ライフサイエンスコーパス: wound infection

1 atient required removal of the system due to wound infection.

2 t rates among patients with an uncomplicated wound infection.

3 %) of all PD readmissions were attributed to wound infection.

4 e generator, transient oscillopsia and minor wound infection.

5 t can accomplish through gastrointestinal or wound infection.

6 n of wound or bone healing or treatment of a wound infection.

7 and culture-confirmed Pythium aphanidermatum wound infection.

8 reimplantation of the neurostimulator due to wound infection.

9 One patient developed a wound infection.

10 on on meat products, and potentially towards wound infection.

11 ia in a model of Pseudomonas aeruginosa burn wound infection.

12 e primary outcome variable was postoperative wound infection.

13 red a permanent neurologic deficit, none had wound infection.

14 clinically relevant murine model of surgical wound infection.

15 ne regeneration, inflammatory reactions, and wound infection.

16 eutrophils in FL-mediated resistance to burn wound infection.

17 cal role in FL-mediated resistance to a burn wound infection.

18 terations in B and T lymphocyte responses to wound infection.

19 outcome of rats in response to a lethal burn wound infection.

20 , almost entirely due to increasing rates of wound infection.

21 wound, and further increased in response to wound infection.

22 se of antibiotics prior to insertion reduces wound infection.

23 in mouse models of abdominal sepsis and burn wound infection.

24 nt with IL-6-deficient MCs failed to control wound infection.

25 immunization of mice against Pf prevents Pa wound infection.

26 nosa in a clinically relevant mouse model of wound infection.

27 ial artery, but did increase risk of sternal wound infection.

28 besity was associated with increased risk of wound infection.

29 he pathogenesis of P. aeruginosa during burn wound infection.

30 or renal replacement therapy or deep sternal wound infection.

31 he primary endpoint was the rate of surgical wound infection.

32 d in wound cultures in patients with post-PD wound infections.

33 tococci are considered predominant causes of wound infections.

34 lated from canine pyoderma and postoperative wound infections.

35 ese ecologic changes may affect the risk for wound infections.

36 nvolvement in nosocomial and especially burn wound infections.

37 omplications is mostly caused by superficial wound infections.

38 s also associated with blood, placental, and wound infections.

39 e were no deaths, strokes, renal failure, or wound infections.

40 treatment of methicillin-resistant SA (MRSA) wound infections.

41 biofilm-forming bacteria frequently found in wound infections.

42 stent biofilm-associated infections, such as wound infections.

43 ureus being a major complication of diabetic wound infections.

44 g human pathogen that causes pharyngitis and wound infections.

45 he treatment of antimicrobial resistant burn wound infections.

46 ice and are associated with chronic human Pa wound infections.

47 ted because three patients developed delayed wound infections.

48 ctions, including pneumonia, bacteremia, and wound infections.

49 spatial and temporal monitoring of potential wound infections.

50 coexist for long periods together in chronic wound infections.

51 re susceptible to horizontal transmission of wound infections.

52 k and 95% CIs were derived for postoperative wound infections.

53 sus 1.7; P=0.03) but a decreased risk of leg wound infections (0.2 versus 1.1; P<0.001).

54 or endometritis, 1.2% (95% CI 1.0%-1.5%) for wound infection, 0.05% (95% CI 0.03%-0.07%) for sepsis,

55 pticemia, 0.5% [n=99]; isolated deep sternal wound infection, 0.5% [n=96]; isolated harvest/cannulati

56 ctious complications (2.6%), of which mainly wound infection (1.6%) and bleeding (1.0%).

57 d significantly (P<0.05) higher incidence of wound infections (12.7% vs. 7.3%), pneumonia (7.6% vs. 3

58 5%] vs 46/749 [6.1%]; P = .77), deep sternal wound infection (14/753 [1.9%] vs 19/749 [2.5%]; P = .37

59 lood (60%), followed by lung (21%), skin and wound infections (14%), abscess (1%), and other (4%).

