark of wound healing and cancer that is commonly attributed to
81 ocrine/paracrine signaling with relevance to wound healing and cellular transformation.
82 ectrotherapy protocol may accelerate palatal wound healing and decrease patient discomfort after FGG
83 s (STZ-induced and db/db) results in delayed wound healing and decreased neutrophil and macrophage re
84 allmarks that might apply to both cancer and wound healing and discuss how wounding, as in biopsy and
85 ity, soft and hyperextensible skin, abnormal wound healing and easy bruising.
86 tory cells, which are active participants in wound healing and fibrogenic processes.
87 r matrix protein that has important roles in wound healing and fibrosis.
88                      RvD6si promotes corneal wound healing and functional recovery by restoring corne
89 d, in so doing, to promote barrier function, wound healing and hair growth, while limiting cancer dev
90 ion of ribosomal proteins, and initiation of wound healing and humoral immune responses.
91 epithelial homeostasis, tissue regeneration, wound healing and immune modulation.
92 INGO2-EGFR complexes, allowing TFF3 to drive wound healing and immunity.
93     We also found roles for Crk in embryonic wound healing and in axon patterning in the nervous syst
94 ial role in organ development and repair, in wound healing and in numerous pathological processes suc
95 chaperone protein, in animal models enhances wound healing and induces tissue regeneration evidenced
96 extracellular polyphosphate as a mediator of wound healing and inflammation and also provide a potent
97                        Neutrophils are early wound healing and inflammation regulators that, due to f
98 enchymal characteristics during development, wound healing and inflammation, and in cancer and fibros
99 s learned from the significant literature on wound healing and macrophage response to implanted bioma
100                                              Wound healing and Matrigel invasion assay, sphere format
101 cidate the mechanisms of efficient embryonic wound healing and may have far-reaching implications for
102                      Besides applications to wound healing and metastatic cancer, these studies are r
103 min D deficiency (VDD) on corneal epithelial wound healing and nerve density in diabetic mice.
104 significantly reduce both corneal epithelial wound healing and nerve density in diabetic mice.
105 t during developmental stages, angiogenesis, wound healing and other processes.
106 ame gland to uncover mechanisms that prevent wound healing and regeneration in mammals.
107 , including development, tissue homeostasis, wound healing and regeneration, immunity, and tumorigene
108 ways underlying angiogenesis, fertilization, wound healing and regeneration.
109 ained popularity in the field of periodontal wound healing and regeneration.
110  protein) plays an important role in cardiac wound healing and remodeling.
111 ween hemostasis and lymphangiogenesis during wound healing and reveal that VEGFC, the dominant lympha
112 able literature regarding the use of CPs for wound healing and skin tissue engineering applications,
113  offer insights into the process of diabetic wound healing and suggest topical mast cell stabilizatio
114 w of the angiogenic process during cutaneous wound healing and the regulatory roles played by catecho
115 almology, and stimulation of angiogenesis in wound healing and tissue engineering.
116 population dynamics that regulate subsequent wound healing and tissue regeneration are still unclear.
117 ory responses, activities that contribute to wound healing and tissue repair.
118 ysics to address biomedical problems such as wound healing and tumor suppression.
119                        We report on in vitro wound-healing and cell-growth studies under the influenc
120 itions, as revealed by significantly delayed wound-healing and impaired tumour growth.
121 l wounding in Hydra, suggesting that Hydra's wound-healing and self-organization capabilities may emp
122 sed two pathway-focused RT-PCR gene arrays ("wound healing" and "neurogenesis") to evaluate tissue sa
123 nning, fragility, wrinkles, laxity, impaired wound healing, and a microenvironment conducive to cance
124 tory roles in hemostasis, tumor development, wound healing, and atherogenesis.
125 ncreased at the inflammation stage of murine wound healing, and bone marrow-derived cells served as a
126 ation is essential to embryonic development, wound healing, and cancer cell dissemination.
127                              In development, wound healing, and cancer metastasis, vertebrate cells m
128 mplicate IL-17 as a link among inflammation, wound healing, and cancer.
129 ulticellular collectives during development, wound healing, and cancer.
130 ic microRNA profile of skin and oral mucosal wound healing, and demonstrate the feasibility of a micr
131 ding on the involvement of adipose tissue in wound healing, and may help to uncover and develop fat-r
132  barrier repair, keratinocyte proliferation, wound healing, and negative regulation of T-cell activat
133 ding of fibroblast heterogeneity during skin wound healing, and on open questions that must be addres
134 es and recruited neutrophils also potentiate wound healing, and polyphosphate also promotes macrophag
135 s such as morphogenesis, tissue homeostasis, wound healing, and tumorigenesis.
