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1 her doses (10-100 nmol), D-SP(1-7) inhibited writhing.
2 0 min) antinociceptive effects of SP(1-7) on writhing.
3  the peritoneal cavity, as well as abdominal writhings.
4 tion ratchet model predicts that growing and writhing actin filaments generate forces and movements,
5     Compound 4 inhibited acetic acid induced writhing after subcutaneous administration in mice via K
6 d good delta antagonist activity in the anti-writhing analgesic assay and also inhibited delta agonis
7 e test, tail-flick test, acetic acid-induced writhing and formalin-induced analgesia) responses at th
8 ntinociceptive activity (acetic acid-induced writhing and hot plate assays), leading to the identific
9 tagonist, prevented thrombin's inhibition of writhing and NMDA-induced behaviours while BQ788, an end
10                       This transition avoids writhing and the ease of this transition may prevent cum
11 and a full agonist effect in the acetic acid writhing assay (A(50) = 7.5 nmol).
12 eased potencies than U50,488, 1 and 2 in the writhing assay and without inducing motor dysfunction af
13 antinociceptive tolerance in the acetic acid writhing assay.
14 ctivity in the mouse abdominal constriction (writhing) assay, a model of persistent pain.
15 ant plants exhibited prolonged searching and writhing behaviors as they unsuccessfully attempted to e
16 bited acetic acid-induced abdominal stretch (writhing) behaviours, which were also sensitive to NMDA
17 profile combining analgesic potency in mouse writhing (ED50 = 0.1 mg/kg) along with potency for norma
18  topological properties such as helicity and writhing have been studied theoretically and in laborato
19 ociceptive tests (tail-flick and acetic acid writhing) in mice, which demonstrated that cyclorphan (3
20 buckled under compressive deformation, and a writhing mode is developed as a result.
21 o the bending and twisting modes, this novel writhing mode of F-actin could also play important roles
22  by the tail-flick test, acetic acid-induced writhing models, hot plate test, and formalin-induced an
23 TAFI deficiency did not alter kaolin-induced writhing response, implying that TAFI does not play a ma
24  decreased the number of acetic acid-induced writhing responses and formalin-induced pain in the late
25 g tool for earlier CP risk assessment in the writhing stage and prior to hospital discharge.
26 uate infants' general movements (GMs) in the writhing stage.
27  and was observed in the acetic acid-induced writhing test (0.6 vs. 0.1-0.5%), in the hot-plate test
28 effective in inducing antinociception in the writhing test in mice after subcutaneous administration,
29 hot plate test; visceral-acetic acid-induced writhing test), and in animal models that are predictive
30 est and the mouse phenylbenzoquinone-induced writhing test, respectively.
31  injection and reduced response to acid in a writhing test.
32 ater tail-withdrawal and acetic acid-induced writhing tests.
33 the tail flick assay and acetic acid-induced writhing tests.
34 ed by exogenous SP(1-7): acetic acid-induced writhing that is inhibited and formalin-induced behavior
35 t superhelical apices, which facilitates DNA writhing to relieve torsional strain.
36 wenty-four hours after injection of SP(1-7), writhing was inhibited and formalin responses were incre
37                                          The writhing was paralleled by an increase in 5-HT which was
38         NT79 blocked the acetic acid-induced writhing with an ED(50) of 0.14 microg/kg while having n
39 ltaneously evaluated for acetic acid-induced writhing with and without pretreatment with NT79.