60 tes of endometritis (3.8% vs. 6.1%, P=0.02), wound infection (2.4% vs. 6.6%, P<0.001), and serious ma

61 ; P = .67), or rehospitalization for sternal wound infection (23/753 [3.1%] vs 24/749 [3.2%]; P = .87

62 was most common (42%), followed by surgical wound infections (29%), mediastinitis (16%), sternal ost

63 The cost of most common complications was wound infection (3.8%, $21,995), renal failure (2.8%, $1

64 ast 1 complication within 90 days, including wound infections (3.6%), pneumonia (2.3%), hemorrhage or

65 group, respectively, in superficial sternal wound infection (49/753 [6.5%] vs 46/749 [6.1%]; P = .77

66 e 4 (3 SE, 1 SA), pouch necrosis 2 (JP), and wound infection 5 (2 JP, 3 SE).

67 %] versus 13 of 414 [3.1%]; P=0.279; sternal wound infection, 7 of 414 [1.7%] versus 13 of 414 [3.1%]

68 the 1623 patients who underwent PD, 133 with wound infections (8.2%) were identified.

69 Of the 133 wound infections, 89 (67.1%) were deep-tissue infection,

70 time, >30 minutes) more often suffered from wound infection (9/63 vs 2/70; P = 0.025), abdominal com

71 Wound infections accounted for 84% of cases, followed by

72 k factors for development of a postoperative wound infection across all procedures.

73 spension (FS) (adjusted OR, 5.86; P < .001), wound infection (adjusted OR, 9.45; P = .02), postoperat

74 Whether obesity is a risk factor for wound infection after laparoscopic colectomy remains unc

75 tenting appears to increase the incidence of wound infection after pancreatoduodenectomy but has no e

76 Wound infections after pancreaticoduodenectomy (PD) are

77 d infections, while 10 (3.2%) developed late wound infections (after 30 days).

78 ial pathogen Vibrio vulnificus causes severe wound infection and fatal septicemia.

79 mouse strains to Pseudomonas aeruginosa skin wound infection and found significantly delayed wound cl

80 erial killing and resulted in the control of wound infection and normal wound healing in vivo.

81 enced 1 or more complications with abdominal wound infection and pulmonary complications being the 2

82 ction was identified in 12 patients: 10 with wound infections and 2 with intra-abdominal infections.

83 asons for readmission after 90 days included wound infections and intra-abdominal abscess (n = 75) an

84 m capable of causing serious and often fatal wound infections and primary septicemia.

85 significant component in some mixed surgical wound infections and that surgical management and antimi

86 ng death, pulmonary embolus, pneumonia, deep wound infection, and acute myocardial infarction) were a

87 cemia managed with oral hypoglycemics, minor wound infection, and hyperuricemia but no infections.

88 ts a higher prevalence of tracheal stenosis, wound infection, and major bleeding for surgical tracheo

89 athologic response, presence of re-operation/wound infection, and no closure of ileostomy/colostomy.

90 age, bile leakage, delayed gastric emptying, wound infection, and pneumonia) with each unfavorable ou

91 osion, port/tube dysfunction, hiatal hernia, wound infection, and pouch dilation.

92 infection such as pneumonia, mediastinitis, wound infection, and sepsis.

93 er surgery, 4 had perianal abscesses, 13 had wound infections, and 1 had C. difficile in a urinary ca

94 sk factors for VGIs include groin incisions, wound infections, and comorbidities.

95 lays an important role in sepsis, pneumonia, wound infections, and cystic fibrosis (CF), which is cau

96 ignificantly more respiratory complications, wound infections, and early postoperative mortality, whe

97 ausing food- and waterborne gastroenteritis, wound infections, and septicemia in humans.

98 problems; body mass index and allergies for wound infections; and patients' age, resection weight fo

99 lin improves outcome following a lethal burn wound infection are not known, the data suggest that imm

100 ital stays as well as the absence of sternal wound infection are the main advantages of this techniqu

101 under the age of 49 and complications due to wound infection are the primary cause of death in the fi

102 Wound infections are a critical healthcare concern world

103 Skin wound infections are a significant health problem, and a

104 Though chronic wound infections are often polymicrobial in nature, much

105 ors associated with delayed healing included wound infection at any point and baseline wound area abo

106 f values were determined for mortality, burn wound infection (at least two infections), sepsis (as de

107 Information on wound infection, axillary seroma, paresthesia, brachial

108 011, the use of SLND + ALND resulted in more wound infections, axillary seromas, and paresthesias tha

109 ed guidelines for the prevention of surgical wound infections based upon review and interpretation of

110 ies higher complication rate than l-ANP with wound infection being the most common.