136 brotic myofibroblast functions and excessive wound healing; and (5) establishes the effectiveness of
137 anti-inflammatory, anticancer, antidiabetic, wound healing, anti-HIV, anti-anxiety and antidepressant
138 eroxide (H(2) O(2) ) mediates the biology of wound healing, apoptosis, inflammation, etc.
139          Morphogenesis, tumor formation, and wound healing are regulated by tissue rigidity.
140 d drug/cell delivery, thrombus ablation, and wound healing are reviewed from these viewpoints.
141 r is introduced for analyzing a standardized wound healing assay by observing cell growth and quantif
142 lial cell migration and repair after scratch-wound healing assay that was associated with reduced cel
143 xperiments of cell proliferation, apoptosis, wound healing assay, as well as reverse-phase protein ar
144                            Using an in vitro wound healing assay, cell migration was evaluated 2, 4,
145 926 cell migration was determined by scratch-wound healing assay.
146                                          The wound-healing assay demonstrated that, at concentrations
147  is evaluated on sparse real-world data from wound healing assays with varying initial cell densities
148 mined by intracellular calcium measurements, wound healing assays, and synthetic activation by gene a
149                          Subsequent in vitro wound-healing assays also confirmed that M2 and M13 acce
150 utic for inflammatory diseases and defective wound healing associated with aging and diabetes.
151 important roles in other settings as sterile wound healing, autoimmunity, cancer, and homeostasis.
152  results suggest that Gal-3 is essential for wound healing because it regulates the dynamics of macro
153 e (EGCG), a polyphenol, influences cutaneous wound healing because of its antiangiogenic, anti-inflam
154 t cell cycle stalling in G2 has key roles in wound healing but becomes detrimental upon chronic JNK o
155 tissue and its up-regulation is required for wound healing but is also involved in fibrosis.
156 for normal processes such as development and wound healing, but can go awry, as in oncogenesis and fi
157 ed fibroblast like cells that participate in wound healing, but little is known about what initiates
158 aling, a process that differs from cutaneous wound healing by being faster and typically scar-free, h
159 ilability, enhance angiogenesis, and improve wound healing by disrupting CCL28-activated CCR10-eNOS i
160 ) have been recently described to accelerate wound healing by enhanced keratinocyte migration and ind
161 al miR-155 inhibition would improve diabetic wound healing by restoring FGF7 expression.
162                     Patients with a surgical wounds healing by secondary intention (an open wound, <3
163 istatin-1 was recently shown to promote oral wound healing, by acting in epithelial and endothelial c
164   After surgery or traumatic injury, corneal wound healing can cause a scarring response that stiffen
165 gh cell plasticity underlying embryogenesis, wound healing, cancer metastasis and drug resistance.
166 process that occurs during tissue formation, wound healing, cancerous invasion, and viral infection.
167                      Moreover, a decrease in wound healing capacity in hTERT-MCPyV gene-expressing HK
168 ding, topical MCS-01 application accelerated wound healing comparable to that achieved with systemic
169                                     Impaired wound healing complicates a wide range of diseases and r
170                                  The risk of wound healing complications (WHCs) and the early use of
171 e) at 3 and 6 months, as well as other local wound healing complications at 30 days.
172 culture and mouse models of angiogenesis and wound healing confirmed these predicted deficiencies and
173 migratory dynamics during episodes of tissue wound healing, development, and plasticity, but is more
174 fferent roles of fibroblasts in ECM biology, wound healing, diseases, and aging.
175 ere utilized as a drug delivery platform for wound healing due to their low cytotoxicity.
176 C cortisol, which is responsible for delayed wound healing during a systemic excess of GC.
177 2-NL, M13-NL) demonstrated excellent in vivo wound-healing effects, and these activities were better
178                                          The wound healing efficacy of the fabricated dressings was e
179                                        After wound healing, emerging fluctuations in the phase shift
180                Striking similarities between wound healing, epimorphic regeneration and the progressi
181                                       During wound healing, expression of AP-1B (and ARH in AP-1B-pos
182 cal processes such as embryonic development, wound healing, fibrosis and cancer.
183 ties to epithelial cells during development, wound-healing, fibrosis and cancer(1-4).
184 as the development or progression of cancer, wound healing, fibrotic disorders, and immune modulation
185 al potential to accelerate extraction socket wound healing for more predictable dental implant recons
186 PAR-gamma was critical for the expression of wound healing genes in AM.
187                                         Poor wound healing has also been observed in in some patients
188  stem cells (ASCs) have potential to improve wound healing; however, their equivalents from domestic
189  treatment with miR-19b and miR-20a improved wound healing in a mouse model of type 2 diabetes.
