コーパス検索結果 (１語後でソート)
通し番号をクリックするとPubMedの該当ページを表示します
1 ent of PD-L1-expressing myeloid cells to the wound site.
2 of the cell in membranes extending over the wound site.
3 an endogenous antiangiogenic factor from the wound site.
4 ytosis of intracellular vesicles next to the wound site.
5 y, and low bioavailability of factors at the wound site.
6 increased leptin production at the ischemic wound site.
7 sponse and increased matrix deposited at the wound site.
8 ence sufficient to withstand the rigors of a wound site.
9 ates of PDL cells and GF to fill an in vitro wound site.
10 the concentration profile of TGF-beta at the wound site.
11 he abilities of these 2 cell types to fill a wound site.
12 ele cells to factors present in serum at the wound site.
13 reases the recruitment of macrophages to the wound site.
14 ions of tissue constituents distant from the wound site.
15 on was localised to a few cells close to the wound site.
16 ) rise in cytosolic Ca2+ was detected at the wound site.
17 d repair, due to the paucity of cells at the wound site.
18 the onset of keratinocyte migration into the wound site.
19 of HB-EGF in the surface epithelium near the wound site.
20 provide a tissue-healing environment at the wound site.
21 established a concentration gradient at the wound site.
22 o produce adventitious roots proximal to the wound site.
23 mage levels reduce once stem cells reach the wound site.
24 ion in Ly6G + neutrophil accumulation at the wound site.
25 d with SMP (Cx-HA + SMP) to release SMP in a wound site.
26 ed to allow successful migration to a distal wound site.
27 ith the formation of an auxin maximum at the wound site.
28 to the anti-inflammatory M2 phenotype at the wound site.
29 s promote the formation of new shoots at the wound site.
30 latworms are required to migrate to a distal wound site.
31 gamma6+ antigen receptors accumulated at the wound site.
32 cruitment of antimicrobial leukocytes to the wound site.
33 ts their recruitment and mobilization at the wound site.
34 y pro-inflammatory mediators released at the wound site.
35 fusion of intracellular compartments at the wound site.
36 information about pathogen behaviour at the wound site.
37 un N-terminal kinase (JNK) signaling, at the wound site.
38 er involving local cell proliferation at the wound site.
39 a polarized repair response targeted to the wound site.
40 local production of mtROS superoxide at the wound site.
41 tro, and deliver them as MSCs on FMBs at the wound site.
42 nt of coe(+) and sim(+) progenitors near the wound site.
43 KC, MIP-2, and MCP-1 chemokine levels at the wound site.
44 i, rapidly melanizes after collection from a wound site.
45 re down-regulated, even far-removed from the wound site.
46 that repopulate the wound and the RPE at the wound site.
47 neutrophil and macrophage recruitment to the wound site.
48 e delivering autologous keratinocytes to the wound site.
49 ioactive form of JA, in leaves distal to the wound site.
50 yo's developmental stage and the subcellular wound site.
51 d fusion of lipid vesicles trafficked to the wound site.
52 zed with fluorescein-labeled fetuin A at the wound site.
53 restoration of skin barrier function at the wound-site.
54 ctin filaments (F-actin) and myosin-2 around wound sites.
55 signals that are thought to be released from wound sites.
56 and subsequent macrophage infiltration into wound sites.
57 ly of phenolic compounds released from plant wound sites.
58 ice had reduced IGF-1 receptor activation at wound sites.
59 pidly deposit callose, a beta-1,3-glucan, at wound sites.
60 inhibiting tumor cell implantation in trocar wound sites.
61 n fibrin gel, a likely provisional matrix at wound sites.
62 es--interleukin 1alpha and interleukin 8--at wound sites.
63 nt petioles and unfertilized flowers, and at wound sites.
64 ds and monosaccharides, synthesized at plant wound sites.
65 r deplete the influx of eosinophils into the wound sites.
66 ea, and led to more normalized tissue in the wound sites.
67 ChvE and monosaccharides released from plant wound sites.
68 onse to chemical signals released from plant wound sites.
69 nd transcription in reprogramming cells near wound sites.
70 y at anterior-facing versus posterior-facing wound sites.
71 esponse genes in epidermal cells surrounding wound sites.
72 -3 expression, and EPIC proteins localize to wound sites.
73 g small GTPase translation in neutrophils at wound sites.
74 nst S. aureus infection and its clearance at wound sites.
75 2 months (12/24; 50%) compared with baseline wound sites.