111 ction, repeat revascularization, and sternal wound infection between propensity score-matched cohorts

112 of 30-day mortality, myocardial infarction, wound infection, bleeding, amputation, or reoperation.

113 n patients at risk of bacteremia or surgical wound infection but failed to reach their clinical endpo

114 nd 13.5% of the dry dressing group developed wound infection, but this was not statistically signific

115 with Escherichia coli and other pathogens in wound infections, but mechanisms that govern polymicrobi

116 rmine the effect of fluid group on AKI, burn wound infections (BWIs), and pneumonia.

117 ance of mice to a subsequent burn injury and wound infection by a dendritic cell-dependent mechanism.

118 fter burn injury decreases susceptibility to wound infections by enhancing global immune cell activat

119 extracts of Arabidopsis leaves subjected to wounding, infection by PstAvr, infection by a virulent s

120 r erysipelas, major cutaneous abscesses, and wound infections, can be life-threatening and may requir

121 acter species, accounted for the majority of wound infections cared for on USNS Comfort during Operat

122 We describe a case of sternal wound infection caused by Trichosporon inkin with a fata

123 Wound infections caused by Staphylococcus aureus are ass

124 new avenue of future topical treatments for wound infections caused by these two important pathogens

125 ociated with nosocomial bloodstream and deep wound infections causing a high mortality rate mainly in

126 hedule, and stratified by type of infection (wound infection, cellulitis or erysipelas, or major absc

127 s of central importance in the prevention of wound infections, colonization of medical devices, and n

128 Robotic surgery had fewer wound infections compared with open.

129 er overall postoperative morbidity and fewer wound infections compared with open.

130 fference of tracheal stenosis, bleeding, and wound infection comparing different techniques.

131 tectomies were associated with high rates of wound infections, complications, and increased recovery

132 vs 3 [4.5%] in institution B; P = .001) and wound infection cultures (predominant microorganism in i

133 ltured from both the intraoperative bile and wound infection cultures (Streptococcus pneumoniae, 114

134 We compared the primary outcome, wound infection cure at 7-14 days, and secondary outcome

135 essing group and 40% of the NPWT group had a wound infection, dehiscence, or both.

136 vent, prolonged hospital length of stay, and wound infection/dehiscence).

137 Major complications remain infrequent: wound infection/dehiscence, 3%, atelectasis/pneumonia, 2

138 ith spontaneous chronic multi drug-resistant wound infections demonstrated clearance of bacteria and

139 sing with the required sensitivity for rapid wound infection detection directly from a clinically rel

140 e a mouse model for investigating E faecalis wound infection determinants, and suggest that both immu

141 c surgical patients at high risk for sternal wound infection (diabetes, body mass index >30, or both)

142 lenged in vivo with the polybacterial bovine wound infection 'digital dermatitis', Zn/Cu-shellac adhe

143 Biofilm formation causes prolonged wound infections due to the dense biofilm structure, dif

144 operties in the prevention and management of wound infections during the conflict.

145 4 expression negatively correlates with burn wound infection episodes per patient.

146 opathy, cerebral radionecrosis) and surgery (wound infections, flap necrosis, fistulas,...).

147 Wound infection following cardiac surgery is well descri

148 mnionitis in labour, puerperal endometritis, wound infection following cesarean section or perineal t

149 iotic prophylaxis to reduce the incidence of wound infections following PD.

150 hylaxis is effective in reducing the risk of wound infection for all types of surgery, even ones wher

151 and an increased risk of major bleeding and wound infection for surgical tracheostomies.

152 P < .001) and an increase in readmission for wound infection from 1.4% (95% CI, 1.3%-1.5%) to 3.0% (9

153 present study, we describe 9 cases of mixed wound infection, from a pool of 400 surgical wound infec

154 colonization (FWC, 121 patients), or fungal wound infection (FWI, 54 patients).