190 e effects of MaR1 on tooth extraction socket wound healing in a preclinical rat model.
191 ue formation and vascularization during skin-wound healing in AD-HIES patients.
192  of reducing bacterial load and accelerating wound healing in an excisional wound model.
193 nd in combination, on the maturation step of wound healing in an ischemic infected delayed healing wo
194       Mast cells have been shown to regulate wound healing in diabetes.
195 ia in diabetic skin and specifically impairs wound healing in diabetes.
196 de of this protein significantly accelerated wound healing in diabetic mice and may be a potential th
197 trategies to improve iPS cell generation and wound healing in elderly individuals.
198 ct of strontium ranelate on tooth-extraction wound healing in estrogen-deficient and estrogen-suffici
199 or targeting CD26(+) fibroblasts to modulate wound healing in human skin.
200 26, which negatively regulates FPR1-mediated wound healing in intestinal epithelial cells.
201 egulatory function of WDR26 in FPR1-mediated wound healing in intestinal epithelial cells.
202  concentration range necessary for arresting wound healing in live cells.
203 n of eicosapentaenoic acid (EPA)-rich oil on wound healing in mice.
204                                              Wound healing in plant tissues, consisting of rigid cell
205        Furthermore, the rapid and consistent wound healing in seedling grafts along with lateral shoo
206 ellular mechanisms by which SOX2 accelerates wound healing in skin.
207 herapeutic evaluation of iron use to promote wound healing in the clinic.
208                                     Impaired wound healing in the diabetic foot is a major problem of
209 ehavior assessed by migration, invasion, and wound healing in vitro.
210 acity of bone marrow-derived MSCs to promote wound healing in vitro.
211 2 is a key contributor to intestinal mucosal wound healing in vivo.
212 ets induce blood clotting (the first step in wound healing) in part by the release of polyphosphate.
213  the analgesic intake and the modified early-wound healing index (MEHI).
214                                    Cutaneous wound healing is associated with the unpleasant sensatio
215        It is unknown whether the chick fetal wound healing is capable of recapitulating this developm
216                                              Wound healing is characterized by cell and extracellular
217                                      Delayed wound healing is commonly associated with diabetes.
218  gene expression in three settings: in vitro wound healing, live lymph node sections and a live tumor
219 dition, favorable modulation of inflammatory wound healing markers occurred when electrotherapy was a
220                   Common culprits of altered wound healing mechanics, including angiogenesis and infl
221 ntain, evolutionarily conserved regenerative wound healing mechanisms in mucosal tissue.
222                We found that IL-7 stimulated wound-healing migration and invasion of PC-3 cells, incr
223 tance, barrier function, actin localization, wound healing, mitochondrial oxygen consumption, and gly
224                              In a Caco2 cell wound healing model, CTB-KDEL, but not CTB or CTB-KDE, f
225                             In an excisional wound healing model, i.p. MV administration accelerated
226             Using an endoscopic biopsy-based wound healing model, we report that RvE1 is locally prod
227 urrounding area to the center in an in vitro wound healing model.
228 eviously shown) and H(2) O(2) in a zebrafish wound healing model.
229 urther exploration of this new approach in a wound-healing model and opens the door for the future us
230 ype 1 and 2 diabetic full-thickness splinted wound healing murine model enhanced the microcirculatory
231 ll as healthy volunteers to study effects on wound healing (NCT04045405; NCT03603431).
232 s showing senescence in postmitotic tissues, wound healing, obesity, and development.
233 evaluate the effect of suturing on the early wound healing of donor sites of subepithelial connective
234 s been shown to regulate the homeostasis and wound healing of the corneal epithelium.
235 ion of this mechanism in studies in vivo, in wound healing or angiogenesis, in which fibrin is contra
236 ective tissue shares features with mammalian wound healing or fibrosis.
237 Because the supplemental diet did not rescue wound healing or nerve density, these effects are likely
238  and non-sutured sites display similar early wound healing outcomes and patient-reported outcomes.
239 ion, with important implications for chronic wound healing pathologies or tumorigenic transformation.
240 /B2 in the clearance of bacterial infection, wound healing, periodontal disease, neurogenic inflammat
241           The most common complications were wound healing problems (n = 270/56%), foreign body react
242                        In characterizing the wound healing process these morphological, mechanical, a
243  to chemoattraction of cells critical to the wound healing process, eCRT induces abundant neo-dermal
244 y the variety of immunological reactions and wound healing process.
245 ted for their potential to contribute to the wound healing process.
246 s, for treating disease states, accelerating wound healing processes and eliminating infections.