76 ry tract, indwelling catheters, and surgical wound sites.
77 itored HF formation at small circular (2 mm) wound sites.
78 ns in response to natural enzymes present at wound sites.
79 ling macrophages, and better angiogenesis at wound sites.
80 extracellular proteases highly expressed at wound sites.
81 e insect-derived elicitor, at a mechanically wounded site.
82 they respond by developing tumors along the wounded site.
83 omoted endogenous stem cell migration to the wound site 1.5 times more effectively than Cx-HA + SDF-1
85 ing at 3 months (21 intact graft sites of 24 wound sites; 87%), 6 months (16/24; 67%), and 12 months
87 pair perfectly without a remnant scar at the wound site, adult tissue repair always leads to formatio
88 , at least in part, to the protection of the wound site against oxidative/nitrosative damage and prev
90 e number of SDF-1-bearing macrophages in the wound sites amplifying the "pull" of mobilized stem cell
91 ation-an initial localized response near the wound site and a subsequent systemic response that varie
92 nd proinflammatory cytokine induction at the wound site and augments intracellular bacterial killing
93 ed expression of NFIA, SOX9 and GLAST at the wound site and in the ventricular zone (VZ) of the injur
94 njury, F4/80(+) myeloid cells infiltrate the wound site and induce smooth muscle actin (SMA) expressi
95 mRNA effectively scavenges excess ROS at the wound site and induces the expression of IL4 in macropha
96 s calcium flash inhibits H2O2 release at the wound site and leads to a reduction in the number of imm
97 JAK/STAT signaling becomes activated at the wound site and promotes regenerative cell proliferation
98 s in TEWL at visit 0 (V0) between the closed wound site and reference skin, with the TEWL score as th
99 administration of CD34(+) cells homed to the wound site and significantly accelerated wound closure.
100 d RA in a spatially restricted region of the wound site and that mucosal fibroblasts responded to thi
101 ) such as hydrogen peroxide are generated at wound sites and act as long-range signals in wound heali
102 homologs are necessary for cell migration to wound sites and for the establishment of migratory cell
103 d in response to wounding can be detected at wound sites and in distal leaf veins within 1 hr after w
104 , accurate detection of biofilm formation on wound sites and that can be translated to point-of-care
105 l known to cause crown gall tumours at plant wound sites and to benefit from this plant association b
106 abscess formation at S. aureus-infected skin wound sites and were also more susceptible to horizontal
107 TEWL measurements were obtained, from closed wound-site and contralateral healthy skin site, starting
109 ymes can inadvertently serve as reporters of wounded sites and constitute an "Achilles heel," allowin
110 es in removing stimulation hardware from the wound site, and absence of means to monitor the healing
111 athogens, clear tissue debris present at the wound site, and orchestrate aspects of tissue remodeling
112 healing, cells collectively migrate into the wound site, and the converging tissue fronts collide and
113 rin-binding ligand was designed to target to wound sites, and the cross-linking of fibrin polymers wa
117 rocyte marker, GFAP, was up-regulated at the wound site, around necrotic areas and cysts, plus in usu
118 ry-induced ependymoglial cell cluster at the wound site as a progenitor cell population for the poten
119 hed in this study showed that GF fill in the wound site at a significantly (P <0.0025) faster rate th
120 G augmented recruitment of macrophage at the wound-site, attenuated pro-inflammatory and promoted ant
123 ifference in fibroblast proliferation at the wound site but Wnt signalling was highly upregulated in
124 y a critical role in leukocyte emigration to wound sites, but differences are evident in different va
125 s, we show that Cxcr1 promotes clustering at wound sites, but is promptly desensitized and internaliz
126 egation contributes to arresting bleeding at wound sites, but may cause occlusion of atherosclerotic
129 ndard wound care) were randomly allocated to wound sites by a computer generated randomisation scheme
130 tudies suggest that IGF-I can concentrate at wound sites by binding to fibrin-immobilized IGFBP-3, an
131 release of drugs or biological agents to the wound site can likewise be modulated through the use of
132 ming is controlled; basal cells close to the wound site can produce adventitious roots, whereas cells
133 EC monolayers, EPCs showed clustering at the wound site, compared with untreated regions (P < 0.001);
134 om the contralateral side of the limb to the wound site concomitantly with nerve deviation, the ectop
135 receptor CX3CR1 were both highly induced at wound sites; CX3CL1 colocalized with macrophages and end
136 lation) phenotype and a heightened basal and wound site DNA damage/repair response that is also commo
137 that excessive and prolonged TGFbeta1 at the wound site does not benefit wound healing, which is part
138 ) three-dimensional scaffold into an in vivo wound site effectively blocks the majority of organized
141 sis factor alpha, and IL-1beta) was lower at wound sites following marital conflicts than after socia
144 the technical difficulties in preparing the wound site for morphologic studies, and the postnatal ph
146 We found that epithelial cells near the wound site fuse to form a giant syncytium, which sends l
147 owth factor-beta (TGF-beta) signaling at the wound site has been implicated in re-epithelization, inf
148 ing of how platelets localize coagulation to wound sites has come mainly from studies of a subpopulat
150 emonstrated accumulation of m-calpain at the wound site in association with the membrane repair prote
152 RhoA and Cdc42 are rapidly activated around wound sites in a calcium-dependent manner and segregate
153 uitment of these inflammatory blood cells to wound sites in both Drosophila and vertebrates [1, 2].