155 5 mg/kg (skin photosensitivity [grade 3] and wound infection [grade 3]); thus, the maximum tolerated

156 The development and treatment of surgical wound infections has always been a limiting factor to th

157 n between nares colonization and concomitant wound infections has not been well established.

158 However, concerns about sternal wound infection have discouraged the use of BIMA graftin

159 Analysis of predictors of wound infection identified standard wound dressings as t

160 Invasive fungal wound infections (IFIs) are an uncommon, but increasingl

161 Trauma-related invasive fungal wound infections (IFIs) are associated with significant

162 l varices haemorrhage, circulatory collapse, wound infection, ileus, cerebrovascular accident [possib

163 nt occurred in 1.8%, hemothorax in 0.3%, and wound infection in 2.9%; 1.4% required surgical drainage

164 Infection types included wound infection in 46.8% of patients, cellulitis/erysipe

165 abetes mellitus occurred in 10% vs. 45%, and wound infection in 6% vs. 31% of steroid-free vs. cortic

166 ere was no significant difference in sternal wound infection in 63 of 753 patients randomized to the

167 We report a case of A. variabilis invasive wound infection in a 21-year-old male after a self-infli

168 report a case of P. aphanidermatum invasive wound infection in a 21-year-old male injured during com

169 fatal case of S. erythrospora invasive burn wound infection in a 26-year-old male injured during com

170 e outcome following a Pseudomonas aeruginosa wound infection in a rodent model of severe burn injury.

171 (4HNE), is important for resistance against wound infection in Drosophila muscle.

172 Pf promote Pa wound infection in mice and are associated with chronic

173 yse both host and pathogen biomarkers during wound infection in near real-time.

174 erious adverse events included a superficial wound infection in one patient that resolved with antibi

175 ependent risk factors for development of any wound infection in patients undergoing mastectomy were a

176 there was an increased risk of rejection and wound infection in the obese group, there was no differe

177 ine model of cutaneous Staphylococcus aureus wound infection in young (3-4 mo) and aged (18-20 mo) BA

178 ents were infections of the target wound: 33 wound infections in 25 (20%) patients of 126 in the sucr

179 ) significantly increases resistance to burn wound infections in a DC-dependent manner that is correl

180 ion days, and 3.5 times the relative risk of wound infections in days 91 to 365 (aHR, 3.55; 95% CI, 1

181 y independent risk factors for postoperative wound infections in each.

182 describe two cases of Clostridium glycolicum wound infections in immunocompetent adults.

183 ate 1990s, an outbreak of tilapia-associated wound infections in Israel was linked to a previously un

184 al venous catheter-associated infection, and wound infections) in HTx, LTx, and MCS device recipients

185 of S. aureus bacteremia and/or deep sternal wound infection (including mediastinitis) through postop

186 Secondary endpoints were postoperative wound infection, intra-abdominal abscess, reoperation, l

187 ween the timing of surgery and postoperative wound infection, intra-abdominal abscess, reoperation, o

188 Secondary outcomes included (1) superficial wound infection (involving subcutaneous tissue but not e

189 ng down to sternal fixation wires), (2) deep wound infection (involving the sternal wires, sternal bo

190 control practices; however, the etiology of wound infection is incompletely understood.

191 procedures affects the rate of postoperative wound infection is unknown.

192 elevant postoperative pancreatic fistula and wound infection, length of stay, or 90-day readmission.

193 n to enhance systemic and local responses to wound infections may be protective after burn injury.

194 Wound infections may result from colonization by feces.

195 Wound infection microbiology analysis and resistance pat

196 In this study, we used an in vivo wound infection model in mice induced by topical inocula

197 Taken together, this S. aureus wound infection model provides a valuable preclinical sc

198 obial therapies are needed, a S. aureus skin wound infection model was developed in which full-thickn

199 n whole blood, human wound fluid, or a mouse wound infection model was in turn increased after antibi

200 Using an open wound infection model, we show that deletion of mGsta4 r

201 in in both the s.c. abscess and the surgical wound infection models in WT mice.

202 ls, (ii) intradermal infection models, (iii) wound infection models, and (iv) epicutaneous infection

203 The most frequent morbid complication was wound infection, more commonly occurring in the mastecto

204 role in the pathogenesis of PA14 during burn wound infection, most likely by contributing to PA14 sur

205 Finally, in an in vivo mixed bacterial wound infection mouse model, S. aureus luc signals could

206 myositis and a clinically relevant S. aureus wound infection murine model.