247 dant activity of honeys may be beneficial in wound healing processes by protecting cells against lipi
248                                           As wound healing progresses, the collagen organization dram
249      In summary, MaR1 accelerated extraction wound healing, promoted socket bone fill, preserved alve
250  starting point for the development of novel wound-healing promoters.
251 ical samples and applied to accelerate tooth wound healing, providing an intriguing insight of how to
252 ied therapeutic could be used to improve the wound healing quality in RDEB and DDEB.
253 ons (1, 2.5, and 5 mM) significantly reduced wound healing rates, whereas nicotine at a concentration
254                                      Reduced wound healing ratios and nerve densities were not fully
255  the immune system is inextricably linked to wound healing/remodeling in the ischemically injured hea
256                                   Epithelial wound healing requires a complex orchestration of cellul
257 o myofibroblasts during liver injury and the wound healing response.
258                               Therefore, the wound-healing response in the infarct area typically yie
259 y is thus due to an acute inflammatory-based wound-healing response that rejuvenates the infarcted ar
260 led that while most of the gland underwent a wound-healing response that resolved by day 14, a small
261    We discuss the concept of metaplasia as a wound-healing response, and how cardiac mucosa might be
262 al cell state marked by low Wnt signaling, a wound-healing response, and transcription factor Klf6 ex
263 that hemostatic responses might be linked to wound healing responses is an old one, but demonstrating
264    Understanding regional differences in the wound-healing responses may ultimately affect regenerati
265 nant cells(3,4) and has a beneficial role in wound-healing responses(5,6).
266  dampen inflammation in favor of homeostatic wound-healing responses, thus potentially contributing t
267 rom bone marrow cells is required for normal wound healing, revealing a physiological role for this g
268  We previously demonstrated that chick fetal wound healing shows a regenerative phenotype regarding t
269                      After 18 to 20 weeks of wound healing, sites were surgically re-entered, ridge d
270 W amplitude, resulting in an increase of the wound-healing speed of up to 135 +/- 85% as compared to
271 and CD4( +) T memory resting cells, but more wound healing than LSCC.
272 mphasize the need for experimental models of wound healing that better approximate human biology, as
273 s of miRNA/mRNA expression during human oral wound healing; the results offer novel insights into the
274 ssibility of SERPINB1 and IL-33 as potential wound healing therapeutic targets.
275 layered wound patch represents an innovative wound healing therapy.
276 zed by gastric cell expression of markers of wound-healing; these cells are called spasmolytic polype
277 he deficit of Gal-3 affected the dynamics of wound healing, thus aggravating the evolution of remodel
278 ons of protein-based adhesives and fibers in wound healing, tissue regeneration, drug delivery, biose
279 asts are fibroblastic cells that function in wound healing, tissue repair and fibrosis, and arise fro
280 on of the cytokine TGF-beta1, which promotes wound healing, to dermal DCs in vitro was sufficient to
281 scaffolds have great potential for improving wound healing treatments by providing controlled drug de
282  may help to uncover and develop fat-related wound healing treatments.
283 , a transglutaminase involved in hemostasis, wound healing, tumor growth, and apoptosis.
284  proliferation, colony formation, migration, wound healing, tumor growth, and lung metastasis.
285  the roles of Smad7 in skin inflammation and wound healing using genetic and pharmacological approach
286 ecific emphasis on perioperative infections, wound healing, vascular complications, cancer propagatio
287  to induce UPR and epithelial restitution or wound healing was corroborated in a dextran sodium sulfa
288 wounds were created using an Algerbrush, and wound healing was monitored over time.
289 ved in the LCD1 patients, corneal epithelial wound healing was significantly delayed in TGFBI-R124C m
290                                              Wound-healing was also impaired in PSW compared to contr
291 ed during epiboly resembles the mechanism of wound healing, we propose actomyosin cable-driven local
292 oteins associated with skin blood supply and wound healing were altered.
293 and it is an essential step during cutaneous wound healing, which supports cells at the wound site wi
294 neal epithelial cells enhanced their rate of wound healing while its ablation using shRNA reduced hea
295 eview provides a brief overview of cutaneous wound healing with discussion on how extracellular matri
296 ic rats significantly enhanced osteochondral wound healing with reduced vascularisation in the repair
297 lteration resembling the remodeling phase of wound healing, with increased matrix metalloproteinase e
298 ferences were found for complicated perineal wound healing within 30 days (RR 1.30; 95% CI 0.92-1.82)
299 nd-free episodes by improving the quality of wound healing would therefore confer substantial benefit
300      Calcium is an important early signal in wound healing, yet how these early signals promote regen

 
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