154 led systemin is a primary signal released at wound sites in response to herbivory that systemically s
155 ith 5 mCi of (64)Cu-PTSM, whereas 96% of the wound sites in the group treated with saline had macrosc
156 ed that Arabidopsides are highly abundant at wounding sites in both wild-type and fer mutant leaves.
157 Direct effects of CX3CR1 disruption at the wound site included marked reduction of macrophages and
158 he growth factor and cytokine profile at the wound site is changed, cell death is reduced, and dying
159 clearance of monocytes/macrophages from the wound site is critical to re-establish tissue functional
161 t the initial response to amputation at both wound sites is identical and includes widespread apoptos
162 e resultant actomyosin array closes over the wound site, junctional F-actin and myosin-2 become mecha
163 in blast injuries, soft tissue injuries, >3 wound sites, loss of limb, abdominal trauma, and higher
165 we reported the physiological conversion of wound-site macrophages to fibroblasts in granulation tis
167 cing inflammation and bacterial infection in wound sites, measuring tension of both the tissue and su
173 recognizes that despite its abundance at the wound site, OSM is inactivated by glycation, and topical
175 We also show that hyperinnervation of the wound site, previously believed to be a consequence of i
177 on of proinflammatory cytokines at an actual wound site, providing in vivo data on the development of
178 e recruitment is restricted to the immediate wound site rather than spilling extensively into the adj
179 olymers that bind subendothelial collagen at wound sites, recruit platelets, and initiate the clottin
180 eutrophils and macrophages, recruited to the wound site, release reactive oxygen species by respirato
181 rmal wounding, tissue-bound cells around the wound site remain sessile and unresponsive, whereas circ
182 ha then recruits these EPCs to the cutaneous wound site, resulting in accelerated wound healing.
183 follicle SCs (HFSCs) that migrated into the wound site revealed activation of an immune-modulatory p
184 g in live mice to monitor macrophage flux at wound sites revealed that macrophage accumulation was pr
185 Histologic analysis of thrombospondin 2-null wound sites revealed the presence of an irregularly orga
186 issue injury, a fibrin network formed at the wound site serves as a scaffold supporting the early mig
187 lls, by cytokines present in the periodontal wound site, such as interleukin-1 (IL-1), may be an impo
188 ation and basement membrane breakdown at the wound site, suggest that MRL mice may share other featur
189 nted dentate gyrus and in macrophages at the wound site, suggesting a role in lesion-induced tissue r
190 ABA levels increased preferentially near the wound site, suggesting that ABA may have accumulated bec
191 wound-associated erythema, tenderness at the wound site, swelling at the site, purulent drainage, and
193 ision and apoptosis were not observed at the wound site, the needle puncture significantly enhanced D
194 atelets bind to exposed vascular matrix at a wound site through a highly specialized surface receptor
200 esulted in persistent PMN trafficking to the wound site via an IL-6-mediated mechanism, and this in t
205 e back of guinea pigs with a 4 mm punch, and wound sites were collected at different time points duri
208 ages, or their equivalents, are drawn to the wound site where they engulf cell and matrix debris and
209 ecruits these cells from the follicle to the wound site, where downstream Hh signal transduction is d
210 and CRK peptides, selectively accumulated at wound sites, where they partially co-localized with bloo
211 nces persisted through month 9 except at the wound site, which had a relatively small sample size.
212 evokes Ca(2+)-regulated exocytosis near the wound site, which is essential for membrane resealing.
213 for wound-responsive release of D-CuP at the wound site while simultaneously attenuating inflammatory