207 Secondary outcomes included wound infection, myocardial infarction, and renal insuff

208 gastrointestinal hemorrhage (n = 5; 12.5%), wound infection (n = 2; 5%) thrombocytopenia (n = 1; 2.5

209 n (n=1), intermittent claudication (n=1) and wound infection (n=1).

210 ia (n = 16, 4%), reexploration (n = 12, 3%), wound infections (n = 12, 3%), and intraabdominal absces

211 0/56%), foreign body reactions (n = 58/12%), wound infections (n = 45/9, 3%) and fat tissue necrosis

212 , pneumonia, sepsis, anastomotic dehiscence, wound infection, noncardiac respiratory failure, atrial

213 Wound infection occurred in 1.4% (17 patients), of whom

214 A wound infection occurred in 4 of the open patients compa

215 Leg-wound infections occurred in 18 patients (3.1%) in the o

216 The primary end point was sternal wound infection occurring through 90 days postoperativel

217 was the strongest predictor of postoperative wound infection (odds ratio, 2.5; 95% CI, 1.58-3.88; P =

218 vity, age, preoperative body mass index, and wound infection on arm volume excess.

219 ificant change in the rates of postoperative wound infection or renal insufficiency during this time

220 splant ascites, posttransplant dialysis, and wound infection or reoperation after transplant should a

221 Composite infection (respiratory or wound infection or septicemia) and ischemic outcomes (my

222 retained stones (OR, 0.5; 95% CI, 0.3-0.9), wound infection (OR, 0.07; 95% CI, 0.04-0.2), reoperatio

223 plications (OR: 3.46; 95% CI: 1.49-8.05) and wound infection (OR: 2.45; 95% CI: 1.01-5.94), longer ho

224 n technique worsened the odds of superficial wound infections (OR, 1.71; P = .02) but not septic shoc

225 y outcome was a serious infection (sepsis or wound infection) or an ischemic event (permanent stroke

226 ary outcome was a composite of endometritis, wound infection, or other infection occurring within 6 w

227 o difference in operative mortality, sternal wound infection, or total complications between matched

228 There were no deaths, wound infections, or instances of pancreatitis.

229 sion, dyspnea, diabetes, renal failure, open wounds/infection, or advanced American Society of Anesth

230 , obese patients displayed increased odds of wound infection: OR (odds ratio) = 1.64 (95% CI: 1.21, 2

231 be an effective intervention for preventing wound infection over a broad range of different surgical

232 ying (P = 0.062), burst abdomen (P = 0.480), wound infection (P = 0.758), and hospital stay (P = 0.48

233 Patients in the SLND + ALND group had more wound infections (P <or= .0016), seromas (P <or= .0001),

234 The occurrence of wound infections pooled by all reviews was lower after l

235 bloodstream, respiratory tract, and surgical wound infections predominated.

236 Additionally, wound infection rate (32.3% vs 12.4%, P < 0.0001) and tr

237 The wound infection rate did not differ significantly across

238 There was no difference in wound infection rate, time to regular diet, length of ho

239 tervention did not reduce the 90-day sternal wound infection rate.

240 tive morbidity (36.4% vs 27.3%, P = 0.3) and wound infection rates (14.5% vs 5.5%, P = 0.13).

241 o treat major trauma-related fractures, deep wound infection rates are high.

242 ates for hernia recurrence and postoperative wound infection rates at 24 months, and the EQ-5D and Sh

243 Wound infection rates were similar in both groups.

244 ients >12 years of age with an uncomplicated wound infection received oral clindamycin 300 mg 4 times

245 technique with no additional risk of sternal wound infection related to age.

246 tion rates in breast cancer surgery are low, wound infections remain the most common complication.

247 y of Thoracic Surgeons measure: deep sternal wound infection, renal failure, prolonged ventilation >2

248 oscopic retrograde cholangiopancreatography, wound infection, reoperation, and mortality.

249 rbidity (stroke, renal failure, deep sternal wound infection, reoperation, prolonged ventilation).

250 A total of 53 (40.0%) of the wound infections required home visiting nurse services o

251 us that cause plague, meningitis, and severe wound infections, respectively.

252 Postoperative ileus, wound infection, respiratory/renal failure, urinary trac

253 t causes food poisoning and life-threatening wound infections, secretes the pore-forming toxin hemoly

254 lism, muscle protein synthesis, incidence of wound infection sepsis, and body composition were obtain

255 n of such drug in a murine model of surgical wound infection significantly reduced the bacterial load

256 evention of surgical infections could reduce wound infections significantly; namely to a target of le

257 4(+) alphabeta T cells homed to the surgical wound infection site of WT animals.

258 ficantly reduced the bacterial burden at the wound infection site.

259 esistant pathogenic bacteria associated with wound infections: Staphylococcus aureus, Klebsiella pneu

260 following 4 organisms commonly implicated in wound infections: Staphylococcus aureus, Pseudomonas aer

261 f prophylactic systemic antibiotics, sternal wound infection still occurs in 5% or more of cardiac su

262 ponse syndrome, sepsis, acute kidney injury, wound infection (superficial and deep), rate of intraope

263 Management of deep sternal wound infection (SWI), a serious complication after card

264 associated with fewer transfusions and fewer wound infections than off-pump CABG.

265 ere burn injury predisposes patients to burn wound infections that can disseminate, lead to uncontrol

266 wound infection, from a pool of 400 surgical wound infections that we have studied, in which S. moore

267 s or erysipelas, major cutaneous abscess, or wound infection) that had a minimum lesion area of 75 cm

268 ve intervention for preventing postoperative wound infection, the level of this effectiveness would a

269 In a subcutaneous infection model to mimic wound infection, the multifunctional autoprocessing RTX

270 During S. aureus surgical wound infection, the presence of IFN-gamma at the infect

271 the PU area of 40% or greater, incidence of wound infections, the total number of dressings at 8 wee

272 r in the treatment of MSSA and MRSA surgical wound infection through enhancement of the local CXC che

273 iews the lessons learned from combat-related wound infections throughout history and in the current c

274 WED dressing was applied within 2 hours of wound infection to test its ability to prevent biofilm f

275 eus causes diseases ranging from superficial wound infections to more invasive manifestations like os

276 as from a patient with a genitourinary tract wound infection, two B. longum isolates were from abdomi

277 With the relative risk of wound infection used as the measure of clinical effectiv

278 ost commonly associated microbial species in wound infections, very little is known about their inter

279 The summary prevalence of wound infection was 53%.

280 The wound infection was cured at 7-14 days in 187 of 203 (92

281 The severity of the wound infection was enhanced by administration of a CXC

282 Risk of wound infection was greater for the surgical tracheostom

283 rdiovascular events, but the risk of sternal wound infection was increased (risk difference, 1.07%; 9

284 was shorter (1 vs. 6 months; P = 0.04), and wound infection was more common in the BMI greater than

285 Local wound infection was the most frequent complication after

286 alters the host immune response to cutaneous wound infection, we developed a murine model of cutaneou

287 In relation to wound infection, we have found a reduction associated wi

288 common, but keratitis, endophthalmitis, and wound infection were less common among CA-MRSA cases tha

289 This paralleled our model of wound infection, where diminished neutrophil and macroph

290 L-1beta contributed to host defense during a wound infection, whereas IL-1beta was more critical duri

291 herwise healthy people can experience severe wound infection, which can lead to sepsis and death.

292 Overall, 14 patients (4.4%) developed early wound infections, while 10 (3.2%) developed late wound i

293 y outcome was the incidence of postoperative wound infection (WI), and secondary outcome was the inci

294 ion of techniques and procedures to decrease wound infections will be highly successful, even in pati

295 mation of biofilms in mice, and treatment of wound infections with CAP 3 was able to clear the bacter

296 tal organism that causes both food-borne and wound infections with high morbidity and mortality in hu

297 ve complications, but a decrease in pain and wound infections with LS.

298 k of overall postoperative complications and wound infection, without a substantial increase in the o

299 second hypothesis that the relative risk of wound infection would substantially vary over different

300 is associated with an increased incidence of wound infections, wound dehiscence, biliary complication